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Artigo de periodico-carta/editorial
Oncogenic effects of PAFR ligands produced in tumours upon chemotherapy and radiotherapy (2017)
Chammas, Roger; Andrade, Luciana Nogueira de Sousa; Jancar, Sonia
Source: Nature reviews cancer. Unidades: FM, ICB
Subjects: APOPTOSE, NEOPLASIAS, ONCOGENES, CÉLULAS CULTIVADAS DE TUMOR
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ABNT
CHAMMAS, Roger e ANDRADE, Luciana Nogueira de Sousa e JANCAR, Sonia. Oncogenic effects of PAFR ligands produced in tumours upon chemotherapy and radiotherapy. Nature reviews cancer. London: Faculdade de Medicina, Universidade de São Paulo. Disponível em: https://doi.org/10.1038/nrc.2017.15. Acesso em: 07 nov. 2024. , 2017
APA
Chammas, R., Andrade, L. N. de S., & Jancar, S. (2017). Oncogenic effects of PAFR ligands produced in tumours upon chemotherapy and radiotherapy. Nature reviews cancer. London: Faculdade de Medicina, Universidade de São Paulo. doi:10.1038/nrc.2017.15
NLM
Chammas R, Andrade LN de S, Jancar S. Oncogenic effects of PAFR ligands produced in tumours upon chemotherapy and radiotherapy [Internet]. Nature reviews cancer. 2017 ; 17( 4):[citado 2024 nov. 07 ] Available from: https://doi.org/10.1038/nrc.2017.15
Vancouver
Chammas R, Andrade LN de S, Jancar S. Oncogenic effects of PAFR ligands produced in tumours upon chemotherapy and radiotherapy [Internet]. Nature reviews cancer. 2017 ; 17( 4):[citado 2024 nov. 07 ] Available from: https://doi.org/10.1038/nrc.2017.15
Artigo de periodico
Modulation of tumor-associated macrophages (TAM) phenotype by platelet-activating factor (PAF) receptor (2017)
Silva Junior, Ildefonso Alves da; Stone, Simone Cardozo; Rossetti, Renata Ariza Marques; Jancar, Sonia ; Lepique, Ana Paula
Source: Journal of Immunology Research. Unidade: ICB
Subjects: IMUNOLOGIA, MACRÓFAGOS, PLAQUETAS SANGUÍNEAS, CÉLULAS CULTIVADAS DE TUMOR, NEUTRÓFILOS, LINFÓCITOS
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SILVA JUNIOR, Ildefonso Alves da et al. Modulation of tumor-associated macrophages (TAM) phenotype by platelet-activating factor (PAF) receptor. Journal of Immunology Research, v. 2017, p. 1-10, 2017Tradução . . Disponível em: https://doi.org/10.1155/2017/5482768. Acesso em: 07 nov. 2024.
APA
Silva Junior, I. A. da, Stone, S. C., Rossetti, R. A. M., Jancar, S., & Lepique, A. P. (2017). Modulation of tumor-associated macrophages (TAM) phenotype by platelet-activating factor (PAF) receptor. Journal of Immunology Research, 2017, 1-10. doi:10.1155/2017/5482768
NLM
Silva Junior IA da, Stone SC, Rossetti RAM, Jancar S, Lepique AP. Modulation of tumor-associated macrophages (TAM) phenotype by platelet-activating factor (PAF) receptor [Internet]. Journal of Immunology Research. 2017 ; 2017 1-10.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1155/2017/5482768
Vancouver
Silva Junior IA da, Stone SC, Rossetti RAM, Jancar S, Lepique AP. Modulation of tumor-associated macrophages (TAM) phenotype by platelet-activating factor (PAF) receptor [Internet]. Journal of Immunology Research. 2017 ; 2017 1-10.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1155/2017/5482768
Artigo de periodico
Platelet-activation factor receptor induces interleukin 10 production through STAT3 activation in dendritic cells (2017)
Koga, Marianna Mainardi; Ribeiro Junior, Luciano Filgueiras; Jancar, Sonia ; Rios, Francisco J.
