ReP USP - Resultado da busca

Artigo de periodico
Ablation of miRNA-22 protects against obesity-induced adipocyte senescence and ameliorates metabolic disorders in middle-aged mice (2023)
Lino, Caroline Antunes ; Silva, Tábatha de Oliveira ; Lunardon, Guilherme ; Silva, Camila Stephanie Balbino ; Lima, Vanessa Morais ; Huang, Zhan-Peng ; Donato Junior, José ; Barreto-Chaves, Maria Luiza Morais ; Diniz, Gabriela Placoná ; Takano, Ana Paula Cremasco
Source: Mechanisms of Ageing and Development. Unidade: ICB
Subjects: FISIOLOGIA, ANATOMIA, ADIPOSIDADE, TECIDO ADIPOSO, OBESIDADE, DIETA ANIMAL, GORDURAS NA DIETA, MICRORNAS, CAMUNDONGOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LINO, Caroline Antunes et al. Ablation of miRNA-22 protects against obesity-induced adipocyte senescence and ameliorates metabolic disorders in middle-aged mice. Mechanisms of Ageing and Development, v. 210, p. 1-14, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.mad.2023.111775. Acesso em: 14 nov. 2024.
APA
Lino, C. A., Silva, T. de O., Lunardon, G., Silva, C. S. B., Lima, V. M., Huang, Z. -P., et al. (2023). Ablation of miRNA-22 protects against obesity-induced adipocyte senescence and ameliorates metabolic disorders in middle-aged mice. Mechanisms of Ageing and Development, 210, 1-14. doi:10.1016/j.mad.2023.111775
NLM
Lino CA, Silva T de O, Lunardon G, Silva CSB, Lima VM, Huang Z-P, Donato Junior J, Barreto-Chaves MLM, Diniz GP, Takano APC. Ablation of miRNA-22 protects against obesity-induced adipocyte senescence and ameliorates metabolic disorders in middle-aged mice [Internet]. Mechanisms of Ageing and Development. 2023 ; 210 1-14.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.mad.2023.111775
Vancouver
Lino CA, Silva T de O, Lunardon G, Silva CSB, Lima VM, Huang Z-P, Donato Junior J, Barreto-Chaves MLM, Diniz GP, Takano APC. Ablation of miRNA-22 protects against obesity-induced adipocyte senescence and ameliorates metabolic disorders in middle-aged mice [Internet]. Mechanisms of Ageing and Development. 2023 ; 210 1-14.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.mad.2023.111775
Artigo de periodico
miRNA-22 is involved in the aortic reactivity in physiological conditions and mediates obesity-induced perivascular adipose tissue dysfunction (2023)
Silva, Camila Stephanie Balbino ; Couto, Gisele Kruger ; Lino, Caroline Antunes ; Silva, Tábatha de Oliveira ; Lunardon, Guilherme ; Huang, Zhan-Peng ; Wang, Da Zhi ; Festuccia, William Tadeu Lara ; Barreto-Chaves, Maria Luiza Morais ; Rossoni, Luciana Venturini ; Diniz, Gabriela Placoná
Source: Life Sciences. Unidade: ICB
Subjects: FISIOLOGIA, ANATOMIA, FISIOLOGIA, AORTA DE ANIMAL, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS, VASOS SANGUÍNEOS, TECIDO ADIPOSO, MICRORNAS, OBESIDADE, REAÇÃO EM CADEIA POR POLIMERASE, WESTERN BLOTTING
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Camila Stephanie Balbino et al. miRNA-22 is involved in the aortic reactivity in physiological conditions and mediates obesity-induced perivascular adipose tissue dysfunction. Life Sciences, v. 316, p. 1-10, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2023.121416. Acesso em: 14 nov. 2024.
APA
Silva, C. S. B., Couto, G. K., Lino, C. A., Silva, T. de O., Lunardon, G., Huang, Z. -P., et al. (2023). miRNA-22 is involved in the aortic reactivity in physiological conditions and mediates obesity-induced perivascular adipose tissue dysfunction. Life Sciences, 316, 1-10. doi:10.1016/j.lfs.2023.121416
NLM
Silva CSB, Couto GK, Lino CA, Silva T de O, Lunardon G, Huang Z-P, Wang DZ, Festuccia WTL, Barreto-Chaves MLM, Rossoni LV, Diniz GP. miRNA-22 is involved in the aortic reactivity in physiological conditions and mediates obesity-induced perivascular adipose tissue dysfunction [Internet]. Life Sciences. 2023 ; 316 1-10.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2023.121416
Vancouver
Silva CSB, Couto GK, Lino CA, Silva T de O, Lunardon G, Huang Z-P, Wang DZ, Festuccia WTL, Barreto-Chaves MLM, Rossoni LV, Diniz GP. miRNA-22 is involved in the aortic reactivity in physiological conditions and mediates obesity-induced perivascular adipose tissue dysfunction [Internet]. Life Sciences. 2023 ; 316 1-10.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2023.121416
Artigo de periodico
The miRNA-143-3p-Sox6-Myh7 pathway is altered in obesogenic diet-induced cardiac hypertrophy (2022)
Silva, Tábatha De Oliveira ; Lino, Caroline Antunes ; Miranda, Juliane Braga ; Silva, Camila Stephanie Balbino ; Lunardon, Guilherme ; Lima, Vanessa Morais ; Jensen, Leonardo ; Donato Junior, José ; Irigoyen, Maria Cláudia Costa ; Barreto-Chaves, Maria Luiza Morais ; Diniz, Gabriela Placoná
Source: Experimental Physiology. Unidades: ICB, FM
Subjects: ANATOMIA, FISIOLOGIA, CARDIOMEGALIA ANIMAL, OBESIDADE, DIETA ANIMAL, HIPERTROFIA VENTRICULAR ESQUERDA, MICRORNAS, FIBRAS MUSCULARES, MIOSINA, RNA MENSAGEIRO, CAMUNDONGOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Tábatha De Oliveira et al. The miRNA-143-3p-Sox6-Myh7 pathway is altered in obesogenic diet-induced cardiac hypertrophy. Experimental Physiology, v. 107, n. 8, p. 892-905, 2022Tradução . . Disponível em: https://doi.org/10.1113/EP090315. Acesso em: 14 nov. 2024.
