Filtros : "FMRP-RCM" "Clinical Endocrinology" Removido: "The Johns Hopkins University School of Medicine, Baltimore, USA" Limpar

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  • Source: Clinical Endocrinology. Unidade: FMRP

    Subjects: INSULINA, SÍNDROME X METABÓLICA, HORMÔNIOS GLICOCORTICOIDES, FENÓTIPOS

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    • ABNT

      RESENDE, Ana Teresa Prata et al. Phenotypic diversity and glucocorticoid sensitivity in patients with familial partial lipodystrophy type 2. Clinical Endocrinology, v. 91, n. 1, p. 94-103, 2019Tradução . . Disponível em: https://doi.org/10.1111/cen.13984. Acesso em: 04 jun. 2024.
    • APA

      Resende, A. T. P., Martins, C. S., Bueno, A. C., Moreira, A. C., Foss-Freitas, M. C., & Castro, M. de. (2019). Phenotypic diversity and glucocorticoid sensitivity in patients with familial partial lipodystrophy type 2. Clinical Endocrinology, 91( 1), 94-103. doi:10.1111/cen.13984
    • NLM

      Resende ATP, Martins CS, Bueno AC, Moreira AC, Foss-Freitas MC, Castro M de. Phenotypic diversity and glucocorticoid sensitivity in patients with familial partial lipodystrophy type 2 [Internet]. Clinical Endocrinology. 2019 ; 91( 1): 94-103.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1111/cen.13984
    • Vancouver

      Resende ATP, Martins CS, Bueno AC, Moreira AC, Foss-Freitas MC, Castro M de. Phenotypic diversity and glucocorticoid sensitivity in patients with familial partial lipodystrophy type 2 [Internet]. Clinical Endocrinology. 2019 ; 91( 1): 94-103.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1111/cen.13984
  • Source: Clinical Endocrinology. Unidades: FFCLRP, FMRP

    Subjects: ANTÍGENOS HLA, POLIMORFISMO, EXPRESSÃO GÊNICA, GLÂNDULA TIREOIDE, NEOPLASIAS

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    • ABNT

      FIGUEIREDO-FEITOSA, Nathalie Lobo de et al. HLA-G 3' untranslated region polymorphic sites associated with increased HLA-G production are more frequent in patients exhibiting differentiated thyroid tumours. Clinical Endocrinology, v. 86, n. 4, p. 597-605, 2017Tradução . . Disponível em: https://doi.org/10.1111/cen.13289. Acesso em: 04 jun. 2024.
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      Figueiredo-Feitosa, N. L. de, Palomino, G. M., Alves, D. C. C., Mendes Júnior, C. T., Donadi, E. A., & Maciel, L. M. Z. (2017). HLA-G 3' untranslated region polymorphic sites associated with increased HLA-G production are more frequent in patients exhibiting differentiated thyroid tumours. Clinical Endocrinology, 86( 4), 597-605. doi:10.1111/cen.13289
    • NLM

      Figueiredo-Feitosa NL de, Palomino GM, Alves DCC, Mendes Júnior CT, Donadi EA, Maciel LMZ. HLA-G 3' untranslated region polymorphic sites associated with increased HLA-G production are more frequent in patients exhibiting differentiated thyroid tumours [Internet]. Clinical Endocrinology. 2017 ; 86( 4): 597-605.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1111/cen.13289
    • Vancouver

      Figueiredo-Feitosa NL de, Palomino GM, Alves DCC, Mendes Júnior CT, Donadi EA, Maciel LMZ. HLA-G 3' untranslated region polymorphic sites associated with increased HLA-G production are more frequent in patients exhibiting differentiated thyroid tumours [Internet]. Clinical Endocrinology. 2017 ; 86( 4): 597-605.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1111/cen.13289
  • Source: Clinical Endocrinology. Unidade: FMRP

