SN-38, the active metabolite of irinotecan, inhibits the acute inflammatory response by targeting toll-like receptor 4 (2019)
- Authors:
- Wong, Deysi Viviana T.
- Ribeiro Filho, Helder Veras
- Wanderley, Carlos Wagner Souza
- Leite, Caio Abner Vitorino Gonçalves
- Lima, Jonilson Berlink
- Assef, Alexia Nathália Brígido
- Cajado, Aurilene Gomes
- Batista, Gabriela Loiola Ponte
- González, Rafael Holanda
- Silva, Karla Oliveira
- Borges, Luis Philipi Carvalho
- Alencar, Nylane Maria Nunes de
- Wilke, Diego Veras
- Cunha, Thiago Mattar
- Figueira, Ana Carolina Migliorini
- Cunha, Fernando de Queiroz
- Lima Júnior, Roberto César P.
- USP affiliated authors: CUNHA, THIAGO MATTAR - FMRP ; CUNHA, FERNANDO DE QUEIROZ - FMRP
- Unidade: FMRP
- DOI: 10.1007/s00280-019-03844-z
- Subjects: INFLAMAÇÃO; FÁRMACOS; RECEPTORES
- Keywords: Inflammation; Irinotecan; Nuclear factor-κB; Toll-like receptor; Topotecan
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Publisher place: Heidelberg
- Date published: 2019
- Source:
- Título: Cancer Chemotherapy and Pharmacology
- ISSN: 0344-5704
- Volume/Número/Paginação/Ano: v. 84, n. 2, p. 287-298, 2019
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
WONG, Deysi Viviana T. et al. SN-38, the active metabolite of irinotecan, inhibits the acute inflammatory response by targeting toll-like receptor 4. Cancer Chemotherapy and Pharmacology, v. 84, n. 2, p. 287-298, 2019Tradução . . Disponível em: https://doi.org/10.1007/s00280-019-03844-z. Acesso em: 27 dez. 2025. -
APA
Wong, D. V. T., Ribeiro Filho, H. V., Wanderley, C. W. S., Leite, C. A. V. G., Lima, J. B., Assef, A. N. B., et al. (2019). SN-38, the active metabolite of irinotecan, inhibits the acute inflammatory response by targeting toll-like receptor 4. Cancer Chemotherapy and Pharmacology, 84( 2), 287-298. doi:10.1007/s00280-019-03844-z -
NLM
Wong DVT, Ribeiro Filho HV, Wanderley CWS, Leite CAVG, Lima JB, Assef ANB, Cajado AG, Batista GLP, González RH, Silva KO, Borges LPC, Alencar NMN de, Wilke DV, Cunha TM, Figueira ACM, Cunha F de Q, Lima Júnior RCP. SN-38, the active metabolite of irinotecan, inhibits the acute inflammatory response by targeting toll-like receptor 4 [Internet]. Cancer Chemotherapy and Pharmacology. 2019 ; 84( 2): 287-298.[citado 2025 dez. 27 ] Available from: https://doi.org/10.1007/s00280-019-03844-z -
Vancouver
Wong DVT, Ribeiro Filho HV, Wanderley CWS, Leite CAVG, Lima JB, Assef ANB, Cajado AG, Batista GLP, González RH, Silva KO, Borges LPC, Alencar NMN de, Wilke DV, Cunha TM, Figueira ACM, Cunha F de Q, Lima Júnior RCP. SN-38, the active metabolite of irinotecan, inhibits the acute inflammatory response by targeting toll-like receptor 4 [Internet]. Cancer Chemotherapy and Pharmacology. 2019 ; 84( 2): 287-298.[citado 2025 dez. 27 ] Available from: https://doi.org/10.1007/s00280-019-03844-z - Agglutinin isolated from the red marine alga Hypnea cervicornis J. Agardh reduces inflammatory hypernociception: involvement of nitric oxide
- Spinal GABA-B receptor modulates neutrophil recruitment to the knee joint in zymosan-induced arthritis
- Clinical-like cryotherapy in acute knee arthritis protects neuromuscular junctions of quadriceps and reduces joint inflammation in mice
- Inhibition of peripheral anion exchanger 3 decreases formalin-induced pain
- Neutrophil recruitment and articular hyperalgesia in antigen-induced arthritis are modulated by the cholinergic anti-inflammatory pathway
- The adaptor protein Myd88 is a key signaling molecule in the pathogenesis of irinotecan-induced intestinal mucositis
- Intense acute swimming induces delayed-onset muscle soreness dependent on spinal cord neuroinflammation
- Trypanosoma cruzi adjuvants potentiate T cell-mediated immunity induced by a NY-ESO-1 based antitumor vaccine
- Essential roles of PKA, iNOS, CD95/CD95L, and terminal caspases in suppression of eosinopoiesis by PGE2 and other cAMP-elevating agents
- The quassinoid isobrucein B reduces inflammatory hyperalgesia and cytokine production by post-transcriptional modulation
Informações sobre o DOI: 10.1007/s00280-019-03844-z (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
