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Artigo de periodico
Hierarchical chitin nanocrystal-based 3D printed dual-layer membranes hydrogels: A dual drug delivery nano-platform for periodontal tissue regeneration (2024)
Santos, Danilo Martins dos; Moon, Jae-I.; Kim, Da-Seul; Bassous, Nicole Joy; Marangon, Crisiane Aparecida; Campana Filho, Sergio Paulo ; Correa, Daniel Souza; Kang, Min-Ho; Kim, Woo-Jin; Shin, Su Ryon
Fonte: ACS Nano. Unidade: IQSC
Assuntos: IMPRESSÃO 3-D, ANATOMIA, REGENERAÇÃO ÓSSEA
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SANTOS, Danilo Martins dos et al. Hierarchical chitin nanocrystal-based 3D printed dual-layer membranes hydrogels: A dual drug delivery nano-platform for periodontal tissue regeneration. ACS Nano, 2024Tradução . . Disponível em: https://doi.org/10.1021/acsnano.4c05558. Acesso em: 06 nov. 2024.
APA
Santos, D. M. dos, Moon, J. -I., Kim, D. -S., Bassous, N. J., Marangon, C. A., Campana Filho, S. P., et al. (2024). Hierarchical chitin nanocrystal-based 3D printed dual-layer membranes hydrogels: A dual drug delivery nano-platform for periodontal tissue regeneration. ACS Nano. doi:10.1021/acsnano.4c05558
NLM
Santos DM dos, Moon J-I, Kim D-S, Bassous NJ, Marangon CA, Campana Filho SP, Correa DS, Kang M-H, Kim W-J, Shin SR. Hierarchical chitin nanocrystal-based 3D printed dual-layer membranes hydrogels: A dual drug delivery nano-platform for periodontal tissue regeneration [Internet]. ACS Nano. 2024 ;[citado 2024 nov. 06 ] Available from: https://doi.org/10.1021/acsnano.4c05558
Vancouver
Santos DM dos, Moon J-I, Kim D-S, Bassous NJ, Marangon CA, Campana Filho SP, Correa DS, Kang M-H, Kim W-J, Shin SR. Hierarchical chitin nanocrystal-based 3D printed dual-layer membranes hydrogels: A dual drug delivery nano-platform for periodontal tissue regeneration [Internet]. ACS Nano. 2024 ;[citado 2024 nov. 06 ] Available from: https://doi.org/10.1021/acsnano.4c05558
Artigo de periodico
Voluntary wheel running prevents formation of membrane attack complexes and myelin degradation after peripheral nerve injury (2024)
Green-Fulgham, Suzanne M. ; Lacagnina, Michael J. ; Willcox, Kendal F.; Li, Jiahe; Harland, Michael E.; Ciena, Adriano Polican ; Rocha, Igor Rafael. Correia ; Ball, Jayson B. ; Dreher, Renee A. ; Zuberi, Younus A.; Dragavon, Joseph M. ; Chacur, Marucia ; Maier, Steven F. ; Watkins, Linda R. ; Grace, Peter M.
Fonte: Brain, Behavior, and Immunity. Unidade: ICB
Assuntos: ANATOMIA, MIELINA DE ANIMAL, BAINHA DE MIELINA, FIBRAS NERVOSAS, NERVOS PERIFÉRICOS DE ANIMAL, NERVO CIÁTICO, DOENÇAS DO SISTEMA NERVOSO PERIFÉRICO, DOR CRÔNICA, RATOS, ROEDORES
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GREEN-FULGHAM, Suzanne M. et al. Voluntary wheel running prevents formation of membrane attack complexes and myelin degradation after peripheral nerve injury. Brain, Behavior, and Immunity, v. 115, p. 419-431, 2024Tradução . . Disponível em: https://doi.org/10.1016/j.bbi.2023.10.027. Acesso em: 06 nov. 2024.
