Imbalance between HDAC and HAT activities drives aberrante STAT1/MyD88 expression in macrophages from type 1 diabetic mice (2017)
- Authors:
- Autor USP: NEGRO, SONIA JANCAR - ICB
- Unidade: ICB
- DOI: 10.1016/j.jdiacomp.2016.08.001
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Publisher place: Philadelphia
- Date published: 2017
- Source:
- Título: Journal of Diabetes and its Complications
- ISSN: 1873-460X
- Volume/Número/Paginação/Ano: v. 31, n. 2, p. 334-339, 2017
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: green
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ABNT
FILGUEIRAS, Luciano Ribeiro et al. Imbalance between HDAC and HAT activities drives aberrante STAT1/MyD88 expression in macrophages from type 1 diabetic mice. Journal of Diabetes and its Complications, v. 31, n. 2, p. 334-339, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.jdiacomp.2016.08.001. Acesso em: 06 out. 2024. -
APA
Filgueiras, L. R., Brandt, S. L., Ramalho, T. R. de O., Jancar, S., & Serezani, C. H. (2017). Imbalance between HDAC and HAT activities drives aberrante STAT1/MyD88 expression in macrophages from type 1 diabetic mice. Journal of Diabetes and its Complications, 31( 2), 334-339. doi:10.1016/j.jdiacomp.2016.08.001 -
NLM
Filgueiras LR, Brandt SL, Ramalho TR de O, Jancar S, Serezani CH. Imbalance between HDAC and HAT activities drives aberrante STAT1/MyD88 expression in macrophages from type 1 diabetic mice [Internet]. Journal of Diabetes and its Complications. 2017 ; 31( 2): 334-339.[citado 2024 out. 06 ] Available from: https://doi.org/10.1016/j.jdiacomp.2016.08.001 -
Vancouver
Filgueiras LR, Brandt SL, Ramalho TR de O, Jancar S, Serezani CH. Imbalance between HDAC and HAT activities drives aberrante STAT1/MyD88 expression in macrophages from type 1 diabetic mice [Internet]. Journal of Diabetes and its Complications. 2017 ; 31( 2): 334-339.[citado 2024 out. 06 ] Available from: https://doi.org/10.1016/j.jdiacomp.2016.08.001 - Are contractions of cerebral vessels induced by arachidonic acid dependent on the presence of intact endothelium?
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Informações sobre o DOI: 10.1016/j.jdiacomp.2016.08.001 (Fonte: oaDOI API)
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