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Artigo de periodico
Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients (2023)
Amaral, Natália Brasil do; Rodrigues, Tamara Silva; Giannini, Marcela Cavichioli; Lopes, Maria Isabel Fernandes; Bonjorno, Letícia Pastorelli; Menezes, Pamella Indira da Silva Oliveira; Dib, Saulo Musse; Gigante, Samara Libich Gusmão; Benatti, Maíra Nilson; Rezek, Uebe C.; Emrich-Filho, Laerte L.; Sousa, Betania A. A.; Almeida, Sérgio Couto Luna de; Luppino-Assad, Rodrigo; Veras, Flávio Protásio ; Schneider, Ayda Henriques; Leiria, Luiz Osório Silveira ; Cunha, Larissa Dias da; Alves Filho, José Carlos Farias ; Cunha, Thiago Mattar ; Arruda Neto, Eurico de; Miranda, Carlos Henrique ; Pazin Filho, Antônio ; Martins, Maria Auxiliadora ; Borges, Marcos de Carvalho ; Fonseca, Benedito Antonio Lopes da ; Bollela, Valdes Roberto ; Del-Ben, Cristina Marta ; Cunha, Fernando de Queiroz ; Santana, Rodrigo de Carvalho ; Vilar, Fernando Crivelenti; Zamboni, Dario Simões ; Louzada Júnior, Paulo ; Oliveira, Renê Donizeti Ribeiro de
Source: Inflammation Research. Unidade: FMRP
Subjects: COVID-19, CITOCINAS, PROTEÍNAS, ALCALOIDES
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ABNT
AMARAL, Natália Brasil do et al. Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients. Inflammation Research, v. 72, n. 5, p. 895-899, 2023Tradução . . Disponível em: https://doi.org/10.1007/s00011-023-01718-y. Acesso em: 22 ago. 2024.
APA
Amaral, N. B. do, Rodrigues, T. S., Giannini, M. C., Lopes, M. I. F., Bonjorno, L. P., Menezes, P. I. da S. O., et al. (2023). Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients. Inflammation Research, 72( 5), 895-899. doi:10.1007/s00011-023-01718-y
NLM
Amaral NB do, Rodrigues TS, Giannini MC, Lopes MIF, Bonjorno LP, Menezes PI da SO, Dib SM, Gigante SLG, Benatti MN, Rezek UC, Emrich-Filho LL, Sousa BAA, Almeida SCL de, Luppino-Assad R, Veras FP, Schneider AH, Leiria LOS, Cunha LD da, Alves Filho JCF, Cunha TM, Arruda Neto E de, Miranda CH, Pazin Filho A, Martins MA, Borges M de C, Fonseca BAL da, Bollela VR, Del-Ben CM, Cunha F de Q, Santana R de C, Vilar FC, Zamboni DS, Louzada Júnior P, Oliveira RDR de. Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients [Internet]. Inflammation Research. 2023 ; 72( 5): 895-899.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s00011-023-01718-y
Vancouver
Amaral NB do, Rodrigues TS, Giannini MC, Lopes MIF, Bonjorno LP, Menezes PI da SO, Dib SM, Gigante SLG, Benatti MN, Rezek UC, Emrich-Filho LL, Sousa BAA, Almeida SCL de, Luppino-Assad R, Veras FP, Schneider AH, Leiria LOS, Cunha LD da, Alves Filho JCF, Cunha TM, Arruda Neto E de, Miranda CH, Pazin Filho A, Martins MA, Borges M de C, Fonseca BAL da, Bollela VR, Del-Ben CM, Cunha F de Q, Santana R de C, Vilar FC, Zamboni DS, Louzada Júnior P, Oliveira RDR de. Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients [Internet]. Inflammation Research. 2023 ; 72( 5): 895-899.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s00011-023-01718-y
Artigo de periodico
FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility (2023)
Borges, Vanessa de Fátima; Galant, Leticia Selinger; Kanashiro, Alexandre ; Castanheira, Fernanda Vargas e Silva; Monteiro, Valter Vinícius Silva; Duarte, Diego Ângelo; Rodrigues, Filipe Camargo; Silva, Camila Meirelles de Souza; Schneider, Ayda Henriques ; Cebinelli, Guilherme Cesar Martelossi ; Lima, Mikhael Haruo Fernandes de; Viola, João Paulo de Biaso; Cunha, Thiago Mattar ; Costa Neto, Claudio Miguel da ; Alves Filho, José Carlos Farias ; Pupo, André Sampaio; Cunha, Fernando de Queiroz
Source: Inflammation Research. Unidade: FMRP
Subjects: NEUTRÓFILOS, SEPSE, FÁRMACOS IMUNOSSUPRESSORES
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ABNT
BORGES, Vanessa de Fátima et al. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility. Inflammation Research, v. 72, n. 2, p. 203-215, 2023Tradução . . Disponível em: https://doi.org/10.1007/s00011-022-01669-w. Acesso em: 22 ago. 2024.
