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Artigo de periodico
CASP4/11 contributes to NLRP3 activation and COVID-19 exacerbation (2023)
Rodrigues, Tamara Silva ; Caetano, Camila C. S.; Sá, Keyla Santos Guedes de ; Almeida, Leticia de; Becerra, Amanda de Matos; Gonçalves, Augusto Velozo ; Lopes, Letícia de Sousa; Oliveira, Samuel; Mascarenhas, Danielle Pini Alves; Batah, Sabrina Setembre ; Silva, Bruna Manuella Souza; Gomes, Giovanni Freitas; Castro, Ricardo de; Martins Júnior, Ronaldo Bragança; Avila, Jonathan; Frantz, Fabiani Gai ; Cunha, Thiago Mattar ; Arruda Neto, Eurico de; Cunha, Fernando Queiroz ; Helder Takashi Imoto Nakaya ; Cunha, Larissa Dias da; Fabro, Alexandre Todorovic ; Louzada Júnior, Paulo ; Oliveira, Renê Donizeti Ribeiro de; Zamboni, Dario Simões
Source: The Journal of Infectious Diseases. Unidades: FMRP, FCFRP
Subjects: COVID-19, MACRÓFAGOS, CORONAVIRUS
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ABNT
RODRIGUES, Tamara Silva et al. CASP4/11 contributes to NLRP3 activation and COVID-19 exacerbation. The Journal of Infectious Diseases, v. 227, n. 12, p. 1364-1375, 2023Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiad037. Acesso em: 02 nov. 2024.
APA
Rodrigues, T. S., Caetano, C. C. S., Sá, K. S. G. de, Almeida, L. de, Becerra, A. de M., Gonçalves, A. V., et al. (2023). CASP4/11 contributes to NLRP3 activation and COVID-19 exacerbation. The Journal of Infectious Diseases, 227( 12), 1364-1375. doi:10.1093/infdis/jiad037
NLM
Rodrigues TS, Caetano CCS, Sá KSG de, Almeida L de, Becerra A de M, Gonçalves AV, Lopes L de S, Oliveira S, Mascarenhas DPA, Batah SS, Silva BMS, Gomes GF, Castro R de, Martins Júnior RB, Avila J, Frantz FG, Cunha TM, Arruda Neto E de, Cunha FQ, Helder Takashi Imoto Nakaya, Cunha LD da, Fabro AT, Louzada Júnior P, Oliveira RDR de, Zamboni DS. CASP4/11 contributes to NLRP3 activation and COVID-19 exacerbation [Internet]. The Journal of Infectious Diseases. 2023 ; 227( 12): 1364-1375.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1093/infdis/jiad037
Vancouver
Rodrigues TS, Caetano CCS, Sá KSG de, Almeida L de, Becerra A de M, Gonçalves AV, Lopes L de S, Oliveira S, Mascarenhas DPA, Batah SS, Silva BMS, Gomes GF, Castro R de, Martins Júnior RB, Avila J, Frantz FG, Cunha TM, Arruda Neto E de, Cunha FQ, Helder Takashi Imoto Nakaya, Cunha LD da, Fabro AT, Louzada Júnior P, Oliveira RDR de, Zamboni DS. CASP4/11 contributes to NLRP3 activation and COVID-19 exacerbation [Internet]. The Journal of Infectious Diseases. 2023 ; 227( 12): 1364-1375.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1093/infdis/jiad037
Artigo de periodico
Rapamycin did not prevent the excessive exercise-induced hepatic fat accumulation (2022)
Pinto, Ana Paula ; Rocha, Alisson Luiz da ; Teixeira, Giovana Rampazzo; Rovina, Rafael Lemes ; Veras, Allice S. C.; Frantz, Fabiani Gai ; Pauli, José Rodrigo; Moura, Leandro Pereira de; Cintra, Dennys Esper; Ropelle, Eduardo Rochete; Quadrilatero, Joe; Silva, Adelino Sanchez Ramos da
Source: Life Sciences. Unidades: FCFRP, EEFERP, FMRP
Subjects: DEGRANULAÇÃO CELULAR, FÍGADO, HEPATOPATIAS, REAÇÃO EM CADEIA POR POLIMERASE
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ABNT
PINTO, Ana Paula et al. Rapamycin did not prevent the excessive exercise-induced hepatic fat accumulation. Life Sciences, v. 306, p. 1-12, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2022.120800. Acesso em: 02 nov. 2024.
APA
Pinto, A. P., Rocha, A. L. da, Teixeira, G. R., Rovina, R. L., Veras, A. S. C., Frantz, F. G., et al. (2022). Rapamycin did not prevent the excessive exercise-induced hepatic fat accumulation. Life Sciences, 306, 1-12. doi:10.1016/j.lfs.2022.120800
NLM
Pinto AP, Rocha AL da, Teixeira GR, Rovina RL, Veras ASC, Frantz FG, Pauli JR, Moura LP de, Cintra DE, Ropelle ER, Quadrilatero J, Silva ASR da. Rapamycin did not prevent the excessive exercise-induced hepatic fat accumulation [Internet]. Life Sciences. 2022 ; 306 1-12.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.lfs.2022.120800
Vancouver
Pinto AP, Rocha AL da, Teixeira GR, Rovina RL, Veras ASC, Frantz FG, Pauli JR, Moura LP de, Cintra DE, Ropelle ER, Quadrilatero J, Silva ASR da. Rapamycin did not prevent the excessive exercise-induced hepatic fat accumulation [Internet]. Life Sciences. 2022 ; 306 1-12.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.lfs.2022.120800
Artigo de periodico
Identification of immunomodulatory drugs that inhibit multiple inflammasomes and impair SARS-CoV-2 infection (2022)
Almeida, Leticia de; Silva, Alexandre Luiz Neves; Rodrigues, Tamara Silva; Oliveira, Samuel; Ishimoto, Adriene Yumi; Seribelli, Amanda Aparecida; Becerra, Amanda; Andrade, Warrison A; Ataíde, Marco Antônio; Caetano, Camila C. S.; Sá, Keyla Santos Guedes de; Pelisson, Natália; Martins, Ronaldo Bragança; Souza, Juliano de Paula; Arruda, Eurico; Batah, Sabrina Setembre; Castro, Ricardo; Frantz, Fabiani Gai; Cunha, Fernando Queiróz; Cunha, Thiago Mattar; Fabro, Alexandre Todorovic; Cunha, Larissa Dias; Louzada-Junior, Paulo; Oliveira, Renê Donizete Ribeiro de; Zamboni, Dario Simões
Source: Science Advances. Unidades: FMRP, FCFRP
Subjects: INFECÇÕES RESPIRATÓRIAS, COVID-19, ADJUVANTES IMUNOLÓGICOS, INFLAMAÇÃO
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ABNT
ALMEIDA, Leticia de et al. Identification of immunomodulatory drugs that inhibit multiple inflammasomes and impair SARS-CoV-2 infection. Science Advances, v. 8, n. 37, 2022Tradução . . Disponível em: https://doi.org/10.1126/sciadv.abo5400. Acesso em: 02 nov. 2024.