APA
Green-Fulgham, S. M., Lacagnina, M. J., Willcox, K. F., Li, J., Harland, M. E., Ciena, A. P., et al. (2024). Voluntary wheel running prevents formation of membrane attack complexes and myelin degradation after peripheral nerve injury. Brain, Behavior, and Immunity, 115, 419-431. doi:10.1016/j.bbi.2023.10.027
NLM
Green-Fulgham SM, Lacagnina MJ, Willcox KF, Li J, Harland ME, Ciena AP, Rocha IRC, Ball JB, Dreher RA, Zuberi YA, Dragavon JM, Chacur M, Maier SF, Watkins LR, Grace PM. Voluntary wheel running prevents formation of membrane attack complexes and myelin degradation after peripheral nerve injury [Internet]. Brain, Behavior, and Immunity. 2024 ; 115 419-431.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.bbi.2023.10.027
Vancouver
Green-Fulgham SM, Lacagnina MJ, Willcox KF, Li J, Harland ME, Ciena AP, Rocha IRC, Ball JB, Dreher RA, Zuberi YA, Dragavon JM, Chacur M, Maier SF, Watkins LR, Grace PM. Voluntary wheel running prevents formation of membrane attack complexes and myelin degradation after peripheral nerve injury [Internet]. Brain, Behavior, and Immunity. 2024 ; 115 419-431.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.bbi.2023.10.027
Artigo de periodico
Central growth hormone action regulates neuroglial and proinflammatory markers in the hypothalamus of male mice (2023)
Wasinski, Frederick ; Tavares, Mariana Rosolen ; Gusmão, Daniela O. ; List, Edward O. ; Kopchick, John J. ; Alves, Guilherme A. ; Frazão, Renata ; Donato Junior, José
Fonte: Neuroscience Letters. Unidade: ICB
Assuntos: ANATOMIA, FISIOLOGIA, HIPOTÁLAMO, ASTRÓCITOS, HORMÔNIO DO CRESCIMENTO, FATOR DE CRESCIMENTO INSULIN-LIKE I, RNA MENSAGEIRO, PROTEÍNAS G
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WASINSKI, Frederick et al. Central growth hormone action regulates neuroglial and proinflammatory markers in the hypothalamus of male mice. Neuroscience Letters, v. 806, p. 1-8, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.neulet.2023.137236. Acesso em: 06 nov. 2024.
APA
Wasinski, F., Tavares, M. R., Gusmão, D. O., List, E. O., Kopchick, J. J., Alves, G. A., et al. (2023). Central growth hormone action regulates neuroglial and proinflammatory markers in the hypothalamus of male mice. Neuroscience Letters, 806, 1-8. doi:10.1016/j.neulet.2023.137236
NLM
Wasinski F, Tavares MR, Gusmão DO, List EO, Kopchick JJ, Alves GA, Frazão R, Donato Junior J. Central growth hormone action regulates neuroglial and proinflammatory markers in the hypothalamus of male mice [Internet]. Neuroscience Letters. 2023 ; 806 1-8.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.neulet.2023.137236
Vancouver
Wasinski F, Tavares MR, Gusmão DO, List EO, Kopchick JJ, Alves GA, Frazão R, Donato Junior J. Central growth hormone action regulates neuroglial and proinflammatory markers in the hypothalamus of male mice [Internet]. Neuroscience Letters. 2023 ; 806 1-8.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.neulet.2023.137236
Artigo de periodico
An AAV-CRISPR/Cas9 strategy for gene editing across divergent rodent species: Targeting neural oxytocin receptors as a proof of concept (2023)
Boender, Arjen J. ; Boon, Marina ; Albers, H. Elliott ; Eck, Samantha R. ; Fricker, Brandon A. ; Kelly, Aubrey M. ; LeDoux, Joseph E. ; Shrestha, Prerana ; Taylor, Jack H. ; Trainor, Brian C. ; Triana-Del Rio, Rodrigo ; Young, Larry J. ; Motta, Simone Cristina
Fonte: Science Advances. Unidade: ICB
Assuntos: ANATOMIA, ROEDORES, OCITOCINA, TOXICIDADE EM ANIMAL, NEUROCIÊNCIAS, DISSECAÇÃO ANIMAL, MODELOS ANIMAIS DE DOENÇAS, CIRCUITOS ELETRÔNICOS, GENÔMICA, GENÉTICA BACTERIANA
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BOENDER, Arjen J. et al. An AAV-CRISPR/Cas9 strategy for gene editing across divergent rodent species: Targeting neural oxytocin receptors as a proof of concept. Science Advances, v. 9, p. 1-16, 2023Tradução . . Disponível em: https://doi.org/10.1126/sciadv.adf4950. Acesso em: 06 nov. 2024.
APA
Boender, A. J., Boon, M., Albers, H. E., Eck, S. R., Fricker, B. A., Kelly, A. M., et al. (2023). An AAV-CRISPR/Cas9 strategy for gene editing across divergent rodent species: Targeting neural oxytocin receptors as a proof of concept. Science Advances, 9, 1-16. doi:10.1126/sciadv.adf4950
NLM
Boender AJ, Boon M, Albers HE, Eck SR, Fricker BA, Kelly AM, LeDoux JE, Shrestha P, Taylor JH, Trainor BC, Triana-Del Rio R, Young LJ, Motta SC. An AAV-CRISPR/Cas9 strategy for gene editing across divergent rodent species: Targeting neural oxytocin receptors as a proof of concept [Internet]. Science Advances. 2023 ; 9 1-16.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1126/sciadv.adf4950
Vancouver
Boender AJ, Boon M, Albers HE, Eck SR, Fricker BA, Kelly AM, LeDoux JE, Shrestha P, Taylor JH, Trainor BC, Triana-Del Rio R, Young LJ, Motta SC. An AAV-CRISPR/Cas9 strategy for gene editing across divergent rodent species: Targeting neural oxytocin receptors as a proof of concept [Internet]. Science Advances. 2023 ; 9 1-16.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1126/sciadv.adf4950
Artigo de periodico
Interactions between cannabinoid and opioid receptors in a mouse model of diabetic neuropathy (2022)
Toniolo, Elaine Flamia ; Gupta, Achla ; Franciosi, Adriano Cardozo ; Gomes, Ivone ; Devi, Lakshmi A.; Dale, Camila Squarzoni
Fonte: PAIN. Unidade: ICB
Assuntos: ANATOMIA, CANABINOIDES, ÓPIO, ANALGESIA, NEUROPATIAS DIABÉTICAS, PROTEÍNAS G, MODELOS ANIMAIS DE DOENÇAS, CAMUNDONGOS
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TONIOLO, Elaine Flamia et al. Interactions between cannabinoid and opioid receptors in a mouse model of diabetic neuropathy. PAIN, v. 163, n. 7, 2022Tradução . . Disponível em: https://doi.org/10.1097/j.pain.0000000000002527. Acesso em: 06 nov. 2024.
