Central growth hormone signaling is not required for the timing of puberty (2019)
- Authors:
- USP affiliated authors: DONATO JÚNIOR, JOSÉ - ICB ; FRAZÃO, RENATA - ICB ; BOHLEN, TABATA MARIZ - ICB ; FURIGO, ISADORA CLIVATTI - ICB ; TEIXEIRA, PRYSCILA DRYELLE SOUSA - ICB
- Unidade: ICB
- DOI: 10.1530/JOE-19-0242
- Subjects: FISIOLOGIA; ANATOMIA; HORMÔNIO DO CRESCIMENTO; PUBERDADE; EXPRESSÃO GÊNICA; LEPTINA; HIPOTÁLAMO; INTERAÇÃO CELULAR; OVÁRIO; SISTEMA NERVOSO CENTRAL
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Journal of Endocrinology
- ISSN: 1479-6805
- Volume/Número/Paginação/Ano: v. 243, n. 3, p. 161–173, 2019
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
BOHLEN, Tabata Mariz et al. Central growth hormone signaling is not required for the timing of puberty. Journal of Endocrinology, v. 243, n. 3, p. 161–173, 2019Tradução . . Disponível em: https://doi.org/10.1530/JOE-19-0242. Acesso em: 12 fev. 2026. -
APA
Bohlen, T. M., Zampieri, T. T., Furigo, I. C., Teixeira, P. D. S., List, E. O., Kopchick, J. J., et al. (2019). Central growth hormone signaling is not required for the timing of puberty. Journal of Endocrinology, 243( 3), 161–173. doi:10.1530/JOE-19-0242 -
NLM
Bohlen TM, Zampieri TT, Furigo IC, Teixeira PDS, List EO, Kopchick JJ, Donato Junior J, Frazão R. Central growth hormone signaling is not required for the timing of puberty [Internet]. Journal of Endocrinology. 2019 ; 243( 3): 161–173.[citado 2026 fev. 12 ] Available from: https://doi.org/10.1530/JOE-19-0242 -
Vancouver
Bohlen TM, Zampieri TT, Furigo IC, Teixeira PDS, List EO, Kopchick JJ, Donato Junior J, Frazão R. Central growth hormone signaling is not required for the timing of puberty [Internet]. Journal of Endocrinology. 2019 ; 243( 3): 161–173.[citado 2026 fev. 12 ] Available from: https://doi.org/10.1530/JOE-19-0242 - Growth hormone enhances the recovery of hypoglycemia via ventromedial hypothalamic neurons
- Socs3 ablation in kisspeptin cells partially prevents lipopolysaccharide-induced body weight loss
- Growth hormone receptor deletion reduces the density of axonal projections from hypothalamic arcuate nucleus neurons
- Central growth hormone action regulates metabolism during pregnancy
- Brain STAT5 modulates long-term metabolic and epigenetic changes induced by pregnancy and lactation in female mice
- STAT5 ablation in AgRP neurons increases female adiposity and blunts food restriction adaptations
- Maternal obesity increases hypothalamic miR-505-5p expression in mouse offspring leading to altered fatty acid sensing and increased intake of high-fat food
- Interactions between prolactin and kisspeptin to control reproduction
- Neuronal STAT5 signaling is required for maintaining lactation but not for postpartum maternal behaviors in mice
- Changes in Leptin Signaling by SOCS3 Modulate Fasting-Induced Hyperphagia and Weight Regain in Mice
Informações sobre o DOI: 10.1530/JOE-19-0242 (Fonte: oaDOI API)
Download do texto completo
| Tipo | Nome | Link | |
|---|---|---|---|
 |
Central_growth_hormone_si... |
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
