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Artigo de periodico
Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats (2019)
Awata, Wanessa Mayumi Carvalho; Gonzaga, Natália de Almeida; Borges, Vanessa de Fátima; Silva, Carla B. P; Tanus-Santos, José Eduardo ; Cunha, Fernando de Queiroz ; Tirapelli, Carlos Renato
Source: European Journal of Pharmacology. Unidades: FMRP, EERP
Subjects: SEPSE, ÓXIDO NÍTRICO
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ABNT
AWATA, Wanessa Mayumi Carvalho et al. Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats. European Journal of Pharmacology, v. 863, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2019.172706. Acesso em: 31 out. 2024.
APA
Awata, W. M. C., Gonzaga, N. de A., Borges, V. de F., Silva, C. B. P., Tanus-Santos, J. E., Cunha, F. de Q., & Tirapelli, C. R. (2019). Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats. European Journal of Pharmacology, 863. doi:10.1016/j.ejphar.2019.172706
NLM
Awata WMC, Gonzaga N de A, Borges V de F, Silva CBP, Tanus-Santos JE, Cunha F de Q, Tirapelli CR. Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats [Internet]. European Journal of Pharmacology. 2019 ; 863[citado 2024 out. 31 ] Available from: https://doi.org/10.1016/j.ejphar.2019.172706
Vancouver
Awata WMC, Gonzaga N de A, Borges V de F, Silva CBP, Tanus-Santos JE, Cunha F de Q, Tirapelli CR. Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats [Internet]. European Journal of Pharmacology. 2019 ; 863[citado 2024 out. 31 ] Available from: https://doi.org/10.1016/j.ejphar.2019.172706
Artigo de periodico
Spinal GABA-B receptor modulates neutrophil recruitment to the knee joint in zymosan-induced arthritis (2016)
Bassi, Gabriel S; Malvar, David do C; Cunha, Thiago Mattar; Cunha, Fernando de Queiroz; Kanashiro, Alexandre
Source: Naunyn-Schmiedeberg's Archives of Pharmacology. Unidade: FMRP
Subjects: ARTRITE, GABA, NEUROIMUNOMODULAÇÃO, NEUTRÓFILOS, SISTEMA NERVOSO SIMPÁTICO
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ABNT
BASSI, Gabriel S et al. Spinal GABA-B receptor modulates neutrophil recruitment to the knee joint in zymosan-induced arthritis. Naunyn-Schmiedeberg's Archives of Pharmacology, v. 389, n. 8, p. 851-861, 2016Tradução . . Disponível em: https://doi.org/10.1007/s00210-016-1248-0. Acesso em: 31 out. 2024.
APA
Bassi, G. S., Malvar, D. do C., Cunha, T. M., Cunha, F. de Q., & Kanashiro, A. (2016). Spinal GABA-B receptor modulates neutrophil recruitment to the knee joint in zymosan-induced arthritis. Naunyn-Schmiedeberg's Archives of Pharmacology, 389( 8), 851-861. doi:10.1007/s00210-016-1248-0
NLM
Bassi GS, Malvar D do C, Cunha TM, Cunha F de Q, Kanashiro A. Spinal GABA-B receptor modulates neutrophil recruitment to the knee joint in zymosan-induced arthritis [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2016 ; 389( 8): 851-861.[citado 2024 out. 31 ] Available from: https://doi.org/10.1007/s00210-016-1248-0
Vancouver
Bassi GS, Malvar D do C, Cunha TM, Cunha F de Q, Kanashiro A. Spinal GABA-B receptor modulates neutrophil recruitment to the knee joint in zymosan-induced arthritis [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2016 ; 389( 8): 851-861.[citado 2024 out. 31 ] Available from: https://doi.org/10.1007/s00210-016-1248-0
Artigo de periodico
Fructose 1,6-bisphosphate, a high-energy intermediate of glycolysis, attenuates experimental arthritis by activating anti-inflammatory adenosinergic pathway (2015)
Veras, Flávio P; Peres, Raphael S; Saraiva, André L. L; Pinto, Larissa G; Louzada Júnior, Paulo; Cunha, Thiago Mattar; Paschoal, Jonas Augusto Rizzato; Cunha, Fernando de Queiroz; Alves Filho, José Carlos Farias
Source: Scientific Reports. Unidades: FMRP, FCFRP
Subjects: ADENOSINA, GLICOSE, INFLAMAÇÃO, ARTRITE REUMATOIDE
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ABNT
VERAS, Flávio P et al. Fructose 1,6-bisphosphate, a high-energy intermediate of glycolysis, attenuates experimental arthritis by activating anti-inflammatory adenosinergic pathway. Scientific Reports, v. 5, 2015Tradução . . Disponível em: https://doi.org/10.1038/srep15171. Acesso em: 31 out. 2024.