APA
Toniolo, E. F., Gupta, A., Franciosi, A. C., Gomes, I., Devi, L. A., & Dale, C. S. (2022). Interactions between cannabinoid and opioid receptors in a mouse model of diabetic neuropathy. PAIN, 163( 7). doi:10.1097/j.pain.0000000000002527
NLM
Toniolo EF, Gupta A, Franciosi AC, Gomes I, Devi LA, Dale CS. Interactions between cannabinoid and opioid receptors in a mouse model of diabetic neuropathy [Internet]. PAIN. 2022 ; 163( 7):[citado 2024 nov. 06 ] Available from: https://doi.org/10.1097/j.pain.0000000000002527
Vancouver
Toniolo EF, Gupta A, Franciosi AC, Gomes I, Devi LA, Dale CS. Interactions between cannabinoid and opioid receptors in a mouse model of diabetic neuropathy [Internet]. PAIN. 2022 ; 163( 7):[citado 2024 nov. 06 ] Available from: https://doi.org/10.1097/j.pain.0000000000002527
Artigo de periodico
Effects of the isolated and combined ablation of growth hormone and IGF-1 receptors in somatostatin neurons (2022)
Chaves, Fernanda Machado; Wasinski, Frederick ; Tavares, Mariana Rosolen ; Mansano, Naira da Silva ; Frazão, Renata ; Gusmão, Daniela O. ; Bergonsi, Paula Gabriele Fernandes Quaresma ; Pedroso, João Alfredo Bolivar ; Elias, Carol F. ; List, Edward O. ; Kopchick, John J. ; Szawka, Raphael E. ; Donato Junior, José
Fonte: Endocrinology. Unidade: ICB
Assuntos: ANATOMIA, FISIOLOGIA, HIPOTÁLAMO, HORMÔNIO DO CRESCIMENTO, FATOR DE CRESCIMENTO INSULIN-LIKE I, NEURÔNIOS, SOMATOSTATINA, EXPRESSÃO GÊNICA, CAMUNDONGOS
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CHAVES, Fernanda Machado et al. Effects of the isolated and combined ablation of growth hormone and IGF-1 receptors in somatostatin neurons. Endocrinology, 2022Tradução . . Disponível em: https://doi.org/10.1210/endocr/bqac045. Acesso em: 06 nov. 2024.
APA
Chaves, F. M., Wasinski, F., Tavares, M. R., Mansano, N. da S., Frazão, R., Gusmão, D. O., et al. (2022). Effects of the isolated and combined ablation of growth hormone and IGF-1 receptors in somatostatin neurons. Endocrinology. doi:10.1210/endocr/bqac045
NLM
Chaves FM, Wasinski F, Tavares MR, Mansano N da S, Frazão R, Gusmão DO, Bergonsi PGFQ, Pedroso JAB, Elias CF, List EO, Kopchick JJ, Szawka RE, Donato Junior J. Effects of the isolated and combined ablation of growth hormone and IGF-1 receptors in somatostatin neurons [Internet]. Endocrinology. 2022 ;[citado 2024 nov. 06 ] Available from: https://doi.org/10.1210/endocr/bqac045
Vancouver
Chaves FM, Wasinski F, Tavares MR, Mansano N da S, Frazão R, Gusmão DO, Bergonsi PGFQ, Pedroso JAB, Elias CF, List EO, Kopchick JJ, Szawka RE, Donato Junior J. Effects of the isolated and combined ablation of growth hormone and IGF-1 receptors in somatostatin neurons [Internet]. Endocrinology. 2022 ;[citado 2024 nov. 06 ] Available from: https://doi.org/10.1210/endocr/bqac045
Artigo de periodico
Mitochondrial fusion, fission, and mitophagy in cardiac diseases: challenges and therapeutic opportunities (2022)
Scheffer, Débora da Luz ; Garcia, Adriana Ann; Lee, Lucia; Rosen, Daria Mochly ; Ferreira, Julio Cesar Batista
Fonte: Antioxidants & Redox Signaling. Unidade: ICB
Assuntos: ANATOMIA, INSUFICIÊNCIA CARDÍACA, MITOCÔNDRIAS, INFARTO DO MIOCÁRDIO, ESTRESSE OXIDATIVO, DOENÇAS CARDIOVASCULARES, FISIOLOGIA CARDIOVASCULAR
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SCHEFFER, Débora da Luz et al. Mitochondrial fusion, fission, and mitophagy in cardiac diseases: challenges and therapeutic opportunities. Antioxidants & Redox Signaling, v. 36, n. 13-15, p. 844-863, 2022Tradução . . Disponível em: https://doi.org/10.1089/ars.2021.0145. Acesso em: 06 nov. 2024.
