Filtros : "BRITTO, LUIZ ROBERTO GIORGETTI DE" "2017" Removidos: "GRU025" "ROMÃO, LUCÍLIA MARIA SOUSA" "ESTHISART" Limpar

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  • Source: Neuroscience. Unidades: IQ, ICB

    Subjects: DOENÇA DE PARKINSON, NEURÔNIOS

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      DATI, L. M et al. Carvacrol promotes neuroprotection in the mouse hemiparkinsonian model. Neuroscience, v. 356, p. 176-181, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2017.05.013. Acesso em: 10 out. 2024.
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      Dati, L. M., Ulrich, H., Real, C. C., Feng, Z. P., Sun, H. S., & Britto, L. R. G. de. (2017). Carvacrol promotes neuroprotection in the mouse hemiparkinsonian model. Neuroscience, 356, 176-181. doi:10.1016/j.neuroscience.2017.05.013
    • NLM

      Dati LM, Ulrich H, Real CC, Feng ZP, Sun HS, Britto LRG de. Carvacrol promotes neuroprotection in the mouse hemiparkinsonian model [Internet]. Neuroscience. 2017 ; 356 176-181.[citado 2024 out. 10 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.05.013
    • Vancouver

      Dati LM, Ulrich H, Real CC, Feng ZP, Sun HS, Britto LRG de. Carvacrol promotes neuroprotection in the mouse hemiparkinsonian model [Internet]. Neuroscience. 2017 ; 356 176-181.[citado 2024 out. 10 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.05.013
  • Source: Journal of Molecular Neuroscience. Unidade: ICB

    Assunto: FISIOLOGIA

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      GARCIA, P. C. e REAL, C. C. e BRITTO, Luiz Roberto Giorgetti de. The impact of short and long-term exercise on the expression of arc and AMPARs during evolution of the 6-Hydroxy-Dopamine animal model of Parkinson's disease. Journal of Molecular Neuroscience, v. 61, n. 4, p. 542-552, 2017Tradução . . Disponível em: https://doi.org/10.1007/s12031-017-0896-y. Acesso em: 10 out. 2024.
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      Garcia, P. C., Real, C. C., & Britto, L. R. G. de. (2017). The impact of short and long-term exercise on the expression of arc and AMPARs during evolution of the 6-Hydroxy-Dopamine animal model of Parkinson's disease. Journal of Molecular Neuroscience, 61( 4), 542-552. doi:10.1007/s12031-017-0896-y
    • NLM

      Garcia PC, Real CC, Britto LRG de. The impact of short and long-term exercise on the expression of arc and AMPARs during evolution of the 6-Hydroxy-Dopamine animal model of Parkinson's disease [Internet]. Journal of Molecular Neuroscience. 2017 ; 61( 4): 542-552.[citado 2024 out. 10 ] Available from: https://doi.org/10.1007/s12031-017-0896-y
    • Vancouver

      Garcia PC, Real CC, Britto LRG de. The impact of short and long-term exercise on the expression of arc and AMPARs during evolution of the 6-Hydroxy-Dopamine animal model of Parkinson's disease [Internet]. Journal of Molecular Neuroscience. 2017 ; 61( 4): 542-552.[citado 2024 out. 10 ] Available from: https://doi.org/10.1007/s12031-017-0896-y
  • Source: Cell Transplantation. Unidades: ICB, IQ

    Subjects: DOENÇA DE PARKINSON, ESTRESSE OXIDATIVO, RECEPTORES DOPAMINÉRGICOS

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      FERRAZOLI, Enéas Galdini et al. Brilliant blue G, but not fenofibrate, treatment reverts hemiparkinsonian behavior and restores dopamine levels in an animal model of Parkinson's disease. Cell Transplantation, v. 26, n. 4, p. 669-677, 2017Tradução . . Disponível em: https://doi.org/10.3727/096368917X695227. Acesso em: 10 out. 2024.
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      Ferrazoli, E. G., Souza, H. D. N. de, Nascimento, I. C. do, Giacomelli, Á. O., Schwindt, T. T., Britto, L. R. G. de, & Ulrich, H. (2017). Brilliant blue G, but not fenofibrate, treatment reverts hemiparkinsonian behavior and restores dopamine levels in an animal model of Parkinson's disease. Cell Transplantation, 26( 4), 669-677. doi:10.3727/096368917X695227
    • NLM

      Ferrazoli EG, Souza HDN de, Nascimento IC do, Giacomelli ÁO, Schwindt TT, Britto LRG de, Ulrich H. Brilliant blue G, but not fenofibrate, treatment reverts hemiparkinsonian behavior and restores dopamine levels in an animal model of Parkinson's disease [Internet]. Cell Transplantation. 2017 ; 26( 4): 669-677.[citado 2024 out. 10 ] Available from: https://doi.org/10.3727/096368917X695227
    • Vancouver