APA
Almeida, L. de, Silva, A. L. N., Rodrigues, T. S., Oliveira, S., Ishimoto, A. Y., Seribelli, A. A., et al. (2022). Identification of immunomodulatory drugs that inhibit multiple inflammasomes and impair SARS-CoV-2 infection. Science Advances, 8( 37). doi:10.1126/sciadv.abo5400
NLM
Almeida L de, Silva ALN, Rodrigues TS, Oliveira S, Ishimoto AY, Seribelli AA, Becerra A, Andrade WA, Ataíde MA, Caetano CCS, Sá KSG de, Pelisson N, Martins RB, Souza J de P, Arruda E, Batah SS, Castro R, Frantz FG, Cunha FQ, Cunha TM, Fabro AT, Cunha LD, Louzada-Junior P, Oliveira RDR de, Zamboni DS. Identification of immunomodulatory drugs that inhibit multiple inflammasomes and impair SARS-CoV-2 infection [Internet]. Science Advances. 2022 ; 8( 37):[citado 2024 nov. 02 ] Available from: https://doi.org/10.1126/sciadv.abo5400
Vancouver
Almeida L de, Silva ALN, Rodrigues TS, Oliveira S, Ishimoto AY, Seribelli AA, Becerra A, Andrade WA, Ataíde MA, Caetano CCS, Sá KSG de, Pelisson N, Martins RB, Souza J de P, Arruda E, Batah SS, Castro R, Frantz FG, Cunha FQ, Cunha TM, Fabro AT, Cunha LD, Louzada-Junior P, Oliveira RDR de, Zamboni DS. Identification of immunomodulatory drugs that inhibit multiple inflammasomes and impair SARS-CoV-2 infection [Internet]. Science Advances. 2022 ; 8( 37):[citado 2024 nov. 02 ] Available from: https://doi.org/10.1126/sciadv.abo5400
Artigo de periodico
Philadelphia-negative myeloproliferative neoplasms display alterations in monocyte subpopulations frequency and immunophenotype (2022)
Bassan, Vitor Leonardo; Barretto, Gabriel Dessotti; Almeida, Felipe Campos de; Palma, Patricia Vianna Bonini; Binelli, Larissa Sarri; Silva, João Paulo Lettieri da; Fontanari, Caroline; Castro, Ricardo Cardoso; Pontes, Lorena Lobo de Fiqueiredo; Frantz, Fabiani Gai; Castro, Fabíola Attié de
Source: Medical Oncology. Unidades: FCFRP, FMRP
Subjects: NEOPLASIAS, MEDULA ÓSSEA, MONÓCITOS, INFLAMAÇÃO, CITOMETRIA DE FLUXO, FISIOPATOLOGIA
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ABNT
BASSAN, Vitor Leonardo et al. Philadelphia-negative myeloproliferative neoplasms display alterations in monocyte subpopulations frequency and immunophenotype. Medical Oncology, v. 39, n. 12, p. 1-12, 2022Tradução . . Disponível em: https://doi.org/10.1007/s12032-022-01825-6. Acesso em: 02 nov. 2024.
APA
Bassan, V. L., Barretto, G. D., Almeida, F. C. de, Palma, P. V. B., Binelli, L. S., Silva, J. P. L. da, et al. (2022). Philadelphia-negative myeloproliferative neoplasms display alterations in monocyte subpopulations frequency and immunophenotype. Medical Oncology, 39( 12), 1-12. doi:10.1007/s12032-022-01825-6
NLM
Bassan VL, Barretto GD, Almeida FC de, Palma PVB, Binelli LS, Silva JPL da, Fontanari C, Castro RC, Pontes LL de F, Frantz FG, Castro FA de. Philadelphia-negative myeloproliferative neoplasms display alterations in monocyte subpopulations frequency and immunophenotype [Internet]. Medical Oncology. 2022 ; 39( 12): 1-12.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1007/s12032-022-01825-6
Vancouver
Bassan VL, Barretto GD, Almeida FC de, Palma PVB, Binelli LS, Silva JPL da, Fontanari C, Castro RC, Pontes LL de F, Frantz FG, Castro FA de. Philadelphia-negative myeloproliferative neoplasms display alterations in monocyte subpopulations frequency and immunophenotype [Internet]. Medical Oncology. 2022 ; 39( 12): 1-12.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1007/s12032-022-01825-6
Artigo de periodico
HIV-1 Gag and Vpr impair the inflammasome activation and contribute to the establishment of chronic infection in human primary macrophages (2022)
Lima, Leonardo Judson Galvão de; Zambuzi, Fabiana Albani; Soares, Luana Silva; Fontanari, Caroline ; Meireles, Aline F. Galvão; Brauer, Verônica Soares; Faccioli, Lucia Helena ; Gama, Lucio; Figueiredo, Luiz Tadeu Moraes ; Bou-Habib, Dumith Chequer; Frantz, Fabiani Gai
Source: Molecular Immunology. Unidades: FCFRP, FMRP
Subjects: INFECÇÕES POR HIV, MACRÓFAGOS, INTERLEUCINA 1
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ABNT
LIMA, Leonardo Judson Galvão de et al. HIV-1 Gag and Vpr impair the inflammasome activation and contribute to the establishment of chronic infection in human primary macrophages. Molecular Immunology, v. 148, p. 68-80, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.molimm.2022.04.018. Acesso em: 02 nov. 2024.