APA
Scheffer, D. da L., Garcia, A. A., Lee, L., Rosen, D. M., & Ferreira, J. C. B. (2022). Mitochondrial fusion, fission, and mitophagy in cardiac diseases: challenges and therapeutic opportunities. Antioxidants & Redox Signaling, 36( 13-15), 844-863. doi:10.1089/ars.2021.0145
NLM
Scheffer D da L, Garcia AA, Lee L, Rosen DM, Ferreira JCB. Mitochondrial fusion, fission, and mitophagy in cardiac diseases: challenges and therapeutic opportunities [Internet]. Antioxidants & Redox Signaling. 2022 ; 36( 13-15): 844-863.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1089/ars.2021.0145
Vancouver
Scheffer D da L, Garcia AA, Lee L, Rosen DM, Ferreira JCB. Mitochondrial fusion, fission, and mitophagy in cardiac diseases: challenges and therapeutic opportunities [Internet]. Antioxidants & Redox Signaling. 2022 ; 36( 13-15): 844-863.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1089/ars.2021.0145
Artigo de periodico
A selective inhibitor of Cardiac Troponin I Phosphorylation by Delta Protein Kinase C (δPKC) as a treatment for Ischemia-reperfusion injury (2022)
Qvit, Nir ; Lin, Amanda J. ; Elezaby, Aly ; Ostberg, Nicolai P. ; Campos, Juliane Cruz ; Ferreira, Julio Cesar Batista ; Rosen, Daria Mochly
Fonte: Pharmaceuticals. Unidade: ICB
Assuntos: ANATOMIA, ISQUEMIA MIOCÁRDICA, TROPONINA I, PROTEÍNAS MUSCULARES, PEPTÍDEOS, INFARTO DO MIOCÁRDIO, CARDIOPATIAS, AMINOÁCIDOS
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QVIT, Nir et al. A selective inhibitor of Cardiac Troponin I Phosphorylation by Delta Protein Kinase C (δPKC) as a treatment for Ischemia-reperfusion injury. Pharmaceuticals, v. 15, n. 3, p. 1-11, 2022Tradução . . Disponível em: https://doi.org/10.3390/ph15030271. Acesso em: 06 nov. 2024.
APA
Qvit, N., Lin, A. J., Elezaby, A., Ostberg, N. P., Campos, J. C., Ferreira, J. C. B., & Rosen, D. M. (2022). A selective inhibitor of Cardiac Troponin I Phosphorylation by Delta Protein Kinase C (δPKC) as a treatment for Ischemia-reperfusion injury. Pharmaceuticals, 15( 3), 1-11. doi:10.3390/ph15030271
NLM
Qvit N, Lin AJ, Elezaby A, Ostberg NP, Campos JC, Ferreira JCB, Rosen DM. A selective inhibitor of Cardiac Troponin I Phosphorylation by Delta Protein Kinase C (δPKC) as a treatment for Ischemia-reperfusion injury [Internet]. Pharmaceuticals. 2022 ; 15( 3): 1-11.[citado 2024 nov. 06 ] Available from: https://doi.org/10.3390/ph15030271
Vancouver
Qvit N, Lin AJ, Elezaby A, Ostberg NP, Campos JC, Ferreira JCB, Rosen DM. A selective inhibitor of Cardiac Troponin I Phosphorylation by Delta Protein Kinase C (δPKC) as a treatment for Ischemia-reperfusion injury [Internet]. Pharmaceuticals. 2022 ; 15( 3): 1-11.[citado 2024 nov. 06 ] Available from: https://doi.org/10.3390/ph15030271
Artigo de periodico
Structural analysis of TrkA mutations in patients with congenital insensitivity to pain reveals PLCy as an analgesic drug target (2022)
Moraes, Beatriz Caroline de ; Ribeiro Filho, Helder Veras ; Roldão, Allan Pradelli ; Toniolo, Elaine Flamia ; Carretero, Gustavo Penteado Battesini ; Sgro, Germán Gustavo ; Batista, Fernanda A. H; Berardi, Damian E ; Oliveira, Victoria Regina da Silva ; Tomasin, Rebeka ; Vieceli, Felipe Monteleone ; Pramio, Dimitrius Tansini ; Bruni-Cardoso, Alexandre ; Figueira, Ana C. M; Farah, Chuck Shaker ; Devi, Lakshmi A. ; Dale, Camila Squarzoni ; Oliveira, Paulo S. L. de; Schechtman, Deborah
Fonte: Science Signaling. Unidades: FCFRP, IQ, ICB
Assuntos: DOR CRÔNICA, ANALGÉSICOS, ANATOMIA, FOSFOLIPASES, MUTAÇÃO GENÉTICA
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MORAES, Beatriz Caroline de et al. Structural analysis of TrkA mutations in patients with congenital insensitivity to pain reveals PLCy as an analgesic drug target. Science Signaling, v. 15, n. 731, p. 1-16, 2022Tradução . . Disponível em: https://doi.org/10.1126/scisignal.abm6046. Acesso em: 06 nov. 2024.
