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Artigo de periodico
Real-time redox monitoring of a nitrosyl ruthenium complex acting as NO-donor agent in a single A549 cancer cell with multiplex Fourier-transform infrared microscopy (2020)
Rodrigues, Fernando Postalli ; Macedo, Lucyano Jefferson Alves de ; Máximo, Leandro Nériton Cândido; Sales, Fernanda Cristina Pena Ferreira ; Silva, Roberto Santana da ; Crespilho, Frank Nelson
Source: Nitric Oxide: Biology and Chemistry. Unidades: FCFRP, IQSC
Subjects: BIOQUÍMICA, NEOPLASIAS, PULMÃO, ÓXIDO NÍTRICO
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RODRIGUES, Fernando Postalli et al. Real-time redox monitoring of a nitrosyl ruthenium complex acting as NO-donor agent in a single A549 cancer cell with multiplex Fourier-transform infrared microscopy. Nitric Oxide: Biology and Chemistry, v. 96, p. 29-34, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.niox.2020.01.005. Acesso em: 18 nov. 2024.
APA
Rodrigues, F. P., Macedo, L. J. A. de, Máximo, L. N. C., Sales, F. C. P. F., Silva, R. S. da, & Crespilho, F. N. (2020). Real-time redox monitoring of a nitrosyl ruthenium complex acting as NO-donor agent in a single A549 cancer cell with multiplex Fourier-transform infrared microscopy. Nitric Oxide: Biology and Chemistry, 96, 29-34. doi:10.1016/j.niox.2020.01.005
NLM
Rodrigues FP, Macedo LJA de, Máximo LNC, Sales FCPF, Silva RS da, Crespilho FN. Real-time redox monitoring of a nitrosyl ruthenium complex acting as NO-donor agent in a single A549 cancer cell with multiplex Fourier-transform infrared microscopy [Internet]. Nitric Oxide: Biology and Chemistry. 2020 ; 96 29-34.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.niox.2020.01.005
Vancouver
Rodrigues FP, Macedo LJA de, Máximo LNC, Sales FCPF, Silva RS da, Crespilho FN. Real-time redox monitoring of a nitrosyl ruthenium complex acting as NO-donor agent in a single A549 cancer cell with multiplex Fourier-transform infrared microscopy [Internet]. Nitric Oxide: Biology and Chemistry. 2020 ; 96 29-34.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.niox.2020.01.005
Artigo de periodico
Photodynamic treatment of melanoma cells using aza-dipyrromethenes as photosensitizers (2020)
Castro, Kelly Aparecida Dias de Freitas; Costa, Letícia D.; Guieu, Samuel; Biazzotto, Juliana Cristina ; Neves, Maria Graça P. M. S. da; Faustino, M. Amparo F.; Silva, Roberto Santana da; Tomé, Augusto C.
Source: Photochemical & Photobiological Sciences. Unidade: FCFRP
Subjects: TERAPIA FOTODINÂMICA, NEOPLASIAS, MELANOMA, COMPOSTOS ORGÂNICOS
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CASTRO, Kelly Aparecida Dias de Freitas et al. Photodynamic treatment of melanoma cells using aza-dipyrromethenes as photosensitizers. Photochemical & Photobiological Sciences, v. 19, n. 7, p. 885-891, 2020Tradução . . Disponível em: https://doi.org/10.1039/d0pp00114g. Acesso em: 18 nov. 2024.
APA
Castro, K. A. D. de F., Costa, L. D., Guieu, S., Biazzotto, J. C., Neves, M. G. P. M. S. da, Faustino, M. A. F., et al. (2020). Photodynamic treatment of melanoma cells using aza-dipyrromethenes as photosensitizers. Photochemical & Photobiological Sciences, 19( 7), 885-891. doi:10.1039/d0pp00114g
NLM
Castro KAD de F, Costa LD, Guieu S, Biazzotto JC, Neves MGPMS da, Faustino MAF, Silva RS da, Tomé AC. Photodynamic treatment of melanoma cells using aza-dipyrromethenes as photosensitizers [Internet]. Photochemical & Photobiological Sciences. 2020 ; 19( 7): 885-891.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1039/d0pp00114g
Vancouver
Castro KAD de F, Costa LD, Guieu S, Biazzotto JC, Neves MGPMS da, Faustino MAF, Silva RS da, Tomé AC. Photodynamic treatment of melanoma cells using aza-dipyrromethenes as photosensitizers [Internet]. Photochemical & Photobiological Sciences. 2020 ; 19( 7): 885-891.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1039/d0pp00114g
Artigo de periodico
Acetylation of cedrelone increases its cytotoxic activity and reverts the malignant phenotype of breast cancer cells in 3D culture (2020)
Becceneri, Amanda Blanque; Fuzer, Angelina Maria; Popolin, Cecília Patrícia; Cazal, Cristiane de Melo; Domingues, Vanessa de Cássia; Fernandes, João Batista; Vieira, Paulo Cezar ; Cominetti, Marcia Regina
Source: Chemico-Biological Interactions. Unidade: FCFRP
Subjects: NEOPLASIAS MAMÁRIAS, NEOPLASIAS, CULTURA DE CÉLULAS, PRODUTOS NATURAIS, FENÓTIPOS, FÁRMACOS, RUTALES
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BECCENERI, Amanda Blanque et al. Acetylation of cedrelone increases its cytotoxic activity and reverts the malignant phenotype of breast cancer cells in 3D culture. Chemico-Biological Interactions, v. 316, p. 1-10, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.cbi.2019.108920. Acesso em: 18 nov. 2024.