APA
Lima, L. J. G. de, Zambuzi, F. A., Soares, L. S., Fontanari, C., Meireles, A. F. G., Brauer, V. S., et al. (2022). HIV-1 Gag and Vpr impair the inflammasome activation and contribute to the establishment of chronic infection in human primary macrophages. Molecular Immunology, 148, 68-80. doi:10.1016/j.molimm.2022.04.018
NLM
Lima LJG de, Zambuzi FA, Soares LS, Fontanari C, Meireles AFG, Brauer VS, Faccioli LH, Gama L, Figueiredo LTM, Bou-Habib DC, Frantz FG. HIV-1 Gag and Vpr impair the inflammasome activation and contribute to the establishment of chronic infection in human primary macrophages [Internet]. Molecular Immunology. 2022 ; 148 68-80.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.molimm.2022.04.018
Vancouver
Lima LJG de, Zambuzi FA, Soares LS, Fontanari C, Meireles AFG, Brauer VS, Faccioli LH, Gama L, Figueiredo LTM, Bou-Habib DC, Frantz FG. HIV-1 Gag and Vpr impair the inflammasome activation and contribute to the establishment of chronic infection in human primary macrophages [Internet]. Molecular Immunology. 2022 ; 148 68-80.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.molimm.2022.04.018
Artigo de periodico
sTREM-1 predicts disease severity and mortality in COVID-19 patients: involvement of peripheral blood leukocytes and MMP-8 activity (2021)
Silva-Neto, Pedro V. da; Carvalho, Jonatan Constança Silva de; Pimentel, Vinícius Eduardo; Pérez, Malena Martínez ; Silva, Thais Fernanda de Campos Fraga da; Fuzo, Carlos Alessandro; Oliveira, Camilla Narjara Simão; Rodrigues, Lilian Cataldi ; Argolo, Jamille Guedes Malta; Carmona-Garcia, Ingryd; Souza, Camila de Oliveira Silva e ; Fernandes, Talita Morais; Bastos, Victor Alexandre Félix; Parra, Rogério Serafim ; Gaspar, Gilberto Gambero ; Rocha, José Joaquim Ribeiro da ; Féres, Omar ; Frantz, Fabiani Gai ; Gerlach, Raquel Fernanda ; Russo, Elisa Maria de Sousa ; Viana, Angelina Lettiere ; Fernandes, Ana Paula Morais ; Santos, Isabel Kinney Ferreira de Miranda ; Bonato, Vânia Luiza Deperon ; Baruffi, Marcelo Dias ; Cardoso, Cristina Ribeiro de Barros ; Faccioli, Lucia Helena ; Sorgi, Carlos Arterio ; Xavier, Ana Carolina; Porcel, Giovanna da Silva; Guarneri, Isabelle Cristina; Zaparoli, Kamila; Garbato, Caroline Tempesta; Ferreira Junior, Angelo Alves; Milanezi, Cristiane Maria
Source: Viruses. Unidades: FCFRP, FMRP, FORP, EERP, FFCLRP, EE/EERP
Subjects: COVID-19, BIOMARCADORES, INFLAMAÇÃO, CÉLULAS MIELOIDES, CUIDADOS INTENSIVOS
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ABNT
SILVA-NETO, Pedro V. da et al. sTREM-1 predicts disease severity and mortality in COVID-19 patients: involvement of peripheral blood leukocytes and MMP-8 activity. Viruses, v. 13, n. 12, p. 1-17, 2021Tradução . . Disponível em: https://doi.org/10.3390/v13122521. Acesso em: 02 nov. 2024.
APA
Silva-Neto, P. V. da, Carvalho, J. C. S. de, Pimentel, V. E., Pérez, M. M., Silva, T. F. de C. F. da, Fuzo, C. A., et al. (2021). sTREM-1 predicts disease severity and mortality in COVID-19 patients: involvement of peripheral blood leukocytes and MMP-8 activity. Viruses, 13( 12), 1-17. doi:10.3390/v13122521
NLM
Silva-Neto PV da, Carvalho JCS de, Pimentel VE, Pérez MM, Silva TF de CF da, Fuzo CA, Oliveira CNS, Rodrigues LC, Argolo JGM, Carmona-Garcia I, Souza C de OS e, Fernandes TM, Bastos VAF, Parra RS, Gaspar GG, Rocha JJR da, Féres O, Frantz FG, Gerlach RF, Russo EM de S, Viana AL, Fernandes APM, Santos IKF de M, Bonato VLD, Baruffi MD, Cardoso CR de B, Faccioli LH, Sorgi CA, Xavier AC, Porcel G da S, Guarneri IC, Zaparoli K, Garbato CT, Ferreira Junior AA, Milanezi CM. sTREM-1 predicts disease severity and mortality in COVID-19 patients: involvement of peripheral blood leukocytes and MMP-8 activity [Internet]. Viruses. 2021 ; 13( 12): 1-17.[citado 2024 nov. 02 ] Available from: https://doi.org/10.3390/v13122521
Vancouver
Silva-Neto PV da, Carvalho JCS de, Pimentel VE, Pérez MM, Silva TF de CF da, Fuzo CA, Oliveira CNS, Rodrigues LC, Argolo JGM, Carmona-Garcia I, Souza C de OS e, Fernandes TM, Bastos VAF, Parra RS, Gaspar GG, Rocha JJR da, Féres O, Frantz FG, Gerlach RF, Russo EM de S, Viana AL, Fernandes APM, Santos IKF de M, Bonato VLD, Baruffi MD, Cardoso CR de B, Faccioli LH, Sorgi CA, Xavier AC, Porcel G da S, Guarneri IC, Zaparoli K, Garbato CT, Ferreira Junior AA, Milanezi CM. sTREM-1 predicts disease severity and mortality in COVID-19 patients: involvement of peripheral blood leukocytes and MMP-8 activity [Internet]. Viruses. 2021 ; 13( 12): 1-17.[citado 2024 nov. 02 ] Available from: https://doi.org/10.3390/v13122521
Artigo de periodico
Notch signaling pathway in infectious diseases: role in the regulation of immune response (2021)
Castro, Ricardo Cardoso; Gonçales, Relber Aguiar; Zambuzi, Fabiana Albani; Frantz, Fabiani Gai
Source: Inflammation Research. Unidades: FCFRP, FMRP
Subjects: DOENÇAS TRANSMISSÍVEIS, RECEPTORES, TRANSDUÇÃO DE SINAL CELULAR
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ABNT
CASTRO, Ricardo Cardoso et al. Notch signaling pathway in infectious diseases: role in the regulation of immune response. Inflammation Research, v. 70, n. 3, p. 261-274, 2021Tradução . . Disponível em: https://doi.org/10.1007/s00011-021-01442-5. Acesso em: 02 nov. 2024.
APA
Castro, R. C., Gonçales, R. A., Zambuzi, F. A., & Frantz, F. G. (2021). Notch signaling pathway in infectious diseases: role in the regulation of immune response. Inflammation Research, 70( 3), 261-274. doi:10.1007/s00011-021-01442-5
NLM
Castro RC, Gonçales RA, Zambuzi FA, Frantz FG. Notch signaling pathway in infectious diseases: role in the regulation of immune response [Internet]. Inflammation Research. 2021 ; 70( 3): 261-274.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1007/s00011-021-01442-5
Vancouver
Castro RC, Gonçales RA, Zambuzi FA, Frantz FG. Notch signaling pathway in infectious diseases: role in the regulation of immune response [Internet]. Inflammation Research. 2021 ; 70( 3): 261-274.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1007/s00011-021-01442-5
Artigo de periodico
Bone marrow soluble mediator signatures of patients with Philadelphia chromosome-negative myeloproliferative neoplasms (2021)
Cominal, Juçara Gastaldi; Cacemiro, Maira da Costa ; Berzoti-Coelho, Maria Gabriela ; Pereira, Illy Enne Gomes ; Frantz, Fabiani Gai ; Souto, Elizabeth Xisto; Covas, Dimas Tadeu; Figueiredo-Pontes, Lorena Lobo de; Oliveira, Maria Carolina; Malmegrim, Kelen Cristina Ribeiro ; Castro, Fabíola Attié de
Source: Frontiers in Oncology. Unidades: FCFRP, FMRP
Subjects: NEOVASCULARIZAÇÃO PATOLÓGICA, MEDULA ÓSSEA, CITOCINAS, INFLAMAÇÃO, NEOPLASIAS
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ABNT
COMINAL, Juçara Gastaldi et al. Bone marrow soluble mediator signatures of patients with Philadelphia chromosome-negative myeloproliferative neoplasms. Frontiers in Oncology, v. 11, p. 1-12 , 2021Tradução . . Disponível em: https://doi.org/10.3389/fonc.2021.665037. Acesso em: 02 nov. 2024.
