Carvacrol promotes neuroprotection in the mouse hemiparkinsonian model (2017)
- Authors:
- USP affiliated authors: ULRICH, ALEXANDER HENNING - IQ ; BRITTO, LUIZ ROBERTO GIORGETTI DE - ICB
- Unidades: IQ; ICB
- DOI: 10.1016/j.neuroscience.2017.05.013
- Subjects: DOENÇA DE PARKINSON; NEURÔNIOS
- Language: Inglês
- Imprenta:
- Source:
- Título: Neuroscience
- ISSN: 0306-4522
- Volume/Número/Paginação/Ano: v. 356, p. 176-181, 2017
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
DATI, L. M et al. Carvacrol promotes neuroprotection in the mouse hemiparkinsonian model. Neuroscience, v. 356, p. 176-181, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2017.05.013. Acesso em: 28 dez. 2025. -
APA
Dati, L. M., Ulrich, H., Real, C. C., Feng, Z. P., Sun, H. S., & Britto, L. R. G. de. (2017). Carvacrol promotes neuroprotection in the mouse hemiparkinsonian model. Neuroscience, 356, 176-181. doi:10.1016/j.neuroscience.2017.05.013 -
NLM
Dati LM, Ulrich H, Real CC, Feng ZP, Sun HS, Britto LRG de. Carvacrol promotes neuroprotection in the mouse hemiparkinsonian model [Internet]. Neuroscience. 2017 ; 356 176-181.[citado 2025 dez. 28 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.05.013 -
Vancouver
Dati LM, Ulrich H, Real CC, Feng ZP, Sun HS, Britto LRG de. Carvacrol promotes neuroprotection in the mouse hemiparkinsonian model [Internet]. Neuroscience. 2017 ; 356 176-181.[citado 2025 dez. 28 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.05.013 - Role of acetylcholine receptors in proliferation and differentiation of P19 embryonal carcinoma cells
- Participation of spontaneous 'CA POT. 2+'-transients and 'CA POT. 2+'-fluxes generated by purinergic and nicotinic acetylcholine receptors in neuronal differentiation of P19 embryonic carcinoma cells
- Brilliant blue G, but not fenofibrate, treatment reverts hemiparkinsonian behavior and restores dopamine levels in an animal model of Parkinson's disease
- Expression and activity of nicotinic and muscarinic acetylcholine receptors during neuronal differentiation In vitro
- Neuronal cell fate determination in P19 embryonic carcinoma cells is dependent on spontaneous calcium oscillations
- Pharmacological properties of purinergic receptors and their effects on proliferation and induction of neuronal differentiation of P19 embryonal carcinoma cells
- Proliferation and differentiation of embryonic cells to neurons depende on purinergic receptor-induced calcium transients
- P19 embryonal carcinoma cells as in vitro model for studying purinergic receptor expression and modulation of N-methyl-D-aspartate-glutamate and acetylcholine receptors during neuronal differentiation
- Mechanism of acetylcholine-induced calcium signaling, proliferation and differentiation effects during In vitro neuronal differentiation of embryonal cells
- Mechanism of acetylcholine-induced calcium signaling during neuronal differentiation of P19 embryonal carcinoma cells In vitro
Informações sobre o DOI: 10.1016/j.neuroscience.2017.05.013 (Fonte: oaDOI API)
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