    Subjects: NEOPLASIAS CRANIANAS, EXPRESSÃO GÊNICA

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      PAULO, Sabrina Soares et al. Sonic Hedgehog mutations are not a common cause of congenital hypopituitarism in the absence of complex midline cerebral defects. Clinical Endocrinology, v. 82, n. 4, p. 562-569, 2015Tradução . . Disponível em: https://doi.org/10.1111/cen.12565. Acesso em: 04 jun. 2024.
    • APA

      Paulo, S. S., Fernandes-Rosa, F. L., Turatti, W., Coeli-Lacchini, F. B., Martinelli Junior, C. E., Nakiri, G. S., et al. (2015). Sonic Hedgehog mutations are not a common cause of congenital hypopituitarism in the absence of complex midline cerebral defects. Clinical Endocrinology, 82( 4), 562-569. doi:10.1111/cen.12565
    • NLM

      Paulo SS, Fernandes-Rosa FL, Turatti W, Coeli-Lacchini FB, Martinelli Junior CE, Nakiri GS, Moreira AC, Santos AC dos, Castro M de, Antonini SRR. Sonic Hedgehog mutations are not a common cause of congenital hypopituitarism in the absence of complex midline cerebral defects [Internet]. Clinical Endocrinology. 2015 ; 82( 4): 562-569.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1111/cen.12565
    • Vancouver

      Paulo SS, Fernandes-Rosa FL, Turatti W, Coeli-Lacchini FB, Martinelli Junior CE, Nakiri GS, Moreira AC, Santos AC dos, Castro M de, Antonini SRR. Sonic Hedgehog mutations are not a common cause of congenital hypopituitarism in the absence of complex midline cerebral defects [Internet]. Clinical Endocrinology. 2015 ; 82( 4): 562-569.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1111/cen.12565
  • Source: Clinical Endocrinology. Unidade: FMRP

    Subjects: GLÂNDULAS SUPRARRENAIS, CORTISOL, ENDOCRINOLOGIA PEDIÁTRICA, CRIANÇAS

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      BALBÃO, Viviane da Mata Pasti et al. Evaluation of adrenal function in critically ill children. Clinical Endocrinology, v. 81, n. 4, p. 559-565, 2014Tradução . . Disponível em: https://doi.org/10.1111/cen.12444. Acesso em: 04 jun. 2024.
    • APA

      Balbão, V. da M. P., Costa, M. M. A., Castro, M. de, & Carlotti, A. P. de C. P. (2014). Evaluation of adrenal function in critically ill children. Clinical Endocrinology, 81( 4), 559-565. doi:10.1111/cen.12444
    • NLM

      Balbão V da MP, Costa MMA, Castro M de, Carlotti AP de CP. Evaluation of adrenal function in critically ill children [Internet]. Clinical Endocrinology. 2014 ; 81( 4): 559-565.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1111/cen.12444
    • Vancouver

      Balbão V da MP, Costa MMA, Castro M de, Carlotti AP de CP. Evaluation of adrenal function in critically ill children [Internet]. Clinical Endocrinology. 2014 ; 81( 4): 559-565.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1111/cen.12444
  • Source: Clinical Endocrinology. Unidade: FMRP

    Subjects: GENES, NEOPLASIAS, PEDIATRIA, ADULTOS

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      MERMEJO, Livia M. et al. Altered expression of noncanonical Wnt pathway genes in paediatric and adult adrenocortical tumours. Clinical Endocrinology, v. 81, n. 4, p. 503-510, 2014Tradução . . Disponível em: https://doi.org/10.1111/cen.12462. Acesso em: 04 jun. 2024.
    • APA

      Mermejo, L. M., Leal, L. F., Colli, L. M., Fragoso, M. C. B. V., Latronico, A. C., Tone, L. G., et al. (2014). Altered expression of noncanonical Wnt pathway genes in paediatric and adult adrenocortical tumours. Clinical Endocrinology, 81( 4), 503-510. doi:10.1111/cen.12462
    • NLM