APA
Cominal, J. G., Cacemiro, M. da C., Berzoti-Coelho, M. G., Pereira, I. E. G., Frantz, F. G., Souto, E. X., et al. (2021). Bone marrow soluble mediator signatures of patients with Philadelphia chromosome-negative myeloproliferative neoplasms. Frontiers in Oncology, 11, 1-12 . doi:10.3389/fonc.2021.665037
NLM
Cominal JG, Cacemiro M da C, Berzoti-Coelho MG, Pereira IEG, Frantz FG, Souto EX, Covas DT, Figueiredo-Pontes LL de, Oliveira MC, Malmegrim KCR, Castro FA de. Bone marrow soluble mediator signatures of patients with Philadelphia chromosome-negative myeloproliferative neoplasms [Internet]. Frontiers in Oncology. 2021 ; 11 1-12 .[citado 2024 nov. 02 ] Available from: https://doi.org/10.3389/fonc.2021.665037
Vancouver
Cominal JG, Cacemiro M da C, Berzoti-Coelho MG, Pereira IEG, Frantz FG, Souto EX, Covas DT, Figueiredo-Pontes LL de, Oliveira MC, Malmegrim KCR, Castro FA de. Bone marrow soluble mediator signatures of patients with Philadelphia chromosome-negative myeloproliferative neoplasms [Internet]. Frontiers in Oncology. 2021 ; 11 1-12 .[citado 2024 nov. 02 ] Available from: https://doi.org/10.3389/fonc.2021.665037
Artigo de periodico
Interleukin-6 ablation does not alter morphofunctional heart characteristics but modulates physiological and inflammatory markers after strenuous exercise (2021)
Rocha, Alisson Luiz da ; Rovina, Rafael Lemes ; Pinto, Ana Paula ; Marafon, Bruno Brieda ; Silva, Lilian Eslaine Costa Mendes da ; Simabuco, Fernando Moreira Simabuco ; Frantz, Fabiani Gai ; Pauli, José Rodrigo; Moura, Leandro Pereira de; Cintra, Dennys Esper; Ropelle, Eduardo Rochete; Tourinho Filho, Hugo ; Freitas, Ellen Cristini de ; Rivas, Donato A.; Silva, Adelino Sanchez Ramos da
Source: Cytokine. Unidades: FMRP, FCFRP, EEFERP
Subjects: BIOMARCADORES, CARDIOMEGALIA, ELETROCARDIOGRAFIA, PERFILAÇÃO DA EXPRESSÃO GÊNICA, CORAÇÃO, DIAGNÓSTICO POR IMAGEM, INTERLEUCINA 6
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ABNT
ROCHA, Alisson Luiz da et al. Interleukin-6 ablation does not alter morphofunctional heart characteristics but modulates physiological and inflammatory markers after strenuous exercise. Cytokine, v. 142, p. 1-11, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.cyto.2021.155494. Acesso em: 02 nov. 2024.
APA
Rocha, A. L. da, Rovina, R. L., Pinto, A. P., Marafon, B. B., Silva, L. E. C. M. da, Simabuco, F. M. S., et al. (2021). Interleukin-6 ablation does not alter morphofunctional heart characteristics but modulates physiological and inflammatory markers after strenuous exercise. Cytokine, 142, 1-11. doi:10.1016/j.cyto.2021.155494
NLM
Rocha AL da, Rovina RL, Pinto AP, Marafon BB, Silva LECM da, Simabuco FMS, Frantz FG, Pauli JR, Moura LP de, Cintra DE, Ropelle ER, Tourinho Filho H, Freitas EC de, Rivas DA, Silva ASR da. Interleukin-6 ablation does not alter morphofunctional heart characteristics but modulates physiological and inflammatory markers after strenuous exercise [Internet]. Cytokine. 2021 ; 142 1-11.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.cyto.2021.155494
Vancouver
Rocha AL da, Rovina RL, Pinto AP, Marafon BB, Silva LECM da, Simabuco FMS, Frantz FG, Pauli JR, Moura LP de, Cintra DE, Ropelle ER, Tourinho Filho H, Freitas EC de, Rivas DA, Silva ASR da. Interleukin-6 ablation does not alter morphofunctional heart characteristics but modulates physiological and inflammatory markers after strenuous exercise [Internet]. Cytokine. 2021 ; 142 1-11.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.cyto.2021.155494
Artigo de periodico
Decitabine promotes modulation in phenotype and function of monocytes and macrophages that drive immune response regulation (2021)
Zambuzi, Fabiana Albani ; Silva, Priscilla Mariane Cardoso; Castro, Ricardo Cardoso ; Fontanari, Caroline ; Emery, Flavio da Silva ; Frantz, Fabiani Gai
Source: Cells. Unidades: FCFRP, FMRP
Subjects: FAGOCITOSE, MONÓCITOS, MACRÓFAGOS, ADJUVANTES IMUNOLÓGICOS, SISTEMA IMUNE, IMUNOLOGIA, FENÓTIPOS, DOENÇAS INFECCIOSAS
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ABNT
ZAMBUZI, Fabiana Albani et al. Decitabine promotes modulation in phenotype and function of monocytes and macrophages that drive immune response regulation. Cells, v. 10, n. 4, p. 1-16, 2021Tradução . . Disponível em: https://doi.org/10.3390/cells10040868. Acesso em: 02 nov. 2024.
