ReP USP - Resultado da busca

Artigo de periodico
PPAR-γ/IL-10 axis inhibits MyD88 expression and ameliorates murine polymicrobial sepsis (2014)
Ferreira, Ana Elisa; Sisti, Flavia; Sônego, Fabiane; Wang, Suojuan; Filgueiras, Luciano Ribeiro; Brandt, Stephanie; Serezani, Ana Paula Moreira; Du, Hong; Cunha, Fernando de Queiroz; Alves Filho, José Carlos Farias; Serezani, Carlos Henrique
Source: Journal of Immunology. Unidade: FMRP
Subjects: EXPRESSÃO GÊNICA, REGULAÇÃO GÊNICA, INTERLEUCINA 10, SEPSE (IMUNOLOGIA), MODELOS ANIMAIS DE DOENÇAS
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ABNT
FERREIRA, Ana Elisa et al. PPAR-γ/IL-10 axis inhibits MyD88 expression and ameliorates murine polymicrobial sepsis. Journal of Immunology, v. 192, n. 5, p. 2357-2365, 2014Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1302375. Acesso em: 18 nov. 2024.
APA
Ferreira, A. E., Sisti, F., Sônego, F., Wang, S., Filgueiras, L. R., Brandt, S., et al. (2014). PPAR-γ/IL-10 axis inhibits MyD88 expression and ameliorates murine polymicrobial sepsis. Journal of Immunology, 192( 5), 2357-2365. doi:10.4049/jimmunol.1302375
NLM
Ferreira AE, Sisti F, Sônego F, Wang S, Filgueiras LR, Brandt S, Serezani APM, Du H, Cunha F de Q, Alves Filho JCF, Serezani CH. PPAR-γ/IL-10 axis inhibits MyD88 expression and ameliorates murine polymicrobial sepsis [Internet]. Journal of Immunology. 2014 ; 192( 5): 2357-2365.[citado 2024 nov. 18 ] Available from: https://doi.org/10.4049/jimmunol.1302375
Vancouver
Ferreira AE, Sisti F, Sônego F, Wang S, Filgueiras LR, Brandt S, Serezani APM, Du H, Cunha F de Q, Alves Filho JCF, Serezani CH. PPAR-γ/IL-10 axis inhibits MyD88 expression and ameliorates murine polymicrobial sepsis [Internet]. Journal of Immunology. 2014 ; 192( 5): 2357-2365.[citado 2024 nov. 18 ] Available from: https://doi.org/10.4049/jimmunol.1302375
Artigo de periodico
Periodontal pathogens directly promote autoimmune experimental arthritis by inducing a TLR2- and IL-1–driven Th17 response (2014)
Aquino, Sabrina G. de; Abdollahi-Roodsaz, Shahla; Koenders, Marije I.; van de Loo, Fons A. J.; Pruijn, Ger J. M.; Marijnissen, Renoud J.; Walgreen, Birgitte; Helsen, Monique M.; van den Bersselaar, Liduine A.; Molon, Rafael S. de; Avila-Campos, Mario Julio; Cunha, Fernando de Queiroz; Cirelli, Joni A.; van den Berg, Wim B.
Source: Journal of Immunology. Unidades: ICB, FMRP
Subjects: DOENÇAS AUTOIMUNES, ARTRITE (IMUNOLOGIA), PERIODONTO, MICROBIOLOGIA ORAL
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ABNT
AQUINO, Sabrina G. de et al. Periodontal pathogens directly promote autoimmune experimental arthritis by inducing a TLR2- and IL-1–driven Th17 response. Journal of Immunology, v. 192, n. 9, p. 4103-4111, 2014Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1301970. Acesso em: 18 nov. 2024.