      Mermejo LM, Leal LF, Colli LM, Fragoso MCBV, Latronico AC, Tone LG, Scrideli CA, Tucci Júnior S, Martinelli Junior CE, Yunes JA, Mastellaro MJ, Seidinger AL, Brandalise SR, Moreira AC, Ramalho LNZ, Antonini SRR, Castro M de. Altered expression of noncanonical Wnt pathway genes in paediatric and adult adrenocortical tumours [Internet]. Clinical Endocrinology. 2014 ; 81( 4): 503-510.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1111/cen.12462
    • Vancouver

      Mermejo LM, Leal LF, Colli LM, Fragoso MCBV, Latronico AC, Tone LG, Scrideli CA, Tucci Júnior S, Martinelli Junior CE, Yunes JA, Mastellaro MJ, Seidinger AL, Brandalise SR, Moreira AC, Ramalho LNZ, Antonini SRR, Castro M de. Altered expression of noncanonical Wnt pathway genes in paediatric and adult adrenocortical tumours [Internet]. Clinical Endocrinology. 2014 ; 81( 4): 503-510.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1111/cen.12462
  • Source: Clinical Endocrinology. Unidade: FMRP

    Subjects: PEDIATRIA, ENDOCRINOLOGIA

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      ESPIÑEIRA, Aniette Renom et al. Postnatal growth and cardiometabolic profile in young adults born large for gestational age. Clinical Endocrinology, v. 75, n. 3, p. 335-341, 2011Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2265.2011.04054.x. Acesso em: 04 jun. 2024.
    • APA

      Espiñeira, A. R., Rosa, F. L. F., Bueno, A. C., Souza, R. M. de, Moreira, A. C., Castro, M. de, et al. (2011). Postnatal growth and cardiometabolic profile in young adults born large for gestational age. Clinical Endocrinology, 75( 3), 335-341. doi:10.1111/j.1365-2265.2011.04054.x
    • NLM

      Espiñeira AR, Rosa FLF, Bueno AC, Souza RM de, Moreira AC, Castro M de, Barbieri MA, Bettiol H, Antonini SR. Postnatal growth and cardiometabolic profile in young adults born large for gestational age [Internet]. Clinical Endocrinology. 2011 ; 75( 3): 335-341.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1111/j.1365-2265.2011.04054.x
    • Vancouver

      Espiñeira AR, Rosa FLF, Bueno AC, Souza RM de, Moreira AC, Castro M de, Barbieri MA, Bettiol H, Antonini SR. Postnatal growth and cardiometabolic profile in young adults born large for gestational age [Internet]. Clinical Endocrinology. 2011 ; 75( 3): 335-341.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1111/j.1365-2265.2011.04054.x
  • Source: Clinical Endocrinology. Unidades: FM, FMRP

    Subjects: ENDOCRINOLOGIA, PUBERDADE PRECOCE

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      ALMEIDA, Madson Queiroz et al. Long-term treatment of familial male-limited precocious puberty (testotoxicosis) with cyproterone acetate or ketoconazole. Clinical Endocrinology, v. 69, n. 1, p. 93-98, 2008Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2265.2007.03160.x. Acesso em: 04 jun. 2024.
    • APA

      Almeida, M. Q., Brito, V. N., Lins, T. S. S., Guerra-Júnior, G., Castro, M. de, Antonini, S. R., et al. (2008). Long-term treatment of familial male-limited precocious puberty (testotoxicosis) with cyproterone acetate or ketoconazole. Clinical Endocrinology, 69( 1), 93-98. doi:10.1111/j.1365-2265.2007.03160.x
    • NLM

      Almeida MQ, Brito VN, Lins TSS, Guerra-Júnior G, Castro M de, Antonini SR, Arnhold IJP, Mendonça BB, Latronico AC. Long-term treatment of familial male-limited precocious puberty (testotoxicosis) with cyproterone acetate or ketoconazole [Internet]. Clinical Endocrinology. 2008 ; 69( 1): 93-98.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1111/j.1365-2265.2007.03160.x
    • Vancouver

      Almeida MQ, Brito VN, Lins TSS, Guerra-Júnior G, Castro M de, Antonini SR, Arnhold IJP, Mendonça BB, Latronico AC. Long-term treatment of familial male-limited precocious puberty (testotoxicosis) with cyproterone acetate or ketoconazole [Internet]. Clinical Endocrinology. 2008 ; 69( 1): 93-98.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1111/j.1365-2265.2007.03160.x
  • Source: Clinical Endocrinology. Unidade: FMRP