APA
Borges, V. de F., Galant, L. S., Kanashiro, A., Castanheira, F. V. e S., Monteiro, V. V. S., Duarte, D. Â., et al. (2023). FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility. Inflammation Research, 72( 2), 203-215. doi:10.1007/s00011-022-01669-w
NLM
Borges V de F, Galant LS, Kanashiro A, Castanheira FV e S, Monteiro VVS, Duarte DÂ, Rodrigues FC, Silva CM de S, Schneider AH, Cebinelli GCM, Lima MHF de, Viola JP de B, Cunha TM, Costa Neto CM da, Alves Filho JCF, Pupo AS, Cunha F de Q. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility [Internet]. Inflammation Research. 2023 ; 72( 2): 203-215.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s00011-022-01669-w
Vancouver
Borges V de F, Galant LS, Kanashiro A, Castanheira FV e S, Monteiro VVS, Duarte DÂ, Rodrigues FC, Silva CM de S, Schneider AH, Cebinelli GCM, Lima MHF de, Viola JP de B, Cunha TM, Costa Neto CM da, Alves Filho JCF, Pupo AS, Cunha F de Q. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility [Internet]. Inflammation Research. 2023 ; 72( 2): 203-215.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s00011-022-01669-w
Artigo de periodico
SARS-CoV-2 productively infects primary human immune system cells in vitro and in COVID-19 patients (2022)
Pontelli, Marjorie C.; Castro, Ítalo A.; Martins, Ronaldo B; La Serra, Leonardo; Veras, Flávio P.; Nascimento, Daniele C; Silva, Camila M.; Cardoso, Ricardo S; Rosales, Roberta Ribeiro Costa; Gomes, Rogério; Lima, Thais M.; Souza, Juliano P.; Vitti, Brenda C.; Caetité, Diego B.; Lima, Mikhael Haruo Fernandes de; Stumpf, Spencer D; Thompson, Cassandra E; Bloyet, Louis-Marie; Toller-Kawahisa, Juliana E.; Giannini, Marcela C.; Bonjorno, Letícia P; Lopes, Maria Isabel Fernandes; Batah, Sabrina Setembre; Siyuan, Li; Luppino-Assad, Rodrigo; Almeida, Sergio C L; Oliveira, Fabiola R; Benatti, Maíra N; Pontes, Lorena Lobo de Figueiredo; Santana, Rodrigo Carvalho; Vilar, Fernando C; Auxiliadora-Martins, Maria; Shi, Pei-Yong; Cunha, Thiago Mattar; Calado, Rodrigo Tocantins; Alves-Filho, José Carlos; Zamboni, Dario Simões; Fabro, Alexandre Todorovic; Louzada Júnior, Paulo; Oliveira, Renê Donizete Ribeiro; Whelan, Sean P J; Cunha, Fernando Queiróz; Arruda, Eurico
Source: Journal of Molecular Cell Biology. Unidades: FCFRP, FMRP
Subjects: COVID-19, LINFÓCITOS, MONÓCITOS, APOPTOSE
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ABNT
PONTELLI, Marjorie C. et al. SARS-CoV-2 productively infects primary human immune system cells in vitro and in COVID-19 patients. Journal of Molecular Cell Biology, v. 14, n. 4, 2022Tradução . . Disponível em: https://doi.org/10.1093/jmcb/mjac021. Acesso em: 22 ago. 2024.
APA
Pontelli, M. C., Castro, Í. A., Martins, R. B., La Serra, L., Veras, F. P., Nascimento, D. C., et al. (2022). SARS-CoV-2 productively infects primary human immune system cells in vitro and in COVID-19 patients. Journal of Molecular Cell Biology, 14( 4). doi:10.1093/jmcb/mjac021
NLM
Pontelli MC, Castro ÍA, Martins RB, La Serra L, Veras FP, Nascimento DC, Silva CM, Cardoso RS, Rosales RRC, Gomes R, Lima TM, Souza JP, Vitti BC, Caetité DB, Lima MHF de, Stumpf SD, Thompson CE, Bloyet L-M, Toller-Kawahisa JE, Giannini MC, Bonjorno LP, Lopes MIF, Batah SS, Siyuan L, Luppino-Assad R, Almeida SCL, Oliveira FR, Benatti MN, Pontes LL de F, Santana RC, Vilar FC, Auxiliadora-Martins M, Shi P-Y, Cunha TM, Calado RT, Alves-Filho JC, Zamboni DS, Fabro AT, Louzada Júnior P, Oliveira RDR, Whelan SPJ, Cunha FQ, Arruda E. SARS-CoV-2 productively infects primary human immune system cells in vitro and in COVID-19 patients [Internet]. Journal of Molecular Cell Biology. 2022 ; 14( 4):[citado 2024 ago. 22 ] Available from: https://doi.org/10.1093/jmcb/mjac021
Vancouver
Pontelli MC, Castro ÍA, Martins RB, La Serra L, Veras FP, Nascimento DC, Silva CM, Cardoso RS, Rosales RRC, Gomes R, Lima TM, Souza JP, Vitti BC, Caetité DB, Lima MHF de, Stumpf SD, Thompson CE, Bloyet L-M, Toller-Kawahisa JE, Giannini MC, Bonjorno LP, Lopes MIF, Batah SS, Siyuan L, Luppino-Assad R, Almeida SCL, Oliveira FR, Benatti MN, Pontes LL de F, Santana RC, Vilar FC, Auxiliadora-Martins M, Shi P-Y, Cunha TM, Calado RT, Alves-Filho JC, Zamboni DS, Fabro AT, Louzada Júnior P, Oliveira RDR, Whelan SPJ, Cunha FQ, Arruda E. SARS-CoV-2 productively infects primary human immune system cells in vitro and in COVID-19 patients [Internet]. Journal of Molecular Cell Biology. 2022 ; 14( 4):[citado 2024 ago. 22 ] Available from: https://doi.org/10.1093/jmcb/mjac021
Artigo de periodico
Nonsurgical periodontal therapy decreases the severity of rheumatoid arthritis and the plasmatic and salivary levels of RANKL and Survivin: a short-term clinical study (2021)
Moura, Marcela Faria; Silva, Tarcília Aparecida ; Cota, Luís Otávio Miranda ; Oliveira, Sicília Rezende ; Cunha, Fernando de Queiroz ; Ferreira, Gilda Aparecida ; Cortelli, José Roberto ; Cortelli, Sheila Cavalca ; Costa, Fernando Oliveira
Source: Clinical Oral Investigations. Unidade: FMRP
Subjects: ARTRITE REUMATOIDE, DOENÇAS PERIODONTAIS, PERIODONTITE, SALIVA
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ABNT
MOURA, Marcela Faria et al. Nonsurgical periodontal therapy decreases the severity of rheumatoid arthritis and the plasmatic and salivary levels of RANKL and Survivin: a short-term clinical study. Clinical Oral Investigations, v. 25, n. 12, p. 6643-6652, 2021Tradução . . Disponível em: https://doi.org/10.1007/s00784-021-03950-4. Acesso em: 22 ago. 2024.