APA
Moraes, B. C. de, Ribeiro Filho, H. V., Roldão, A. P., Toniolo, E. F., Carretero, G. P. B., Sgro, G. G., et al. (2022). Structural analysis of TrkA mutations in patients with congenital insensitivity to pain reveals PLCy as an analgesic drug target. Science Signaling, 15( 731), 1-16. doi:10.1126/scisignal.abm6046
NLM
Moraes BC de, Ribeiro Filho HV, Roldão AP, Toniolo EF, Carretero GPB, Sgro GG, Batista FAH, Berardi DE, Oliveira VR da S, Tomasin R, Vieceli FM, Pramio DT, Bruni-Cardoso A, Figueira ACM, Farah CS, Devi LA, Dale CS, Oliveira PSL de, Schechtman D. Structural analysis of TrkA mutations in patients with congenital insensitivity to pain reveals PLCy as an analgesic drug target [Internet]. Science Signaling. 2022 ; 15( 731): 1-16.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1126/scisignal.abm6046
Vancouver
Moraes BC de, Ribeiro Filho HV, Roldão AP, Toniolo EF, Carretero GPB, Sgro GG, Batista FAH, Berardi DE, Oliveira VR da S, Tomasin R, Vieceli FM, Pramio DT, Bruni-Cardoso A, Figueira ACM, Farah CS, Devi LA, Dale CS, Oliveira PSL de, Schechtman D. Structural analysis of TrkA mutations in patients with congenital insensitivity to pain reveals PLCy as an analgesic drug target [Internet]. Science Signaling. 2022 ; 15( 731): 1-16.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1126/scisignal.abm6046
Artigo de periodico
Surface-directed mineralization of fibrous collagen scaffolds in simulated body fluid for tissue engineering applications (2021)
Bim-Júnior, Odair; Curylofo-Zotti, Fabiana de Almeida ; Reis, Mariana; Alania, Yvette; Lisboa-Filho, Paulo N.; Bedran-Russo, Ana K.
Fonte: ACS Applied Bio Materials. Unidade: FORP
Assuntos: ANATOMIA, BIOPOLÍMEROS, MINERAIS, MATERIAIS NANOESTRUTURADOS, ENGENHARIA TECIDUAL, COLÁGENO
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BIM-JÚNIOR, Odair et al. Surface-directed mineralization of fibrous collagen scaffolds in simulated body fluid for tissue engineering applications. ACS Applied Bio Materials, v. 4, n. 3, p. 2514-2522, 2021Tradução . . Disponível em: https://doi.org/10.1021/acsabm.0c01507. Acesso em: 06 nov. 2024.
APA
Bim-Júnior, O., Curylofo-Zotti, F. de A., Reis, M., Alania, Y., Lisboa-Filho, P. N., & Bedran-Russo, A. K. (2021). Surface-directed mineralization of fibrous collagen scaffolds in simulated body fluid for tissue engineering applications. ACS Applied Bio Materials, 4( 3), 2514-2522. doi:10.1021/acsabm.0c01507
NLM
Bim-Júnior O, Curylofo-Zotti F de A, Reis M, Alania Y, Lisboa-Filho PN, Bedran-Russo AK. Surface-directed mineralization of fibrous collagen scaffolds in simulated body fluid for tissue engineering applications [Internet]. ACS Applied Bio Materials. 2021 ; 4( 3): 2514-2522.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1021/acsabm.0c01507
Vancouver
Bim-Júnior O, Curylofo-Zotti F de A, Reis M, Alania Y, Lisboa-Filho PN, Bedran-Russo AK. Surface-directed mineralization of fibrous collagen scaffolds in simulated body fluid for tissue engineering applications [Internet]. ACS Applied Bio Materials. 2021 ; 4( 3): 2514-2522.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1021/acsabm.0c01507
Artigo de periodico
Mild mitochondrial impairment enhances innate immunity and longevity through ATFS-1 and p38 signaling (2021)
Campos, Juliane Cruz ; Wu, Ziyun ; Rudich, Paige ; Soo, Sonja K.; Mistry, Meeta ; Ferreira, Julio Cesar Batista ; Blackwell, T. Keith ; Raamsdonk, Jeremy M. Van
Fonte: EMBO Reports. Unidade: ICB
Assuntos: ANATOMIA, NEMATOIDES, MITOCÔNDRIAS, ENVELHECIMENTO, IMUNIDADE, PROTEÍNAS, ELÉTRONS, REGULAÇÃO GÊNICA
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CAMPOS, Juliane Cruz et al. Mild mitochondrial impairment enhances innate immunity and longevity through ATFS-1 and p38 signaling. EMBO Reports, p. 1-19, 2021Tradução . . Disponível em: https://doi.org/10.15252/embr.202152964. Acesso em: 06 nov. 2024.