APA
Aquino, S. G. de, Abdollahi-Roodsaz, S., Koenders, M. I., van de Loo, F. A. J., Pruijn, G. J. M., Marijnissen, R. J., et al. (2014). Periodontal pathogens directly promote autoimmune experimental arthritis by inducing a TLR2- and IL-1–driven Th17 response. Journal of Immunology, 192( 9), 4103-4111. doi:10.4049/jimmunol.1301970
NLM
Aquino SG de, Abdollahi-Roodsaz S, Koenders MI, van de Loo FAJ, Pruijn GJM, Marijnissen RJ, Walgreen B, Helsen MM, van den Bersselaar LA, Molon RS de, Avila-Campos MJ, Cunha F de Q, Cirelli JA, van den Berg WB. Periodontal pathogens directly promote autoimmune experimental arthritis by inducing a TLR2- and IL-1–driven Th17 response [Internet]. Journal of Immunology. 2014 ; 192( 9): 4103-4111.[citado 2024 nov. 18 ] Available from: https://doi.org/10.4049/jimmunol.1301970
Vancouver
Aquino SG de, Abdollahi-Roodsaz S, Koenders MI, van de Loo FAJ, Pruijn GJM, Marijnissen RJ, Walgreen B, Helsen MM, van den Bersselaar LA, Molon RS de, Avila-Campos MJ, Cunha F de Q, Cirelli JA, van den Berg WB. Periodontal pathogens directly promote autoimmune experimental arthritis by inducing a TLR2- and IL-1–driven Th17 response [Internet]. Journal of Immunology. 2014 ; 192( 9): 4103-4111.[citado 2024 nov. 18 ] Available from: https://doi.org/10.4049/jimmunol.1301970
Artigo de periodico
Histamine H2 receptor signaling in the pathogenesis of sepsis: studies in a murine Diabetes model (2013)
Carlos, Daniela; Spiller, Fernando; Souto, Fabrício O.; Trevelin, Silvia C.; Borges, Vanessa F.; Freitas, Andressa de; Alves Filho, José Carlos Farias; Silva, João Santana da; Ryffel, Bernhard; Cunha, Fernando de Queiroz
Source: Journal of Immunology. Unidade: FMRP
Subjects: DIABETES MELLITUS (EXPERIMENTAÇÃO), SEPSE, RECEPTORES (METABOLISMO), BACTEREMIA
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ABNT
CARLOS, Daniela et al. Histamine H2 receptor signaling in the pathogenesis of sepsis: studies in a murine Diabetes model. Journal of Immunology, v. 191, n. 3, p. 1373-1382, 2013Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1202907. Acesso em: 18 nov. 2024.
APA
Carlos, D., Spiller, F., Souto, F. O., Trevelin, S. C., Borges, V. F., Freitas, A. de, et al. (2013). Histamine H2 receptor signaling in the pathogenesis of sepsis: studies in a murine Diabetes model. Journal of Immunology, 191( 3), 1373-1382. doi:10.4049/jimmunol.1202907
NLM
Carlos D, Spiller F, Souto FO, Trevelin SC, Borges VF, Freitas A de, Alves Filho JCF, Silva JS da, Ryffel B, Cunha F de Q. Histamine H2 receptor signaling in the pathogenesis of sepsis: studies in a murine Diabetes model [Internet]. Journal of Immunology. 2013 ; 191( 3): 1373-1382.[citado 2024 nov. 18 ] Available from: https://doi.org/10.4049/jimmunol.1202907
Vancouver
Carlos D, Spiller F, Souto FO, Trevelin SC, Borges VF, Freitas A de, Alves Filho JCF, Silva JS da, Ryffel B, Cunha F de Q. Histamine H2 receptor signaling in the pathogenesis of sepsis: studies in a murine Diabetes model [Internet]. Journal of Immunology. 2013 ; 191( 3): 1373-1382.[citado 2024 nov. 18 ] Available from: https://doi.org/10.4049/jimmunol.1202907
Artigo de periodico
Joint NOD2/RIPK2 signaling regulates IL-17 axis and contributes to the development of experimental arthritis (2012)
Vieira, Sílvio M.; Cunha, Thiago Mattar; França, Rafael F. O.; Pinto, Larissa G.; Talbot, Jhimmy; Turato, Walter M.; Lemos, Henrique P.; Lima, Jonilson B.; Verri Júnior, Waldiceu A.; Almeida, Sérgio C. L.; Ferreira, Sérgio Henrique; Louzada Junior, Paulo; Zamboni, Dario Simões; Cunha, Fernando de Queiroz
Source: Journal of Immunology. Unidade: FMRP
Subjects: ARTRITE (EXPERIMENTAÇÃO), TRANSDUÇÃO DE SINAL CELULAR, ALBUMINAS
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ABNT
VIEIRA, Sílvio M. et al. Joint NOD2/RIPK2 signaling regulates IL-17 axis and contributes to the development of experimental arthritis. Journal of Immunology, v. 188, n. 10, p. 5116-5122, 2012Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1004190. Acesso em: 18 nov. 2024.