APA
Becceneri, A. B., Fuzer, A. M., Popolin, C. P., Cazal, C. de M., Domingues, V. de C., Fernandes, J. B., et al. (2020). Acetylation of cedrelone increases its cytotoxic activity and reverts the malignant phenotype of breast cancer cells in 3D culture. Chemico-Biological Interactions, 316, 1-10. doi:10.1016/j.cbi.2019.108920
NLM
Becceneri AB, Fuzer AM, Popolin CP, Cazal C de M, Domingues V de C, Fernandes JB, Vieira PC, Cominetti MR. Acetylation of cedrelone increases its cytotoxic activity and reverts the malignant phenotype of breast cancer cells in 3D culture [Internet]. Chemico-Biological Interactions. 2020 ; 316 1-10.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.cbi.2019.108920
Vancouver
Becceneri AB, Fuzer AM, Popolin CP, Cazal C de M, Domingues V de C, Fernandes JB, Vieira PC, Cominetti MR. Acetylation of cedrelone increases its cytotoxic activity and reverts the malignant phenotype of breast cancer cells in 3D culture [Internet]. Chemico-Biological Interactions. 2020 ; 316 1-10.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.cbi.2019.108920
Trabalho de evento-resumo periodico
Beyond hemostasis: a potassium channel blocker snake venom serine protease with potential antitumor activity (2020)
Boldrini-França, Johara; Pinheiro-Júnior, Ernesto Lopes ; Peigneur, Steve; Costa, Tássia Rafaella; Borges, Rafael Junqueira; Pucca, Manuela Berto; Cerni, Felipe Augusto; Carone, Sante Emmanuel Imai; Mattos Fontes, Marcos Roberto de; Sampaio, Suely Vilela ; Arantes, Eliane Candiani ; Tytgat, Jan
Source: Toxicon. Conference titles: World Congress of the International Society on Toxinology. Unidade: FCFRP
Subjects: HOMEOSTASE, VENENOS DE ORIGEM ANIMAL, CANAIS DE POTÁSSIO, SERPENTES, NEOPLASIAS
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BOLDRINI-FRANÇA, Johara et al. Beyond hemostasis: a potassium channel blocker snake venom serine protease with potential antitumor activity. Toxicon. Oxford: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. Disponível em: https://doi.org/10.1016/j.toxicon.2019.10.040. Acesso em: 18 nov. 2024. , 2020
APA
Boldrini-França, J., Pinheiro-Júnior, E. L., Peigneur, S., Costa, T. R., Borges, R. J., Pucca, M. B., et al. (2020). Beyond hemostasis: a potassium channel blocker snake venom serine protease with potential antitumor activity. Toxicon. Oxford: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. doi:10.1016/j.toxicon.2019.10.040
NLM
Boldrini-França J, Pinheiro-Júnior EL, Peigneur S, Costa TR, Borges RJ, Pucca MB, Cerni FA, Carone SEI, Mattos Fontes MR de, Sampaio SV, Arantes EC, Tytgat J. Beyond hemostasis: a potassium channel blocker snake venom serine protease with potential antitumor activity [Internet]. Toxicon. 2020 ; 177 S8-S9.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.toxicon.2019.10.040
Vancouver
Boldrini-França J, Pinheiro-Júnior EL, Peigneur S, Costa TR, Borges RJ, Pucca MB, Cerni FA, Carone SEI, Mattos Fontes MR de, Sampaio SV, Arantes EC, Tytgat J. Beyond hemostasis: a potassium channel blocker snake venom serine protease with potential antitumor activity [Internet]. Toxicon. 2020 ; 177 S8-S9.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.toxicon.2019.10.040
Artigo de periodico
Engineering of galectin-3 for glycan-binding optical imaging (2020)
Leo, Thais Canassa de ; Santos, Sofia Nascimento dos; Andrade, Camillo Del Cistia; Ricci, Eduardo; Turato, Walter Miguel; Lopes, Norberto Peporine ; Oliveira, Ralph Santos; Bernardes, Emerson Soares; Dias-Baruffi, Marcelo
Source: Biochemical and Biophysical Research Communication. Unidades: FCFRP, FCF, IPEN
Subjects: BIOLUMINESCÊNCIA, PROTEÍNAS RECOMBINANTES, BIOMARCADORES, NEOPLASIAS
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LEO, Thais Canassa de et al. Engineering of galectin-3 for glycan-binding optical imaging. Biochemical and Biophysical Research Communication, v. 521, n. 3, p. 674-680, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.bbrc.2019.10.161. Acesso em: 18 nov. 2024.