Source: Journal of Immunobiology. Unidade: ICB
Subjects: IMUNOLOGIA, INTERLEUCINA 10, CÉLULAS DENDRÍTICAS, PLAQUETAS SANGUÍNEAS, MACRÓFAGOS, PROSTAGLANDINAS
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KOGA, Marianna Mainardi et al. Platelet-activation factor receptor induces interleukin 10 production through STAT3 activation in dendritic cells. Journal of Immunobiology, v. 2, n. 2, p. 1-9, 2017Tradução . . Disponível em: https://doi.org/10.4172/2476-1966.1000123. Acesso em: 07 nov. 2024.
APA
Koga, M. M., Ribeiro Junior, L. F., Jancar, S., & Rios, F. J. (2017). Platelet-activation factor receptor induces interleukin 10 production through STAT3 activation in dendritic cells. Journal of Immunobiology, 2( 2), 1-9. doi:10.4172/2476-1966.1000123
NLM
Koga MM, Ribeiro Junior LF, Jancar S, Rios FJ. Platelet-activation factor receptor induces interleukin 10 production through STAT3 activation in dendritic cells [Internet]. Journal of Immunobiology. 2017 ; 2( 2): 1-9.[citado 2024 nov. 07 ] Available from: https://doi.org/10.4172/2476-1966.1000123
Vancouver
Koga MM, Ribeiro Junior LF, Jancar S, Rios FJ. Platelet-activation factor receptor induces interleukin 10 production through STAT3 activation in dendritic cells [Internet]. Journal of Immunobiology. 2017 ; 2( 2): 1-9.[citado 2024 nov. 07 ] Available from: https://doi.org/10.4172/2476-1966.1000123
Artigo de periodico
Imbalance between HDAC and HAT activities drives aberrante STAT1/MyD88 expression in macrophages from type 1 diabetic mice (2017)
Filgueiras, Luciano Ribeiro; Brandt, Stephanie L.; Ramalho, Theresa Raquel de Oliveira; Jancar, Sonia ; Serezani, C. Henrique
Source: Journal of Diabetes and its Complications. Unidade: ICB
Assunto: IMUNOLOGIA
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FILGUEIRAS, Luciano Ribeiro et al. Imbalance between HDAC and HAT activities drives aberrante STAT1/MyD88 expression in macrophages from type 1 diabetic mice. Journal of Diabetes and its Complications, v. 31, n. 2, p. 334-339, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.jdiacomp.2016.08.001. Acesso em: 07 nov. 2024.
APA
Filgueiras, L. R., Brandt, S. L., Ramalho, T. R. de O., Jancar, S., & Serezani, C. H. (2017). Imbalance between HDAC and HAT activities drives aberrante STAT1/MyD88 expression in macrophages from type 1 diabetic mice. Journal of Diabetes and its Complications, 31( 2), 334-339. doi:10.1016/j.jdiacomp.2016.08.001
NLM
Filgueiras LR, Brandt SL, Ramalho TR de O, Jancar S, Serezani CH. Imbalance between HDAC and HAT activities drives aberrante STAT1/MyD88 expression in macrophages from type 1 diabetic mice [Internet]. Journal of Diabetes and its Complications. 2017 ; 31( 2): 334-339.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.jdiacomp.2016.08.001
Vancouver
Filgueiras LR, Brandt SL, Ramalho TR de O, Jancar S, Serezani CH. Imbalance between HDAC and HAT activities drives aberrante STAT1/MyD88 expression in macrophages from type 1 diabetic mice [Internet]. Journal of Diabetes and its Complications. 2017 ; 31( 2): 334-339.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.jdiacomp.2016.08.001
Artigo de periodico
PAFR in adipose tissue macrophages is associated with anti-inflammatory phenotype and metabolic homoeostasis (2016)
Filgueiras, Luciano Ribeiro; Koga, Marianna Mainardi; Quaresma, Paula G.; Ishizuka, Edson Kiyotaka; Montes, Marlise B. A.; Prada, Patricia O.; Saad, Mario J.; Jancar, Sonia ; Rios, Francisco J.
Source: Clinical Science. Unidade: ICB
Assunto: IMUNOLOGIA
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FILGUEIRAS, Luciano Ribeiro et al. PAFR in adipose tissue macrophages is associated with anti-inflammatory phenotype and metabolic homoeostasis. Clinical Science, v. 130, n. 8, p. 601-612, 2016Tradução . . Disponível em: https://doi.org/10.1042/CS20150538. Acesso em: 07 nov. 2024.