APA
Silva, T. D. O., Lino, C. A., Miranda, J. B., Silva, C. S. B., Lunardon, G., Lima, V. M., et al. (2022). The miRNA-143-3p-Sox6-Myh7 pathway is altered in obesogenic diet-induced cardiac hypertrophy. Experimental Physiology, 107( 8), 892-905. doi:10.1113/EP090315
NLM
Silva TDO, Lino CA, Miranda JB, Silva CSB, Lunardon G, Lima VM, Jensen L, Donato Junior J, Irigoyen MCC, Barreto-Chaves MLM, Diniz GP. The miRNA-143-3p-Sox6-Myh7 pathway is altered in obesogenic diet-induced cardiac hypertrophy [Internet]. Experimental Physiology. 2022 ; 107( 8): 892-905.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1113/EP090315
Vancouver
Silva TDO, Lino CA, Miranda JB, Silva CSB, Lunardon G, Lima VM, Jensen L, Donato Junior J, Irigoyen MCC, Barreto-Chaves MLM, Diniz GP. The miRNA-143-3p-Sox6-Myh7 pathway is altered in obesogenic diet-induced cardiac hypertrophy [Internet]. Experimental Physiology. 2022 ; 107( 8): 892-905.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1113/EP090315
Artigo de periodico
Set7 deletion prevents glucose intolerance and improves the recovery of cardiac function after ischemia and reperfusion in obese female mice (2022)
Miranda, Juliane Braga ; Lunardon, Guilherme ; Lima, Vanessa Morais ; Silva, Tábatha de Oliveira ; Lino, Caroline Antunes ; Jensen, Leonardo ; Irigoyen, Maria Cláudia Costa ; Donato Junior, José ; Barreto-Chaves, Maria Luiza Morais ; Diniz, Gabriela Placoná
Source: Cellular Physiology and Biochemistry. Unidades: FM, ICB
Subjects: FISIOLOGIA, ANATOMIA, DIETA ANIMAL, GLICOSE, ISQUEMIA, OBESIDADE, CAMUNDONGOS, DOENÇAS CARDIOVASCULARES EM ANIMAL, DOENÇAS METABÓLICAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MIRANDA, Juliane Braga et al. Set7 deletion prevents glucose intolerance and improves the recovery of cardiac function after ischemia and reperfusion in obese female mice. Cellular Physiology and Biochemistry, v. 56, n. 3, p. 293-309, 2022Tradução . . Disponível em: https://doi.org/10.33594/000000535. Acesso em: 14 nov. 2024.
APA
Miranda, J. B., Lunardon, G., Lima, V. M., Silva, T. de O., Lino, C. A., Jensen, L., et al. (2022). Set7 deletion prevents glucose intolerance and improves the recovery of cardiac function after ischemia and reperfusion in obese female mice. Cellular Physiology and Biochemistry, 56( 3), 293-309. doi:10.33594/000000535
NLM
Miranda JB, Lunardon G, Lima VM, Silva T de O, Lino CA, Jensen L, Irigoyen MCC, Donato Junior J, Barreto-Chaves MLM, Diniz GP. Set7 deletion prevents glucose intolerance and improves the recovery of cardiac function after ischemia and reperfusion in obese female mice [Internet]. Cellular Physiology and Biochemistry. 2022 ; 56( 3): 293-309.[citado 2024 nov. 14 ] Available from: https://doi.org/10.33594/000000535
Vancouver
Miranda JB, Lunardon G, Lima VM, Silva T de O, Lino CA, Jensen L, Irigoyen MCC, Donato Junior J, Barreto-Chaves MLM, Diniz GP. Set7 deletion prevents glucose intolerance and improves the recovery of cardiac function after ischemia and reperfusion in obese female mice [Internet]. Cellular Physiology and Biochemistry. 2022 ; 56( 3): 293-309.[citado 2024 nov. 14 ] Available from: https://doi.org/10.33594/000000535
Artigo de periodico
Set7 deletion attenuates isoproterenol-induced cardiac fibrosis and delays cardiac dysfunction (2022)
Lunardon, Guilherme ; Silva, Tábatha de Oliveira ; Lino, Caroline Antunes ; Miranda, Juliane Braga ; Silva, Amanda de Almeida; Irigoyen, Maria Cláudia Costa ; Takano, Ana Paula Cremasco ; Barreto-Chaves, Maria Luiza Morais ; Diniz, Gabriela Placoná
Source: Clinical Science (Online): an international journal of translational. Unidades: FM, ICB
Subjects: ANATOMIA, HIPERTROFIA VENTRICULAR ESQUERDA, CARDIOMEGALIA, CARDIOPATIAS, MIOCARDIOPATIAS, CÉLULAS MUSCULARES, CAMUNDONGOS, DNA MITOCONDRIAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LUNARDON, Guilherme et al. Set7 deletion attenuates isoproterenol-induced cardiac fibrosis and delays cardiac dysfunction. Clinical Science (Online): an international journal of translational, v. 136, p. 1537–1554, 2022Tradução . . Disponível em: https://doi.org/10.1042/CS20220466. Acesso em: 14 nov. 2024.