APA
Vieira, S. M., Cunha, T. M., França, R. F. O., Pinto, L. G., Talbot, J., Turato, W. M., et al. (2012). Joint NOD2/RIPK2 signaling regulates IL-17 axis and contributes to the development of experimental arthritis. Journal of Immunology, 188( 10), 5116-5122. doi:10.4049/jimmunol.1004190
NLM
Vieira SM, Cunha TM, França RFO, Pinto LG, Talbot J, Turato WM, Lemos HP, Lima JB, Verri Júnior WA, Almeida SCL, Ferreira SH, Louzada Junior P, Zamboni DS, Cunha F de Q. Joint NOD2/RIPK2 signaling regulates IL-17 axis and contributes to the development of experimental arthritis [Internet]. Journal of Immunology. 2012 ; 188( 10): 5116-5122.[citado 2024 nov. 18 ] Available from: https://doi.org/10.4049/jimmunol.1004190
Vancouver
Vieira SM, Cunha TM, França RFO, Pinto LG, Talbot J, Turato WM, Lemos HP, Lima JB, Verri Júnior WA, Almeida SCL, Ferreira SH, Louzada Junior P, Zamboni DS, Cunha F de Q. Joint NOD2/RIPK2 signaling regulates IL-17 axis and contributes to the development of experimental arthritis [Internet]. Journal of Immunology. 2012 ; 188( 10): 5116-5122.[citado 2024 nov. 18 ] Available from: https://doi.org/10.4049/jimmunol.1004190
Artigo de periodico
Nucleosides from Phlebotomus papatasi salivary gland ameliorate murine collagen-induced arthritis by impairing dendritic cell functions (2011)
Carregaro, Vanessa; Nunes, Anderson de Sá ; Cunha, Thiago Mattar; Grespan, Renata; Oliveira, Carlos J. F.; Lima-Junior, Djalma S.; Costa, Diego L.; Verri Jr, Waldiceu A.; Milanezi, Cristiane M.; Pham, Van My; Brand, David D.; Venezuela, Jesus G.; Silva, João Santana da; Ribeiro, José M. C.; Cunha, Fernando Queiroz
Source: Journal of Immunology. Unidades: ICB, FMRP
Assunto: IMUNOLOGIA
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ABNT
CARREGARO, Vanessa et al. Nucleosides from Phlebotomus papatasi salivary gland ameliorate murine collagen-induced arthritis by impairing dendritic cell functions. Journal of Immunology, v. 187, n. 8, p. 4347-4359, 2011Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1003404. Acesso em: 18 nov. 2024.