APA
Filgueiras, L. R., Koga, M. M., Quaresma, P. G., Ishizuka, E. K., Montes, M. B. A., Prada, P. O., et al. (2016). PAFR in adipose tissue macrophages is associated with anti-inflammatory phenotype and metabolic homoeostasis. Clinical Science, 130( 8), 601-612. doi:10.1042/CS20150538
NLM
Filgueiras LR, Koga MM, Quaresma PG, Ishizuka EK, Montes MBA, Prada PO, Saad MJ, Jancar S, Rios FJ. PAFR in adipose tissue macrophages is associated with anti-inflammatory phenotype and metabolic homoeostasis [Internet]. Clinical Science. 2016 ; 130( 8): 601-612.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1042/CS20150538
Vancouver
Filgueiras LR, Koga MM, Quaresma PG, Ishizuka EK, Montes MBA, Prada PO, Saad MJ, Jancar S, Rios FJ. PAFR in adipose tissue macrophages is associated with anti-inflammatory phenotype and metabolic homoeostasis [Internet]. Clinical Science. 2016 ; 130( 8): 601-612.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1042/CS20150538
Artigo de periodico
Boosting adaptive immunity:: a new role for PAFR antagonists (2016)
Koga, Marianna M.; Bizarro, Bruna; Nunes, Anderson de Sá ; Rios, Francisco J.; Jancar, Sonia
Source: Scientific Reports. Unidade: ICB
Assunto: IMUNOLOGIA
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KOGA, Marianna M. et al. Boosting adaptive immunity:: a new role for PAFR antagonists. Scientific Reports, v. 6, n. 39146, p. 1-9, 2016Tradução . . Disponível em: https://doi.org/10.1038/srep39146. Acesso em: 07 nov. 2024.
APA
Koga, M. M., Bizarro, B., Nunes, A. de S., Rios, F. J., & Jancar, S. (2016). Boosting adaptive immunity:: a new role for PAFR antagonists. Scientific Reports, 6( 39146), 1-9. doi:10.1038/srep39146
NLM
Koga MM, Bizarro B, Nunes A de S, Rios FJ, Jancar S. Boosting adaptive immunity:: a new role for PAFR antagonists [Internet]. Scientific Reports. 2016 ; 6( 39146): 1-9.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1038/srep39146
Vancouver
Koga MM, Bizarro B, Nunes A de S, Rios FJ, Jancar S. Boosting adaptive immunity:: a new role for PAFR antagonists [Internet]. Scientific Reports. 2016 ; 6( 39146): 1-9.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1038/srep39146
Artigo de periodico
PAFR activation of NF-κB p65 or p105 precursor dictates pro- and anti-inflammatory responses during TLR activation in murine macrophages (2016)
Ishizuka, Edson Kiyotaka; Ribeiro Junior, Luciano Filgueiras; Rios, Francisco José Oliveira; Serezani, Carlos Henrique Cardoso; Jancar, Sonia
Source: Scientific Reports. Unidade: ICB
Subjects: MACRÓFAGOS, RECEPTORES, INFLAMAÇÃO, IMUNOLOGIA
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ISHIZUKA, Edson Kiyotaka et al. PAFR activation of NF-κB p65 or p105 precursor dictates pro- and anti-inflammatory responses during TLR activation in murine macrophages. Scientific Reports, v. 6, n. 32092, p. 11 , 2016Tradução . . Disponível em: https://doi.org/10.1038/srep32092. Acesso em: 07 nov. 2024.