APA
Lunardon, G., Silva, T. de O., Lino, C. A., Miranda, J. B., Silva, A. de A., Irigoyen, M. C. C., et al. (2022). Set7 deletion attenuates isoproterenol-induced cardiac fibrosis and delays cardiac dysfunction. Clinical Science (Online): an international journal of translational, 136, 1537–1554. doi:10.1042/CS20220466
NLM
Lunardon G, Silva T de O, Lino CA, Miranda JB, Silva A de A, Irigoyen MCC, Takano APC, Barreto-Chaves MLM, Diniz GP. Set7 deletion attenuates isoproterenol-induced cardiac fibrosis and delays cardiac dysfunction [Internet]. Clinical Science (Online): an international journal of translational. 2022 ; 136 1537–1554.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1042/CS20220466
Vancouver
Lunardon G, Silva T de O, Lino CA, Miranda JB, Silva A de A, Irigoyen MCC, Takano APC, Barreto-Chaves MLM, Diniz GP. Set7 deletion attenuates isoproterenol-induced cardiac fibrosis and delays cardiac dysfunction [Internet]. Clinical Science (Online): an international journal of translational. 2022 ; 136 1537–1554.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1042/CS20220466
Artigo de periodico
Beta-arrestins in the context of cardiovascular diseases: focusing on angiotensin II type 1 receptor (AT1R) (2022)
Lino, Caroline Antunes ; Barreto-Chaves, Maria Luiza Morais
Source: Cellular Signalling. Unidade: ICB
Subjects: ANATOMIA, DOENÇAS CARDIOVASCULARES, SISTEMA RENINA-ANGIOTENSINA, HIPERTROFIA VENTRICULAR ESQUERDA, INFARTO DO MIOCÁRDIO, ALDOSTERONA, INFECÇÕES POR CORONAVIRUS, ANGIOTENSINA II, OLIGOPEPTÍDEOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LINO, Caroline Antunes e BARRETO-CHAVES, Maria Luiza Morais. Beta-arrestins in the context of cardiovascular diseases: focusing on angiotensin II type 1 receptor (AT1R). Cellular Signalling, v. 92, p. 1-9, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.cellsig.2022.110253. Acesso em: 14 nov. 2024.
APA
Lino, C. A., & Barreto-Chaves, M. L. M. (2022). Beta-arrestins in the context of cardiovascular diseases: focusing on angiotensin II type 1 receptor (AT1R). Cellular Signalling, 92, 1-9. doi:10.1016/j.cellsig.2022.110253
NLM
Lino CA, Barreto-Chaves MLM. Beta-arrestins in the context of cardiovascular diseases: focusing on angiotensin II type 1 receptor (AT1R) [Internet]. Cellular Signalling. 2022 ; 92 1-9.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.cellsig.2022.110253
Vancouver
Lino CA, Barreto-Chaves MLM. Beta-arrestins in the context of cardiovascular diseases: focusing on angiotensin II type 1 receptor (AT1R) [Internet]. Cellular Signalling. 2022 ; 92 1-9.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.cellsig.2022.110253
Artigo de periodico
miRNA-22 deletion limits white adipose expansion and activates brown fat to attenuate high-fat diet-induced fat mass accumulation (2021)
Lima, Vanessa Morais; Liu, Jianming; Brandão, Bruna B. ; Lino, Caroline Antunes ; Silva, Camila Stephanie Balbino ; Ribeiro, Márcio Augusto Campos; Silva, Tiago Eugênio Oliveira da; Gregório, Caroline Cristiano Real ; Faria, Daniele de Paula ; Cederquist, Carly; Huang, Zhan-Peng; Hu, Xiaoyun ; Barreto-Chaves, Maria Luiza Morais ; Ferreira, Julio Cesar Batista ; Festuccia, William Tadeu Lara ; Mori, Marcelo A. ; Kahn, C. Ronald ; Wang, Da-Zhi ; Diniz, Gabriela Placoná
Source: Metabolism. Unidades: FM, ICB
Subjects: ANATOMIA, FISIOLOGIA, TECIDO ADIPOSO, DIETA, OBESIDADE, TERMOGÊNESE, METABOLISMO DE GORDURA, MICRORNAS, CAMUNDONGOS, ADIPOSIDADE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LIMA, Vanessa Morais et al. miRNA-22 deletion limits white adipose expansion and activates brown fat to attenuate high-fat diet-induced fat mass accumulation. Metabolism, v. 117, p. 13 , 2021Tradução . . Disponível em: https://doi.org/10.1016/j.metabol.2021.154723. Acesso em: 14 nov. 2024.
