Brilliant blue G, but not fenofibrate, treatment reverts hemiparkinsonian behavior and restores dopamine levels in an animal model of Parkinson's disease (2017)
- Authors:
- USP affiliated authors: BRITTO, LUIZ ROBERTO GIORGETTI DE - ICB ; ULRICH, ALEXANDER HENNING - IQ
- Unidades: ICB; IQ
- DOI: 10.3727/096368917X695227
- Subjects: DOENÇA DE PARKINSON; ESTRESSE OXIDATIVO; RECEPTORES DOPAMINÉRGICOS
- Language: Inglês
- Imprenta:
- Source:
- Título: Cell Transplantation
- ISSN: 0963-6897
- Volume/Número/Paginação/Ano: v. 26, n. 4, p. 669-677, 2017
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by-nc
-
ABNT
FERRAZOLI, Enéas Galdini et al. Brilliant blue G, but not fenofibrate, treatment reverts hemiparkinsonian behavior and restores dopamine levels in an animal model of Parkinson's disease. Cell Transplantation, v. 26, n. 4, p. 669-677, 2017Tradução . . Disponível em: https://doi.org/10.3727/096368917X695227. Acesso em: 28 dez. 2025. -
APA
Ferrazoli, E. G., Souza, H. D. N. de, Nascimento, I. C. do, Giacomelli, Á. O., Schwindt, T. T., Britto, L. R. G. de, & Ulrich, H. (2017). Brilliant blue G, but not fenofibrate, treatment reverts hemiparkinsonian behavior and restores dopamine levels in an animal model of Parkinson's disease. Cell Transplantation, 26( 4), 669-677. doi:10.3727/096368917X695227 -
NLM
Ferrazoli EG, Souza HDN de, Nascimento IC do, Giacomelli ÁO, Schwindt TT, Britto LRG de, Ulrich H. Brilliant blue G, but not fenofibrate, treatment reverts hemiparkinsonian behavior and restores dopamine levels in an animal model of Parkinson's disease [Internet]. Cell Transplantation. 2017 ; 26( 4): 669-677.[citado 2025 dez. 28 ] Available from: https://doi.org/10.3727/096368917X695227 -
Vancouver
Ferrazoli EG, Souza HDN de, Nascimento IC do, Giacomelli ÁO, Schwindt TT, Britto LRG de, Ulrich H. Brilliant blue G, but not fenofibrate, treatment reverts hemiparkinsonian behavior and restores dopamine levels in an animal model of Parkinson's disease [Internet]. Cell Transplantation. 2017 ; 26( 4): 669-677.[citado 2025 dez. 28 ] Available from: https://doi.org/10.3727/096368917X695227 - Role of acetylcholine receptors in proliferation and differentiation of P19 embryonal carcinoma cells
- Participation of spontaneous 'CA POT. 2+'-transients and 'CA POT. 2+'-fluxes generated by purinergic and nicotinic acetylcholine receptors in neuronal differentiation of P19 embryonic carcinoma cells
- Expression and activity of nicotinic and muscarinic acetylcholine receptors during neuronal differentiation In vitro
- Neuronal cell fate determination in P19 embryonic carcinoma cells is dependent on spontaneous calcium oscillations
- Pharmacological properties of purinergic receptors and their effects on proliferation and induction of neuronal differentiation of P19 embryonal carcinoma cells
- Proliferation and differentiation of embryonic cells to neurons depende on purinergic receptor-induced calcium transients
- P19 embryonal carcinoma cells as in vitro model for studying purinergic receptor expression and modulation of N-methyl-D-aspartate-glutamate and acetylcholine receptors during neuronal differentiation
- Mechanism of acetylcholine-induced calcium signaling, proliferation and differentiation effects during In vitro neuronal differentiation of embryonal cells
- Carvacrol promotes neuroprotection in the mouse hemiparkinsonian model
- Mechanism of acetylcholine-induced calcium signaling during neuronal differentiation of P19 embryonal carcinoma cells In vitro
Informações sobre o DOI: 10.3727/096368917X695227 (Fonte: oaDOI API)
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