APA
Moura, M. F., Silva, T. A., Cota, L. O. M., Oliveira, S. R., Cunha, F. de Q., Ferreira, G. A., et al. (2021). Nonsurgical periodontal therapy decreases the severity of rheumatoid arthritis and the plasmatic and salivary levels of RANKL and Survivin: a short-term clinical study. Clinical Oral Investigations, 25( 12), 6643-6652. doi:10.1007/s00784-021-03950-4
NLM
Moura MF, Silva TA, Cota LOM, Oliveira SR, Cunha F de Q, Ferreira GA, Cortelli JR, Cortelli SC, Costa FO. Nonsurgical periodontal therapy decreases the severity of rheumatoid arthritis and the plasmatic and salivary levels of RANKL and Survivin: a short-term clinical study [Internet]. Clinical Oral Investigations. 2021 ; 25( 12): 6643-6652.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s00784-021-03950-4
Vancouver
Moura MF, Silva TA, Cota LOM, Oliveira SR, Cunha F de Q, Ferreira GA, Cortelli JR, Cortelli SC, Costa FO. Nonsurgical periodontal therapy decreases the severity of rheumatoid arthritis and the plasmatic and salivary levels of RANKL and Survivin: a short-term clinical study [Internet]. Clinical Oral Investigations. 2021 ; 25( 12): 6643-6652.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s00784-021-03950-4
Artigo de periodico
IL-33 enhances macrophage release of IL-1β and promotes pain and inflammation in gouty arthritis (2020)
Fattori, Victor; Staurengo-Ferrari, Larissa; Zaninelli, Tiago H.; Casagrande, Rúbia; Oliveira, Renê Donizeti Ribeiro de; Louzada Júnior, Paulo; Cunha, Thiago Mattar; Alves Filho, José Carlos Farias ; Teixeira, Mauro Martins; Cunha, Fernando de Queiroz ; Amaral, Flávio Almeida; Verri Júnior, Waldiceu Aparecido
Source: Inflammation Research. Unidade: FMRP
Subjects: ARTRITE, INFLAMAÇÃO, INTERLEUCINA 1, MACRÓFAGOS, DOR, PEROXIDASE, MEMBRANA SINOVIAL, ÁCIDO ÚRICO
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ABNT
FATTORI, Victor et al. IL-33 enhances macrophage release of IL-1β and promotes pain and inflammation in gouty arthritis. Inflammation Research, v. 69, n. 12, p. 1271-1282, 2020Tradução . . Disponível em: https://doi.org/10.1007/s00011-020-01399-x. Acesso em: 22 ago. 2024.
APA
Fattori, V., Staurengo-Ferrari, L., Zaninelli, T. H., Casagrande, R., Oliveira, R. D. R. de, Louzada Júnior, P., et al. (2020). IL-33 enhances macrophage release of IL-1β and promotes pain and inflammation in gouty arthritis. Inflammation Research, 69( 12), 1271-1282. doi:10.1007/s00011-020-01399-x
NLM
Fattori V, Staurengo-Ferrari L, Zaninelli TH, Casagrande R, Oliveira RDR de, Louzada Júnior P, Cunha TM, Alves Filho JCF, Teixeira MM, Cunha F de Q, Amaral FA, Verri Júnior WA. IL-33 enhances macrophage release of IL-1β and promotes pain and inflammation in gouty arthritis [Internet]. Inflammation Research. 2020 ; 69( 12): 1271-1282.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s00011-020-01399-x
Vancouver
Fattori V, Staurengo-Ferrari L, Zaninelli TH, Casagrande R, Oliveira RDR de, Louzada Júnior P, Cunha TM, Alves Filho JCF, Teixeira MM, Cunha F de Q, Amaral FA, Verri Júnior WA. IL-33 enhances macrophage release of IL-1β and promotes pain and inflammation in gouty arthritis [Internet]. Inflammation Research. 2020 ; 69( 12): 1271-1282.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s00011-020-01399-x
Artigo de periodico
Non-peptidergic nociceptive neurons are essential for mechanical inflammatory hypersensitivity in mice (2019)
Pinto, Larissa G.; Souza, Guilherme R.; Kusuda, Ricardo; Lopes, Alexandre Hashimoto Pereira; Sant’Anna, Morena Brazil; Cunha, Fernando de Queiroz ; Ferreira, Sérgio Henrique; Cunha, Thiago Mattar
Source: Molecular Neurobiology. Unidade: FMRP
Subjects: DOR, INFLAMAÇÃO, HIPERSENSIBILIDADE, NOCICEPTORES
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ABNT
PINTO, Larissa G. et al. Non-peptidergic nociceptive neurons are essential for mechanical inflammatory hypersensitivity in mice. Molecular Neurobiology, v. 56, n. 8, p. 5715-5728, 2019Tradução . . Disponível em: https://doi.org/10.1007/s12035-019-1494-5. Acesso em: 22 ago. 2024.
APA
Pinto, L. G., Souza, G. R., Kusuda, R., Lopes, A. H. P., Sant’Anna, M. B., Cunha, F. de Q., et al. (2019). Non-peptidergic nociceptive neurons are essential for mechanical inflammatory hypersensitivity in mice. Molecular Neurobiology, 56( 8), 5715-5728. doi:10.1007/s12035-019-1494-5
NLM
Pinto LG, Souza GR, Kusuda R, Lopes AHP, Sant’Anna MB, Cunha F de Q, Ferreira SH, Cunha TM. Non-peptidergic nociceptive neurons are essential for mechanical inflammatory hypersensitivity in mice [Internet]. Molecular Neurobiology. 2019 ; 56( 8): 5715-5728.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s12035-019-1494-5
Vancouver
Pinto LG, Souza GR, Kusuda R, Lopes AHP, Sant’Anna MB, Cunha F de Q, Ferreira SH, Cunha TM. Non-peptidergic nociceptive neurons are essential for mechanical inflammatory hypersensitivity in mice [Internet]. Molecular Neurobiology. 2019 ; 56( 8): 5715-5728.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s12035-019-1494-5
Artigo de periodico
SN-38, the active metabolite of irinotecan, inhibits the acute inflammatory response by targeting toll-like receptor 4 (2019)
Wong, Deysi Viviana T.; Ribeiro Filho, Helder Veras; Wanderley, Carlos Wagner Souza; Leite, Caio Abner Vitorino Gonçalves; Lima, Jonilson Berlink; Assef, Alexia Nathália Brígido; Cajado, Aurilene Gomes; Batista, Gabriela Loiola Ponte; González, Rafael Holanda; Silva, Karla Oliveira; Borges, Luis Philipi Carvalho; Alencar, Nylane Maria Nunes de; Wilke, Diego Veras; Cunha, Thiago Mattar ; Figueira, Ana Carolina Migliorini; Cunha, Fernando de Queiroz ; Lima Júnior, Roberto César P.
Source: Cancer Chemotherapy and Pharmacology. Unidade: FMRP
Subjects: INFLAMAÇÃO, FÁRMACOS, RECEPTORES
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ABNT
WONG, Deysi Viviana T. et al. SN-38, the active metabolite of irinotecan, inhibits the acute inflammatory response by targeting toll-like receptor 4. Cancer Chemotherapy and Pharmacology, v. 84, n. 2, p. 287-298, 2019Tradução . . Disponível em: https://doi.org/10.1007/s00280-019-03844-z. Acesso em: 22 ago. 2024.