    Subjects: HORMÔNIOS TIREOIDIANOS, DOENÇAS GENÉTICAS

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      MAGALHÃES, Patrícia Künzle Ribeiro et al. Clinical features and genetic analysis of four brazilian kindreds with resistance to thyroid hormone. Clinical Endocrinology, v. 67, p. 748-753, 2007Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2265.2007.02956.x. Acesso em: 04 jun. 2024.
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      Magalhães, P. K. R., Daré, G. L. R., Santos, S. R. dos, Nogueira, C. R., Castro, M. de, & Maciel, L. M. Z. (2007). Clinical features and genetic analysis of four brazilian kindreds with resistance to thyroid hormone. Clinical Endocrinology, 67, 748-753. doi:10.1111/j.1365-2265.2007.02956.x
    • NLM

      Magalhães PKR, Daré GLR, Santos SR dos, Nogueira CR, Castro M de, Maciel LMZ. Clinical features and genetic analysis of four brazilian kindreds with resistance to thyroid hormone [Internet]. Clinical Endocrinology. 2007 ; 67 748-753.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1111/j.1365-2265.2007.02956.x
    • Vancouver

      Magalhães PKR, Daré GLR, Santos SR dos, Nogueira CR, Castro M de, Maciel LMZ. Clinical features and genetic analysis of four brazilian kindreds with resistance to thyroid hormone [Internet]. Clinical Endocrinology. 2007 ; 67 748-753.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1111/j.1365-2265.2007.02956.x
  • Source: Clinical Endocrinology. Unidade: FMRP

    Subjects: RITMO CIRCADIANO, GÊMEOS, PEDIATRIA

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      CUSTÓDIO, Rodrigo José et al. The emergence of the cortizol circadian rhythm in monozygotic and dizygotic twin infants: the twin-pair synchrony. Clinical Endocrinology, v. 66, n. 2, p. 192-197, 2007Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2265.2006.02706.x. Acesso em: 04 jun. 2024.
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      Custódio, R. J., Martinelli Júnior, C. E., Milani, S. L. S., Simões, A. L., Castro, M. de, & Moreira, A. C. (2007). The emergence of the cortizol circadian rhythm in monozygotic and dizygotic twin infants: the twin-pair synchrony. Clinical Endocrinology, 66( 2), 192-197. doi:10.1111/j.1365-2265.2006.02706.x
    • NLM

      Custódio RJ, Martinelli Júnior CE, Milani SLS, Simões AL, Castro M de, Moreira AC. The emergence of the cortizol circadian rhythm in monozygotic and dizygotic twin infants: the twin-pair synchrony [Internet]. Clinical Endocrinology. 2007 ; 66( 2): 192-197.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1111/j.1365-2265.2006.02706.x
    • Vancouver

      Custódio RJ, Martinelli Júnior CE, Milani SLS, Simões AL, Castro M de, Moreira AC. The emergence of the cortizol circadian rhythm in monozygotic and dizygotic twin infants: the twin-pair synchrony [Internet]. Clinical Endocrinology. 2007 ; 66( 2): 192-197.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1111/j.1365-2265.2006.02706.x
  • Source: Clinical Endocrinology. Unidade: FMRP

    Subjects: ENDOCRINOLOGIA, HIPOGONADISMO

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    • ABNT

      VERSIANI, Beatriz R. et al. Clinical assessment and molecular analysis of GnRHR and KAL1 genes in males with idiopathic hypogonadotrophic hypogonadism. Clinical Endocrinology, v. 66, p. 173-179, 2007Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2265.2006.02702.x. Acesso em: 04 jun. 2024.
    • APA

      Versiani, B. R., Trarbach, E., Koenigkam-Santos, M., Santos, A. C. dos, Elias, L. L. K., Moreira, A. C., et al. (2007). Clinical assessment and molecular analysis of GnRHR and KAL1 genes in males with idiopathic hypogonadotrophic hypogonadism. Clinical Endocrinology, 66, 173-179. doi:10.1111/j.1365-2265.2006.02702.x
    • NLM