APA
Leo, T. C. de, Santos, S. N. dos, Andrade, C. D. C., Ricci, E., Turato, W. M., Lopes, N. P., et al. (2020). Engineering of galectin-3 for glycan-binding optical imaging. Biochemical and Biophysical Research Communication, 521( 3), 674-680. doi:10.1016/j.bbrc.2019.10.161
NLM
Leo TC de, Santos SN dos, Andrade CDC, Ricci E, Turato WM, Lopes NP, Oliveira RS, Bernardes ES, Dias-Baruffi M. Engineering of galectin-3 for glycan-binding optical imaging [Internet]. Biochemical and Biophysical Research Communication. 2020 ; 521( 3): 674-680.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.bbrc.2019.10.161
Vancouver
Leo TC de, Santos SN dos, Andrade CDC, Ricci E, Turato WM, Lopes NP, Oliveira RS, Bernardes ES, Dias-Baruffi M. Engineering of galectin-3 for glycan-binding optical imaging [Internet]. Biochemical and Biophysical Research Communication. 2020 ; 521( 3): 674-680.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.bbrc.2019.10.161
Artigo de periodico
Rapanone, a naturally occurring benzoquinone, inhibits mitochondrial respiration and induces HepG2 cell death (2020)
Andreu, Gilberto L. Pardo; Reis, Felipe Henrique Zuccolotto dos ; González-Durruthy, Michael; Hernández, René Delgado; D'Vries, Richard F.; Vanden Berghe, Wim; Alberici, Luciane Carla
Source: Toxicology in Vitro. Unidades: FCFRP, FMRP
Subjects: ANTINEOPLÁSICOS, TOXICOLOGIA, NEOPLASIAS, MITOCÔNDRIAS, CARCINOMA HEPATOCELULAR
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ANDREU, Gilberto L. Pardo et al. Rapanone, a naturally occurring benzoquinone, inhibits mitochondrial respiration and induces HepG2 cell death. Toxicology in Vitro, v. 63, p. 1-10, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.tiv.2019.104737. Acesso em: 18 nov. 2024.
APA
Andreu, G. L. P., Reis, F. H. Z. dos, González-Durruthy, M., Hernández, R. D., D'Vries, R. F., Vanden Berghe, W., & Alberici, L. C. (2020). Rapanone, a naturally occurring benzoquinone, inhibits mitochondrial respiration and induces HepG2 cell death. Toxicology in Vitro, 63, 1-10. doi:10.1016/j.tiv.2019.104737
NLM
Andreu GLP, Reis FHZ dos, González-Durruthy M, Hernández RD, D'Vries RF, Vanden Berghe W, Alberici LC. Rapanone, a naturally occurring benzoquinone, inhibits mitochondrial respiration and induces HepG2 cell death [Internet]. Toxicology in Vitro. 2020 ; 63 1-10.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.tiv.2019.104737
Vancouver
Andreu GLP, Reis FHZ dos, González-Durruthy M, Hernández RD, D'Vries RF, Vanden Berghe W, Alberici LC. Rapanone, a naturally occurring benzoquinone, inhibits mitochondrial respiration and induces HepG2 cell death [Internet]. Toxicology in Vitro. 2020 ; 63 1-10.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.tiv.2019.104737
Artigo de periodico
Effects of DNA methyltransferase inhibition on pattern separation performance in mice (2019)
Argyrousi, Elentina K.; de Nijs, Laurence; Lagatta, Davi Campos ; Schlütter, Anna; Weidner, Magdalena T.; Zöller, Johanna; van Goethem, Nick P.; Wegener, Sâmia Regiane Lourenço Joca ; van den Hove, Daniel L. A.; Prickaerts, Jos
Source: Neurobiology of Learning and Memory. Unidades: FCFRP, FMRP
Subjects: METILAÇÃO DE DNA, EPIGÊNESE GENÉTICA, NEOPLASIAS
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ARGYROUSI, Elentina K. et al. Effects of DNA methyltransferase inhibition on pattern separation performance in mice. Neurobiology of Learning and Memory, v. 159, p. 6-15, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.nlm.2019.02.003. Acesso em: 18 nov. 2024.
APA
Argyrousi, E. K., de Nijs, L., Lagatta, D. C., Schlütter, A., Weidner, M. T., Zöller, J., et al. (2019). Effects of DNA methyltransferase inhibition on pattern separation performance in mice. Neurobiology of Learning and Memory, 159, 6-15. doi:10.1016/j.nlm.2019.02.003
NLM
Argyrousi EK, de Nijs L, Lagatta DC, Schlütter A, Weidner MT, Zöller J, van Goethem NP, Wegener SRLJ, van den Hove DLA, Prickaerts J. Effects of DNA methyltransferase inhibition on pattern separation performance in mice [Internet]. Neurobiology of Learning and Memory. 2019 ; 159 6-15.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.nlm.2019.02.003
Vancouver
Argyrousi EK, de Nijs L, Lagatta DC, Schlütter A, Weidner MT, Zöller J, van Goethem NP, Wegener SRLJ, van den Hove DLA, Prickaerts J. Effects of DNA methyltransferase inhibition on pattern separation performance in mice [Internet]. Neurobiology of Learning and Memory. 2019 ; 159 6-15.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.nlm.2019.02.003
Artigo de periodico
The cytotoxic effects of VE-3N, a novel 1,4-dihydropyridine derivative, involve the mitochondrial bioenergetic disruption via uncoupling mechanisms (2017)
Prida, Javier Marín; Andreu, Gilberto L. Pardo; Rossignoli, Camila Pederiva; Durruthy, Michael González; Rodríguez, Estael Ochoa; Reyes, Yamila Verdecia; Acosta, Roberto Fernández; Uyemura, Sérgio Akira; Alberici, Luciane Carla
Source: Toxicology in Vitro. Unidade: FCFRP
Subjects: NEOPLASIAS, CITOTOXICIDADE IMUNOLÓGICA, MITOCÔNDRIAS, FÁRMACOS
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PRIDA, Javier Marín et al. The cytotoxic effects of VE-3N, a novel 1,4-dihydropyridine derivative, involve the mitochondrial bioenergetic disruption via uncoupling mechanisms. Toxicology in Vitro, v. 42, p. 21-30, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.tiv.2017.03.011. Acesso em: 18 nov. 2024.