APA
Ishizuka, E. K., Ribeiro Junior, L. F., Rios, F. J. O., Serezani, C. H. C., & Jancar, S. (2016). PAFR activation of NF-κB p65 or p105 precursor dictates pro- and anti-inflammatory responses during TLR activation in murine macrophages. Scientific Reports, 6( 32092), 11 . doi:10.1038/srep32092
NLM
Ishizuka EK, Ribeiro Junior LF, Rios FJO, Serezani CHC, Jancar S. PAFR activation of NF-κB p65 or p105 precursor dictates pro- and anti-inflammatory responses during TLR activation in murine macrophages [Internet]. Scientific Reports. 2016 ; 6( 32092): 11 .[citado 2024 nov. 07 ] Available from: https://doi.org/10.1038/srep32092
Vancouver
Ishizuka EK, Ribeiro Junior LF, Rios FJO, Serezani CHC, Jancar S. PAFR activation of NF-κB p65 or p105 precursor dictates pro- and anti-inflammatory responses during TLR activation in murine macrophages [Internet]. Scientific Reports. 2016 ; 6( 32092): 11 .[citado 2024 nov. 07 ] Available from: https://doi.org/10.1038/srep32092
Artigo de periodico
Leukotriene B4-mediated sterile inflammation promotes susceptibility to sepsis in a mouse model of type 1 diabetes (2015)
Filgueiras, Luciano Ribeiro; Brandt, Stephanie L.; Wang, Soujuan; Wang, Zhuo; Morris, David L.; Evans-Molina, Carmella; Mirmira, Raghavendra G.; Jancar, Sonia ; Serezani, Carlos Henrique Cardoso
Source: Science Signaling. Unidade: ICB
Subjects: IMUNOLOGIA, LEUCOTRIENOS B, INFLAMAÇÃO, SEPSE, CAMUNDONGOS
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FILGUEIRAS, Luciano Ribeiro et al. Leukotriene B4-mediated sterile inflammation promotes susceptibility to sepsis in a mouse model of type 1 diabetes. Science Signaling, v. 8, n. 361, p. 1-9, 2015Tradução . . Disponível em: https://doi.org/10.1126/scisignal.2005568. Acesso em: 07 nov. 2024.
APA
Filgueiras, L. R., Brandt, S. L., Wang, S., Wang, Z., Morris, D. L., Evans-Molina, C., et al. (2015). Leukotriene B4-mediated sterile inflammation promotes susceptibility to sepsis in a mouse model of type 1 diabetes. Science Signaling, 8( 361), 1-9. doi:10.1126/scisignal.2005568
NLM
Filgueiras LR, Brandt SL, Wang S, Wang Z, Morris DL, Evans-Molina C, Mirmira RG, Jancar S, Serezani CHC. Leukotriene B4-mediated sterile inflammation promotes susceptibility to sepsis in a mouse model of type 1 diabetes [Internet]. Science Signaling. 2015 ; 8( 361): 1-9.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1126/scisignal.2005568
Vancouver
Filgueiras LR, Brandt SL, Wang S, Wang Z, Morris DL, Evans-Molina C, Mirmira RG, Jancar S, Serezani CHC. Leukotriene B4-mediated sterile inflammation promotes susceptibility to sepsis in a mouse model of type 1 diabetes [Internet]. Science Signaling. 2015 ; 8( 361): 1-9.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1126/scisignal.2005568
Artigo de periodico
Essential role of leukotriene B4 on Leishmania (Viannia) braziliensis killing by human macrophages (2014)
Morato, Camila I.; Silva Jr, Ildefonso A. da; Borges, Arissa F.; Dorta, Miriam L.; Oliveira, Milton A. P.; Jancar, Sonia ; Serezani, Carlos H.; Ribeiro-Dias, Fátima
Source: Microbes and Infection. Unidade: ICB
Subjects: IMUNOLOGIA, LEISHMANIA BRASILIENSIS, MACRÓFAGOS, LEUCOTRIENOS
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MORATO, Camila I. et al. Essential role of leukotriene B4 on Leishmania (Viannia) braziliensis killing by human macrophages. Microbes and Infection, v. 16, n. 11, p. 945-953, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2014.08.015. Acesso em: 07 nov. 2024.
APA
Morato, C. I., Silva Jr, I. A. da, Borges, A. F., Dorta, M. L., Oliveira, M. A. P., Jancar, S., et al. (2014). Essential role of leukotriene B4 on Leishmania (Viannia) braziliensis killing by human macrophages. Microbes and Infection, 16( 11), 945-953. doi:10.1016/j.micinf.2014.08.015
NLM
Morato CI, Silva Jr IA da, Borges AF, Dorta ML, Oliveira MAP, Jancar S, Serezani CH, Ribeiro-Dias F. Essential role of leukotriene B4 on Leishmania (Viannia) braziliensis killing by human macrophages [Internet]. Microbes and Infection. 2014 ; 16( 11): 945-953.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.micinf.2014.08.015
Vancouver
Morato CI, Silva Jr IA da, Borges AF, Dorta ML, Oliveira MAP, Jancar S, Serezani CH, Ribeiro-Dias F. Essential role of leukotriene B4 on Leishmania (Viannia) braziliensis killing by human macrophages [Internet]. Microbes and Infection. 2014 ; 16( 11): 945-953.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.micinf.2014.08.015
Artigo de periodico
Lung remodeling in a mouse model of asthma involves a balance between TGF-β1 and BMP-7 (2014)
Stumm, Camila Leindecker; Halcsik, Erik; Landgraf, Richardt Gama; Câmara, Niels Olsen Saraiva ; Sogayar, Mari Cleide ; Jancar, Sonia
Source: PLOS ONE. Unidades: ICB, IQ
Subjects: ASMA, FIBROBLASTOS
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STUMM, Camila Leindecker et al. Lung remodeling in a mouse model of asthma involves a balance between TGF-β1 and BMP-7. PLOS ONE, v. 9, n. 4, p. 1-9 art. e95959, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0095959. Acesso em: 07 nov. 2024.