APA
Campos, J. C., Wu, Z., Rudich, P., Soo, S. K., Mistry, M., Ferreira, J. C. B., et al. (2021). Mild mitochondrial impairment enhances innate immunity and longevity through ATFS-1 and p38 signaling. EMBO Reports, 1-19. doi:10.15252/embr.202152964
NLM
Campos JC, Wu Z, Rudich P, Soo SK, Mistry M, Ferreira JCB, Blackwell TK, Raamsdonk JMV. Mild mitochondrial impairment enhances innate immunity and longevity through ATFS-1 and p38 signaling [Internet]. EMBO Reports. 2021 ; 1-19.[citado 2024 nov. 06 ] Available from: https://doi.org/10.15252/embr.202152964
Vancouver
Campos JC, Wu Z, Rudich P, Soo SK, Mistry M, Ferreira JCB, Blackwell TK, Raamsdonk JMV. Mild mitochondrial impairment enhances innate immunity and longevity through ATFS-1 and p38 signaling [Internet]. EMBO Reports. 2021 ; 1-19.[citado 2024 nov. 06 ] Available from: https://doi.org/10.15252/embr.202152964
Artigo de periodico
Dorsal premammillary projection to periaqueductal gray controls escape vigor from innate and conditioned threats (2021)
Wang, Weisheng; Schuette, Peter J. ; Vu, Mimi Q. La; Torossian, Anita ; Tobias, Brooke C. ; Ceko, Marta ; Kragel, Philip A. ; Reis, Fernando Midea C. V. ; Ji, Shiyu ; Sehgal, Megha ; Pereira, Sandra Maesta ; Chakerian, Meghmik; Silva, Alcino J. ; Canteras, Newton Sabino ; Wager, Tor ; Kao, Jonathan C. ; Adhikari, Avishek
Fonte: eLife. Unidade: ICB
Assuntos: ANATOMIA, SUBSTÂNCIA CINZENTA PERIAQUEDUTAL, NEUROCIÊNCIAS, CAMUNDONGOS, COLECISTOCININA, HORMÔNIOS DO TRATO GASTROINTESTINAL, HIPOTÁLAMO
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WANG, Weisheng et al. Dorsal premammillary projection to periaqueductal gray controls escape vigor from innate and conditioned threats. eLife, v. 10, p. 1-30, 2021Tradução . . Disponível em: https://doi.org/10.7554/eLife.69178. Acesso em: 06 nov. 2024.
APA
Wang, W., Schuette, P. J., Vu, M. Q. L., Torossian, A., Tobias, B. C., Ceko, M., et al. (2021). Dorsal premammillary projection to periaqueductal gray controls escape vigor from innate and conditioned threats. eLife, 10, 1-30. doi:10.7554/eLife.69178
NLM
Wang W, Schuette PJ, Vu MQL, Torossian A, Tobias BC, Ceko M, Kragel PA, Reis FMCV, Ji S, Sehgal M, Pereira SM, Chakerian M, Silva AJ, Canteras NS, Wager T, Kao JC, Adhikari A. Dorsal premammillary projection to periaqueductal gray controls escape vigor from innate and conditioned threats [Internet]. eLife. 2021 ; 10 1-30.[citado 2024 nov. 06 ] Available from: https://doi.org/10.7554/eLife.69178
Vancouver
Wang W, Schuette PJ, Vu MQL, Torossian A, Tobias BC, Ceko M, Kragel PA, Reis FMCV, Ji S, Sehgal M, Pereira SM, Chakerian M, Silva AJ, Canteras NS, Wager T, Kao JC, Adhikari A. Dorsal premammillary projection to periaqueductal gray controls escape vigor from innate and conditioned threats [Internet]. eLife. 2021 ; 10 1-30.[citado 2024 nov. 06 ] Available from: https://doi.org/10.7554/eLife.69178
Artigo de periodico
Activation of PKCε-ALDH2 Axis Prevents 4-HNE-induced pain in mice (2021)
Martins, Bárbara B.; Hösch, Natália G. ; Alcantara, Queren Apuque ; Budas, Grant R.; Chen, Che-Hong ; Rosen, Daria Mochly ; Ferreira, Julio Cesar Batista ; Zambelli, Vanessa O.