APA
Wong, D. V. T., Ribeiro Filho, H. V., Wanderley, C. W. S., Leite, C. A. V. G., Lima, J. B., Assef, A. N. B., et al. (2019). SN-38, the active metabolite of irinotecan, inhibits the acute inflammatory response by targeting toll-like receptor 4. Cancer Chemotherapy and Pharmacology, 84( 2), 287-298. doi:10.1007/s00280-019-03844-z
NLM
Wong DVT, Ribeiro Filho HV, Wanderley CWS, Leite CAVG, Lima JB, Assef ANB, Cajado AG, Batista GLP, González RH, Silva KO, Borges LPC, Alencar NMN de, Wilke DV, Cunha TM, Figueira ACM, Cunha F de Q, Lima Júnior RCP. SN-38, the active metabolite of irinotecan, inhibits the acute inflammatory response by targeting toll-like receptor 4 [Internet]. Cancer Chemotherapy and Pharmacology. 2019 ; 84( 2): 287-298.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s00280-019-03844-z
Vancouver
Wong DVT, Ribeiro Filho HV, Wanderley CWS, Leite CAVG, Lima JB, Assef ANB, Cajado AG, Batista GLP, González RH, Silva KO, Borges LPC, Alencar NMN de, Wilke DV, Cunha TM, Figueira ACM, Cunha F de Q, Lima Júnior RCP. SN-38, the active metabolite of irinotecan, inhibits the acute inflammatory response by targeting toll-like receptor 4 [Internet]. Cancer Chemotherapy and Pharmacology. 2019 ; 84( 2): 287-298.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s00280-019-03844-z
Artigo de periodico
Spinal GABA-B receptor modulates neutrophil recruitment to the knee joint in zymosan-induced arthritis (2016)
Bassi, Gabriel S; Malvar, David do C; Cunha, Thiago Mattar; Cunha, Fernando de Queiroz; Kanashiro, Alexandre
Source: Naunyn-Schmiedeberg's Archives of Pharmacology. Unidade: FMRP
Subjects: ARTRITE, GABA, NEUROIMUNOMODULAÇÃO, NEUTRÓFILOS, SISTEMA NERVOSO SIMPÁTICO
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ABNT
BASSI, Gabriel S et al. Spinal GABA-B receptor modulates neutrophil recruitment to the knee joint in zymosan-induced arthritis. Naunyn-Schmiedeberg's Archives of Pharmacology, v. 389, n. 8, p. 851-861, 2016Tradução . . Disponível em: https://doi.org/10.1007/s00210-016-1248-0. Acesso em: 22 ago. 2024.
APA
Bassi, G. S., Malvar, D. do C., Cunha, T. M., Cunha, F. de Q., & Kanashiro, A. (2016). Spinal GABA-B receptor modulates neutrophil recruitment to the knee joint in zymosan-induced arthritis. Naunyn-Schmiedeberg's Archives of Pharmacology, 389( 8), 851-861. doi:10.1007/s00210-016-1248-0
NLM
Bassi GS, Malvar D do C, Cunha TM, Cunha F de Q, Kanashiro A. Spinal GABA-B receptor modulates neutrophil recruitment to the knee joint in zymosan-induced arthritis [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2016 ; 389( 8): 851-861.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s00210-016-1248-0
Vancouver
Bassi GS, Malvar D do C, Cunha TM, Cunha F de Q, Kanashiro A. Spinal GABA-B receptor modulates neutrophil recruitment to the knee joint in zymosan-induced arthritis [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2016 ; 389( 8): 851-861.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s00210-016-1248-0
Artigo de periodico
Irinotecan- and 5-fluorouracil-induced intestinal mucositis: insights into pathogenesis and therapeutic perspectives (2016)
Ribeiro, Ronaldo A.; Wanderley, Carlos W. S.; Wong, Deysi V. T.; Mota, José Maurício S. C.; Leite, Caio A. V. G.; Souza, Marcellus H. L. P.; Cunha, Fernando de Queiroz; Lima Junior, Roberto César P.
Source: Cancer Chemotherapy and Pharmacology. Unidade: FMRP
Subjects: ENTEROPATIAS, ANTINEOPLÁSICOS, CITOCINAS, RECEPTORES
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ABNT
RIBEIRO, Ronaldo A. et al. Irinotecan- and 5-fluorouracil-induced intestinal mucositis: insights into pathogenesis and therapeutic perspectives. Cancer Chemotherapy and Pharmacology, v. 78, n. 5, p. 881-893, 2016Tradução . . Disponível em: https://doi.org/10.1007/s00280-016-3139-y. Acesso em: 22 ago. 2024.
APA
Ribeiro, R. A., Wanderley, C. W. S., Wong, D. V. T., Mota, J. M. S. C., Leite, C. A. V. G., Souza, M. H. L. P., et al. (2016). Irinotecan- and 5-fluorouracil-induced intestinal mucositis: insights into pathogenesis and therapeutic perspectives. Cancer Chemotherapy and Pharmacology, 78( 5), 881-893. doi:10.1007/s00280-016-3139-y
NLM
Ribeiro RA, Wanderley CWS, Wong DVT, Mota JMSC, Leite CAVG, Souza MHLP, Cunha F de Q, Lima Junior RCP. Irinotecan- and 5-fluorouracil-induced intestinal mucositis: insights into pathogenesis and therapeutic perspectives [Internet]. Cancer Chemotherapy and Pharmacology. 2016 ; 78( 5): 881-893.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s00280-016-3139-y
Vancouver
Ribeiro RA, Wanderley CWS, Wong DVT, Mota JMSC, Leite CAVG, Souza MHLP, Cunha F de Q, Lima Junior RCP. Irinotecan- and 5-fluorouracil-induced intestinal mucositis: insights into pathogenesis and therapeutic perspectives [Internet]. Cancer Chemotherapy and Pharmacology. 2016 ; 78( 5): 881-893.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s00280-016-3139-y
Artigo de periodico
Modulation of monocytes in septic patients: preserved phagocytic activity, increased ROS and NO generation, and decreased production of inflammatory cytokines (2016)
Santos, Sidnéia S.; Carmo, Amanda M.; Brunialti, Milena K. C.; Machado, Flavia R.; Azevedo, Luciano C.; Assunção, Murillo; Trevelin, Silvia C.; Cunha, Fernando de Queiroz; Salomão, Reinaldo
Source: Intensive Care Medicine Experimental. Unidade: FMRP
Subjects: ÓXIDO NÍTRICO, CITOCINAS, INFLAMAÇÃO
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ABNT
SANTOS, Sidnéia S. et al. Modulation of monocytes in septic patients: preserved phagocytic activity, increased ROS and NO generation, and decreased production of inflammatory cytokines. Intensive Care Medicine Experimental, v. 4, 2016Tradução . . Disponível em: https://doi.org/10.1186/s40635-016-0078-1. Acesso em: 22 ago. 2024.