      Ferrazoli EG, Souza HDN de, Nascimento IC do, Giacomelli ÁO, Schwindt TT, Britto LRG de, Ulrich H. Brilliant blue G, but not fenofibrate, treatment reverts hemiparkinsonian behavior and restores dopamine levels in an animal model of Parkinson's disease [Internet]. Cell Transplantation. 2017 ; 26( 4): 669-677.[citado 2024 out. 10 ] Available from: https://doi.org/10.3727/096368917X695227
  • Source: Neuroscience. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      RAVELLI, Katherine Garcia et al. NADPH oxidase contributes to streptozotocin-induced neurodegeneration. Neuroscience, v. 358, p. 227-237, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2017.06.050. Acesso em: 10 out. 2024.
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      Ravelli, K. G., Rosário, B. dos A., Vasconcelos, A. R., Scavone, C., Camarini, R., Hernandes, M. S., & Britto, L. R. G. de. (2017). NADPH oxidase contributes to streptozotocin-induced neurodegeneration. Neuroscience, 358, 227-237. doi:10.1016/j.neuroscience.2017.06.050
    • NLM

      Ravelli KG, Rosário B dos A, Vasconcelos AR, Scavone C, Camarini R, Hernandes MS, Britto LRG de. NADPH oxidase contributes to streptozotocin-induced neurodegeneration [Internet]. Neuroscience. 2017 ; 358 227-237.[citado 2024 out. 10 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.06.050
    • Vancouver

      Ravelli KG, Rosário B dos A, Vasconcelos AR, Scavone C, Camarini R, Hernandes MS, Britto LRG de. NADPH oxidase contributes to streptozotocin-induced neurodegeneration [Internet]. Neuroscience. 2017 ; 358 227-237.[citado 2024 out. 10 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.06.050
  • Source: Journal of Biological Regulators and Homeostatic Agents. Unidades: FO, ICB

    Subjects: GÂNGLIO TRIGÊMEO, INJÚRIA

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      MARTINS, D. O et al. Neurochemical effects of photobiostimulation in the trigeminal ganglion after inferior alveolar nerve injury. Journal of Biological Regulators and Homeostatic Agents, v. 31, n. Ja 2017, p. 147-152, 2017Tradução . . Acesso em: 10 out. 2024.
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      Martins, D. O., Santos, F. M., Britto, L. R. G. de, Lemos, J. B. D., & Chacur, M. (2017). Neurochemical effects of photobiostimulation in the trigeminal ganglion after inferior alveolar nerve injury. Journal of Biological Regulators and Homeostatic Agents, 31( Ja 2017), 147-152.
    • NLM

      Martins DO, Santos FM, Britto LRG de, Lemos JBD, Chacur M. Neurochemical effects of photobiostimulation in the trigeminal ganglion after inferior alveolar nerve injury. Journal of Biological Regulators and Homeostatic Agents. 2017 ; 31( Ja 2017): 147-152.[citado 2024 out. 10 ]
    • Vancouver

      Martins DO, Santos FM, Britto LRG de, Lemos JBD, Chacur M. Neurochemical effects of photobiostimulation in the trigeminal ganglion after inferior alveolar nerve injury. Journal of Biological Regulators and Homeostatic Agents. 2017 ; 31( Ja 2017): 147-152.[citado 2024 out. 10 ]
  • Source: Lasers in Medical Science. Unidades: FO, ICB

    Subjects: NERVO TRIGÊMEO, RATOS

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      MARTINS, Daniel Oliveira et al. Neuropeptide expression and morphometric differences in crushed alveolar inferior nerve of rats: effects of photobiomodulation. Lasers in Medical Science, p. 1-8, 2017Tradução . . Disponível em: https://doi.org/10.1007/s10103-017-2181-2. Acesso em: 10 out. 2024.
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      Martins, D. O., Santos, F. M., Ciena, A. P., Watanabe, I. -S., Britto, L. R. G. de, Lemos, J. B. D., & Chacur, M. (2017). Neuropeptide expression and morphometric differences in crushed alveolar inferior nerve of rats: effects of photobiomodulation. Lasers in Medical Science, 1-8. doi:10.1007/s10103-017-2181-2
    • NLM

      Martins DO, Santos FM, Ciena AP, Watanabe I-S, Britto LRG de, Lemos JBD, Chacur M. Neuropeptide expression and morphometric differences in crushed alveolar inferior nerve of rats: effects of photobiomodulation [Internet]. Lasers in Medical Science. 2017 ; 1-8.[citado 2024 out. 10 ] Available from: https://doi.org/10.1007/s10103-017-2181-2
    • Vancouver