APA
Prida, J. M., Andreu, G. L. P., Rossignoli, C. P., Durruthy, M. G., Rodríguez, E. O., Reyes, Y. V., et al. (2017). The cytotoxic effects of VE-3N, a novel 1,4-dihydropyridine derivative, involve the mitochondrial bioenergetic disruption via uncoupling mechanisms. Toxicology in Vitro, 42, 21-30. doi:10.1016/j.tiv.2017.03.011
NLM
Prida JM, Andreu GLP, Rossignoli CP, Durruthy MG, Rodríguez EO, Reyes YV, Acosta RF, Uyemura SA, Alberici LC. The cytotoxic effects of VE-3N, a novel 1,4-dihydropyridine derivative, involve the mitochondrial bioenergetic disruption via uncoupling mechanisms [Internet]. Toxicology in Vitro. 2017 ; 42 21-30.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.tiv.2017.03.011
Vancouver
Prida JM, Andreu GLP, Rossignoli CP, Durruthy MG, Rodríguez EO, Reyes YV, Acosta RF, Uyemura SA, Alberici LC. The cytotoxic effects of VE-3N, a novel 1,4-dihydropyridine derivative, involve the mitochondrial bioenergetic disruption via uncoupling mechanisms [Internet]. Toxicology in Vitro. 2017 ; 42 21-30.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.tiv.2017.03.011
Artigo de periodico
SET oncoprotein accumulation regulates transcription through DNA demethylation and histone hypoacetylation (2017)
Almeida, Luciana O.; Cavalcanti Neto, Marinaldo P.; Sousa, Lucas Oliveira; Tannous, Maryna A.; Curti, Carlos; Leopoldino, Andréia Machado
Source: Oncotarget. Unidade: FCFRP
Subjects: EPIGÊNESE GENÉTICA, NEOPLASIAS, METILAÇÃO DE DNA, PROTEÍNAS, CROMATINA
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ALMEIDA, Luciana O. et al. SET oncoprotein accumulation regulates transcription through DNA demethylation and histone hypoacetylation. Oncotarget, v. 8, n. 16, p. 26802-26818, 2017Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.15818. Acesso em: 18 nov. 2024.
APA
Almeida, L. O., Cavalcanti Neto, M. P., Sousa, L. O., Tannous, M. A., Curti, C., & Leopoldino, A. M. (2017). SET oncoprotein accumulation regulates transcription through DNA demethylation and histone hypoacetylation. Oncotarget, 8( 16), 26802-26818. doi:10.18632/oncotarget.15818
NLM
Almeida LO, Cavalcanti Neto MP, Sousa LO, Tannous MA, Curti C, Leopoldino AM. SET oncoprotein accumulation regulates transcription through DNA demethylation and histone hypoacetylation [Internet]. Oncotarget. 2017 ; 8( 16): 26802-26818.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.15818
Vancouver
Almeida LO, Cavalcanti Neto MP, Sousa LO, Tannous MA, Curti C, Leopoldino AM. SET oncoprotein accumulation regulates transcription through DNA demethylation and histone hypoacetylation [Internet]. Oncotarget. 2017 ; 8( 16): 26802-26818.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.15818
Artigo de periodico
Incorporation of a ruthenium nitrosyl complex into liposomes, the nitric oxide released from these liposomes and HepG2 cell death mechanism (2016)
Rodrigues, Fernando P.; Carneiro, Zumira, A.; Mascharak, Pradip; Curti, Carlos; Silva, Roberto Santana da
Source: Coordination Chemistry Reviews. Unidade: FCFRP
Subjects: RUTÊNIO, FÁRMACOS, CARCINOMA HEPATOCELULAR, NEOPLASIAS, CITOTOXINAS
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RODRIGUES, Fernando P. et al. Incorporation of a ruthenium nitrosyl complex into liposomes, the nitric oxide released from these liposomes and HepG2 cell death mechanism. Coordination Chemistry Reviews, v. 306, p. 701-707, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.ccr.2015.03.028. Acesso em: 18 nov. 2024.