APA
Veras, F. P., Peres, R. S., Saraiva, A. L. L., Pinto, L. G., Louzada Júnior, P., Cunha, T. M., et al. (2015). Fructose 1,6-bisphosphate, a high-energy intermediate of glycolysis, attenuates experimental arthritis by activating anti-inflammatory adenosinergic pathway. Scientific Reports, 5. doi:10.1038/srep15171
NLM
Veras FP, Peres RS, Saraiva ALL, Pinto LG, Louzada Júnior P, Cunha TM, Paschoal JAR, Cunha F de Q, Alves Filho JCF. Fructose 1,6-bisphosphate, a high-energy intermediate of glycolysis, attenuates experimental arthritis by activating anti-inflammatory adenosinergic pathway [Internet]. Scientific Reports. 2015 ; 5[citado 2024 out. 31 ] Available from: https://doi.org/10.1038/srep15171
Vancouver
Veras FP, Peres RS, Saraiva ALL, Pinto LG, Louzada Júnior P, Cunha TM, Paschoal JAR, Cunha F de Q, Alves Filho JCF. Fructose 1,6-bisphosphate, a high-energy intermediate of glycolysis, attenuates experimental arthritis by activating anti-inflammatory adenosinergic pathway [Internet]. Scientific Reports. 2015 ; 5[citado 2024 out. 31 ] Available from: https://doi.org/10.1038/srep15171
Artigo de periodico
Anti-inflammatory and anti-nociceptive activity of ruthenium complexes with isonicotinic and nicotinic acids (Niacin) as ligands (2015)
Freitas, Cristina S; Roveda Júnior, Antonio Carlos; Truzzi, Daniela Ramos ; Garcia, André Castilho; Cunha, Thiago Mattar; Cunha, Fernando Queiróz; Franco, Douglas Wagner
Source: Journal of Medicinal Chemistry. Unidades: FMRP, IQSC
Subjects: ANTI-INFLAMATÓRIOS NÃO ESTEROIDES, QUÍMICA
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ABNT
FREITAS, Cristina S et al. Anti-inflammatory and anti-nociceptive activity of ruthenium complexes with isonicotinic and nicotinic acids (Niacin) as ligands. Journal of Medicinal Chemistry, 2015Tradução . . Disponível em: https://doi.org/10.1021/acs.jmedchem.5b00133. Acesso em: 31 out. 2024.