APA
Lima, V. M., Liu, J., Brandão, B. B., Lino, C. A., Silva, C. S. B., Ribeiro, M. A. C., et al. (2021). miRNA-22 deletion limits white adipose expansion and activates brown fat to attenuate high-fat diet-induced fat mass accumulation. Metabolism, 117, 13 . doi:10.1016/j.metabol.2021.154723
NLM
Lima VM, Liu J, Brandão BB, Lino CA, Silva CSB, Ribeiro MAC, Silva TEO da, Gregório CCR, Faria D de P, Cederquist C, Huang Z-P, Hu X, Barreto-Chaves MLM, Ferreira JCB, Festuccia WTL, Mori MA, Kahn CR, Wang D-Z, Diniz GP. miRNA-22 deletion limits white adipose expansion and activates brown fat to attenuate high-fat diet-induced fat mass accumulation [Internet]. Metabolism. 2021 ; 117 13 .[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.metabol.2021.154723
Vancouver
Lima VM, Liu J, Brandão BB, Lino CA, Silva CSB, Ribeiro MAC, Silva TEO da, Gregório CCR, Faria D de P, Cederquist C, Huang Z-P, Hu X, Barreto-Chaves MLM, Ferreira JCB, Festuccia WTL, Mori MA, Kahn CR, Wang D-Z, Diniz GP. miRNA-22 deletion limits white adipose expansion and activates brown fat to attenuate high-fat diet-induced fat mass accumulation [Internet]. Metabolism. 2021 ; 117 13 .[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.metabol.2021.154723
Artigo de periodico
Renin-angiotensin system overactivation in perivascular adipose tissue contributes to vascular dysfunction in heart failure (2020)
Fontes, Milene Tavares ; Paula, Suliana Mesquita ; Lino, Caroline Antunes ; Senger, Nathalia ; Couto, Gisele Kruger; Barreto-Chaves, Maria Luiza Morais ; Mill, José Geraldo ; Rossoni, Luciana Venturini
Source: Clinical Science. Unidade: ICB
Subjects: ANATOMIA, FISIOLOGIA, SISTEMA RENINA-ANGIOTENSINA, TECIDO ADIPOSO, INSUFICIÊNCIA CARDÍACA ANIMAL, DOENÇAS CARDIOVASCULARES EM ANIMAL, ENDOTÉLIO VASCULAR, INFARTO DO MIOCÁRDIO, RATOS, ANGIOTENSINAS, AORTA DE ANIMAL, ESTRESSE OXIDATIVO, ÓXIDO NÍTRICO, AORTA TORÁCICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FONTES, Milene Tavares et al. Renin-angiotensin system overactivation in perivascular adipose tissue contributes to vascular dysfunction in heart failure. Clinical Science, 2020Tradução . . Disponível em: https://doi.org/10.1042/CS20201099. Acesso em: 14 nov. 2024.
APA
Fontes, M. T., Paula, S. M., Lino, C. A., Senger, N., Couto, G. K., Barreto-Chaves, M. L. M., et al. (2020). Renin-angiotensin system overactivation in perivascular adipose tissue contributes to vascular dysfunction in heart failure. Clinical Science. doi:10.1042/CS20201099
NLM
Fontes MT, Paula SM, Lino CA, Senger N, Couto GK, Barreto-Chaves MLM, Mill JG, Rossoni LV. Renin-angiotensin system overactivation in perivascular adipose tissue contributes to vascular dysfunction in heart failure [Internet]. Clinical Science. 2020 ;[citado 2024 nov. 14 ] Available from: https://doi.org/10.1042/CS20201099
Vancouver
Fontes MT, Paula SM, Lino CA, Senger N, Couto GK, Barreto-Chaves MLM, Mill JG, Rossoni LV. Renin-angiotensin system overactivation in perivascular adipose tissue contributes to vascular dysfunction in heart failure [Internet]. Clinical Science. 2020 ;[citado 2024 nov. 14 ] Available from: https://doi.org/10.1042/CS20201099
Artigo de periodico
Beta‐arrestin 2 mediates cardiac hypertrophy induced by thyroid hormones via AT1R (2020)
Lino, Caroline Antunes ; Teixeira, Larissa de Bortoli ; Simões, Sarah Capelupe; Silva, Tábatha de Oliveira ; Diniz, Gabriela Placoná ; Costa Neto, Claudio Miguel da ; Barreto-Chaves, Maria Luiza Morais
Source: Journal of Cellular Physiology. Unidades: FMRP, ICB
Subjects: ANATOMIA, HIPERTROFIA VENTRICULAR ESQUERDA, HORMÔNIOS TIREOIDIANOS, ANGIOTENSINA II, SISTEMA CARDIOVASCULAR, IMUNOHISTOQUÍMICA, PROTEÍNAS QUINASES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LINO, Caroline Antunes et al. Beta‐arrestin 2 mediates cardiac hypertrophy induced by thyroid hormones via AT1R. Journal of Cellular Physiology, v. 236, n. 6, p. 4640-4654, 2020Tradução . . Disponível em: https://doi.org/10.1002/jcp.30187. Acesso em: 14 nov. 2024.