APA
Rodrigues, F. P., Carneiro, Z., Mascharak, P., Curti, C., & Silva, R. S. da. (2016). Incorporation of a ruthenium nitrosyl complex into liposomes, the nitric oxide released from these liposomes and HepG2 cell death mechanism. Coordination Chemistry Reviews, 306, 701-707. doi:10.1016/j.ccr.2015.03.028
NLM
Rodrigues FP, Carneiro Z, Mascharak P, Curti C, Silva RS da. Incorporation of a ruthenium nitrosyl complex into liposomes, the nitric oxide released from these liposomes and HepG2 cell death mechanism [Internet]. Coordination Chemistry Reviews. 2016 ; 306 701-707.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.ccr.2015.03.028
Vancouver
Rodrigues FP, Carneiro Z, Mascharak P, Curti C, Silva RS da. Incorporation of a ruthenium nitrosyl complex into liposomes, the nitric oxide released from these liposomes and HepG2 cell death mechanism [Internet]. Coordination Chemistry Reviews. 2016 ; 306 701-707.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.ccr.2015.03.028
Artigo de periodico
CR-LAAO antileukemic effect against Bcr-Abl+ cells is mediated by apoptosis and hydrogen peroxide (2016)
Burin, Sandra Mara; Ghisla, Sandro; Ouchida, Amanda Tomie; Aissa, Alexandre Ferro; Coelho, Maria Gabriela Berzoti; Costa, Tássia Rafaella; Marsola, Ana Paula Zambuzi Cardoso; Simões, Belinda Pinto; Antunes, Lusânia Maria Greggi; Curti, Carlos; Sampaio, Suely Vilela; Castro, Fabíola Attié de
Source: International Journal of Biological Macromolecules. Unidades: FMRP, FCFRP
Subjects: APOPTOSE, LEUCEMIA MIELOIDE, NEOPLASIAS, VENENOS DE ORIGEM ANIMAL, COBRAS, VIPERIDAE, CITOTOXICIDADE IMUNOLÓGICA
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BURIN, Sandra Mara et al. CR-LAAO antileukemic effect against Bcr-Abl+ cells is mediated by apoptosis and hydrogen peroxide. International Journal of Biological Macromolecules, v. 86, p. 309-320, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.ijbiomac.2016.01.069. Acesso em: 18 nov. 2024.
APA
Burin, S. M., Ghisla, S., Ouchida, A. T., Aissa, A. F., Coelho, M. G. B., Costa, T. R., et al. (2016). CR-LAAO antileukemic effect against Bcr-Abl+ cells is mediated by apoptosis and hydrogen peroxide. International Journal of Biological Macromolecules, 86, 309-320. doi:10.1016/j.ijbiomac.2016.01.069
NLM
Burin SM, Ghisla S, Ouchida AT, Aissa AF, Coelho MGB, Costa TR, Marsola APZC, Simões BP, Antunes LMG, Curti C, Sampaio SV, Castro FA de. CR-LAAO antileukemic effect against Bcr-Abl+ cells is mediated by apoptosis and hydrogen peroxide [Internet]. International Journal of Biological Macromolecules. 2016 ; 86 309-320.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.ijbiomac.2016.01.069
Vancouver
Burin SM, Ghisla S, Ouchida AT, Aissa AF, Coelho MGB, Costa TR, Marsola APZC, Simões BP, Antunes LMG, Curti C, Sampaio SV, Castro FA de. CR-LAAO antileukemic effect against Bcr-Abl+ cells is mediated by apoptosis and hydrogen peroxide [Internet]. International Journal of Biological Macromolecules. 2016 ; 86 309-320.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.ijbiomac.2016.01.069
Artigo de periodico
Metabolic profile and safety of piperlongumine (2016)
Moreira, Fernanda de Lima; Habenschus, Maísa D.; Barth, Thiago; Marques, Lucas M. M.; Pilon, Alan Cesar; Bolzani, Vanderlan da Silva; Vessecchi, Ricardo; Lopes, Norberto Peporine; Oliveira, Anderson Rodrigo Moraes de
Source: Scientific Reports. Unidades: FCFRP, FFCLRP
Subjects: METABOLISMO, PRODUTOS NATURAIS, PLANTAS MEDICINAIS, PIMENTA, NEOPLASIAS
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ABNT
MOREIRA, Fernanda de Lima et al. Metabolic profile and safety of piperlongumine. Scientific Reports, v. 6, 2016Tradução . . Disponível em: https://doi.org/10.1038/srep33646. Acesso em: 18 nov. 2024.