APA
Freitas, C. S., Roveda Júnior, A. C., Truzzi, D. R., Garcia, A. C., Cunha, T. M., Cunha, F. Q., & Franco, D. W. (2015). Anti-inflammatory and anti-nociceptive activity of ruthenium complexes with isonicotinic and nicotinic acids (Niacin) as ligands. Journal of Medicinal Chemistry. doi:10.1021/acs.jmedchem.5b00133
NLM
Freitas CS, Roveda Júnior AC, Truzzi DR, Garcia AC, Cunha TM, Cunha FQ, Franco DW. Anti-inflammatory and anti-nociceptive activity of ruthenium complexes with isonicotinic and nicotinic acids (Niacin) as ligands [Internet]. Journal of Medicinal Chemistry. 2015 ;[citado 2024 out. 31 ] Available from: https://doi.org/10.1021/acs.jmedchem.5b00133
Vancouver
Freitas CS, Roveda Júnior AC, Truzzi DR, Garcia AC, Cunha TM, Cunha FQ, Franco DW. Anti-inflammatory and anti-nociceptive activity of ruthenium complexes with isonicotinic and nicotinic acids (Niacin) as ligands [Internet]. Journal of Medicinal Chemistry. 2015 ;[citado 2024 out. 31 ] Available from: https://doi.org/10.1021/acs.jmedchem.5b00133
Artigo de periodico
Inhibition of peripheral anion exchanger 3 decreases formalin-induced pain (2014)
Barragán-Iglesias, Paulino; Rocha-González, Héctor I; Pineda-Farias , Jorge Baruch; Murbartián, Janet; Godínez-Chaparro, Beatriz; Reinach, Peter S; Cunha, Thiago Mattar; Cunha, Fernando de Queiroz; Granados-Soto, Vinicio
Source: European Journal of Pharmacology. Unidade: FMRP
Subjects: GÂNGLIOS ESPINHAIS DE ANIMAL (EFEITOS DE DROGAS), EXPRESSÃO GÊNICA, NOCICEPTORES, DOR (INDUZIDO QUIMICAMENTE)
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ABNT
BARRAGÁN-IGLESIAS, Paulino et al. Inhibition of peripheral anion exchanger 3 decreases formalin-induced pain. European Journal of Pharmacology, v. 738, p. 91-100, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2014.05.029. Acesso em: 31 out. 2024.
APA
Barragán-Iglesias, P., Rocha-González, H. I., Pineda-Farias , J. B., Murbartián, J., Godínez-Chaparro, B., Reinach, P. S., et al. (2014). Inhibition of peripheral anion exchanger 3 decreases formalin-induced pain. European Journal of Pharmacology, 738, 91-100. doi:10.1016/j.ejphar.2014.05.029
NLM
Barragán-Iglesias P, Rocha-González HI, Pineda-Farias JB, Murbartián J, Godínez-Chaparro B, Reinach PS, Cunha TM, Cunha F de Q, Granados-Soto V. Inhibition of peripheral anion exchanger 3 decreases formalin-induced pain [Internet]. European Journal of Pharmacology. 2014 ; 738 91-100.[citado 2024 out. 31 ] Available from: https://doi.org/10.1016/j.ejphar.2014.05.029
Vancouver
Barragán-Iglesias P, Rocha-González HI, Pineda-Farias JB, Murbartián J, Godínez-Chaparro B, Reinach PS, Cunha TM, Cunha F de Q, Granados-Soto V. Inhibition of peripheral anion exchanger 3 decreases formalin-induced pain [Internet]. European Journal of Pharmacology. 2014 ; 738 91-100.[citado 2024 out. 31 ] Available from: https://doi.org/10.1016/j.ejphar.2014.05.029
Artigo de periodico
The role of PAF/PAFR signaling in zymosan-induced articular inflammatory hyperalgesia (2013)
Guerrero, Ana T.; Zaperlon, Ana C; Vieira, Silvio M; Pinto, Larissa G; Ferreira, Sérgio Henrique; Cunha, Fernando de Queiroz; Verri Junior, Waldiceu A; Cunha, Thiago Mattar
Source: Naunyn-Schmiedeberg's Archives of Pharmacology. Unidade: FMRP
Subjects: DOR, ARTICULAÇÕES, NEUTRÓFILOS (METABOLISMO), DOENÇAS IMUNOLÓGICAS, MODELOS ANIMAIS DE DOENÇAS
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ABNT
GUERRERO, Ana T. et al. The role of PAF/PAFR signaling in zymosan-induced articular inflammatory hyperalgesia. Naunyn-Schmiedeberg's Archives of Pharmacology, v. 386, n. 1, p. 51-59, 2013Tradução . . Disponível em: https://doi.org/10.1007/s00210-012-0813-4. Acesso em: 31 out. 2024.