APA
Stumm, C. L., Halcsik, E., Landgraf, R. G., Câmara, N. O. S., Sogayar, M. C., & Jancar, S. (2014). Lung remodeling in a mouse model of asthma involves a balance between TGF-β1 and BMP-7. PLOS ONE, 9( 4), 1-9 art. e95959. doi:10.1371/journal.pone.0095959
NLM
Stumm CL, Halcsik E, Landgraf RG, Câmara NOS, Sogayar MC, Jancar S. Lung remodeling in a mouse model of asthma involves a balance between TGF-β1 and BMP-7 [Internet]. PLOS ONE. 2014 ; 9( 4): 1-9 art. e95959.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1371/journal.pone.0095959
Vancouver
Stumm CL, Halcsik E, Landgraf RG, Câmara NOS, Sogayar MC, Jancar S. Lung remodeling in a mouse model of asthma involves a balance between TGF-β1 and BMP-7 [Internet]. PLOS ONE. 2014 ; 9( 4): 1-9 art. e95959.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1371/journal.pone.0095959
Artigo de periodico
PAF receptor and tumor growth (2014)
Jancar, Sonia ; Chammas, Roger
Source: Current Drug Targets. Unidades: ICB, FM
Subjects: IMUNOLOGIA, CÉLULAS CULTIVADAS DE TUMOR, PLAQUETAS SANGUÍNEAS
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JANCAR, Sonia e CHAMMAS, Roger. PAF receptor and tumor growth. Current Drug Targets, v. 15, n. 10, p. 982-987, 2014Tradução . . Acesso em: 07 nov. 2024.
APA
Jancar, S., & Chammas, R. (2014). PAF receptor and tumor growth. Current Drug Targets, 15( 10), 982-987.
NLM
Jancar S, Chammas R. PAF receptor and tumor growth. Current Drug Targets. 2014 ; 15( 10): 982-987.[citado 2024 nov. 07 ]
Vancouver
Jancar S, Chammas R. PAF receptor and tumor growth. Current Drug Targets. 2014 ; 15( 10): 982-987.[citado 2024 nov. 07 ]
Artigo de periodico
Leukotriene B4 enhances the generation of proinflammatory MicroRNAs to promote MyD88-dependent macrophage activation (2014)
Wang, Zhuo; Filgueiras, Luciano Ribeiro; Wang, Soujuan; Serezani, Ana Paula Moreira; Peters-Golden, Marc; Jancar, Sonia ; Serezani, C. Henrique
Source: Journal of Immunology. Unidade: ICB
Subjects: IMUNOLOGIA, LEUCOTRIENOS B, MACRÓFAGOS
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ABNT
WANG, Zhuo et al. Leukotriene B4 enhances the generation of proinflammatory MicroRNAs to promote MyD88-dependent macrophage activation. Journal of Immunology, v. 192, n. 5, p. 2349-2356, 2014Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1302982. Acesso em: 07 nov. 2024.