      Versiani BR, Trarbach E, Koenigkam-Santos M, Santos AC dos, Elias LLK, Moreira AC, Latronico AC, Castro M de. Clinical assessment and molecular analysis of GnRHR and KAL1 genes in males with idiopathic hypogonadotrophic hypogonadism [Internet]. Clinical Endocrinology. 2007 ; 66 173-179.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1111/j.1365-2265.2006.02702.x
    • Vancouver

      Versiani BR, Trarbach E, Koenigkam-Santos M, Santos AC dos, Elias LLK, Moreira AC, Latronico AC, Castro M de. Clinical assessment and molecular analysis of GnRHR and KAL1 genes in males with idiopathic hypogonadotrophic hypogonadism [Internet]. Clinical Endocrinology. 2007 ; 66 173-179.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1111/j.1365-2265.2006.02702.x
  • Source: Clinical Endocrinology. Unidade: FMRP

    Subjects: ENDOCRINOPATIAS, PEDIATRIA

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      CUNHA, Heloisa Marcelina et al. Different states of clinical control are associated with changes in IGF-I and IGFBPs in children with congenital adrenal hyperplasia due to 21-hydroxylase deficiency. Clinical Endocrinology, v. 61, p. 94-101, 2004Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2265.2004.02075.x. Acesso em: 04 jun. 2024.
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      Cunha, H. M., Elias, L. L. K., Camacho-Hubner, C., Moreira, A. C., & Martinelli Júnior, C. E. (2004). Different states of clinical control are associated with changes in IGF-I and IGFBPs in children with congenital adrenal hyperplasia due to 21-hydroxylase deficiency. Clinical Endocrinology, 61, 94-101. doi:10.1111/j.1365-2265.2004.02075.x
    • NLM

      Cunha HM, Elias LLK, Camacho-Hubner C, Moreira AC, Martinelli Júnior CE. Different states of clinical control are associated with changes in IGF-I and IGFBPs in children with congenital adrenal hyperplasia due to 21-hydroxylase deficiency [Internet]. Clinical Endocrinology. 2004 ; 61 94-101.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1111/j.1365-2265.2004.02075.x
    • Vancouver

      Cunha HM, Elias LLK, Camacho-Hubner C, Moreira AC, Martinelli Júnior CE. Different states of clinical control are associated with changes in IGF-I and IGFBPs in children with congenital adrenal hyperplasia due to 21-hydroxylase deficiency [Internet]. Clinical Endocrinology. 2004 ; 61 94-101.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1111/j.1365-2265.2004.02075.x
  • Source: Clinical Endocrinology. Unidade: FMRP

    Assunto: ENDOCRINOLOGIA

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      CASTRO, Margaret et al. A dose-response study of salivary cortisol after dexamethasone suppression test in Cushing's disease and its potential use in the differential diagnosis of Cushing's syndrome. Clinical Endocrinology, v. 59, p. 800-805, 2003Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2265.2003.01908.x. Acesso em: 04 jun. 2024.
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      Castro, M., Elias, L. L. K., Elias, P. C. L., & Moreira, A. C. (2003). A dose-response study of salivary cortisol after dexamethasone suppression test in Cushing's disease and its potential use in the differential diagnosis of Cushing's syndrome. Clinical Endocrinology, 59, 800-805. doi:10.1046/j.1365-2265.2003.01908.x
    • NLM

      Castro M, Elias LLK, Elias PCL, Moreira AC. A dose-response study of salivary cortisol after dexamethasone suppression test in Cushing's disease and its potential use in the differential diagnosis of Cushing's syndrome [Internet]. Clinical Endocrinology. 2003 ; 59 800-805.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1046/j.1365-2265.2003.01908.x
    • Vancouver