APA
Santos, S. S., Carmo, A. M., Brunialti, M. K. C., Machado, F. R., Azevedo, L. C., Assunção, M., et al. (2016). Modulation of monocytes in septic patients: preserved phagocytic activity, increased ROS and NO generation, and decreased production of inflammatory cytokines. Intensive Care Medicine Experimental, 4. doi:10.1186/s40635-016-0078-1
NLM
Santos SS, Carmo AM, Brunialti MKC, Machado FR, Azevedo LC, Assunção M, Trevelin SC, Cunha F de Q, Salomão R. Modulation of monocytes in septic patients: preserved phagocytic activity, increased ROS and NO generation, and decreased production of inflammatory cytokines [Internet]. Intensive Care Medicine Experimental. 2016 ; 4[citado 2024 ago. 22 ] Available from: https://doi.org/10.1186/s40635-016-0078-1
Vancouver
Santos SS, Carmo AM, Brunialti MKC, Machado FR, Azevedo LC, Assunção M, Trevelin SC, Cunha F de Q, Salomão R. Modulation of monocytes in septic patients: preserved phagocytic activity, increased ROS and NO generation, and decreased production of inflammatory cytokines [Internet]. Intensive Care Medicine Experimental. 2016 ; 4[citado 2024 ago. 22 ] Available from: https://doi.org/10.1186/s40635-016-0078-1
Artigo de periodico
Mast cells control insulitis and increase Treg cells to confer protection against STZ-induced type 1 diabetes in mice (2015)
Carlos, Daniela; Yaochite, Juliana Navarro Ueda; Rocha, Fernanda Agostini; Toso, Vanina Danuza; Malmegrim, Kelen C. R.; Ramos, Simone Gusmão; Jamur, Maria Célia; Oliver, Constance; Câmara, Niels Olsen Saraiva ; Andrade, Marcus Vinicius Melo de; Cunha, Fernando de Queiróz; Silva, João Santana da
Source: European Journal of Immunology. Unidades: FMRP, ICB
Subjects: MASTÓCITOS, DIABETES MELLITUS, IMUNOLOGIA
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ABNT
CARLOS, Daniela et al. Mast cells control insulitis and increase Treg cells to confer protection against STZ-induced type 1 diabetes in mice. European Journal of Immunology, v. 45, n. 10, p. 2873–2885, 2015Tradução . . Disponível em: https://doi.org/10.1002/eji.201545498. Acesso em: 22 ago. 2024.
APA
Carlos, D., Yaochite, J. N. U., Rocha, F. A., Toso, V. D., Malmegrim, K. C. R., Ramos, S. G., et al. (2015). Mast cells control insulitis and increase Treg cells to confer protection against STZ-induced type 1 diabetes in mice. European Journal of Immunology, 45( 10), 2873–2885. doi:10.1002/eji.201545498
NLM
Carlos D, Yaochite JNU, Rocha FA, Toso VD, Malmegrim KCR, Ramos SG, Jamur MC, Oliver C, Câmara NOS, Andrade MVM de, Cunha F de Q, Silva JS da. Mast cells control insulitis and increase Treg cells to confer protection against STZ-induced type 1 diabetes in mice [Internet]. European Journal of Immunology. 2015 ; 45( 10): 2873–2885.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1002/eji.201545498
Vancouver
Carlos D, Yaochite JNU, Rocha FA, Toso VD, Malmegrim KCR, Ramos SG, Jamur MC, Oliver C, Câmara NOS, Andrade MVM de, Cunha F de Q, Silva JS da. Mast cells control insulitis and increase Treg cells to confer protection against STZ-induced type 1 diabetes in mice [Internet]. European Journal of Immunology. 2015 ; 45( 10): 2873–2885.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1002/eji.201545498
Artigo de periodico
Bosentan, a mixed endothelin receptor antagonist, inhibits superoxide anion-induced pain and inflammation in mice (2015)
Serafim, Karla G. G.; Navarro, Suelen A.; Zarpelon, Ana C.; Pinho-Ribeiro, Felipe A.; Fattori, Victor; Cunha, Thiago Mattar; Alves Filho, José Carlos Farias; Cunha, Fernando de Queiroz; Casagrande, Rubia; Verri Júnior, Waldiceu Aparecido
Source: Naunyn-Schmiedeberg's Archives of Pharmacology. Unidade: FMRP
Subjects: HIPERTENSÃO PULMONAR, ESTRESSE OXIDATIVO, DOR, ENDOTELINAS, ANTI-INFLAMATÓRIOS
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ABNT
SERAFIM, Karla G. G. et al. Bosentan, a mixed endothelin receptor antagonist, inhibits superoxide anion-induced pain and inflammation in mice. Naunyn-Schmiedeberg's Archives of Pharmacology, v. 388, n. 11, p. 1211-1221, 2015Tradução . . Disponível em: https://doi.org/10.1007/s00210-015-1160-z. Acesso em: 22 ago. 2024.
APA
Serafim, K. G. G., Navarro, S. A., Zarpelon, A. C., Pinho-Ribeiro, F. A., Fattori, V., Cunha, T. M., et al. (2015). Bosentan, a mixed endothelin receptor antagonist, inhibits superoxide anion-induced pain and inflammation in mice. Naunyn-Schmiedeberg's Archives of Pharmacology, 388( 11), 1211-1221. doi:10.1007/s00210-015-1160-z
NLM
Serafim KGG, Navarro SA, Zarpelon AC, Pinho-Ribeiro FA, Fattori V, Cunha TM, Alves Filho JCF, Cunha F de Q, Casagrande R, Verri Júnior WA. Bosentan, a mixed endothelin receptor antagonist, inhibits superoxide anion-induced pain and inflammation in mice [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2015 ; 388( 11): 1211-1221.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s00210-015-1160-z
Vancouver
Serafim KGG, Navarro SA, Zarpelon AC, Pinho-Ribeiro FA, Fattori V, Cunha TM, Alves Filho JCF, Cunha F de Q, Casagrande R, Verri Júnior WA. Bosentan, a mixed endothelin receptor antagonist, inhibits superoxide anion-induced pain and inflammation in mice [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2015 ; 388( 11): 1211-1221.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s00210-015-1160-z
Artigo de periodico
Mechanisms affecting neutrophil migration capacity in breast cancer patients before and after chemotherapy (2014)
Mendonça, Maria Angélica Oliveira; Souto, Fabrício O.; Micheli, Douglas C.; Alves-Filho, José Carlos; Cunha, Fernando de Queiroz; Murta, Eddie Fernando C.; Tavares-Murta, Beatriz M.