APA
Guerrero, A. T., Zaperlon, A. C., Vieira, S. M., Pinto, L. G., Ferreira, S. H., Cunha, F. de Q., et al. (2013). The role of PAF/PAFR signaling in zymosan-induced articular inflammatory hyperalgesia. Naunyn-Schmiedeberg's Archives of Pharmacology, 386( 1), 51-59. doi:10.1007/s00210-012-0813-4
NLM
Guerrero AT, Zaperlon AC, Vieira SM, Pinto LG, Ferreira SH, Cunha F de Q, Verri Junior WA, Cunha TM. The role of PAF/PAFR signaling in zymosan-induced articular inflammatory hyperalgesia [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2013 ; 386( 1): 51-59.[citado 2024 out. 31 ] Available from: https://doi.org/10.1007/s00210-012-0813-4
Vancouver
Guerrero AT, Zaperlon AC, Vieira SM, Pinto LG, Ferreira SH, Cunha F de Q, Verri Junior WA, Cunha TM. The role of PAF/PAFR signaling in zymosan-induced articular inflammatory hyperalgesia [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2013 ; 386( 1): 51-59.[citado 2024 out. 31 ] Available from: https://doi.org/10.1007/s00210-012-0813-4
Artigo de periodico
NDP-MSH inhibits neutrophil migration through nicotinic and adrenergic receptors in experimental peritonitis (2013)
Figueiredo, Jozi; Ferreira, Ana Elisa; Silva, Rangel Leal; Ulloa, Luís; Grieco, Paolo; Cunha, Thiago Mattar; Ferreira, Sérgio Henrique; Cunha, Fernando de Queiroz; Kanashiro, Alexandre
Source: Naunyn-Schmiedeberg's Archives of Pharmacology. Unidade: FMRP
Subjects: NEUTRÓFILOS (EFEITOS DE DROGAS), RECEPTORES ADRENÉRGICOS, NEUROIMUNOMODULAÇÃO, PERITONITE
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ABNT
FIGUEIREDO, Jozi et al. NDP-MSH inhibits neutrophil migration through nicotinic and adrenergic receptors in experimental peritonitis. Naunyn-Schmiedeberg's Archives of Pharmacology, v. 386, n. 4, p. 311-318, 2013Tradução . . Disponível em: https://doi.org/10.1007/s00210-013-0834-7. Acesso em: 31 out. 2024.
APA
Figueiredo, J., Ferreira, A. E., Silva, R. L., Ulloa, L., Grieco, P., Cunha, T. M., et al. (2013). NDP-MSH inhibits neutrophil migration through nicotinic and adrenergic receptors in experimental peritonitis. Naunyn-Schmiedeberg's Archives of Pharmacology, 386( 4), 311-318. doi:10.1007/s00210-013-0834-7
NLM
Figueiredo J, Ferreira AE, Silva RL, Ulloa L, Grieco P, Cunha TM, Ferreira SH, Cunha F de Q, Kanashiro A. NDP-MSH inhibits neutrophil migration through nicotinic and adrenergic receptors in experimental peritonitis [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2013 ; 386( 4): 311-318.[citado 2024 out. 31 ] Available from: https://doi.org/10.1007/s00210-013-0834-7
Vancouver
Figueiredo J, Ferreira AE, Silva RL, Ulloa L, Grieco P, Cunha TM, Ferreira SH, Cunha F de Q, Kanashiro A. NDP-MSH inhibits neutrophil migration through nicotinic and adrenergic receptors in experimental peritonitis [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2013 ; 386( 4): 311-318.[citado 2024 out. 31 ] Available from: https://doi.org/10.1007/s00210-013-0834-7
Artigo de periodico
Inhibition of neutrophil migration by hemopexin leads to increased mortality due to sepsis in mice (2011)
Spiller, Fernando; Costa, Carlotta; Souto, Fabrício Oliveira; Vinchi, Francesca; Mestriner, Fabiola Leslie Antunes Cardoso; Laure, Hélen Julie; Alves Filho, José C.; Freitas, Andressa; Rosa, José César; Ferreira, Sérgio Henrique; Altruda, Fiorella; Hirsch, Emilio; Greene, Lewis Joel; Tolosano, Emanuela; Cunha, Fernando de Queiroz
Source: American Journal of Respiratory and Critical Care Medicine. Unidade: FMRP
Subjects: NEUTRÓFILOS, SEPSE
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ABNT
SPILLER, Fernando et al. Inhibition of neutrophil migration by hemopexin leads to increased mortality due to sepsis in mice. American Journal of Respiratory and Critical Care Medicine, v. 183, n. 7, p. 922-931, 2011Tradução . . Acesso em: 31 out. 2024.