      Martins DO, Santos FM, Ciena AP, Watanabe I-S, Britto LRG de, Lemos JBD, Chacur M. Neuropeptide expression and morphometric differences in crushed alveolar inferior nerve of rats: effects of photobiomodulation [Internet]. Lasers in Medical Science. 2017 ; 1-8.[citado 2024 out. 10 ] Available from: https://doi.org/10.1007/s10103-017-2181-2
  • Source: Journal of Molecular Neuroscience. Unidade: ICB

    Subjects: FISIOLOGIA, DOENÇA DE PARKINSON, PREVENÇÃO DE DOENÇAS ANIMAIS, RATOS WISTAR, ASTRÓCITOS, RECEPTORES DE DOPAMINA, NEURÔNIOS, MODELOS ANIMAIS DE DOENÇAS

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      GREGÓRIO, Caroline Cristiano Real e GARCIA, Priscila Crespo e BRITTO, Luiz Roberto Giorgetti de. Treadmill exercise prevents increase of neuroinflammation markers involved in the dopaminergic damage of the 6-OHDA Parkinson's disease model. Journal of Molecular Neuroscience, v. 63, p. 36-49, 2017Tradução . . Disponível em: https://doi.org/10.1007/s12031-017-0955-4. Acesso em: 10 out. 2024.
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      Gregório, C. C. R., Garcia, P. C., & Britto, L. R. G. de. (2017). Treadmill exercise prevents increase of neuroinflammation markers involved in the dopaminergic damage of the 6-OHDA Parkinson's disease model. Journal of Molecular Neuroscience, 63, 36-49. doi:10.1007/s12031-017-0955-4
    • NLM

      Gregório CCR, Garcia PC, Britto LRG de. Treadmill exercise prevents increase of neuroinflammation markers involved in the dopaminergic damage of the 6-OHDA Parkinson's disease model. [Internet]. Journal of Molecular Neuroscience. 2017 ; 63 36-49.[citado 2024 out. 10 ] Available from: https://doi.org/10.1007/s12031-017-0955-4
    • Vancouver

      Gregório CCR, Garcia PC, Britto LRG de. Treadmill exercise prevents increase of neuroinflammation markers involved in the dopaminergic damage of the 6-OHDA Parkinson's disease model. [Internet]. Journal of Molecular Neuroscience. 2017 ; 63 36-49.[citado 2024 out. 10 ] Available from: https://doi.org/10.1007/s12031-017-0955-4
  • Source: Journal of Proteomics. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      CAFÉ-MENDES, C. C. et al. Peptidomic analysis of the anterior temporal lobe and corpus callosum from schizophrenia patients. Journal of Proteomics, v. 151, p. 97-105, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.jprot.2016.05.025. Acesso em: 10 out. 2024.
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      Café-Mendes, C. C., Ferro, E. S., Torrão, A. da S., Crunfli, F., Rioli, V., Schmitt, A., et al. (2017). Peptidomic analysis of the anterior temporal lobe and corpus callosum from schizophrenia patients. Journal of Proteomics, 151, 97-105. doi:10.1016/j.jprot.2016.05.025
    • NLM

      Café-Mendes CC, Ferro ES, Torrão A da S, Crunfli F, Rioli V, Schmitt A, Falkai P, Britto LRG de, Turck CW, Martins-de-Souza D. Peptidomic analysis of the anterior temporal lobe and corpus callosum from schizophrenia patients [Internet]. Journal of Proteomics. 2017 ; 151 97-105.[citado 2024 out. 10 ] Available from: https://doi.org/10.1016/j.jprot.2016.05.025
    • Vancouver

      Café-Mendes CC, Ferro ES, Torrão A da S, Crunfli F, Rioli V, Schmitt A, Falkai P, Britto LRG de, Turck CW, Martins-de-Souza D. Peptidomic analysis of the anterior temporal lobe and corpus callosum from schizophrenia patients [Internet]. Journal of Proteomics. 2017 ; 151 97-105.[citado 2024 out. 10 ] Available from: https://doi.org/10.1016/j.jprot.2016.05.025
  • Source: CNS Neuroscience & Therapeutics. Unidade: ICB