Source: Cancer Chemotherapy and Pharmacology. Unidade: FMRP
Subjects: NEOPLASIAS MAMÁRIAS, QUIMIOTERAPIA, CITOCINAS, ÓXIDO NÍTRICO, RECEPTORES CELULARES
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ABNT
MENDONÇA, Maria Angélica Oliveira et al. Mechanisms affecting neutrophil migration capacity in breast cancer patients before and after chemotherapy. Cancer Chemotherapy and Pharmacology, v. 73, n. 2, p. 317-324, 2014Tradução . . Disponível em: https://doi.org/10.1007/s00280-013-2348-x. Acesso em: 22 ago. 2024.
APA
Mendonça, M. A. O., Souto, F. O., Micheli, D. C., Alves-Filho, J. C., Cunha, F. de Q., Murta, E. F. C., & Tavares-Murta, B. M. (2014). Mechanisms affecting neutrophil migration capacity in breast cancer patients before and after chemotherapy. Cancer Chemotherapy and Pharmacology, 73( 2), 317-324. doi:10.1007/s00280-013-2348-x
NLM
Mendonça MAO, Souto FO, Micheli DC, Alves-Filho JC, Cunha F de Q, Murta EFC, Tavares-Murta BM. Mechanisms affecting neutrophil migration capacity in breast cancer patients before and after chemotherapy [Internet]. Cancer Chemotherapy and Pharmacology. 2014 ; 73( 2): 317-324.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s00280-013-2348-x
Vancouver
Mendonça MAO, Souto FO, Micheli DC, Alves-Filho JC, Cunha F de Q, Murta EFC, Tavares-Murta BM. Mechanisms affecting neutrophil migration capacity in breast cancer patients before and after chemotherapy [Internet]. Cancer Chemotherapy and Pharmacology. 2014 ; 73( 2): 317-324.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s00280-013-2348-x
Artigo de periodico
The role of PAF/PAFR signaling in zymosan-induced articular inflammatory hyperalgesia (2013)
Guerrero, Ana T.; Zaperlon, Ana C; Vieira, Silvio M; Pinto, Larissa G; Ferreira, Sérgio Henrique; Cunha, Fernando de Queiroz; Verri Junior, Waldiceu A; Cunha, Thiago Mattar
Source: Naunyn-Schmiedeberg's Archives of Pharmacology. Unidade: FMRP
Subjects: DOR, ARTICULAÇÕES, NEUTRÓFILOS (METABOLISMO), DOENÇAS IMUNOLÓGICAS, MODELOS ANIMAIS DE DOENÇAS
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ABNT
GUERRERO, Ana T. et al. The role of PAF/PAFR signaling in zymosan-induced articular inflammatory hyperalgesia. Naunyn-Schmiedeberg's Archives of Pharmacology, v. 386, n. 1, p. 51-59, 2013Tradução . . Disponível em: https://doi.org/10.1007/s00210-012-0813-4. Acesso em: 22 ago. 2024.
APA
Guerrero, A. T., Zaperlon, A. C., Vieira, S. M., Pinto, L. G., Ferreira, S. H., Cunha, F. de Q., et al. (2013). The role of PAF/PAFR signaling in zymosan-induced articular inflammatory hyperalgesia. Naunyn-Schmiedeberg's Archives of Pharmacology, 386( 1), 51-59. doi:10.1007/s00210-012-0813-4
NLM
Guerrero AT, Zaperlon AC, Vieira SM, Pinto LG, Ferreira SH, Cunha F de Q, Verri Junior WA, Cunha TM. The role of PAF/PAFR signaling in zymosan-induced articular inflammatory hyperalgesia [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2013 ; 386( 1): 51-59.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s00210-012-0813-4
Vancouver
Guerrero AT, Zaperlon AC, Vieira SM, Pinto LG, Ferreira SH, Cunha F de Q, Verri Junior WA, Cunha TM. The role of PAF/PAFR signaling in zymosan-induced articular inflammatory hyperalgesia [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2013 ; 386( 1): 51-59.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s00210-012-0813-4
Artigo de periodico
Reappraisal of total body irradiation followed by bone marrow transplantation as a therapy for inflammatory bowel disease (2013)
Godoi, Dannielle Fernandes; Cardoso, Cristina Ribeiro; Silva, Marcelo José Barbosa; Ferraz, Denise Brufato; Provinciatto, Pauline Rossetti; Cunha, Fernando de Queiroz; Silva, João Santana da; Voltarelli, Julio Cesar
Source: Immunobiology. Unidades: FCFRP, FMRP
Subjects: TRANSPLANTE DE MEDULA ÓSSEA, COLITE, INFLAMAÇÃO, MUCOSA INTESTINAL (IMUNOLOGIA)
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ABNT
GODOI, Dannielle Fernandes et al. Reappraisal of total body irradiation followed by bone marrow transplantation as a therapy for inflammatory bowel disease. Immunobiology, v. 218, n. 3, p. 317-324, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.imbio.2012.05.012. Acesso em: 22 ago. 2024.