APA
Spiller, F., Costa, C., Souto, F. O., Vinchi, F., Mestriner, F. L. A. C., Laure, H. J., et al. (2011). Inhibition of neutrophil migration by hemopexin leads to increased mortality due to sepsis in mice. American Journal of Respiratory and Critical Care Medicine, 183( 7), 922-931.
NLM
Spiller F, Costa C, Souto FO, Vinchi F, Mestriner FLAC, Laure HJ, Alves Filho JC, Freitas A, Rosa JC, Ferreira SH, Altruda F, Hirsch E, Greene LJ, Tolosano E, Cunha F de Q. Inhibition of neutrophil migration by hemopexin leads to increased mortality due to sepsis in mice. American Journal of Respiratory and Critical Care Medicine. 2011 ; 183( 7): 922-931.[citado 2024 out. 31 ]
Vancouver
Spiller F, Costa C, Souto FO, Vinchi F, Mestriner FLAC, Laure HJ, Alves Filho JC, Freitas A, Rosa JC, Ferreira SH, Altruda F, Hirsch E, Greene LJ, Tolosano E, Cunha F de Q. Inhibition of neutrophil migration by hemopexin leads to increased mortality due to sepsis in mice. American Journal of Respiratory and Critical Care Medicine. 2011 ; 183( 7): 922-931.[citado 2024 out. 31 ]
Artigo de periodico
IL-33 induces neutrophil migration in rheumatoid arthrits and is a target of anti-TNF therapy (2010)
Verri Júnior, Waldiceu Aparecido; Souto, Fabrício Oliveira; Teixeira, Mauro Martins; Silva, João Santana da; Mclnnes, Iain B.; Ferreira, Sérgio Henrique; Louzada Junior, Paulo; Liew, Foo Y.; Cunha, Fernando de Queiroz; Vieira, Sílvio Manfredo; Almeida, Sérgio C. L.; Fukada, Sandra Yasuyo; Xu, Damo; Alves-Filho, José Carlos; Cunha, Thiago Mattar; Guerrero, Ana Tereza Gomes; Guimarães, Rafaela B. Mattos; Oliveira, Fabíola Reis de
Source: Annals of the Rheumatic Diseses. Unidade: FMRP
Subjects: ARTRITE REUMATOIDE, NEUTRÓFILOS
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ABNT
VERRI JÚNIOR, Waldiceu Aparecido et al. IL-33 induces neutrophil migration in rheumatoid arthrits and is a target of anti-TNF therapy. Annals of the Rheumatic Diseses, v. 69, n. 9, p. 1697-1703, 2010Tradução . . Disponível em: https://doi.org/10.1136/ard.2009.122655. Acesso em: 31 out. 2024.