APA
Carregaro, V., Nunes, A. de S., Cunha, T. M., Grespan, R., Oliveira, C. J. F., Lima-Junior, D. S., et al. (2011). Nucleosides from Phlebotomus papatasi salivary gland ameliorate murine collagen-induced arthritis by impairing dendritic cell functions. Journal of Immunology, 187( 8), 4347-4359. doi:10.4049/jimmunol.1003404
NLM
Carregaro V, Nunes A de S, Cunha TM, Grespan R, Oliveira CJF, Lima-Junior DS, Costa DL, Verri Jr WA, Milanezi CM, Pham VM, Brand DD, Venezuela JG, Silva JS da, Ribeiro JMC, Cunha FQ. Nucleosides from Phlebotomus papatasi salivary gland ameliorate murine collagen-induced arthritis by impairing dendritic cell functions [Internet]. Journal of Immunology. 2011 ; 187( 8): 4347-4359.[citado 2024 nov. 18 ] Available from: https://doi.org/10.4049/jimmunol.1003404
Vancouver
Carregaro V, Nunes A de S, Cunha TM, Grespan R, Oliveira CJF, Lima-Junior DS, Costa DL, Verri Jr WA, Milanezi CM, Pham VM, Brand DD, Venezuela JG, Silva JS da, Ribeiro JMC, Cunha FQ. Nucleosides from Phlebotomus papatasi salivary gland ameliorate murine collagen-induced arthritis by impairing dendritic cell functions [Internet]. Journal of Immunology. 2011 ; 187( 8): 4347-4359.[citado 2024 nov. 18 ] Available from: https://doi.org/10.4049/jimmunol.1003404
Artigo de periodico
Crucial role of TNF receptors 1 and 2 in the control of polymicrobial sepsis (2009)
Secher, Thomas; Vasseur, Virginie; Poisson, Didier Marc; Mitchell, jane A.; Cunha, Fernando de Queiroz; Alves-Filho, José Carlos; Ryffel, Bernhard
Source: Journal of Immunology. Unidade: FMRP
Subjects: NEUTRÓFILOS, SEPSE (TRATAMENTO)
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ABNT
SECHER, Thomas et al. Crucial role of TNF receptors 1 and 2 in the control of polymicrobial sepsis. Journal of Immunology, v. 182, n. 12, p. 7855-7864, 2009Tradução . . Acesso em: 18 nov. 2024.
APA
Secher, T., Vasseur, V., Poisson, D. M., Mitchell, jane A., Cunha, F. de Q., Alves-Filho, J. C., & Ryffel, B. (2009). Crucial role of TNF receptors 1 and 2 in the control of polymicrobial sepsis. Journal of Immunology, 182( 12), 7855-7864.
NLM
Secher T, Vasseur V, Poisson DM, Mitchell jane A, Cunha F de Q, Alves-Filho JC, Ryffel B. Crucial role of TNF receptors 1 and 2 in the control of polymicrobial sepsis. Journal of Immunology. 2009 ; 182( 12): 7855-7864.[citado 2024 nov. 18 ]
Vancouver
Secher T, Vasseur V, Poisson DM, Mitchell jane A, Cunha F de Q, Alves-Filho JC, Ryffel B. Crucial role of TNF receptors 1 and 2 in the control of polymicrobial sepsis. Journal of Immunology. 2009 ; 182( 12): 7855-7864.[citado 2024 nov. 18 ]
Artigo de periodico
IL-17 receptor signaling is required to control polymicrobial sepsis (2009)
Freitas, Andressa; Alves-Filho, José C.; Victoni, Tatiana; Secher, Thomas; Lemos, Henrique P.; Sônego, Fabiane; Cunha, Fernando de Queiroz; Ryffel, Bernhard
Source: Journal of Immunology. Unidade: FMRP
Subjects: NEUTRÓFILOS, SEPSE
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ABNT
FREITAS, Andressa et al. IL-17 receptor signaling is required to control polymicrobial sepsis. Journal of Immunology, v. 182, n. 12, p. 7846-7854, 2009Tradução . . Disponível em: https://doi.org/10.1186/cc7810. Acesso em: 18 nov. 2024.