APA
Moreira, F. de L., Habenschus, M. D., Barth, T., Marques, L. M. M., Pilon, A. C., Bolzani, V. da S., et al. (2016). Metabolic profile and safety of piperlongumine. Scientific Reports, 6. doi:10.1038/srep33646
NLM
Moreira F de L, Habenschus MD, Barth T, Marques LMM, Pilon AC, Bolzani V da S, Vessecchi R, Lopes NP, Oliveira ARM de. Metabolic profile and safety of piperlongumine [Internet]. Scientific Reports. 2016 ; 6[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/srep33646
Vancouver
Moreira F de L, Habenschus MD, Barth T, Marques LMM, Pilon AC, Bolzani V da S, Vessecchi R, Lopes NP, Oliveira ARM de. Metabolic profile and safety of piperlongumine [Internet]. Scientific Reports. 2016 ; 6[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/srep33646
Trabalho de evento-resumo
Evaluation of anticancer and cytotoxic activities of toxins from Rhinella schneideri toad poison on HepG2 cells and primary hepatocytes (2015)
Fornari-Baldo, Elisa Correa; Baldo, Mateus Amaral; Pereira, Lílian Cristina; Reis, Felippe Henrique Zuccolotto dos; Alberici, Luciane Carla; Dorta, Daniel Junqueira; Arantes, Eliane Candiani
Source: Abstracts. Conference titles: Congress of the Brazilian Society of Toxinology. Unidades: FCFRP, FFCLRP
Subjects: BUFONIDAE, TOXINAS, CARCINOMA, NEOPLASIAS, TERAPÊUTICA
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ABNT
FORNARI-BALDO, Elisa Correa et al. Evaluation of anticancer and cytotoxic activities of toxins from Rhinella schneideri toad poison on HepG2 cells and primary hepatocytes. 2015, Anais.. Campos do Jordão: SBTx, 2015. . Acesso em: 18 nov. 2024.
APA
Fornari-Baldo, E. C., Baldo, M. A., Pereira, L. C., Reis, F. H. Z. dos, Alberici, L. C., Dorta, D. J., & Arantes, E. C. (2015). Evaluation of anticancer and cytotoxic activities of toxins from Rhinella schneideri toad poison on HepG2 cells and primary hepatocytes. In Abstracts. Campos do Jordão: SBTx.
NLM
Fornari-Baldo EC, Baldo MA, Pereira LC, Reis FHZ dos, Alberici LC, Dorta DJ, Arantes EC. Evaluation of anticancer and cytotoxic activities of toxins from Rhinella schneideri toad poison on HepG2 cells and primary hepatocytes. Abstracts. 2015 ;[citado 2024 nov. 18 ]
Vancouver
Fornari-Baldo EC, Baldo MA, Pereira LC, Reis FHZ dos, Alberici LC, Dorta DJ, Arantes EC. Evaluation of anticancer and cytotoxic activities of toxins from Rhinella schneideri toad poison on HepG2 cells and primary hepatocytes. Abstracts. 2015 ;[citado 2024 nov. 18 ]
Artigo de periodico
SET overexpression in HEK293 cells regulates mitochondrial uncoupling proteins levels within a mitochondrial fission/reduced autophagic flux scenario (2015)
Almeida, Luciana O; Goto, Renata N; Cavalcanti Neto, Marinaldo P.; Sousa, Lucas Oliveira; Curti, Carlos; Leopoldino, Andréia Machado
Source: Biochemical and Biophysical Research Communications. Unidade: FCFRP
Subjects: MITOCÔNDRIAS, PROTEÍNAS, NEOPLASIAS, DOENÇA DE ALZHEIMER
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ALMEIDA, Luciana O et al. SET overexpression in HEK293 cells regulates mitochondrial uncoupling proteins levels within a mitochondrial fission/reduced autophagic flux scenario. Biochemical and Biophysical Research Communications, v. 458, n. 2, p. 300-306, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.bbrc.2015.01.104. Acesso em: 18 nov. 2024.
APA
Almeida, L. O., Goto, R. N., Cavalcanti Neto, M. P., Sousa, L. O., Curti, C., & Leopoldino, A. M. (2015). SET overexpression in HEK293 cells regulates mitochondrial uncoupling proteins levels within a mitochondrial fission/reduced autophagic flux scenario. Biochemical and Biophysical Research Communications, 458( 2), 300-306. doi:10.1016/j.bbrc.2015.01.104
NLM
Almeida LO, Goto RN, Cavalcanti Neto MP, Sousa LO, Curti C, Leopoldino AM. SET overexpression in HEK293 cells regulates mitochondrial uncoupling proteins levels within a mitochondrial fission/reduced autophagic flux scenario [Internet]. Biochemical and Biophysical Research Communications. 2015 ; 458( 2): 300-306.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.bbrc.2015.01.104
Vancouver
Almeida LO, Goto RN, Cavalcanti Neto MP, Sousa LO, Curti C, Leopoldino AM. SET overexpression in HEK293 cells regulates mitochondrial uncoupling proteins levels within a mitochondrial fission/reduced autophagic flux scenario [Internet]. Biochemical and Biophysical Research Communications. 2015 ; 458( 2): 300-306.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.bbrc.2015.01.104
Trabalho de evento-resumo
Epigenetic regulation by SET protein in HNSCC: histone acetylation versus DNA methylation (2013)
Almeida, Luciana; Tannous, Maryna; Matsumoto, Camila; Sobral, Lais; Curti, Carlos; Leopoldino, Andréia Machado
Source: Proceedings. Conference titles: Annual Meeting of American Association for Cancer Research. Unidade: FCFRP
Subjects: NEOPLASIAS, EXPRESSÃO GÊNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ALMEIDA, Luciana et al. Epigenetic regulation by SET protein in HNSCC: histone acetylation versus DNA methylation. 2013, Anais.. Washington: AACR, 2013. . Acesso em: 18 nov. 2024.