    Assunto: FISIOLOGIA

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      HUANG, Sammen et al. GSK-3β inhibitor TDZD-8 reduces neonatal hypoxic-ischemic brain injury in mice. CNS Neuroscience & Therapeutics, v. 23, n. 5, p. 405-415, 2017Tradução . . Disponível em: https://doi.org/10.1111/cns.12683. Acesso em: 10 out. 2024.
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      Huang, S., Wang, H., Turlova, E., Abussaud, A., Ji, X., Britto, L. R. G. de, et al. (2017). GSK-3β inhibitor TDZD-8 reduces neonatal hypoxic-ischemic brain injury in mice. CNS Neuroscience & Therapeutics, 23( 5), 405-415. doi:10.1111/cns.12683
    • NLM

      Huang S, Wang H, Turlova E, Abussaud A, Ji X, Britto LRG de, Miller SP, Martinez A, Sun H‐S, Feng Z‐P. GSK-3β inhibitor TDZD-8 reduces neonatal hypoxic-ischemic brain injury in mice [Internet]. CNS Neuroscience & Therapeutics. 2017 ; 23( 5): 405-415.[citado 2024 out. 10 ] Available from: https://doi.org/10.1111/cns.12683
    • Vancouver

      Huang S, Wang H, Turlova E, Abussaud A, Ji X, Britto LRG de, Miller SP, Martinez A, Sun H‐S, Feng Z‐P. GSK-3β inhibitor TDZD-8 reduces neonatal hypoxic-ischemic brain injury in mice [Internet]. CNS Neuroscience & Therapeutics. 2017 ; 23( 5): 405-415.[citado 2024 out. 10 ] Available from: https://doi.org/10.1111/cns.12683
  • Source: Scientific reports. Unidades: ICB, FM, FMRP

    Subjects: PEPTÍDEOS, METABOLISMO, SISTEMA NERVOSO CENTRAL, FARMACOLOGIA, FISIOLOGIA

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      RECKZIEGEL, Patricia et al. A novel peptide that improves metabolic parameters without adverse central nervous system effects. Scientific reports, v. 7, 2017Tradução . . Disponível em: https://doi.org/10.1038/s41598-017-13690-9. Acesso em: 10 out. 2024.
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      Reckziegel, P., Festuccia, W. T. L., Britto, L. R. G. de, Heimann, J. C., Guimarães, F. S., Eichler, R. A. dos S., & Ferro, E. S. (2017). A novel peptide that improves metabolic parameters without adverse central nervous system effects. Scientific reports, 7. doi:10.1038/s41598-017-13690-9
    • NLM

      Reckziegel P, Festuccia WTL, Britto LRG de, Heimann JC, Guimarães FS, Eichler RA dos S, Ferro ES. A novel peptide that improves metabolic parameters without adverse central nervous system effects [Internet]. Scientific reports. 2017 ; 7[citado 2024 out. 10 ] Available from: https://doi.org/10.1038/s41598-017-13690-9
    • Vancouver

      Reckziegel P, Festuccia WTL, Britto LRG de, Heimann JC, Guimarães FS, Eichler RA dos S, Ferro ES. A novel peptide that improves metabolic parameters without adverse central nervous system effects [Internet]. Scientific reports. 2017 ; 7[citado 2024 out. 10 ] Available from: https://doi.org/10.1038/s41598-017-13690-9
  • Source: Neurotoxicity Research. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      RAVELLI, Katherine Garcia et al. Intracerebroventricular streptozotocin as a model of Alzheimer’s disease: neurochemical and behavioral characterization in mice. Neurotoxicity Research, v. 31, n. 3, p. 327-333, 2017Tradução . . Disponível em: https://doi.org/10.1007/s12640-016-9684-7. Acesso em: 10 out. 2024.
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      Ravelli, K. G., Rosário, B. dos A., Camarini, R., Hernandes, M. S., & Britto, L. R. G. de. (2017). Intracerebroventricular streptozotocin as a model of Alzheimer’s disease: neurochemical and behavioral characterization in mice. Neurotoxicity Research, 31( 3), 327-333. doi:10.1007/s12640-016-9684-7
    • NLM

      Ravelli KG, Rosário B dos A, Camarini R, Hernandes MS, Britto LRG de. Intracerebroventricular streptozotocin as a model of Alzheimer’s disease: neurochemical and behavioral characterization in mice [Internet]. Neurotoxicity Research. 2017 ; 31( 3): 327-333.[citado 2024 out. 10 ] Available from: https://doi.org/10.1007/s12640-016-9684-7
    • Vancouver

      Ravelli KG, Rosário B dos A, Camarini R, Hernandes MS, Britto LRG de. Intracerebroventricular streptozotocin as a model of Alzheimer’s disease: neurochemical and behavioral characterization in mice [Internet]. Neurotoxicity Research. 2017 ; 31( 3): 327-333.[citado 2024 out. 10 ] Available from: https://doi.org/10.1007/s12640-016-9684-7

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