      Castro M, Elias LLK, Elias PCL, Moreira AC. A dose-response study of salivary cortisol after dexamethasone suppression test in Cushing's disease and its potential use in the differential diagnosis of Cushing's syndrome [Internet]. Clinical Endocrinology. 2003 ; 59 800-805.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1046/j.1365-2265.2003.01908.x
  • Source: Clinical Endocrinology. Unidade: FMRP

    Assunto: ENDOCRINOLOGIA

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      ELIAS, Paula Conde Lamparelli et al. Progressive decline of vasopressin secretion in familial autossomal dominant neurohypophyseal diabetes insipidus presenting a novel mutation in the vasopressin-neurophysin II gene. Clinical Endocrinology, v. 39, p. 511-518, 2003Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2265.2003.01834.x. Acesso em: 04 jun. 2024.
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      Elias, P. C. L., Elias, L. L. K., Torres, N., Moreira, A. C., Antunes-Rodrigues, J., & Castro, M. de. (2003). Progressive decline of vasopressin secretion in familial autossomal dominant neurohypophyseal diabetes insipidus presenting a novel mutation in the vasopressin-neurophysin II gene. Clinical Endocrinology, 39, 511-518. doi:10.1046/j.1365-2265.2003.01834.x
    • NLM

      Elias PCL, Elias LLK, Torres N, Moreira AC, Antunes-Rodrigues J, Castro M de. Progressive decline of vasopressin secretion in familial autossomal dominant neurohypophyseal diabetes insipidus presenting a novel mutation in the vasopressin-neurophysin II gene [Internet]. Clinical Endocrinology. 2003 ; 39 511-518.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1046/j.1365-2265.2003.01834.x
    • Vancouver

      Elias PCL, Elias LLK, Torres N, Moreira AC, Antunes-Rodrigues J, Castro M de. Progressive decline of vasopressin secretion in familial autossomal dominant neurohypophyseal diabetes insipidus presenting a novel mutation in the vasopressin-neurophysin II gene [Internet]. Clinical Endocrinology. 2003 ; 39 511-518.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1046/j.1365-2265.2003.01834.x
  • Source: Clinical Endocrinology. Unidades: FMRP, FM

    Assunto: ENDOCRINOLOGIA

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      ANTONINI, S. R. R. et al. Glucocorticoid receptor gene polymorphysms in ACTH-secreting pituitary tumors. Clinical Endocrinology, v. 57, p. 657-662, 2002Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2265.2002.01639.x. Acesso em: 04 jun. 2024.
    • APA

      Antonini, S. R. R., Latronicot, A. C., ELIAS, L. L. K., Machado, H. R., Liberman, B., Mendonça, B. B. de, et al. (2002). Glucocorticoid receptor gene polymorphysms in ACTH-secreting pituitary tumors. Clinical Endocrinology, 57, 657-662. doi:10.1046/j.1365-2265.2002.01639.x
    • NLM

      Antonini SRR, Latronicot AC, ELIAS LLK, Machado HR, Liberman B, Mendonça BB de, Moreira AC, Castro M de. Glucocorticoid receptor gene polymorphysms in ACTH-secreting pituitary tumors [Internet]. Clinical Endocrinology. 2002 ; 57 657-662.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1046/j.1365-2265.2002.01639.x
    • Vancouver

      Antonini SRR, Latronicot AC, ELIAS LLK, Machado HR, Liberman B, Mendonça BB de, Moreira AC, Castro M de. Glucocorticoid receptor gene polymorphysms in ACTH-secreting pituitary tumors [Internet]. Clinical Endocrinology. 2002 ; 57 657-662.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1046/j.1365-2265.2002.01639.x
  • Source: Clinical Endocrinology. Unidade: FMRP

    Subjects: ENDOCRINOLOGIA, HORMÔNIOS (METABOLISMO), DESENVOLVIMENTO FÍSICO

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      MARUI, S. et al. Mutations in the type II 3'beta'-hydroxysteroid dehydrogenase (HSD3B2) gene can cause premature pubarche in girls. Clinical Endocrinology, v. 52, p. 67-75, 2000Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2265.2000.00873.x. Acesso em: 04 jun. 2024.
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      Marui, S., Castro, M. de, Latronico, A. C., Elias, L. L. K., Arnhold, I. J. P., Moreira, A. C., & Mendonça, B. B. (2000). Mutations in the type II 3'beta'-hydroxysteroid dehydrogenase (HSD3B2) gene can cause premature pubarche in girls. Clinical Endocrinology, 52, 67-75. doi:10.1046/j.1365-2265.2000.00873.x
    • NLM