APA
Freitas, A., Alves-Filho, J. C., Victoni, T., Secher, T., Lemos, H. P., Sônego, F., et al. (2009). IL-17 receptor signaling is required to control polymicrobial sepsis. Journal of Immunology, 182( 12), 7846-7854. doi:10.1186/cc7810
NLM
Freitas A, Alves-Filho JC, Victoni T, Secher T, Lemos HP, Sônego F, Cunha F de Q, Ryffel B. IL-17 receptor signaling is required to control polymicrobial sepsis [Internet]. Journal of Immunology. 2009 ; 182( 12): 7846-7854.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1186/cc7810
Vancouver
Freitas A, Alves-Filho JC, Victoni T, Secher T, Lemos HP, Sônego F, Cunha F de Q, Ryffel B. IL-17 receptor signaling is required to control polymicrobial sepsis [Internet]. Journal of Immunology. 2009 ; 182( 12): 7846-7854.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1186/cc7810
Artigo de periodico
Peroxisome proliferator-activated receptor-'gama' ligand, '15-deoxy-'delta' POT.12,14'-prostaglandin 'J IND.2', reduces neutrophil migration via a nitric oxide pathway (2008)
Napimoga, Marcelo H.; Vieira, Silvio M.; Dal-Secco, Daniela; Freitas, Andressa; Souto, Fabrício O.; Mestriner, Fabiola Leslie Antunes Cardoso; Alves-Filho, José C.; Grespan, Renata; Kawai, Toshihisa; Ferreira, Sérgio Henrique; Cunha, Fernando de Queiroz
Source: Journal of Immunology. Unidade: FMRP
Subjects: NEUTRÓFILOS, ÓXIDO NÍTRICO
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ABNT
NAPIMOGA, Marcelo H. et al. Peroxisome proliferator-activated receptor-'gama' ligand, '15-deoxy-'delta' POT.12,14'-prostaglandin 'J IND.2', reduces neutrophil migration via a nitric oxide pathway. Journal of Immunology, v. 180, n. 1, p. 609-617, 2008Tradução . . Acesso em: 18 nov. 2024.
APA
Napimoga, M. H., Vieira, S. M., Dal-Secco, D., Freitas, A., Souto, F. O., Mestriner, F. L. A. C., et al. (2008). Peroxisome proliferator-activated receptor-'gama' ligand, '15-deoxy-'delta' POT.12,14'-prostaglandin 'J IND.2', reduces neutrophil migration via a nitric oxide pathway. Journal of Immunology, 180( 1), 609-617.
NLM
Napimoga MH, Vieira SM, Dal-Secco D, Freitas A, Souto FO, Mestriner FLAC, Alves-Filho JC, Grespan R, Kawai T, Ferreira SH, Cunha F de Q. Peroxisome proliferator-activated receptor-'gama' ligand, '15-deoxy-'delta' POT.12,14'-prostaglandin 'J IND.2', reduces neutrophil migration via a nitric oxide pathway. Journal of Immunology. 2008 ; 180( 1): 609-617.[citado 2024 nov. 18 ]
Vancouver
Napimoga MH, Vieira SM, Dal-Secco D, Freitas A, Souto FO, Mestriner FLAC, Alves-Filho JC, Grespan R, Kawai T, Ferreira SH, Cunha F de Q. Peroxisome proliferator-activated receptor-'gama' ligand, '15-deoxy-'delta' POT.12,14'-prostaglandin 'J IND.2', reduces neutrophil migration via a nitric oxide pathway. Journal of Immunology. 2008 ; 180( 1): 609-617.[citado 2024 nov. 18 ]
Artigo de periodico
Hydrogen sulfide augments neutrophil migration through enhancement of adhesion molecule expression and prevention of CXCR2 internalization: role of ATP-Sensitive potassium channels (2008)
Dal-Secco, Daniela; Cunha, Thiago M.; Freitas, Andressa; Alves-Filho, José Carlos; Souto, Fabrício O.; Fukada, Sandra Y.; Grespan, Renata; Alencar, Nylane M. N.; Neto, Alberto F.; Rossi, Marcos Antonio; Ferreira, S. H.; Hothersall, John S.; Cunha, Fernando de Queiroz
Source: Journal of Immunology. Unidade: FMRP
Subjects: NEUTRÓFILOS, IMUNOLOGIA
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ABNT
DAL-SECCO, Daniela et al. Hydrogen sulfide augments neutrophil migration through enhancement of adhesion molecule expression and prevention of CXCR2 internalization: role of ATP-Sensitive potassium channels. Journal of Immunology, v. 181, n. 6, p. 4287-4298, 2008Tradução . . Acesso em: 18 nov. 2024.
APA
Dal-Secco, D., Cunha, T. M., Freitas, A., Alves-Filho, J. C., Souto, F. O., Fukada, S. Y., et al. (2008). Hydrogen sulfide augments neutrophil migration through enhancement of adhesion molecule expression and prevention of CXCR2 internalization: role of ATP-Sensitive potassium channels. Journal of Immunology, 181( 6), 4287-4298.