APA
Wang, Z., Filgueiras, L. R., Wang, S., Serezani, A. P. M., Peters-Golden, M., Jancar, S., & Serezani, C. H. (2014). Leukotriene B4 enhances the generation of proinflammatory MicroRNAs to promote MyD88-dependent macrophage activation. Journal of Immunology, 192( 5), 2349-2356. doi:10.4049/jimmunol.1302982
NLM
Wang Z, Filgueiras LR, Wang S, Serezani APM, Peters-Golden M, Jancar S, Serezani CH. Leukotriene B4 enhances the generation of proinflammatory MicroRNAs to promote MyD88-dependent macrophage activation [Internet]. Journal of Immunology. 2014 ; 192( 5): 2349-2356.[citado 2024 nov. 07 ] Available from: https://doi.org/10.4049/jimmunol.1302982
Vancouver
Wang Z, Filgueiras LR, Wang S, Serezani APM, Peters-Golden M, Jancar S, Serezani CH. Leukotriene B4 enhances the generation of proinflammatory MicroRNAs to promote MyD88-dependent macrophage activation [Internet]. Journal of Immunology. 2014 ; 192( 5): 2349-2356.[citado 2024 nov. 07 ] Available from: https://doi.org/10.4049/jimmunol.1302982
Artigo de periodico
Activation of platelet-activating factor receptor exacerbates renal inflammation and promotes fibrosis (2014)
Correa-Costa, Matheus; Andrade-Oliveira, Vinicius; Braga, Tárcio Teodoro; Castoldi, Angela; Aguiar, Cristhiane Favero de; Origassa, Clarice S. T.; Rodas, Andrea C. D.; Hiyane, Meire Ioshie; Malheiros, Denise Maria Avancini Costa; Rios, Francisco J. O.; Jancar, Sonia ; Câmara, Niels Olsen Saraiva
Source: Laboratory Investigation. Unidades: ICB, FM
Subjects: IMUNOLOGIA, PLAQUETAS SANGUINEAS, INFLAMAÇÃO
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ABNT
CORREA-COSTA, Matheus et al. Activation of platelet-activating factor receptor exacerbates renal inflammation and promotes fibrosis. Laboratory Investigation, v. 94, n. 4, p. 455-466, 2014Tradução . . Disponível em: https://doi.org/10.1038/labinvest.2013.155. Acesso em: 07 nov. 2024.
APA
Correa-Costa, M., Andrade-Oliveira, V., Braga, T. T., Castoldi, A., Aguiar, C. F. de, Origassa, C. S. T., et al. (2014). Activation of platelet-activating factor receptor exacerbates renal inflammation and promotes fibrosis. Laboratory Investigation, 94( 4), 455-466. doi:10.1038/labinvest.2013.155
NLM
Correa-Costa M, Andrade-Oliveira V, Braga TT, Castoldi A, Aguiar CF de, Origassa CST, Rodas ACD, Hiyane MI, Malheiros DMAC, Rios FJO, Jancar S, Câmara NOS. Activation of platelet-activating factor receptor exacerbates renal inflammation and promotes fibrosis [Internet]. Laboratory Investigation. 2014 ; 94( 4): 455-466.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1038/labinvest.2013.155
Vancouver
Correa-Costa M, Andrade-Oliveira V, Braga TT, Castoldi A, Aguiar CF de, Origassa CST, Rodas ACD, Hiyane MI, Malheiros DMAC, Rios FJO, Jancar S, Câmara NOS. Activation of platelet-activating factor receptor exacerbates renal inflammation and promotes fibrosis [Internet]. Laboratory Investigation. 2014 ; 94( 4): 455-466.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1038/labinvest.2013.155
Artigo de periodico
Oxidized LDL induces alternative macrophage phenotype through activation of CD36 and PAFR (2013)
Rios, Francisco J.; Koga, Marianna Mainardi; Pecenin, Mateus; Ferracini, Matheus; Gidlund, Magnus Ake ; Jancar, Sonia
Source: Mediators of Inflammation. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
RIOS, Francisco J. et al. Oxidized LDL induces alternative macrophage phenotype through activation of CD36 and PAFR. Mediators of Inflammation, v. 2013, p. 1-8, 2013Tradução . . Disponível em: https://doi.org/10.1155/2013/198193. Acesso em: 07 nov. 2024.