      Marui S, Castro M de, Latronico AC, Elias LLK, Arnhold IJP, Moreira AC, Mendonça BB. Mutations in the type II 3'beta'-hydroxysteroid dehydrogenase (HSD3B2) gene can cause premature pubarche in girls [Internet]. Clinical Endocrinology. 2000 ; 52 67-75.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1046/j.1365-2265.2000.00873.x
    • Vancouver

      Marui S, Castro M de, Latronico AC, Elias LLK, Arnhold IJP, Moreira AC, Mendonça BB. Mutations in the type II 3'beta'-hydroxysteroid dehydrogenase (HSD3B2) gene can cause premature pubarche in girls [Internet]. Clinical Endocrinology. 2000 ; 52 67-75.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1046/j.1365-2265.2000.00873.x
  • Source: Clinical Endocrinology. Unidade: FMRP

    Subjects: ENDOCRINOLOGIA, PEDIATRIA

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      ANTONINI, Sonir Roberto Rauber e JORGE, Salim Moysés e MOREIRA, Ayrton Custódio. The emergence of salivary cortisol circadin rhythm and its relationship to sleep activity in preterm infants. Clinical Endocrinology, v. 52, p. 423-426, 2000Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2265.2000.tb00561.x. Acesso em: 04 jun. 2024.
    • APA

      Antonini, S. R. R., Jorge, S. M., & Moreira, A. C. (2000). The emergence of salivary cortisol circadin rhythm and its relationship to sleep activity in preterm infants. Clinical Endocrinology, 52, 423-426. doi:10.1111/j.1365-2265.2000.tb00561.x
    • NLM

      Antonini SRR, Jorge SM, Moreira AC. The emergence of salivary cortisol circadin rhythm and its relationship to sleep activity in preterm infants [Internet]. Clinical Endocrinology. 2000 ; 52 423-426.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1111/j.1365-2265.2000.tb00561.x
    • Vancouver

      Antonini SRR, Jorge SM, Moreira AC. The emergence of salivary cortisol circadin rhythm and its relationship to sleep activity in preterm infants [Internet]. Clinical Endocrinology. 2000 ; 52 423-426.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1111/j.1365-2265.2000.tb00561.x
  • Source: Clinical Endocrinology. Unidade: FMRP

    Subjects: ENDOCRINOPATIAS, ENDOCRINOLOGIA

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    • ABNT

      MARTINELLI, Carlos Eduardo et al. Correlation between cortisol and insulin-like growth factor-binding proteins (IGFBPs) under physiological conditions in children. Clinical Endocrinology, v. 50, p. 767-774, 1999Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2265.1999.00724.x. Acesso em: 04 jun. 2024.
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      Martinelli, C. E., Yateman, M., Cotterill, A. M., Moreira, A. C., & Camacho-Hübsner, C. (1999). Correlation between cortisol and insulin-like growth factor-binding proteins (IGFBPs) under physiological conditions in children. Clinical Endocrinology, 50, 767-774. doi:10.1046/j.1365-2265.1999.00724.x
    • NLM

      Martinelli CE, Yateman M, Cotterill AM, Moreira AC, Camacho-Hübsner C. Correlation between cortisol and insulin-like growth factor-binding proteins (IGFBPs) under physiological conditions in children [Internet]. Clinical Endocrinology. 1999 ; 50 767-774.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1046/j.1365-2265.1999.00724.x
    • Vancouver

      Martinelli CE, Yateman M, Cotterill AM, Moreira AC, Camacho-Hübsner C. Correlation between cortisol and insulin-like growth factor-binding proteins (IGFBPs) under physiological conditions in children [Internet]. Clinical Endocrinology. 1999 ; 50 767-774.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1046/j.1365-2265.1999.00724.x
  • Source: Clinical Endocrinology. Unidade: FMRP