APA
Zambuzi, F. A., Silva, P. M. C., Castro, R. C., Fontanari, C., Emery, F. da S., & Frantz, F. G. (2021). Decitabine promotes modulation in phenotype and function of monocytes and macrophages that drive immune response regulation. Cells, 10( 4), 1-16. doi:10.3390/cells10040868
NLM
Zambuzi FA, Silva PMC, Castro RC, Fontanari C, Emery F da S, Frantz FG. Decitabine promotes modulation in phenotype and function of monocytes and macrophages that drive immune response regulation [Internet]. Cells. 2021 ; 10( 4): 1-16.[citado 2024 nov. 02 ] Available from: https://doi.org/10.3390/cells10040868
Vancouver
Zambuzi FA, Silva PMC, Castro RC, Fontanari C, Emery F da S, Frantz FG. Decitabine promotes modulation in phenotype and function of monocytes and macrophages that drive immune response regulation [Internet]. Cells. 2021 ; 10( 4): 1-16.[citado 2024 nov. 02 ] Available from: https://doi.org/10.3390/cells10040868
Artigo de periodico
Inflammasomes are activated in response to SARS-CoV-2 infection and are associated with COVID-19 severity in patients (2021)
Rodrigues, Tamara Silva ; Sá, Keyla Santos Guedes de ; Ishimoto, Adriene Yumi; Becerra, Amanda; Oliveira, Samuel; Almeida, Leticia; Gonçalves, Augusto V.; Perucello, Debora B.; Andrade, Warrison A. ; Castro, Ricardo; Veras, Flavio P.; Toller-Kawahisa, Juliana E.; Nascimento, Daniele C.; Lima, Mikhael H. F. de; Silva, Camila M. S.; Caetite, Diego B.; Martins, Ronaldo Bragança ; Castro, Italo A.; Pontelli, Marjorie C.; Barros, Fabio C. de; Amaral, Natália B. do; Giannini, Marcela C.; Bonjorno, Letícia P.; Lopes, Maria Isabel F.; Santana, Rodrigo de Carvalho ; Vilar, Fernando C.; Auxiliadora-Martins, Maria ; Luppino-Assad, Rodrigo; Almeida, Sergio C. L. de; Oliveira, Fabiola R. de; Batah, Sabrina Setembre ; Siyuan, Li; Benatti, Maira N.; Cunha, Thiago Mattar ; Alves Filho, José Carlos Farias ; Cunha, Fernando de Queiroz ; Cunha, Larissa D.; Frantz, Fabiani Gai ; Kohlsdorf, Tiana ; Fabro, Alexandre Todorovic ; Arruda, Eurico ; Oliveira, Renê D. R. de; Louzada Júnior, Paulo ; Zamboni, Dario Simões
Source: Journal of Experimental Medicine. Unidades: FMRP, FCFRP, FFCLRP
Subjects: COVID-19, INFLAMAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RODRIGUES, Tamara Silva et al. Inflammasomes are activated in response to SARS-CoV-2 infection and are associated with COVID-19 severity in patients. Journal of Experimental Medicine, v. 218, n. 3, 2021Tradução . . Disponível em: https://doi.org/10.1084/jem.20201707. Acesso em: 02 nov. 2024.
APA
Rodrigues, T. S., Sá, K. S. G. de, Ishimoto, A. Y., Becerra, A., Oliveira, S., Almeida, L., et al. (2021). Inflammasomes are activated in response to SARS-CoV-2 infection and are associated with COVID-19 severity in patients. Journal of Experimental Medicine, 218( 3). doi:10.1084/jem.20201707
NLM
Rodrigues TS, Sá KSG de, Ishimoto AY, Becerra A, Oliveira S, Almeida L, Gonçalves AV, Perucello DB, Andrade WA, Castro R, Veras FP, Toller-Kawahisa JE, Nascimento DC, Lima MHF de, Silva CMS, Caetite DB, Martins RB, Castro IA, Pontelli MC, Barros FC de, Amaral NB do, Giannini MC, Bonjorno LP, Lopes MIF, Santana R de C, Vilar FC, Auxiliadora-Martins M, Luppino-Assad R, Almeida SCL de, Oliveira FR de, Batah SS, Siyuan L, Benatti MN, Cunha TM, Alves Filho JCF, Cunha F de Q, Cunha LD, Frantz FG, Kohlsdorf T, Fabro AT, Arruda E, Oliveira RDR de, Louzada Júnior P, Zamboni DS. Inflammasomes are activated in response to SARS-CoV-2 infection and are associated with COVID-19 severity in patients [Internet]. Journal of Experimental Medicine. 2021 ; 218( 3):[citado 2024 nov. 02 ] Available from: https://doi.org/10.1084/jem.20201707
Vancouver
Rodrigues TS, Sá KSG de, Ishimoto AY, Becerra A, Oliveira S, Almeida L, Gonçalves AV, Perucello DB, Andrade WA, Castro R, Veras FP, Toller-Kawahisa JE, Nascimento DC, Lima MHF de, Silva CMS, Caetite DB, Martins RB, Castro IA, Pontelli MC, Barros FC de, Amaral NB do, Giannini MC, Bonjorno LP, Lopes MIF, Santana R de C, Vilar FC, Auxiliadora-Martins M, Luppino-Assad R, Almeida SCL de, Oliveira FR de, Batah SS, Siyuan L, Benatti MN, Cunha TM, Alves Filho JCF, Cunha F de Q, Cunha LD, Frantz FG, Kohlsdorf T, Fabro AT, Arruda E, Oliveira RDR de, Louzada Júnior P, Zamboni DS. Inflammasomes are activated in response to SARS-CoV-2 infection and are associated with COVID-19 severity in patients [Internet]. Journal of Experimental Medicine. 2021 ; 218( 3):[citado 2024 nov. 02 ] Available from: https://doi.org/10.1084/jem.20201707
Artigo de periodico
The influence of dehydroepiandrosterone on effector functions of neutrophils (2021)
Brauer, Verônica Soares; Zambuzi, Fabiana Albani; Espíndola, Milena Sobral; Cavalcanti Neto, Marinaldo Pacífico; Prado, Morgana Kelly Borges; Silva, Priscilla Mariane Cardoso; Soares, Luana Silva; Lima, Leonardo Judson Galvão de; Leopoldino, Andréia Machado ; Cardoso, Cristina Ribeiro de Barros; Frantz, Fabiani Gai
Source: Brazilian Journal of Pharmaceutical Sciences. Unidade: FCFRP
Subjects: FAGOCITOSE, NEUTRÓFILOS, HORMÔNIOS SEXUAIS, SALMONELLA TYPHIMURIUM
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ABNT
BRAUER, Verônica Soares et al. The influence of dehydroepiandrosterone on effector functions of neutrophils. Brazilian Journal of Pharmaceutical Sciences, v. 57, p. 1-15, 2021Tradução . . Disponível em: https://doi.org/10.1590/s2175-97902020000419139. Acesso em: 02 nov. 2024.