APA
Rios, F. J., Koga, M. M., Pecenin, M., Ferracini, M., Gidlund, M. A., & Jancar, S. (2013). Oxidized LDL induces alternative macrophage phenotype through activation of CD36 and PAFR. Mediators of Inflammation, 2013, 1-8. doi:10.1155/2013/198193
NLM
Rios FJ, Koga MM, Pecenin M, Ferracini M, Gidlund MA, Jancar S. Oxidized LDL induces alternative macrophage phenotype through activation of CD36 and PAFR [Internet]. Mediators of Inflammation. 2013 ; 2013 1-8.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1155/2013/198193
Vancouver
Rios FJ, Koga MM, Pecenin M, Ferracini M, Gidlund MA, Jancar S. Oxidized LDL induces alternative macrophage phenotype through activation of CD36 and PAFR [Internet]. Mediators of Inflammation. 2013 ; 2013 1-8.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1155/2013/198193
Artigo de periodico
Clearance of apoptotic cells by macrophages induces regulatory phenotype and involves stimulation of CD36 and platelet-activating factor receptor (2013)
Ferracini, Matheus; Rios, Francisco J. O.; Pecenin, Mateus; Jancar, Sonia
Source: Mediators of Inflammation. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
FERRACINI, Matheus et al. Clearance of apoptotic cells by macrophages induces regulatory phenotype and involves stimulation of CD36 and platelet-activating factor receptor. Mediators of Inflammation, v. 2013, p. 1-8, 2013Tradução . . Disponível em: https://doi.org/10.1155/2013/950273. Acesso em: 07 nov. 2024.
APA
Ferracini, M., Rios, F. J. O., Pecenin, M., & Jancar, S. (2013). Clearance of apoptotic cells by macrophages induces regulatory phenotype and involves stimulation of CD36 and platelet-activating factor receptor. Mediators of Inflammation, 2013, 1-8. doi:10.1155/2013/950273
NLM
Ferracini M, Rios FJO, Pecenin M, Jancar S. Clearance of apoptotic cells by macrophages induces regulatory phenotype and involves stimulation of CD36 and platelet-activating factor receptor [Internet]. Mediators of Inflammation. 2013 ; 2013 1-8.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1155/2013/950273
Vancouver
Ferracini M, Rios FJO, Pecenin M, Jancar S. Clearance of apoptotic cells by macrophages induces regulatory phenotype and involves stimulation of CD36 and platelet-activating factor receptor [Internet]. Mediators of Inflammation. 2013 ; 2013 1-8.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1155/2013/950273
Artigo de periodico
Uptake of oxLDL and IL-10 production by macrophages requires PAFR and CD36 recruitment into the same lipid rafts (2013)
Francisco J. O. Rios; Ferracini, Matheus; Pecenin, Mateus; Koga, Marianna M.; Wang, Yajuan; Ketelhuth, Daniel F. J.; Jancar, Sonia
Source: PLOS ONE. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FRANCISCO J. O. RIOS, et al. Uptake of oxLDL and IL-10 production by macrophages requires PAFR and CD36 recruitment into the same lipid rafts. PLOS ONE, v. 8, n. 10, p. 1-13, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0076893. Acesso em: 07 nov. 2024.
APA
Francisco J. O. Rios,, Ferracini, M., Pecenin, M., Koga, M. M., Wang, Y., Ketelhuth, D. F. J., & Jancar, S. (2013). Uptake of oxLDL and IL-10 production by macrophages requires PAFR and CD36 recruitment into the same lipid rafts. PLOS ONE, 8( 10), 1-13. doi:10.1371/journal.pone.0076893
NLM
Francisco J. O. Rios, Ferracini M, Pecenin M, Koga MM, Wang Y, Ketelhuth DFJ, Jancar S. Uptake of oxLDL and IL-10 production by macrophages requires PAFR and CD36 recruitment into the same lipid rafts [Internet]. PLOS ONE. 2013 ;8( 10): 1-13.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1371/journal.pone.0076893
Vancouver
Francisco J. O. Rios, Ferracini M, Pecenin M, Koga MM, Wang Y, Ketelhuth DFJ, Jancar S. Uptake of oxLDL and IL-10 production by macrophages requires PAFR and CD36 recruitment into the same lipid rafts [Internet]. PLOS ONE. 2013 ;8( 10): 1-13.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1371/journal.pone.0076893
Artigo de periodico
Sepsis-induced acute lung injury (ALI) is milder in diabetic rats and correlates with impaired NFkB activation (2012)
Filgueiras Jr, Luciano R.; Martins, Joilson de Oliveira ; Serezani, Carlos H.; Capelozzi, Vera Luiza; Montes, Marlise Bonetti Agostinho; Jancar, Sonia
Source: PLOS ONE. Unidades: ICB, FCF, FM
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FILGUEIRAS JR, Luciano R. et al. Sepsis-induced acute lung injury (ALI) is milder in diabetic rats and correlates with impaired NFkB activation. PLOS ONE, v. 7, n. 9, p. art. e44987 1-9, 2012Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0044987. Acesso em: 07 nov. 2024.