    Subjects: ENDOCRINOPATIAS, ENDOCRINOLOGIA

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    • ABNT

      MARTINELLI JÚNIOR, Carlos Eduardo et al. Salivary cortisol for screening of Cushing's syndrome in children. Clinical Endocrinology, v. 51, p. 67-71, 1999Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2265.1999.00749.x. Acesso em: 04 jun. 2024.
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      Martinelli Júnior, C. E., Sader, S. L., Oliveira, E. B. de, Daneluzzi, J. C., & Moreira, A. C. (1999). Salivary cortisol for screening of Cushing's syndrome in children. Clinical Endocrinology, 51, 67-71. doi:10.1046/j.1365-2265.1999.00749.x
    • NLM

      Martinelli Júnior CE, Sader SL, Oliveira EB de, Daneluzzi JC, Moreira AC. Salivary cortisol for screening of Cushing's syndrome in children [Internet]. Clinical Endocrinology. 1999 ; 51 67-71.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1046/j.1365-2265.1999.00749.x
    • Vancouver

      Martinelli Júnior CE, Sader SL, Oliveira EB de, Daneluzzi JC, Moreira AC. Salivary cortisol for screening of Cushing's syndrome in children [Internet]. Clinical Endocrinology. 1999 ; 51 67-71.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1046/j.1365-2265.1999.00749.x
  • Source: Clinical Endocrinology. Unidade: FMRP

    Assunto: ENDOCRINOPATIAS

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      FOSS, M C et al. Peripheral glucose metabolism in human hiperprolactinemia. Clinical Endocrinology, v. 43, p. 721-6, 1996Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2265.1995.tb00541.x. Acesso em: 04 jun. 2024.
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      Foss, M. C., Paula, F. J. A., Paccola, G. M. G. F., & Piccinato, C. E. (1996). Peripheral glucose metabolism in human hiperprolactinemia. Clinical Endocrinology, 43, 721-6. doi:10.1111/j.1365-2265.1995.tb00541.x
    • NLM

      Foss MC, Paula FJA, Paccola GMGF, Piccinato CE. Peripheral glucose metabolism in human hiperprolactinemia [Internet]. Clinical Endocrinology. 1996 ;43 721-6.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1111/j.1365-2265.1995.tb00541.x
    • Vancouver

      Foss MC, Paula FJA, Paccola GMGF, Piccinato CE. Peripheral glucose metabolism in human hiperprolactinemia [Internet]. Clinical Endocrinology. 1996 ;43 721-6.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1111/j.1365-2265.1995.tb00541.x
  • Source: Clinical Endocrinology. Unidade: FMRP

    Assunto: ENDOCRINOPATIAS

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      SANTIAGO, L B e JORGE, S M e MOREIRA, Ayrton Custódio. Longitudinal evaluation of development of salivary cortisol circadian rhythm in infancy. Clinical Endocrinology, v. 44, p. 157-161, 1996Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2265.1996.645466.x. Acesso em: 04 jun. 2024.
    • APA

      Santiago, L. B., Jorge, S. M., & Moreira, A. C. (1996). Longitudinal evaluation of development of salivary cortisol circadian rhythm in infancy. Clinical Endocrinology, 44, 157-161. doi:10.1046/j.1365-2265.1996.645466.x
    • NLM

      Santiago LB, Jorge SM, Moreira AC. Longitudinal evaluation of development of salivary cortisol circadian rhythm in infancy [Internet]. Clinical Endocrinology. 1996 ; 44 157-161.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1046/j.1365-2265.1996.645466.x
    • Vancouver

      Santiago LB, Jorge SM, Moreira AC. Longitudinal evaluation of development of salivary cortisol circadian rhythm in infancy [Internet]. Clinical Endocrinology. 1996 ; 44 157-161.[citado 2024 jun. 04 ] Available from: https://doi.org/10.1046/j.1365-2265.1996.645466.x

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