APA
Brauer, V. S., Zambuzi, F. A., Espíndola, M. S., Cavalcanti Neto, M. P., Prado, M. K. B., Silva, P. M. C., et al. (2021). The influence of dehydroepiandrosterone on effector functions of neutrophils. Brazilian Journal of Pharmaceutical Sciences, 57, 1-15. doi:10.1590/s2175-97902020000419139
NLM
Brauer VS, Zambuzi FA, Espíndola MS, Cavalcanti Neto MP, Prado MKB, Silva PMC, Soares LS, Lima LJG de, Leopoldino AM, Cardoso CR de B, Frantz FG. The influence of dehydroepiandrosterone on effector functions of neutrophils [Internet]. Brazilian Journal of Pharmaceutical Sciences. 2021 ; 57 1-15.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1590/s2175-97902020000419139
Vancouver
Brauer VS, Zambuzi FA, Espíndola MS, Cavalcanti Neto MP, Prado MKB, Silva PMC, Soares LS, Lima LJG de, Leopoldino AM, Cardoso CR de B, Frantz FG. The influence of dehydroepiandrosterone on effector functions of neutrophils [Internet]. Brazilian Journal of Pharmaceutical Sciences. 2021 ; 57 1-15.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1590/s2175-97902020000419139
Artigo de periodico
TLR4 deletion increases basal energy expenditure and attenuates heart apoptosis and ER stress but mitigates the training-induced cardiac function and performance improvement (2021)
Vicente, Larissa Gaioto de; Muñoz, Vitor Rosetto; Pinto, Ana Paula ; Rovina, Rafael Lemes ; Rocha, Alisson Luiz da ; Marafon, Bruno Brieda ; Tavares, Maria Eduarda de Almeida; Teixeira, Giovana Rampazzo; Ferrari, Gustavo Duarte; Alberici, Luciane Carla ; Frantz, Fabiani Gai ; Simabuco, Fernando Moreira; Ropelle, Eduardo Rochete; Moura, Leandro Pereira de; Cintra, Dennys Esper; Pauli, José Rodrigo; Silva, Adelino Sanchez Ramos da
Source: Life Sciences. Unidades: FCFRP, EEFERP, FMRP
Subjects: BIOINFORMÁTICA, COMPUTAÇÃO APLICADA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VICENTE, Larissa Gaioto de et al. TLR4 deletion increases basal energy expenditure and attenuates heart apoptosis and ER stress but mitigates the training-induced cardiac function and performance improvement. Life Sciences, v. 285, p. 1-10, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2021.119988. Acesso em: 02 nov. 2024.
APA
Vicente, L. G. de, Muñoz, V. R., Pinto, A. P., Rovina, R. L., Rocha, A. L. da, Marafon, B. B., et al. (2021). TLR4 deletion increases basal energy expenditure and attenuates heart apoptosis and ER stress but mitigates the training-induced cardiac function and performance improvement. Life Sciences, 285, 1-10. doi:10.1016/j.lfs.2021.119988
NLM
Vicente LG de, Muñoz VR, Pinto AP, Rovina RL, Rocha AL da, Marafon BB, Tavares ME de A, Teixeira GR, Ferrari GD, Alberici LC, Frantz FG, Simabuco FM, Ropelle ER, Moura LP de, Cintra DE, Pauli JR, Silva ASR da. TLR4 deletion increases basal energy expenditure and attenuates heart apoptosis and ER stress but mitigates the training-induced cardiac function and performance improvement [Internet]. Life Sciences. 2021 ; 285 1-10.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.lfs.2021.119988
Vancouver
Vicente LG de, Muñoz VR, Pinto AP, Rovina RL, Rocha AL da, Marafon BB, Tavares ME de A, Teixeira GR, Ferrari GD, Alberici LC, Frantz FG, Simabuco FM, Ropelle ER, Moura LP de, Cintra DE, Pauli JR, Silva ASR da. TLR4 deletion increases basal energy expenditure and attenuates heart apoptosis and ER stress but mitigates the training-induced cardiac function and performance improvement [Internet]. Life Sciences. 2021 ; 285 1-10.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.lfs.2021.119988
Artigo de periodico
Publisher Correction: Leishmania RNA virus exacerbates Leishmaniasis by subverting innate immunity via TLR3-mediated NLRP3 inflammasome inhibition (2020)
Carvalho, Renan Villanova Homem de; Lima Júnior, Djalma Souza; Silva, Marcus Vinícius Gomes da; Dilucca, Marisa; Rodrigues, Tamara Silva; Horta, Catarina Veltrini; Silva, Alexandre Luiz Neves; Silva, Patrick Fernandes da; Frantz, Fabiani Gai ; Lorenzon, Lucas Bigolin; Souza, Marcos Michel de; Almeida, Fausto Bruno dos Reis ; Cantanhêde, Lilian Motta; Ferreira, Ricardo de Godoi Mattos; Cruz, Angela Kaysel ; Zamboni, Dario Simões
Source: Nature Communications. Unidades: FCFRP, FMRP
Subjects: IMUNOLOGIA, VÍRUS DE RNA, LEISHMANIA, MACRÓFAGOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CARVALHO, Renan Villanova Homem de et al. Publisher Correction: Leishmania RNA virus exacerbates Leishmaniasis by subverting innate immunity via TLR3-mediated NLRP3 inflammasome inhibition. Nature Communications, v. 11, 2020Tradução . . Disponível em: https://doi.org/10.1038/s41467-019-13356-2. Acesso em: 02 nov. 2024.
APA
Carvalho, R. V. H. de, Lima Júnior, D. S., Silva, M. V. G. da, Dilucca, M., Rodrigues, T. S., Horta, C. V., et al. (2020). Publisher Correction: Leishmania RNA virus exacerbates Leishmaniasis by subverting innate immunity via TLR3-mediated NLRP3 inflammasome inhibition. Nature Communications, 11. doi:10.1038/s41467-019-13356-2
NLM
Carvalho RVH de, Lima Júnior DS, Silva MVG da, Dilucca M, Rodrigues TS, Horta CV, Silva ALN, Silva PF da, Frantz FG, Lorenzon LB, Souza MM de, Almeida FB dos R, Cantanhêde LM, Ferreira R de GM, Cruz AK, Zamboni DS. Publisher Correction: Leishmania RNA virus exacerbates Leishmaniasis by subverting innate immunity via TLR3-mediated NLRP3 inflammasome inhibition [Internet]. Nature Communications. 2020 ; 11[citado 2024 nov. 02 ] Available from: https://doi.org/10.1038/s41467-019-13356-2
Vancouver
Carvalho RVH de, Lima Júnior DS, Silva MVG da, Dilucca M, Rodrigues TS, Horta CV, Silva ALN, Silva PF da, Frantz FG, Lorenzon LB, Souza MM de, Almeida FB dos R, Cantanhêde LM, Ferreira R de GM, Cruz AK, Zamboni DS. Publisher Correction: Leishmania RNA virus exacerbates Leishmaniasis by subverting innate immunity via TLR3-mediated NLRP3 inflammasome inhibition [Internet]. Nature Communications. 2020 ; 11[citado 2024 nov. 02 ] Available from: https://doi.org/10.1038/s41467-019-13356-2
Artigo de periodico
Eicosanoid pathway on host resistance and inflammation during Mycobacterium tuberculosis infection is comprised by LTB4 reduction but not PGE2 increment (2020)
Sorgi, Carlos Arterio ; Soares, Elyara Maria; Rosada, Rogério Silva; Bitencourt, Claudia Silva; Zoccal, Karina Furlani ; Pereira, Priscilla Aparecida Tártari; Fontanari, Caroline; Brandão, Izaira Tincani; Masson, Ana Paula ; Ramos, Simone Gusmão ; Silva, Célio Lopes ; Frantz, Fabiani Gai ; Faccioli, Lucia Helena
Source: Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. Unidades: FFCLRP, FCFRP, FMRP
Subjects: TUBERCULOSE, LEUCOTRIENOS, PROSTAGLANDINAS, INFLAMAÇÃO, IMUNOPATOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SORGI, Carlos Arterio et al. Eicosanoid pathway on host resistance and inflammation during Mycobacterium tuberculosis infection is comprised by LTB4 reduction but not PGE2 increment. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, v. 1866, n. 3, p. [10] , 2020Tradução . . Disponível em: https://doi.org/10.1016/j.bbadis.2019.165574. Acesso em: 02 nov. 2024.