APA
Almeida, L., Tannous, M., Matsumoto, C., Sobral, L., Curti, C., & Leopoldino, A. M. (2013). Epigenetic regulation by SET protein in HNSCC: histone acetylation versus DNA methylation. In Proceedings. Washington: AACR.
NLM
Almeida L, Tannous M, Matsumoto C, Sobral L, Curti C, Leopoldino AM. Epigenetic regulation by SET protein in HNSCC: histone acetylation versus DNA methylation. Proceedings. 2013 ;[citado 2024 nov. 18 ]
Vancouver
Almeida L, Tannous M, Matsumoto C, Sobral L, Curti C, Leopoldino AM. Epigenetic regulation by SET protein in HNSCC: histone acetylation versus DNA methylation. Proceedings. 2013 ;[citado 2024 nov. 18 ]
Artigo de periodico
SET overexpression decreases cell detoxification efficiency: ALDH2 and GSTP1 are dowregulated, DDR is impaired and DNA damage accumulates (2012)
Almeida, Luciana O.; Goto, Renata N.; Pestana, Cézar R.; Uyemura, Sérgio Akira; Gutkind, Silvio; Curti, Carlos; Leopoldino, Andréia Machado
Source: FEBS Journal. Unidade: FCFRP
Subjects: ENZIMAS (GENÉTICA), DANO AO DNA, FATORES DE TRANSCRIÇÃO, MARCADOR MOLECULAR, NEOPLASIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ALMEIDA, Luciana O. et al. SET overexpression decreases cell detoxification efficiency: ALDH2 and GSTP1 are dowregulated, DDR is impaired and DNA damage accumulates. FEBS Journal, v. 279, n. 24, p. 4615-4628, 2012Tradução . . Disponível em: https://doi.org/10.1111/febs.12047. Acesso em: 18 nov. 2024.
APA
Almeida, L. O., Goto, R. N., Pestana, C. R., Uyemura, S. A., Gutkind, S., Curti, C., & Leopoldino, A. M. (2012). SET overexpression decreases cell detoxification efficiency: ALDH2 and GSTP1 are dowregulated, DDR is impaired and DNA damage accumulates. FEBS Journal, 279( 24), 4615-4628. doi:10.1111/febs.12047
NLM
Almeida LO, Goto RN, Pestana CR, Uyemura SA, Gutkind S, Curti C, Leopoldino AM. SET overexpression decreases cell detoxification efficiency: ALDH2 and GSTP1 are dowregulated, DDR is impaired and DNA damage accumulates [Internet]. FEBS Journal. 2012 ; 279( 24): 4615-4628.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1111/febs.12047
Vancouver
Almeida LO, Goto RN, Pestana CR, Uyemura SA, Gutkind S, Curti C, Leopoldino AM. SET overexpression decreases cell detoxification efficiency: ALDH2 and GSTP1 are dowregulated, DDR is impaired and DNA damage accumulates [Internet]. FEBS Journal. 2012 ; 279( 24): 4615-4628.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1111/febs.12047
Trabalho de evento-resumo
ATP production in HnRNP K-overexpressing HEK293 cells (2012)
Garcia, C. B.; Reis, F. H. Z.; Alberici, L. C.; Curti, Carlos; Leopoldino, Andréia Machado
Conference titles: Reunião Anual da Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq). Unidade: FCFRP
Subjects: PROTEÍNAS, BIOQUÍMICA CELULAR, NEOPLASIAS, MITOCÔNDRIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GARCIA, C. B. et al. ATP production in HnRNP K-overexpressing HEK293 cells. 2012, Anais.. Foz do Iguaçu: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo, 2012. . Acesso em: 18 nov. 2024.
APA
Garcia, C. B., Reis, F. H. Z., Alberici, L. C., Curti, C., & Leopoldino, A. M. (2012). ATP production in HnRNP K-overexpressing HEK293 cells. In . Foz do Iguaçu: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo.
NLM
Garcia CB, Reis FHZ, Alberici LC, Curti C, Leopoldino AM. ATP production in HnRNP K-overexpressing HEK293 cells. 2012 ;[citado 2024 nov. 18 ]
Vancouver
Garcia CB, Reis FHZ, Alberici LC, Curti C, Leopoldino AM. ATP production in HnRNP K-overexpressing HEK293 cells. 2012 ;[citado 2024 nov. 18 ]
Trabalho de evento-resumo
Evaluation of the cytotoxic activity of Rhinella schneideri toad poison on tumor cells and its effect on mitochondria (2012)
Baldo, Elisa C. Fornari; Pereira, Lílian Cristina; Silva, Cássio Prinholato da; Sampaio, Suely Vilela; Dorta, Daniel Junqueira; Arantes, Eliane Candiani
Source: Anais. Conference titles: Simpósio Internacional de Pós-Graduação e Pesquisa (SINPOSPq). Unidades: FCFRP, FFCLRP
Subjects: VENENOS DE ORIGEM ANIMAL, BUFONIDAE, NEOPLASIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BALDO, Elisa C. Fornari et al. Evaluation of the cytotoxic activity of Rhinella schneideri toad poison on tumor cells and its effect on mitochondria. 2012, Anais.. Ribeirão Preto: FCFRP-USP, 2012. . Acesso em: 18 nov. 2024.