Fonte: Biomolecules. Unidade: ICB
Assuntos: ANATOMIA, CAMUNDONGOS, ESTRESSE OXIDATIVO, PROTEÍNAS QUINASES, DOR CRÔNICA, COMPORTAMENTO ANIMAL, CARRAGENINA, MITOCÔNDRIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MARTINS, Bárbara B. et al. Activation of PKCε-ALDH2 Axis Prevents 4-HNE-induced pain in mice. Biomolecules, v. 11, n. 12, p. 1-13, 2021Tradução . . Disponível em: https://doi.org/10.3390/biom11121798. Acesso em: 06 nov. 2024.
APA
Martins, B. B., Hösch, N. G., Alcantara, Q. A., Budas, G. R., Chen, C. -H., Rosen, D. M., et al. (2021). Activation of PKCε-ALDH2 Axis Prevents 4-HNE-induced pain in mice. Biomolecules, 11( 12), 1-13. doi:10.3390/biom11121798
NLM
Martins BB, Hösch NG, Alcantara QA, Budas GR, Chen C-H, Rosen DM, Ferreira JCB, Zambelli VO. Activation of PKCε-ALDH2 Axis Prevents 4-HNE-induced pain in mice [Internet]. Biomolecules. 2021 ; 11( 12): 1-13.[citado 2024 nov. 06 ] Available from: https://doi.org/10.3390/biom11121798
Vancouver
Martins BB, Hösch NG, Alcantara QA, Budas GR, Chen C-H, Rosen DM, Ferreira JCB, Zambelli VO. Activation of PKCε-ALDH2 Axis Prevents 4-HNE-induced pain in mice [Internet]. Biomolecules. 2021 ; 11( 12): 1-13.[citado 2024 nov. 06 ] Available from: https://doi.org/10.3390/biom11121798
Artigo de periodico
Treatment strategies for glucose-6-phosphate dehydrogenase deficiency: past and future perspectives (2021)
Garcia, Adriana A. ; Koperniku, Ana ; Ferreira, Julio Cesar Batista ; Rosen, Daria Mochly
Fonte: Trends in Pharmacological Sciences. Unidade: ICB
Assuntos: ANATOMIA, AMINOÁCIDOS, ATIVAÇÃO ENZIMÁTICA, GLICOSE, ERITRÓCITOS, ESTRESSE OXIDATIVO, MUTAÇÃO GENÉTICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GARCIA, Adriana A. et al. Treatment strategies for glucose-6-phosphate dehydrogenase deficiency: past and future perspectives. Trends in Pharmacological Sciences, v. 42, n. 10, p. 829-844, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.tips.2021.07.002. Acesso em: 06 nov. 2024.
APA
Garcia, A. A., Koperniku, A., Ferreira, J. C. B., & Rosen, D. M. (2021). Treatment strategies for glucose-6-phosphate dehydrogenase deficiency: past and future perspectives. Trends in Pharmacological Sciences, 42( 10), 829-844. doi:10.1016/j.tips.2021.07.002
NLM
Garcia AA, Koperniku A, Ferreira JCB, Rosen DM. Treatment strategies for glucose-6-phosphate dehydrogenase deficiency: past and future perspectives [Internet]. Trends in Pharmacological Sciences. 2021 ; 42( 10): 829-844.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.tips.2021.07.002
Vancouver
Garcia AA, Koperniku A, Ferreira JCB, Rosen DM. Treatment strategies for glucose-6-phosphate dehydrogenase deficiency: past and future perspectives [Internet]. Trends in Pharmacological Sciences. 2021 ; 42( 10): 829-844.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.tips.2021.07.002
Artigo de periodico
Dorsal periaqueductal gray ensembles represent approach and avoidance states (2021)
Reis, Fernando M. C. V.; Lee, Johannes Y.; Pereira, Sandra Maesta ; Schuette, Peter J. ; Chakerian, Meghmik; Liu, Jinhan; Vu, Mimi Q. La; Tobias, Brooke C. ; Ikebara, Juliane M. ; Kihara, Alexandre Hiroaki ; Canteras, Newton Sabino ; Kao, Jonathan C.; Adhikari, Avishek
Fonte: eLife. Unidade: ICB
Assuntos: ANATOMIA, ANIMAIS PREDADORES, CAMUNDONGOS, COMPORTAMENTO ANIMAL, SUBSTÂNCIA CINZENTA PERIAQUEDUTAL, DORSO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
REIS, Fernando M. C. V. et al. Dorsal periaqueductal gray ensembles represent approach and avoidance states. eLife, v. 10, p. 1-22, 2021Tradução . . Disponível em: https://doi.org/10.7554/eLife.64934. Acesso em: 06 nov. 2024.