APA
Sorgi, C. A., Soares, E. M., Rosada, R. S., Bitencourt, C. S., Zoccal, K. F., Pereira, P. A. T., et al. (2020). Eicosanoid pathway on host resistance and inflammation during Mycobacterium tuberculosis infection is comprised by LTB4 reduction but not PGE2 increment. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 1866( 3), [10] . doi:10.1016/j.bbadis.2019.165574
NLM
Sorgi CA, Soares EM, Rosada RS, Bitencourt CS, Zoccal KF, Pereira PAT, Fontanari C, Brandão IT, Masson AP, Ramos SG, Silva CL, Frantz FG, Faccioli LH. Eicosanoid pathway on host resistance and inflammation during Mycobacterium tuberculosis infection is comprised by LTB4 reduction but not PGE2 increment [Internet]. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 2020 ; 1866( 3): [10] .[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.bbadis.2019.165574
Vancouver
Sorgi CA, Soares EM, Rosada RS, Bitencourt CS, Zoccal KF, Pereira PAT, Fontanari C, Brandão IT, Masson AP, Ramos SG, Silva CL, Frantz FG, Faccioli LH. Eicosanoid pathway on host resistance and inflammation during Mycobacterium tuberculosis infection is comprised by LTB4 reduction but not PGE2 increment [Internet]. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 2020 ; 1866( 3): [10] .[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.bbadis.2019.165574
Artigo de periodico
Type I and II interferon are associated with high expression of the hippo pathway family members (2020)
Sciescia, Bianca; Tognon, Raquel; Nunes, Natália de Souza; Malta, Tathiane Maistro; Frantz, Fabiani Gai ; Castro, Fabíola Attié de ; Cacemiro, Maira da Costa
Source: Journal of Clinical & Cellular Immunology. Unidade: FCFRP
Subjects: INFLAMAÇÃO, CITOCINAS, QUIMIOCINAS, PROLIFERAÇÃO CELULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SCIESCIA, Bianca et al. Type I and II interferon are associated with high expression of the hippo pathway family members. Journal of Clinical & Cellular Immunology, v. 11, n. 1, 2020Tradução . . Disponível em: https://doi.org/10.35248/2155-9899.20.11:585. Acesso em: 02 nov. 2024.
APA
Sciescia, B., Tognon, R., Nunes, N. de S., Malta, T. M., Frantz, F. G., Castro, F. A. de, & Cacemiro, M. da C. (2020). Type I and II interferon are associated with high expression of the hippo pathway family members. Journal of Clinical & Cellular Immunology, 11( 1). doi:10.35248/2155-9899.20.11:585
NLM
Sciescia B, Tognon R, Nunes N de S, Malta TM, Frantz FG, Castro FA de, Cacemiro M da C. Type I and II interferon are associated with high expression of the hippo pathway family members [Internet]. Journal of Clinical & Cellular Immunology. 2020 ; 11( 1):[citado 2024 nov. 02 ] Available from: https://doi.org/10.35248/2155-9899.20.11:585
Vancouver
Sciescia B, Tognon R, Nunes N de S, Malta TM, Frantz FG, Castro FA de, Cacemiro M da C. Type I and II interferon are associated with high expression of the hippo pathway family members [Internet]. Journal of Clinical & Cellular Immunology. 2020 ; 11( 1):[citado 2024 nov. 02 ] Available from: https://doi.org/10.35248/2155-9899.20.11:585
Artigo de periodico
Role of crotoxin in coagulation: novel insights into anticoagulant mechanisms and impairment of inflammation-induced coagulation (2020)
Gimenez, Bruna Terada; Cezarette, Gabriel Neves; Bomfim, Aline de Sousa; Monteiro, Wuelton Marcelo; Russo, Elisa Maria de Sousa ; Frantz, Fabiani Gai ; Sampaio, Suely Vilela ; Sartim, Marco Aurélio
Source: Journal of Venomous Animals and Toxins including Tropical Diseases. Unidade: FCFRP
Subjects: COBRAS, VENENOS, FOSFOLIPASES A, ANTI-INFLAMATÓRIOS, FATORES DE COAGULAÇÃO SANGUÍNEA, CITOCINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GIMENEZ, Bruna Terada et al. Role of crotoxin in coagulation: novel insights into anticoagulant mechanisms and impairment of inflammation-induced coagulation. Journal of Venomous Animals and Toxins including Tropical Diseases, v. 26, 2020Tradução . . Disponível em: https://doi.org/10.1590/1678-9199-JVATITD-2020-0076. Acesso em: 02 nov. 2024.
APA
Gimenez, B. T., Cezarette, G. N., Bomfim, A. de S., Monteiro, W. M., Russo, E. M. de S., Frantz, F. G., et al. (2020). Role of crotoxin in coagulation: novel insights into anticoagulant mechanisms and impairment of inflammation-induced coagulation. Journal of Venomous Animals and Toxins including Tropical Diseases, 26. doi:10.1590/1678-9199-JVATITD-2020-0076
NLM
Gimenez BT, Cezarette GN, Bomfim A de S, Monteiro WM, Russo EM de S, Frantz FG, Sampaio SV, Sartim MA. Role of crotoxin in coagulation: novel insights into anticoagulant mechanisms and impairment of inflammation-induced coagulation [Internet]. Journal of Venomous Animals and Toxins including Tropical Diseases. 2020 ; 26[citado 2024 nov. 02 ] Available from: https://doi.org/10.1590/1678-9199-JVATITD-2020-0076
Vancouver
Gimenez BT, Cezarette GN, Bomfim A de S, Monteiro WM, Russo EM de S, Frantz FG, Sampaio SV, Sartim MA. Role of crotoxin in coagulation: novel insights into anticoagulant mechanisms and impairment of inflammation-induced coagulation [Internet]. Journal of Venomous Animals and Toxins including Tropical Diseases. 2020 ; 26[citado 2024 nov. 02 ] Available from: https://doi.org/10.1590/1678-9199-JVATITD-2020-0076
Artigo de periodico
Regional cerebral blood flow at rest in obesity (2020)
Silvah, José Henrique da; Marchini, Julio Sergio ; Lima, Cristiane Maria Mártires de; Nicoletti, Carolina Ferreira; Santos, Leonardo Alexandre; Itikawa, Emerson Nobuyuki; Trevisan, Ana Carolina; Pitella, Felipe Arriva; Kato, Mery; Iucif Junior, Nelson; Frantz, Fabiani Gai ; Cunha, Selma Freire de Carvalho da; Buchpiguel, Carlos Alberto; Wichert-Ana, Lauro
Source: Nutrition. Unidades: FMRP, FCFRP, FM
Subjects: COMPORTAMENTO ALIMENTAR, INSULINA, OBESIDADE, ANORMALIDADES DO SISTEMA NERVOSO
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ABNT
SILVAH, José Henrique da et al. Regional cerebral blood flow at rest in obesity. Nutrition, v. 79-80, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.nut.2020.110888. Acesso em: 02 nov. 2024.