APA
Godoi, D. F., Cardoso, C. R., Silva, M. J. B., Ferraz, D. B., Provinciatto, P. R., Cunha, F. de Q., et al. (2013). Reappraisal of total body irradiation followed by bone marrow transplantation as a therapy for inflammatory bowel disease. Immunobiology, 218( 3), 317-324. doi:10.1016/j.imbio.2012.05.012
NLM
Godoi DF, Cardoso CR, Silva MJB, Ferraz DB, Provinciatto PR, Cunha F de Q, Silva JS da, Voltarelli JC. Reappraisal of total body irradiation followed by bone marrow transplantation as a therapy for inflammatory bowel disease [Internet]. Immunobiology. 2013 ; 218( 3): 317-324.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1016/j.imbio.2012.05.012
Vancouver
Godoi DF, Cardoso CR, Silva MJB, Ferraz DB, Provinciatto PR, Cunha F de Q, Silva JS da, Voltarelli JC. Reappraisal of total body irradiation followed by bone marrow transplantation as a therapy for inflammatory bowel disease [Internet]. Immunobiology. 2013 ; 218( 3): 317-324.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1016/j.imbio.2012.05.012
Artigo de periodico
Dynamic changes of the Th17/Tc17 and regulatory T cell populations interfere in the experimental autoimmune diabetes pathogenesis (2013)
Yaochite, Juliana Navarro Ueda; Oliveira, Carolina Caliari; Davanso, Mariana Rodrigues; Carlos, Daniela; Malmegrimb, Kelen Cristina Ribeiro; Cardoso, Cristina Ribeiro de Barros; Ramalho, Leandra Naíra Zambelli; Palma, Patricia Vianna Bonini; Silva, João Santana da; Cunha, Fernando Queiróz; Covas, Dimas Tadeu; Voltarelli, Julio Cesar
Source: Immunobiology. Unidades: FCFRP, FMRP
Subjects: DIABETES MELLITUS, QUÍMICA, CITOTOXINAS, LINFÓCITOS T
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ABNT
YAOCHITE, Juliana Navarro Ueda et al. Dynamic changes of the Th17/Tc17 and regulatory T cell populations interfere in the experimental autoimmune diabetes pathogenesis. Immunobiology, v. 218, n. 3, p. 338-352, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.imbio.2012.05.010. Acesso em: 22 ago. 2024.
APA
Yaochite, J. N. U., Oliveira, C. C., Davanso, M. R., Carlos, D., Malmegrimb, K. C. R., Cardoso, C. R. de B., et al. (2013). Dynamic changes of the Th17/Tc17 and regulatory T cell populations interfere in the experimental autoimmune diabetes pathogenesis. Immunobiology, 218( 3), 338-352. doi:10.1016/j.imbio.2012.05.010
NLM
Yaochite JNU, Oliveira CC, Davanso MR, Carlos D, Malmegrimb KCR, Cardoso CR de B, Ramalho LNZ, Palma PVB, Silva JS da, Cunha FQ, Covas DT, Voltarelli JC. Dynamic changes of the Th17/Tc17 and regulatory T cell populations interfere in the experimental autoimmune diabetes pathogenesis [Internet]. Immunobiology. 2013 ; 218( 3): 338-352.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1016/j.imbio.2012.05.010
Vancouver
Yaochite JNU, Oliveira CC, Davanso MR, Carlos D, Malmegrimb KCR, Cardoso CR de B, Ramalho LNZ, Palma PVB, Silva JS da, Cunha FQ, Covas DT, Voltarelli JC. Dynamic changes of the Th17/Tc17 and regulatory T cell populations interfere in the experimental autoimmune diabetes pathogenesis [Internet]. Immunobiology. 2013 ; 218( 3): 338-352.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1016/j.imbio.2012.05.010
Artigo de periodico
NDP-MSH inhibits neutrophil migration through nicotinic and adrenergic receptors in experimental peritonitis (2013)
Figueiredo, Jozi; Ferreira, Ana Elisa; Silva, Rangel Leal; Ulloa, Luís; Grieco, Paolo; Cunha, Thiago Mattar; Ferreira, Sérgio Henrique; Cunha, Fernando de Queiroz; Kanashiro, Alexandre
Source: Naunyn-Schmiedeberg's Archives of Pharmacology. Unidade: FMRP
Subjects: NEUTRÓFILOS (EFEITOS DE DROGAS), RECEPTORES ADRENÉRGICOS, NEUROIMUNOMODULAÇÃO, PERITONITE
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ABNT
FIGUEIREDO, Jozi et al. NDP-MSH inhibits neutrophil migration through nicotinic and adrenergic receptors in experimental peritonitis. Naunyn-Schmiedeberg's Archives of Pharmacology, v. 386, n. 4, p. 311-318, 2013Tradução . . Disponível em: https://doi.org/10.1007/s00210-013-0834-7. Acesso em: 22 ago. 2024.
APA
Figueiredo, J., Ferreira, A. E., Silva, R. L., Ulloa, L., Grieco, P., Cunha, T. M., et al. (2013). NDP-MSH inhibits neutrophil migration through nicotinic and adrenergic receptors in experimental peritonitis. Naunyn-Schmiedeberg's Archives of Pharmacology, 386( 4), 311-318. doi:10.1007/s00210-013-0834-7
NLM
Figueiredo J, Ferreira AE, Silva RL, Ulloa L, Grieco P, Cunha TM, Ferreira SH, Cunha F de Q, Kanashiro A. NDP-MSH inhibits neutrophil migration through nicotinic and adrenergic receptors in experimental peritonitis [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2013 ; 386( 4): 311-318.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s00210-013-0834-7
Vancouver
Figueiredo J, Ferreira AE, Silva RL, Ulloa L, Grieco P, Cunha TM, Ferreira SH, Cunha F de Q, Kanashiro A. NDP-MSH inhibits neutrophil migration through nicotinic and adrenergic receptors in experimental peritonitis [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2013 ; 386( 4): 311-318.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s00210-013-0834-7
Artigo de periodico
A crucial role for IL-6 in the CNS of rats during fever induced by the injection of live E. coli (2012)
Soares, Denis de Melo; Figueiredo, Maria Jose; Martins, Juliano Manvailer; Machado, Renes de Resende; Sorgi, Carlos Arterio; Faccioli, Lucia Helena; Alves-Filho, José Carlos; Cunha, Fernando de Queiroz; Souza, Glória Emília Petto de
Source: Medical Microbiology and Immunology. Unidades: FCFRP, FMRP
Subjects: ESCHERICHIA COLI, FEBRE, CITOCINAS, INFECÇÕES BACTERIANAS
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ABNT
SOARES, Denis de Melo et al. A crucial role for IL-6 in the CNS of rats during fever induced by the injection of live E. coli. Medical Microbiology and Immunology, v. 201, n. 1, p. 47-60, 2012Tradução . . Disponível em: https://doi.org/10.1007/s00430-011-0204-3. Acesso em: 22 ago. 2024.