NLM
Dal-Secco D, Cunha TM, Freitas A, Alves-Filho JC, Souto FO, Fukada SY, Grespan R, Alencar NMN, Neto AF, Rossi MA, Ferreira SH, Hothersall JS, Cunha F de Q. Hydrogen sulfide augments neutrophil migration through enhancement of adhesion molecule expression and prevention of CXCR2 internalization: role of ATP-Sensitive potassium channels. Journal of Immunology. 2008 ; 181( 6): 4287-4298.[citado 2024 nov. 18 ]
Vancouver
Dal-Secco D, Cunha TM, Freitas A, Alves-Filho JC, Souto FO, Fukada SY, Grespan R, Alencar NMN, Neto AF, Rossi MA, Ferreira SH, Hothersall JS, Cunha F de Q. Hydrogen sulfide augments neutrophil migration through enhancement of adhesion molecule expression and prevention of CXCR2 internalization: role of ATP-Sensitive potassium channels. Journal of Immunology. 2008 ; 181( 6): 4287-4298.[citado 2024 nov. 18 ]
Artigo de periodico
Treatment of Paracoccidioides brasiliensis-infected mice with a nitric oxide inhibitor prevents the failure of cell-mediated immune response (1998)
Bocca, Anamelia Lorenzetti; Hayashi, Emília E; Pinheiro, Artur G; Furlanetto, Alessandra B; Campanelli, Ana Paula; Cunha, Fernando de Queiroz; Figueiredo, Florêncio
Source: Journal of Immunology. Unidade: FMRP
Assunto: IMUNOLOGIA
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ABNT
BOCCA, Anamelia Lorenzetti et al. Treatment of Paracoccidioides brasiliensis-infected mice with a nitric oxide inhibitor prevents the failure of cell-mediated immune response. Journal of Immunology, v. 161, n. 6, p. 3056-3063, 1998Tradução . . Acesso em: 18 nov. 2024.
APA
Bocca, A. L., Hayashi, E. E., Pinheiro, A. G., Furlanetto, A. B., Campanelli, A. P., Cunha, F. de Q., & Figueiredo, F. (1998). Treatment of Paracoccidioides brasiliensis-infected mice with a nitric oxide inhibitor prevents the failure of cell-mediated immune response. Journal of Immunology, 161( 6), 3056-3063.
NLM
Bocca AL, Hayashi EE, Pinheiro AG, Furlanetto AB, Campanelli AP, Cunha F de Q, Figueiredo F. Treatment of Paracoccidioides brasiliensis-infected mice with a nitric oxide inhibitor prevents the failure of cell-mediated immune response. Journal of Immunology. 1998 ; 161( 6): 3056-3063.[citado 2024 nov. 18 ]
Vancouver
Bocca AL, Hayashi EE, Pinheiro AG, Furlanetto AB, Campanelli AP, Cunha F de Q, Figueiredo F. Treatment of Paracoccidioides brasiliensis-infected mice with a nitric oxide inhibitor prevents the failure of cell-mediated immune response. Journal of Immunology. 1998 ; 161( 6): 3056-3063.[citado 2024 nov. 18 ]
Artigo de periodico
Treatment of paracoccidioides brasiliensis-infected mice with a nitric oxide inhibitor prevents the failure of cell-mediated immune response (1998)
Bocca, Anamélia Lorenzetti; Hayashi, Emília E; Pinheiro, Artur G; Furlanetto, Alessandra B; Campanelli, Ana Paula; Cunha, Fernando de Queiroz; Figueiredo, Florêncio
Source: Journal of Immunology. Unidade: FMRP
Subjects: IMUNOLOGIA MÉDICA, MICOLOGIA
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ABNT
BOCCA, Anamélia Lorenzetti et al. Treatment of paracoccidioides brasiliensis-infected mice with a nitric oxide inhibitor prevents the failure of cell-mediated immune response. Journal of Immunology, v. 161, p. 3054-3063, 1998Tradução . . Acesso em: 18 nov. 2024.