APA
Lino, C. A., Teixeira, L. de B., Simões, S. C., Silva, T. de O., Diniz, G. P., Costa Neto, C. M. da, & Barreto-Chaves, M. L. M. (2020). Beta‐arrestin 2 mediates cardiac hypertrophy induced by thyroid hormones via AT1R. Journal of Cellular Physiology, 236( 6), 4640-4654. doi:10.1002/jcp.30187
NLM
Lino CA, Teixeira L de B, Simões SC, Silva T de O, Diniz GP, Costa Neto CM da, Barreto-Chaves MLM. Beta‐arrestin 2 mediates cardiac hypertrophy induced by thyroid hormones via AT1R [Internet]. Journal of Cellular Physiology. 2020 ; 236( 6): 4640-4654.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1002/jcp.30187
Vancouver
Lino CA, Teixeira L de B, Simões SC, Silva T de O, Diniz GP, Costa Neto CM da, Barreto-Chaves MLM. Beta‐arrestin 2 mediates cardiac hypertrophy induced by thyroid hormones via AT1R [Internet]. Journal of Cellular Physiology. 2020 ; 236( 6): 4640-4654.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1002/jcp.30187
Artigo de periodico
Deletion of miRNA-22 induces cardiac hypertrophy in females but attenuates obesogenic diet-mediated metabolic disorders (2020)
Silva, Tábatha de Oliveira ; Lino, Caroline Antunes ; Buzatto, Vanessa C.; Asprino, Paula Fontes ; Luc, Yao Wei; Lima, Vanessa Morais; Fonseca, Renata I. B.; Jensen, Leonardo; Murata, Gilson Masahiro ; Filho, Sidney Verissimo; Ribeiro, Márcio Augusto Campos; Irigoyen, Maria Claudia Costa ; Huangg, Zhan-Peng; Galante, Pedro Alexandre Favoretto ; Wang, Da‐Zhi ; Diniz, Gabriela Placoná ; Donato Junior, José ; Ferreira, Julio Cesar Batista ; Rodrigues, Alice Cristina ; Barreto-Chaves, Maria Luiza Morais
Source: Cellular Physiology and Biochemistry. Unidades: FM, ICB
Subjects: ANATOMIA, FARMACOLOGIA, FISIOLOGIA, OBESIDADE, MICRORNAS, HIPERTROFIA VENTRICULAR ESQUERDA, DOENÇAS METABÓLICAS, ADIPOSIDADE, CAMUNDONGOS, TECIDO ADIPOSO, ECOCARDIOGRAFIA, HORMÔNIOS TIREOIDIANOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Tábatha de Oliveira et al. Deletion of miRNA-22 induces cardiac hypertrophy in females but attenuates obesogenic diet-mediated metabolic disorders. Cellular Physiology and Biochemistry, v. 54, n. 6, p. 1199-1217, 2020Tradução . . Disponível em: https://doi.org/10.33594/000000309. Acesso em: 14 nov. 2024.
APA
Silva, T. de O., Lino, C. A., Buzatto, V. C., Asprino, P. F., Luc, Y. W., Lima, V. M., et al. (2020). Deletion of miRNA-22 induces cardiac hypertrophy in females but attenuates obesogenic diet-mediated metabolic disorders. Cellular Physiology and Biochemistry, 54( 6), 1199-1217. doi:10.33594/000000309
NLM
Silva T de O, Lino CA, Buzatto VC, Asprino PF, Luc YW, Lima VM, Fonseca RIB, Jensen L, Murata GM, Filho SV, Ribeiro MAC, Irigoyen MCC, Huangg Z-P, Galante PAF, Wang D‐Z, Diniz GP, Donato Junior J, Ferreira JCB, Rodrigues AC, Barreto-Chaves MLM. Deletion of miRNA-22 induces cardiac hypertrophy in females but attenuates obesogenic diet-mediated metabolic disorders [Internet]. Cellular Physiology and Biochemistry. 2020 ; 54( 6): 1199-1217.[citado 2024 nov. 14 ] Available from: https://doi.org/10.33594/000000309
Vancouver
Silva T de O, Lino CA, Buzatto VC, Asprino PF, Luc YW, Lima VM, Fonseca RIB, Jensen L, Murata GM, Filho SV, Ribeiro MAC, Irigoyen MCC, Huangg Z-P, Galante PAF, Wang D‐Z, Diniz GP, Donato Junior J, Ferreira JCB, Rodrigues AC, Barreto-Chaves MLM. Deletion of miRNA-22 induces cardiac hypertrophy in females but attenuates obesogenic diet-mediated metabolic disorders [Internet]. Cellular Physiology and Biochemistry. 2020 ; 54( 6): 1199-1217.[citado 2024 nov. 14 ] Available from: https://doi.org/10.33594/000000309
Artigo de periodico
Ubiquitin proteasome system (UPS) activation in the cardiac hypertrophy ofhyperthyroidism (2019)
Lino, Caroline Antunes; Demasi, Marilene; Barreto-Chaves, Maria Luiza Morais
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Subjects: ANATOMIA, METABOLISMO DE PROTEÍNA, HORMÔNIOS TIREOIDIANOS, CORAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LINO, Caroline Antunes e DEMASI, Marilene e BARRETO-CHAVES, Maria Luiza Morais. Ubiquitin proteasome system (UPS) activation in the cardiac hypertrophy ofhyperthyroidism. Molecular and Cellular Endocrinology, v. 493, p. 1-9, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2019.110451. Acesso em: 14 nov. 2024.