APA
Silvah, J. H. da, Marchini, J. S., Lima, C. M. M. de, Nicoletti, C. F., Santos, L. A., Itikawa, E. N., et al. (2020). Regional cerebral blood flow at rest in obesity. Nutrition, 79-80. doi:10.1016/j.nut.2020.110888
NLM
Silvah JH da, Marchini JS, Lima CMM de, Nicoletti CF, Santos LA, Itikawa EN, Trevisan AC, Pitella FA, Kato M, Iucif Junior N, Frantz FG, Cunha SF de C da, Buchpiguel CA, Wichert-Ana L. Regional cerebral blood flow at rest in obesity [Internet]. Nutrition. 2020 ; 79-80[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.nut.2020.110888
Vancouver
Silvah JH da, Marchini JS, Lima CMM de, Nicoletti CF, Santos LA, Itikawa EN, Trevisan AC, Pitella FA, Kato M, Iucif Junior N, Frantz FG, Cunha SF de C da, Buchpiguel CA, Wichert-Ana L. Regional cerebral blood flow at rest in obesity [Internet]. Nutrition. 2020 ; 79-80[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.nut.2020.110888
Artigo de periodico
Tlr4 participates in the responses of markers of apoptosis, inflammation, and ER stress to different acute exercise intensities in mice hearts (2020)
Vicente, Larissa Gaioto de; Pinto, Ana Paula; Muñoz, Vitor R.; Rovina, Rafael Lemes ; Rocha, Alisson Luiz da ; Gaspar, Rafael C.; Silva, Lilian E. C. M. da; Simabuco, Fernando M.; Frantz, Fabiani Gai ; Pauli, José R.; Moura, Leandro P. de; Cintra, Dennys E.; Ropelle, Eduardo R.; Silva, Adelino Sanchez Ramos da
Source: Life Sciences. Unidades: FCFRP, EEFERP, FMRP
Subjects: ECOCARDIOGRAFIA, EXPRESSÃO GÊNICA, BIOINFORMÁTICA, APOPTOSE, EXERCÍCIO FÍSICO, PROTEÍNAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VICENTE, Larissa Gaioto de et al. Tlr4 participates in the responses of markers of apoptosis, inflammation, and ER stress to different acute exercise intensities in mice hearts. Life Sciences, v. 240, p. 1-9, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2019.117107. Acesso em: 02 nov. 2024.
APA
Vicente, L. G. de, Pinto, A. P., Muñoz, V. R., Rovina, R. L., Rocha, A. L. da, Gaspar, R. C., et al. (2020). Tlr4 participates in the responses of markers of apoptosis, inflammation, and ER stress to different acute exercise intensities in mice hearts. Life Sciences, 240, 1-9. doi:10.1016/j.lfs.2019.117107
NLM
Vicente LG de, Pinto AP, Muñoz VR, Rovina RL, Rocha AL da, Gaspar RC, Silva LECM da, Simabuco FM, Frantz FG, Pauli JR, Moura LP de, Cintra DE, Ropelle ER, Silva ASR da. Tlr4 participates in the responses of markers of apoptosis, inflammation, and ER stress to different acute exercise intensities in mice hearts [Internet]. Life Sciences. 2020 ; 240 1-9.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.lfs.2019.117107
Vancouver
Vicente LG de, Pinto AP, Muñoz VR, Rovina RL, Rocha AL da, Gaspar RC, Silva LECM da, Simabuco FM, Frantz FG, Pauli JR, Moura LP de, Cintra DE, Ropelle ER, Silva ASR da. Tlr4 participates in the responses of markers of apoptosis, inflammation, and ER stress to different acute exercise intensities in mice hearts [Internet]. Life Sciences. 2020 ; 240 1-9.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.lfs.2019.117107
Artigo de periodico
Pulmonary surfactant phosphatidylcholines induce immunological adaptation of alveolar macrophages (2020)
Loureiro, Luma da Costa; Loureiro, Luana da Costa; Gabriel-Junior, Edson Alves; Zambuzi, Fabiana Albani; Fontanari, Caroline ; Sales-Campos, Helioswilton; Frantz, Fabiani Gai ; Faccioli, Lucia Helena ; Sorgi, Carlos Arterio
Source: Molecular Immunology. Unidades: FCFRP, FFCLRP
Subjects: MACRÓFAGOS, EPIGÊNESE GENÉTICA, INFLAMAÇÃO, IMUNIDADE, PULMÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LOUREIRO, Luma da Costa et al. Pulmonary surfactant phosphatidylcholines induce immunological adaptation of alveolar macrophages. Molecular Immunology, v. 122, p. 163-172, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.molimm.2020.04.010. Acesso em: 02 nov. 2024.
APA
Loureiro, L. da C., Loureiro, L. da C., Gabriel-Junior, E. A., Zambuzi, F. A., Fontanari, C., Sales-Campos, H., et al. (2020). Pulmonary surfactant phosphatidylcholines induce immunological adaptation of alveolar macrophages. Molecular Immunology, 122, 163-172. doi:10.1016/j.molimm.2020.04.010
NLM
Loureiro L da C, Loureiro L da C, Gabriel-Junior EA, Zambuzi FA, Fontanari C, Sales-Campos H, Frantz FG, Faccioli LH, Sorgi CA. Pulmonary surfactant phosphatidylcholines induce immunological adaptation of alveolar macrophages [Internet]. Molecular Immunology. 2020 ; 122 163-172.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.molimm.2020.04.010
Vancouver
Loureiro L da C, Loureiro L da C, Gabriel-Junior EA, Zambuzi FA, Fontanari C, Sales-Campos H, Frantz FG, Faccioli LH, Sorgi CA. Pulmonary surfactant phosphatidylcholines induce immunological adaptation of alveolar macrophages [Internet]. Molecular Immunology. 2020 ; 122 163-172.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.molimm.2020.04.010

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