APA
Soares, D. de M., Figueiredo, M. J., Martins, J. M., Machado, R. de R., Sorgi, C. A., Faccioli, L. H., et al. (2012). A crucial role for IL-6 in the CNS of rats during fever induced by the injection of live E. coli. Medical Microbiology and Immunology, 201( 1), 47-60. doi:10.1007/s00430-011-0204-3
NLM
Soares D de M, Figueiredo MJ, Martins JM, Machado R de R, Sorgi CA, Faccioli LH, Alves-Filho JC, Cunha F de Q, Souza GEP de. A crucial role for IL-6 in the CNS of rats during fever induced by the injection of live E. coli [Internet]. Medical Microbiology and Immunology. 2012 ; 201( 1): 47-60.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s00430-011-0204-3
Vancouver
Soares D de M, Figueiredo MJ, Martins JM, Machado R de R, Sorgi CA, Faccioli LH, Alves-Filho JC, Cunha F de Q, Souza GEP de. A crucial role for IL-6 in the CNS of rats during fever induced by the injection of live E. coli [Internet]. Medical Microbiology and Immunology. 2012 ; 201( 1): 47-60.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s00430-011-0204-3
Artigo de periodico
Involvement of nitric oxide on the pathogenesis of irinotecan-induced intestinal mucositis: role of cytokines on inducible nitric oxide synthase activation (2012)
Lima Júnior, Roberto César P.; Figueiredo, Aline A.; Freitas, Helano C.; Melo, Maria Luisa P.; Wong, Deysi Viviana T.; Leite, Caio Abner V. G.; Medeiros, Raul P.; Marques Neto, Raphael D.; Vale, Mariana L.; Brito, Gerly Anne C.; Oriá, Reinaldo B.; Souza, Marcellus H. L. P.; Cunha, Fernando de Queiroz; Ribeiro, Ronaldo A.
Source: Cancer Chemotherapy and Pharmacology. Unidade: FMRP
Subjects: ANTINEOPLÁSICOS (TOXICIDADE), CITOCINAS (METABOLISMO), MUCOSA INTESTINAL (EFEITOS DE DROGAS), ÓXIDO NÍTRICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LIMA JÚNIOR, Roberto César P. et al. Involvement of nitric oxide on the pathogenesis of irinotecan-induced intestinal mucositis: role of cytokines on inducible nitric oxide synthase activation. Cancer Chemotherapy and Pharmacology, v. 69, n. 4, p. 931-942, 2012Tradução . . Disponível em: https://doi.org/10.1007/s00280-011-1780-z. Acesso em: 22 ago. 2024.
APA
Lima Júnior, R. C. P., Figueiredo, A. A., Freitas, H. C., Melo, M. L. P., Wong, D. V. T., Leite, C. A. V. G., et al. (2012). Involvement of nitric oxide on the pathogenesis of irinotecan-induced intestinal mucositis: role of cytokines on inducible nitric oxide synthase activation. Cancer Chemotherapy and Pharmacology, 69( 4), 931-942. doi:10.1007/s00280-011-1780-z
NLM
Lima Júnior RCP, Figueiredo AA, Freitas HC, Melo MLP, Wong DVT, Leite CAVG, Medeiros RP, Marques Neto RD, Vale ML, Brito GAC, Oriá RB, Souza MHLP, Cunha F de Q, Ribeiro RA. Involvement of nitric oxide on the pathogenesis of irinotecan-induced intestinal mucositis: role of cytokines on inducible nitric oxide synthase activation [Internet]. Cancer Chemotherapy and Pharmacology. 2012 ; 69( 4): 931-942.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s00280-011-1780-z
Vancouver
Lima Júnior RCP, Figueiredo AA, Freitas HC, Melo MLP, Wong DVT, Leite CAVG, Medeiros RP, Marques Neto RD, Vale ML, Brito GAC, Oriá RB, Souza MHLP, Cunha F de Q, Ribeiro RA. Involvement of nitric oxide on the pathogenesis of irinotecan-induced intestinal mucositis: role of cytokines on inducible nitric oxide synthase activation [Internet]. Cancer Chemotherapy and Pharmacology. 2012 ; 69( 4): 931-942.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s00280-011-1780-z
Artigo de periodico
Activation pattern of neutrophils from blood of elderly individuals with Candida-related denture stomatitis (2012)
Gasparoto, Thais Helena ; Oliveira, Carine Ervolino de; Vieira, Narciso Almeida; Porto, Vinícius Carvalho ; Cunha, Fernando de Queiroz ; Garlet, Gustavo Pompermaier ; Campanelli, Ana Paula ; Lara, Vanessa Soares
Source: European Journal of Clinical Microbiology and Infectious Diseases. Unidades: FOB, FMRP
Subjects: NEUTRÓFILOS, IDOSOS, ESTOMATITE SOB PRÓTESE, CANDIDA ALBICANS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GASPAROTO, Thais Helena et al. Activation pattern of neutrophils from blood of elderly individuals with Candida-related denture stomatitis. European Journal of Clinical Microbiology and Infectious Diseases, v. 31, n. 6, p. 1271-1277, 2012Tradução . . Disponível em: https://doi.org/10.1007/s10096-011-1439-z. Acesso em: 22 ago. 2024.
APA
Gasparoto, T. H., Oliveira, C. E. de, Vieira, N. A., Porto, V. C., Cunha, F. de Q., Garlet, G. P., et al. (2012). Activation pattern of neutrophils from blood of elderly individuals with Candida-related denture stomatitis. European Journal of Clinical Microbiology and Infectious Diseases, 31( 6), 1271-1277. doi:10.1007/s10096-011-1439-z
NLM
Gasparoto TH, Oliveira CE de, Vieira NA, Porto VC, Cunha F de Q, Garlet GP, Campanelli AP, Lara VS. Activation pattern of neutrophils from blood of elderly individuals with Candida-related denture stomatitis [Internet]. European Journal of Clinical Microbiology and Infectious Diseases. 2012 ; 31( 6): 1271-1277.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s10096-011-1439-z
Vancouver
Gasparoto TH, Oliveira CE de, Vieira NA, Porto VC, Cunha F de Q, Garlet GP, Campanelli AP, Lara VS. Activation pattern of neutrophils from blood of elderly individuals with Candida-related denture stomatitis [Internet]. European Journal of Clinical Microbiology and Infectious Diseases. 2012 ; 31( 6): 1271-1277.[citado 2024 ago. 22 ] Available from: https://doi.org/10.1007/s10096-011-1439-z

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