APA
Lino, C. A., Demasi, M., & Barreto-Chaves, M. L. M. (2019). Ubiquitin proteasome system (UPS) activation in the cardiac hypertrophy ofhyperthyroidism. Molecular and Cellular Endocrinology, 493, 1-9. doi:10.1016/j.mce.2019.110451
NLM
Lino CA, Demasi M, Barreto-Chaves MLM. Ubiquitin proteasome system (UPS) activation in the cardiac hypertrophy ofhyperthyroidism [Internet]. Molecular and Cellular Endocrinology. 2019 ; 493 1-9.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.mce.2019.110451
Vancouver
Lino CA, Demasi M, Barreto-Chaves MLM. Ubiquitin proteasome system (UPS) activation in the cardiac hypertrophy ofhyperthyroidism [Internet]. Molecular and Cellular Endocrinology. 2019 ; 493 1-9.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.mce.2019.110451
Artigo de periodico
Mutations of cys and Ser residues in the α5-subunit of the 20S proteasome from Saccharomyces cerevisiae affects gating and chronological lifespan (2019)
Leme, Janaina M. M.; Ohar, Erina; Santiago, Veronica Feijoli ; Barros, Mário Henrique de ; Netto, Luis Eduardo Soares ; Pimenta, Daniel Carvalho ; Mariano, Douglas Oscar Ceolin ; Oliveira, Cristiano Luis Pinto de ; Bicev, Renata Naporano ; Barreto-Chaves, Maria Luiza Morais ; Lino, Caroline Antunes ; Demasi, Marilene
Source: Archives of Biochemistry and Biophysics. Unidades: ICB, IF
Subjects: MUTAÇÃO, ANATOMIA, MICROBIOLOGIA, CRONOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LEME, Janaina M. M. et al. Mutations of cys and Ser residues in the α5-subunit of the 20S proteasome from Saccharomyces cerevisiae affects gating and chronological lifespan. Archives of Biochemistry and Biophysics, v. 666, p. 63-72, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.abb.2019.03.012. Acesso em: 14 nov. 2024.
APA
Leme, J. M. M., Ohar, E., Santiago, V. F., Barros, M. H. de, Netto, L. E. S., Pimenta, D. C., et al. (2019). Mutations of cys and Ser residues in the α5-subunit of the 20S proteasome from Saccharomyces cerevisiae affects gating and chronological lifespan. Archives of Biochemistry and Biophysics, 666, 63-72. doi:10.1016/j.abb.2019.03.012
NLM
Leme JMM, Ohar E, Santiago VF, Barros MH de, Netto LES, Pimenta DC, Mariano DOC, Oliveira CLP de, Bicev RN, Barreto-Chaves MLM, Lino CA, Demasi M. Mutations of cys and Ser residues in the α5-subunit of the 20S proteasome from Saccharomyces cerevisiae affects gating and chronological lifespan [Internet]. Archives of Biochemistry and Biophysics. 2019 ; 666 63-72.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.abb.2019.03.012
Vancouver
Leme JMM, Ohar E, Santiago VF, Barros MH de, Netto LES, Pimenta DC, Mariano DOC, Oliveira CLP de, Bicev RN, Barreto-Chaves MLM, Lino CA, Demasi M. Mutations of cys and Ser residues in the α5-subunit of the 20S proteasome from Saccharomyces cerevisiae affects gating and chronological lifespan [Internet]. Archives of Biochemistry and Biophysics. 2019 ; 666 63-72.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.abb.2019.03.012
Artigo de periodico
Angiotensin II type 2 receptor mediates high fat diet-induced cardiomyocyte hypertrophy and hypercholesterolemia (2019)
Lima, Vanessa Morais ; Lino, Caroline Antunes ; Senger, Nathalia ; Silva, Tábatha de Oliveira ; Fonseca, Renata Inzinna Bernardo; Bader, Michael ; Santos, Robson Augusto Sousa ; Donato Junior, José ; Barreto-Chaves, Maria Luiza Morais ; Diniz, Gabriela Placoná
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Subjects: ANATOMIA, FISIOLOGIA, ANGIOTENSINA II, METABOLISMO, OBESIDADE, GLICOSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LIMA, Vanessa Morais et al. Angiotensin II type 2 receptor mediates high fat diet-induced cardiomyocyte hypertrophy and hypercholesterolemia. Molecular and Cellular Endocrinology, v. 498, p. 1-10, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2019.110576. Acesso em: 14 nov. 2024.