APA
Reis, F. M. C. V., Lee, J. Y., Pereira, S. M., Schuette, P. J., Chakerian, M., Liu, J., et al. (2021). Dorsal periaqueductal gray ensembles represent approach and avoidance states. eLife, 10, 1-22. doi:10.7554/eLife.64934
NLM
Reis FMCV, Lee JY, Pereira SM, Schuette PJ, Chakerian M, Liu J, Vu MQL, Tobias BC, Ikebara JM, Kihara AH, Canteras NS, Kao JC, Adhikari A. Dorsal periaqueductal gray ensembles represent approach and avoidance states [Internet]. eLife. 2021 ; 10 1-22.[citado 2024 nov. 06 ] Available from: https://doi.org/10.7554/eLife.64934
Vancouver
Reis FMCV, Lee JY, Pereira SM, Schuette PJ, Chakerian M, Liu J, Vu MQL, Tobias BC, Ikebara JM, Kihara AH, Canteras NS, Kao JC, Adhikari A. Dorsal periaqueductal gray ensembles represent approach and avoidance states [Internet]. eLife. 2021 ; 10 1-22.[citado 2024 nov. 06 ] Available from: https://doi.org/10.7554/eLife.64934
Artigo de periodico
The role of co-neurotransmitters in sleep and wake regulation (2019)
Oh, Jun ; Petersen, Cathrine ; Walsh, Christine M.; Bittencourt, Jackson Cioni ; Neylan, Thomas C. ; Grinberg, Lea T.
Fonte: Molecular Psychiatry. Unidade: ICB
Assuntos: ANATOMIA, CÉREBRO, TRONCO CEREBRAL, DIENCÉFALO, HIPOTÁLAMO, NEUROTRANSMISSORES, SONO, VIGÍLIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
OH, Jun et al. The role of co-neurotransmitters in sleep and wake regulation. Molecular Psychiatry, v. 24, p. 1284–1295, 2019Tradução . . Disponível em: https://doi.org/10.1038/s41380-018-0291-2. Acesso em: 06 nov. 2024.
APA
Oh, J., Petersen, C., Walsh, C. M., Bittencourt, J. C., Neylan, T. C., & Grinberg, L. T. (2019). The role of co-neurotransmitters in sleep and wake regulation. Molecular Psychiatry, 24, 1284–1295. doi:10.1038/s41380-018-0291-2
NLM
Oh J, Petersen C, Walsh CM, Bittencourt JC, Neylan TC, Grinberg LT. The role of co-neurotransmitters in sleep and wake regulation [Internet]. Molecular Psychiatry. 2019 ; 24 1284–1295.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1038/s41380-018-0291-2
Vancouver
Oh J, Petersen C, Walsh CM, Bittencourt JC, Neylan TC, Grinberg LT. The role of co-neurotransmitters in sleep and wake regulation [Internet]. Molecular Psychiatry. 2019 ; 24 1284–1295.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1038/s41380-018-0291-2
Artigo de periodico
Central growth hormone signaling is not required for the timing of puberty (2019)
Bohlen, Tabata Mariz ; Zampieri, Thais Tessari ; Furigo, Isadora Clivatti ; Teixeira, Pryscila Dryelle Sousa ; List, Edward O. ; Kopchick, John J. ; Donato Junior, José ; Frazão, Renata
Fonte: Journal of Endocrinology. Unidade: ICB
Assuntos: FISIOLOGIA, ANATOMIA, HORMÔNIO DO CRESCIMENTO, PUBERDADE, EXPRESSÃO GÊNICA, LEPTINA, HIPOTÁLAMO, INTERAÇÃO CELULAR, OVÁRIO, SISTEMA NERVOSO CENTRAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BOHLEN, Tabata Mariz et al. Central growth hormone signaling is not required for the timing of puberty. Journal of Endocrinology, v. 243, n. 3, p. 161–173, 2019Tradução . . Disponível em: https://doi.org/10.1530/JOE-19-0242. Acesso em: 06 nov. 2024.
APA
Bohlen, T. M., Zampieri, T. T., Furigo, I. C., Teixeira, P. D. S., List, E. O., Kopchick, J. J., et al. (2019). Central growth hormone signaling is not required for the timing of puberty. Journal of Endocrinology, 243( 3), 161–173. doi:10.1530/JOE-19-0242
NLM
Bohlen TM, Zampieri TT, Furigo IC, Teixeira PDS, List EO, Kopchick JJ, Donato Junior J, Frazão R. Central growth hormone signaling is not required for the timing of puberty [Internet]. Journal of Endocrinology. 2019 ; 243( 3): 161–173.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1530/JOE-19-0242
Vancouver
Bohlen TM, Zampieri TT, Furigo IC, Teixeira PDS, List EO, Kopchick JJ, Donato Junior J, Frazão R. Central growth hormone signaling is not required for the timing of puberty [Internet]. Journal of Endocrinology. 2019 ; 243( 3): 161–173.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1530/JOE-19-0242

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