APA
Baldo, E. C. F., Pereira, L. C., Silva, C. P. da, Sampaio, S. V., Dorta, D. J., & Arantes, E. C. (2012). Evaluation of the cytotoxic activity of Rhinella schneideri toad poison on tumor cells and its effect on mitochondria. In Anais. Ribeirão Preto: FCFRP-USP.
NLM
Baldo ECF, Pereira LC, Silva CP da, Sampaio SV, Dorta DJ, Arantes EC. Evaluation of the cytotoxic activity of Rhinella schneideri toad poison on tumor cells and its effect on mitochondria. Anais. 2012 ;[citado 2024 nov. 18 ]
Vancouver
Baldo ECF, Pereira LC, Silva CP da, Sampaio SV, Dorta DJ, Arantes EC. Evaluation of the cytotoxic activity of Rhinella schneideri toad poison on tumor cells and its effect on mitochondria. Anais. 2012 ;[citado 2024 nov. 18 ]
Artigo de periodico
The anti-cancer agent nemorosone is a new potent protonophoric mitochondrial uncoupler (2011)
Andreu, Gilberto Lázaro Pardo; Figueredo, Yanier Nuñez; Tudella, Valéria Gomes; Rubio, Osmany Cuesta; Rodrigues, Fernando P.; Pestana, Cezar Rangel; Uyemura, Sérgio Akira; Leopoldino, Andréia Machado; Alberici, Luciane Carla; Curti, Carlos
Source: Mitochondrion. Unidade: FCFRP
Subjects: NEOPLASIAS, MITOCÔNDRIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ANDREU, Gilberto Lázaro Pardo et al. The anti-cancer agent nemorosone is a new potent protonophoric mitochondrial uncoupler. Mitochondrion, v. 11, n. 2, p. 255-263, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.mito.2010.10.008. Acesso em: 18 nov. 2024.
APA
Andreu, G. L. P., Figueredo, Y. N., Tudella, V. G., Rubio, O. C., Rodrigues, F. P., Pestana, C. R., et al. (2011). The anti-cancer agent nemorosone is a new potent protonophoric mitochondrial uncoupler. Mitochondrion, 11( 2), 255-263. doi:10.1016/j.mito.2010.10.008
NLM
Andreu GLP, Figueredo YN, Tudella VG, Rubio OC, Rodrigues FP, Pestana CR, Uyemura SA, Leopoldino AM, Alberici LC, Curti C. The anti-cancer agent nemorosone is a new potent protonophoric mitochondrial uncoupler [Internet]. Mitochondrion. 2011 ; 11( 2): 255-263.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.mito.2010.10.008
Vancouver
Andreu GLP, Figueredo YN, Tudella VG, Rubio OC, Rodrigues FP, Pestana CR, Uyemura SA, Leopoldino AM, Alberici LC, Curti C. The anti-cancer agent nemorosone is a new potent protonophoric mitochondrial uncoupler [Internet]. Mitochondrion. 2011 ; 11( 2): 255-263.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.mito.2010.10.008
Trabalho de evento-resumo
A nitrosyl ruthenium complex [Ru"(terpy)(bdqi)NO]³+ with nitric oxide release and cytotoxic effects against B16-F10 tumor cell line (2011)
Heinrich, Tassiele Andréa; Rodrigues, Gerson Jhonatan; Bendhack, Lusiane Maria; Barufi, Marcelo Dias; Silva, Roberto Santana da
Source: Oficial Program. Conference titles: International Congress of Pharmaceutical Sciences (CIFARP). Unidade: FCFRP
Subjects: ÓXIDO NÍTRICO, RUTÊNIO, NEOPLASIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
HEINRICH, Tassiele Andréa et al. A nitrosyl ruthenium complex [Ru"(terpy)(bdqi)NO]³+ with nitric oxide release and cytotoxic effects against B16-F10 tumor cell line. 2011, Anais.. Ribeirão Preto: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo, 2011. . Acesso em: 18 nov. 2024.
APA
Heinrich, T. A., Rodrigues, G. J., Bendhack, L. M., Barufi, M. D., & Silva, R. S. da. (2011). A nitrosyl ruthenium complex [Ru"(terpy)(bdqi)NO]³+ with nitric oxide release and cytotoxic effects against B16-F10 tumor cell line. In Oficial Program. Ribeirão Preto: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo.
NLM
Heinrich TA, Rodrigues GJ, Bendhack LM, Barufi MD, Silva RS da. A nitrosyl ruthenium complex [Ru"(terpy)(bdqi)NO]³+ with nitric oxide release and cytotoxic effects against B16-F10 tumor cell line. Oficial Program. 2011 ;[citado 2024 nov. 18 ]
Vancouver
Heinrich TA, Rodrigues GJ, Bendhack LM, Barufi MD, Silva RS da. A nitrosyl ruthenium complex [Ru"(terpy)(bdqi)NO]³+ with nitric oxide release and cytotoxic effects against B16-F10 tumor cell line. Oficial Program. 2011 ;[citado 2024 nov. 18 ]

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