APA
Lima, V. M., Lino, C. A., Senger, N., Silva, T. de O., Fonseca, R. I. B., Bader, M., et al. (2019). Angiotensin II type 2 receptor mediates high fat diet-induced cardiomyocyte hypertrophy and hypercholesterolemia. Molecular and Cellular Endocrinology, 498, 1-10. doi:10.1016/j.mce.2019.110576
NLM
Lima VM, Lino CA, Senger N, Silva T de O, Fonseca RIB, Bader M, Santos RAS, Donato Junior J, Barreto-Chaves MLM, Diniz GP. Angiotensin II type 2 receptor mediates high fat diet-induced cardiomyocyte hypertrophy and hypercholesterolemia [Internet]. Molecular and Cellular Endocrinology. 2019 ; 498 1-10.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.mce.2019.110576
Vancouver
Lima VM, Lino CA, Senger N, Silva T de O, Fonseca RIB, Bader M, Santos RAS, Donato Junior J, Barreto-Chaves MLM, Diniz GP. Angiotensin II type 2 receptor mediates high fat diet-induced cardiomyocyte hypertrophy and hypercholesterolemia [Internet]. Molecular and Cellular Endocrinology. 2019 ; 498 1-10.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.mce.2019.110576
Artigo de periodico
MicroRNA-1 overexpression blunts cardiomyocyte hypertrophy elicited by thyroid hormone (2017)
Diniz, Gabriela Placoná ; Lino, Caroline Antunes; Moreno, Camila Rodrigues; Senger, Nathalia; Barreto-Chaves, Maria Luiza Morais
Source: Journal of Cellular Physiology. Unidade: ICB
Subjects: ANATOMIA, HIPERTIREOIDISMO, HORMÔNIOS TIREOIDIANOS, RNA DE TRANSFERÊNCIA, SISTEMA RENINA-ANGIOTENSINA, RATOS WISTAR, BLOQUEIO CARDÍACO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DINIZ, Gabriela Placoná et al. MicroRNA-1 overexpression blunts cardiomyocyte hypertrophy elicited by thyroid hormone. Journal of Cellular Physiology, v. 232, n. 12, p. 3360-3368, 2017Tradução . . Disponível em: https://doi.org/10.1002/jcp.25781. Acesso em: 14 nov. 2024.
APA
Diniz, G. P., Lino, C. A., Moreno, C. R., Senger, N., & Barreto-Chaves, M. L. M. (2017). MicroRNA-1 overexpression blunts cardiomyocyte hypertrophy elicited by thyroid hormone. Journal of Cellular Physiology, 232( 12), 3360-3368. doi:10.1002/jcp.25781
NLM
Diniz GP, Lino CA, Moreno CR, Senger N, Barreto-Chaves MLM. MicroRNA-1 overexpression blunts cardiomyocyte hypertrophy elicited by thyroid hormone [Internet]. Journal of Cellular Physiology. 2017 ; 232( 12): 3360-3368.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1002/jcp.25781
Vancouver
Diniz GP, Lino CA, Moreno CR, Senger N, Barreto-Chaves MLM. MicroRNA-1 overexpression blunts cardiomyocyte hypertrophy elicited by thyroid hormone [Internet]. Journal of Cellular Physiology. 2017 ; 232( 12): 3360-3368.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1002/jcp.25781
Artigo de periodico
MicroRNA expression signature is altered in the cardiac remodeling induced by high fat diets (2016)
Guedes, Elaine Castilho ; França, Gustavo Starvaggi ; Lino, Caroline Antunes ; Koyama, Fernanda Christtanini ; Moreira, Luana do Nascimento; Alexandre, Juliana Gomes ; Galante, Pedro Alexandre Favoretto ; Barreto-Chaves, Maria Luiza Morais ; Diniz, Gabriela Placoná
Source: Journal of Cellular Physiology. Unidades: ICB, IQ
Subjects: ANATOMIA, MICRORNAS, EXPRESSÃO GÊNICA, HIPERTROFIA VENTRICULAR ESQUERDA, MIOCÁRDIO, DIETA ANIMAL, CAMUNDONGOS, GLICEMIA, JEJUNO DE ANIMAL, INSULINA, RNA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GUEDES, Elaine Castilho et al. MicroRNA expression signature is altered in the cardiac remodeling induced by high fat diets. Journal of Cellular Physiology, v. 231, n. 8, p. 1771-1783, 2016Tradução . . Disponível em: https://doi.org/10.1002/jcp.25280. Acesso em: 14 nov. 2024.
APA
Guedes, E. C., França, G. S., Lino, C. A., Koyama, F. C., Moreira, L. do N., Alexandre, J. G., et al. (2016). MicroRNA expression signature is altered in the cardiac remodeling induced by high fat diets. Journal of Cellular Physiology, 231( 8), 1771-1783. doi:10.1002/jcp.25280
NLM
Guedes EC, França GS, Lino CA, Koyama FC, Moreira L do N, Alexandre JG, Galante PAF, Barreto-Chaves MLM, Diniz GP. MicroRNA expression signature is altered in the cardiac remodeling induced by high fat diets [Internet]. Journal of Cellular Physiology. 2016 ; 231( 8): 1771-1783.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1002/jcp.25280
Vancouver
Guedes EC, França GS, Lino CA, Koyama FC, Moreira L do N, Alexandre JG, Galante PAF, Barreto-Chaves MLM, Diniz GP. MicroRNA expression signature is altered in the cardiac remodeling induced by high fat diets [Internet]. Journal of Cellular Physiology. 2016 ; 231( 8): 1771-1783.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1002/jcp.25280
Artigo de periodico
Maternal hyperthyroidism increases the susceptibility of rat adult offspring to cardiovascular disorders (2015)
Lino, Caroline Antunes; Silva, Ivson Bezerra da; Shibata, Caroline Emy Rodrigues; Barreto-Chaves, Maria Luiza Morais
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Subjects: ANATOMIA, HIPERTIREOIDISMO, RATOS WISTAR, DOENÇAS CARDIOVASCULARES EM ANIMAL, SISTEMA RENINA-ANGIOTENSINA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LINO, Caroline Antunes et al. Maternal hyperthyroidism increases the susceptibility of rat adult offspring to cardiovascular disorders. Molecular and Cellular Endocrinology, v. 416, p. 1-8, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2015.08.015. Acesso em: 14 nov. 2024.