APA
Filgueiras Jr, L. R., Martins, J. de O., Serezani, C. H., Capelozzi, V. L., Montes, M. B. A., & Jancar, S. (2012). Sepsis-induced acute lung injury (ALI) is milder in diabetic rats and correlates with impaired NFkB activation. PLOS ONE, 7( 9), art. e44987 1-9. doi:10.1371/journal.pone.0044987
NLM
Filgueiras Jr LR, Martins J de O, Serezani CH, Capelozzi VL, Montes MBA, Jancar S. Sepsis-induced acute lung injury (ALI) is milder in diabetic rats and correlates with impaired NFkB activation [Internet]. PLOS ONE. 2012 ; 7( 9): art. e44987 1-9.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1371/journal.pone.0044987
Vancouver
Filgueiras Jr LR, Martins J de O, Serezani CH, Capelozzi VL, Montes MBA, Jancar S. Sepsis-induced acute lung injury (ALI) is milder in diabetic rats and correlates with impaired NFkB activation [Internet]. PLOS ONE. 2012 ; 7( 9): art. e44987 1-9.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1371/journal.pone.0044987
Artigo de periodico
Co-stimulation of PAFR and CD36 is required for oxLDLinduced human macrophages activation (2012)
Rios, Francisco José Oliveira; Koga, Mariana Mainardi; Ferracini, Matheus; Jancar, Sonia
Source: PLOS ONE. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RIOS, Francisco José Oliveira et al. Co-stimulation of PAFR and CD36 is required for oxLDLinduced human macrophages activation. PLOS ONE, v. 7, n. 5, p. 1-10, 2012Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0036632. Acesso em: 07 nov. 2024.
APA
Rios, F. J. O., Koga, M. M., Ferracini, M., & Jancar, S. (2012). Co-stimulation of PAFR and CD36 is required for oxLDLinduced human macrophages activation. PLOS ONE, 7( 5), 1-10. doi:10.1371/journal.pone.0036632
NLM
Rios FJO, Koga MM, Ferracini M, Jancar S. Co-stimulation of PAFR and CD36 is required for oxLDLinduced human macrophages activation [Internet]. PLOS ONE. 2012 ; 7( 5): 1-10.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1371/journal.pone.0036632
Vancouver
Rios FJO, Koga MM, Ferracini M, Jancar S. Co-stimulation of PAFR and CD36 is required for oxLDLinduced human macrophages activation [Internet]. PLOS ONE. 2012 ; 7( 5): 1-10.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1371/journal.pone.0036632
Artigo de periodico
Bradykinin B1 receptor antagonist R954 inhibits eosinophil activation/proliferation/migration and increase TGF-β and VEGF in a murine model of asthma (2010)
Vasquez-Pinto, Luciana Miranda Chaves.; Nantel, Francois; Sirois, Pierre; Jancar, Sonia
Source: Neuropeptides. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VASQUEZ-PINTO, Luciana Miranda Chaves. et al. Bradykinin B1 receptor antagonist R954 inhibits eosinophil activation/proliferation/migration and increase TGF-β and VEGF in a murine model of asthma. Neuropeptides, v. 44, n. 2, p. 107-113, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.npep.2009.11.001. Acesso em: 07 nov. 2024.
APA
Vasquez-Pinto, L. M. C., Nantel, F., Sirois, P., & Jancar, S. (2010). Bradykinin B1 receptor antagonist R954 inhibits eosinophil activation/proliferation/migration and increase TGF-β and VEGF in a murine model of asthma. Neuropeptides, 44( 2), 107-113. doi:10.1016/j.npep.2009.11.001
NLM
Vasquez-Pinto LMC, Nantel F, Sirois P, Jancar S. Bradykinin B1 receptor antagonist R954 inhibits eosinophil activation/proliferation/migration and increase TGF-β and VEGF in a murine model of asthma [Internet]. Neuropeptides. 2010 ; 44( 2): 107-113.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.npep.2009.11.001
Vancouver
Vasquez-Pinto LMC, Nantel F, Sirois P, Jancar S. Bradykinin B1 receptor antagonist R954 inhibits eosinophil activation/proliferation/migration and increase TGF-β and VEGF in a murine model of asthma [Internet]. Neuropeptides. 2010 ; 44( 2): 107-113.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1016/j.npep.2009.11.001

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