APA
Bocca, A. L., Hayashi, E. E., Pinheiro, A. G., Furlanetto, A. B., Campanelli, A. P., Cunha, F. de Q., & Figueiredo, F. (1998). Treatment of paracoccidioides brasiliensis-infected mice with a nitric oxide inhibitor prevents the failure of cell-mediated immune response. Journal of Immunology, 161, 3054-3063.
NLM
Bocca AL, Hayashi EE, Pinheiro AG, Furlanetto AB, Campanelli AP, Cunha F de Q, Figueiredo F. Treatment of paracoccidioides brasiliensis-infected mice with a nitric oxide inhibitor prevents the failure of cell-mediated immune response. Journal of Immunology. 1998 ; 161 3054-3063.[citado 2024 nov. 18 ]
Vancouver
Bocca AL, Hayashi EE, Pinheiro AG, Furlanetto AB, Campanelli AP, Cunha F de Q, Figueiredo F. Treatment of paracoccidioides brasiliensis-infected mice with a nitric oxide inhibitor prevents the failure of cell-mediated immune response. Journal of Immunology. 1998 ; 161 3054-3063.[citado 2024 nov. 18 ]
Artigo de periodico
Recombinant migration inhibitory factor induces nitric oxide synthase in murine macrophages (1993)
Cunha, Fernando de Queiroz; Weiser, W Y; David, J R; Moss, D W; Moncada, S; Liew, F Y
Source: Journal of Immunology. Unidade: FMRP
Assunto: IMUNOLOGIA
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ABNT
CUNHA, Fernando de Queiroz et al. Recombinant migration inhibitory factor induces nitric oxide synthase in murine macrophages. Journal of Immunology, v. 150, p. 1908-12, 1993Tradução . . Acesso em: 18 nov. 2024.
APA
Cunha, F. de Q., Weiser, W. Y., David, J. R., Moss, D. W., Moncada, S., & Liew, F. Y. (1993). Recombinant migration inhibitory factor induces nitric oxide synthase in murine macrophages. Journal of Immunology, 150, 1908-12.
NLM
Cunha F de Q, Weiser WY, David JR, Moss DW, Moncada S, Liew FY. Recombinant migration inhibitory factor induces nitric oxide synthase in murine macrophages. Journal of Immunology. 1993 ;150 1908-12.[citado 2024 nov. 18 ]
Vancouver
Cunha F de Q, Weiser WY, David JR, Moss DW, Moncada S, Liew FY. Recombinant migration inhibitory factor induces nitric oxide synthase in murine macrophages. Journal of Immunology. 1993 ;150 1908-12.[citado 2024 nov. 18 ]
Artigo de periodico
Il-10 inhibits the synthesis of migration inhibitory factor and migration inhibitory factor-mediated macrophage activation (1993)
Wu, J; Cunha, Fernando de Queiroz; Liew, F Y; Weiser, W Y
Source: Journal of Immunology. Unidade: FMRP
Assunto: LIMNOLOGIA
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ABNT
WU, J et al. Il-10 inhibits the synthesis of migration inhibitory factor and migration inhibitory factor-mediated macrophage activation. Journal of Immunology, v. 151, p. 4325-32, 1993Tradução . . Acesso em: 18 nov. 2024.
APA
Wu, J., Cunha, F. de Q., Liew, F. Y., & Weiser, W. Y. (1993). Il-10 inhibits the synthesis of migration inhibitory factor and migration inhibitory factor-mediated macrophage activation. Journal of Immunology, 151, 4325-32.
NLM
Wu J, Cunha F de Q, Liew FY, Weiser WY. Il-10 inhibits the synthesis of migration inhibitory factor and migration inhibitory factor-mediated macrophage activation. Journal of Immunology. 1993 ;151 4325-32.[citado 2024 nov. 18 ]
Vancouver
Wu J, Cunha F de Q, Liew FY, Weiser WY. Il-10 inhibits the synthesis of migration inhibitory factor and migration inhibitory factor-mediated macrophage activation. Journal of Immunology. 1993 ;151 4325-32.[citado 2024 nov. 18 ]

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