APA
Lino, C. A., Silva, I. B. da, Shibata, C. E. R., & Barreto-Chaves, M. L. M. (2015). Maternal hyperthyroidism increases the susceptibility of rat adult offspring to cardiovascular disorders. Molecular and Cellular Endocrinology, 416, 1-8. doi:10.1016/j.mce.2015.08.015
NLM
Lino CA, Silva IB da, Shibata CER, Barreto-Chaves MLM. Maternal hyperthyroidism increases the susceptibility of rat adult offspring to cardiovascular disorders [Internet]. Molecular and Cellular Endocrinology. 2015 ; 416 1-8.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.mce.2015.08.015
Vancouver
Lino CA, Silva IB da, Shibata CER, Barreto-Chaves MLM. Maternal hyperthyroidism increases the susceptibility of rat adult offspring to cardiovascular disorders [Internet]. Molecular and Cellular Endocrinology. 2015 ; 416 1-8.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.mce.2015.08.015
Artigo de periodico
Cardiac microRNA-133 is down-regulated in thyroid hormone-mediated cardiac hypertrophy partially via Type 1 Angiotensin II receptor (2015)
Diniz, Gabriela Placoná ; Lino, Caroline Antunes; Guedes, Elaine Castilho; Moreira, Luana do Nascimento; Barreto-Chaves, Maria Luiza Morais
Source: Basic Research in Cardiology. Unidade: ICB
Subjects: ANATOMIA, HORMÔNIOS TIREOIDIANOS, SISTEMA RENINA-ANGIOTENSINA, ANGIOTENSINA II
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DINIZ, Gabriela Placoná et al. Cardiac microRNA-133 is down-regulated in thyroid hormone-mediated cardiac hypertrophy partially via Type 1 Angiotensin II receptor. Basic Research in Cardiology, v. 110, n. 5, p. 1-11, 2015Tradução . . Disponível em: https://doi.org/10.1007/s00395-015-0504-7. Acesso em: 14 nov. 2024.
APA
Diniz, G. P., Lino, C. A., Guedes, E. C., Moreira, L. do N., & Barreto-Chaves, M. L. M. (2015). Cardiac microRNA-133 is down-regulated in thyroid hormone-mediated cardiac hypertrophy partially via Type 1 Angiotensin II receptor. Basic Research in Cardiology, 110( 5), 1-11. doi:10.1007/s00395-015-0504-7
NLM
Diniz GP, Lino CA, Guedes EC, Moreira L do N, Barreto-Chaves MLM. Cardiac microRNA-133 is down-regulated in thyroid hormone-mediated cardiac hypertrophy partially via Type 1 Angiotensin II receptor [Internet]. Basic Research in Cardiology. 2015 ; 110( 5): 1-11.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1007/s00395-015-0504-7
Vancouver
Diniz GP, Lino CA, Guedes EC, Moreira L do N, Barreto-Chaves MLM. Cardiac microRNA-133 is down-regulated in thyroid hormone-mediated cardiac hypertrophy partially via Type 1 Angiotensin II receptor [Internet]. Basic Research in Cardiology. 2015 ; 110( 5): 1-11.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1007/s00395-015-0504-7
Trabalho de evento-resumo periodico
Influence of maternal hypothyroidism in fetal cardiac renin-angiotensin system (RAS) (2010)
Lino, Caroline Antunes; Shibata, Caroline Emy Rodrigues; Barreto-Chaves, Maria Luiza Morais
Source: Faseb Journal. Conference titles: Experimental Biology Meeting. Unidade: ICB
Assunto: ANATOMIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LINO, Caroline Antunes e SHIBATA, Caroline Emy Rodrigues e BARRETO-CHAVES, Maria Luiza Morais. Influence of maternal hypothyroidism in fetal cardiac renin-angiotensin system (RAS). Faseb Journal. Califórnia: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 14 nov. 2024. , 2010
APA
Lino, C. A., Shibata, C. E. R., & Barreto-Chaves, M. L. M. (2010). Influence of maternal hypothyroidism in fetal cardiac renin-angiotensin system (RAS). Faseb Journal. Califórnia: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
Lino CA, Shibata CER, Barreto-Chaves MLM. Influence of maternal hypothyroidism in fetal cardiac renin-angiotensin system (RAS). Faseb Journal. 2010 ; 24[citado 2024 nov. 14 ]
Vancouver
Lino CA, Shibata CER, Barreto-Chaves MLM. Influence of maternal hypothyroidism in fetal cardiac renin-angiotensin system (RAS). Faseb Journal. 2010 ; 24[citado 2024 nov. 14 ]

USP Schools

ReP

Filtros

Departament

Authors

USP affiliated authors

Date published

Subjects

Source title

Funding Agencies

Indexado em

Non normalized affiliation