APA
Verri Júnior, W. A., Souto, F. O., Teixeira, M. M., Silva, J. S. da, Mclnnes, I. B., Ferreira, S. H., et al. (2010). IL-33 induces neutrophil migration in rheumatoid arthrits and is a target of anti-TNF therapy. Annals of the Rheumatic Diseses, 69( 9), 1697-1703. doi:10.1136/ard.2009.122655
NLM
Verri Júnior WA, Souto FO, Teixeira MM, Silva JS da, Mclnnes IB, Ferreira SH, Louzada Junior P, Liew FY, Cunha F de Q, Vieira SM, Almeida SCL, Fukada SY, Xu D, Alves-Filho JC, Cunha TM, Guerrero ATG, Guimarães RBM, Oliveira FR de. IL-33 induces neutrophil migration in rheumatoid arthrits and is a target of anti-TNF therapy [Internet]. Annals of the Rheumatic Diseses. 2010 ; 69( 9): 1697-1703.[citado 2024 out. 31 ] Available from: https://doi.org/10.1136/ard.2009.122655
Vancouver
Verri Júnior WA, Souto FO, Teixeira MM, Silva JS da, Mclnnes IB, Ferreira SH, Louzada Junior P, Liew FY, Cunha F de Q, Vieira SM, Almeida SCL, Fukada SY, Xu D, Alves-Filho JC, Cunha TM, Guerrero ATG, Guimarães RBM, Oliveira FR de. IL-33 induces neutrophil migration in rheumatoid arthrits and is a target of anti-TNF therapy [Internet]. Annals of the Rheumatic Diseses. 2010 ; 69( 9): 1697-1703.[citado 2024 out. 31 ] Available from: https://doi.org/10.1136/ard.2009.122655
Artigo de periodico
Interleukin-17 and kidney allograft outcome (2009)
Crispim, Janaína Cristiana de Oliveira; Palomino, Gustavo Martelli; Rassi, Diane Meyre; Costa, Roberto Silva; Saber, L. T.; Cunha, Fernando de Queiroz; Donadi, Eduardo Antônio; Grespan, Renata
Source: Transplantation Proceedings. Unidade: FMRP
Subjects: TRANSPLANTE DE RIM, FISIOPATOLOGIA, CITOCINAS
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ABNT
CRISPIM, Janaína Cristiana de Oliveira et al. Interleukin-17 and kidney allograft outcome. Transplantation Proceedings, v. 41, n. 5, p. 1562-1564, 2009Tradução . . Acesso em: 31 out. 2024.
APA
Crispim, J. C. de O., Palomino, G. M., Rassi, D. M., Costa, R. S., Saber, L. T., Cunha, F. de Q., et al. (2009). Interleukin-17 and kidney allograft outcome. Transplantation Proceedings, 41( 5), 1562-1564.
NLM
Crispim JC de O, Palomino GM, Rassi DM, Costa RS, Saber LT, Cunha F de Q, Donadi EA, Grespan R. Interleukin-17 and kidney allograft outcome. Transplantation Proceedings. 2009 ; 41( 5): 1562-1564.[citado 2024 out. 31 ]
Vancouver
Crispim JC de O, Palomino GM, Rassi DM, Costa RS, Saber LT, Cunha F de Q, Donadi EA, Grespan R. Interleukin-17 and kidney allograft outcome. Transplantation Proceedings. 2009 ; 41( 5): 1562-1564.[citado 2024 out. 31 ]
Artigo de periodico
Cannabidiol decreases bone resorption by inhibiting RANK/RANKL expression and pro-inflammatory cytrokines during experimental periodontitis in rats (2009)
Napimoga, Marcelo H.; Benatti, Bruno B.; Lima, Flávia Oliveira de; Alves, Polyanna Miranda; Campos, Alline Cristina de; Pena-dos-Santos, Diego R.; Severino, Fernando P.; Cunha, Fernando de Queiroz; Guimarães, Francisco Silveira
Source: International Immunopharmacology. Unidade: FMRP
Subjects: DOENÇAS PERIODONTAIS, INFLAMAÇÃO, REABSORÇÃO ÓSSEA ALVEOLAR
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ABNT
NAPIMOGA, Marcelo H. et al. Cannabidiol decreases bone resorption by inhibiting RANK/RANKL expression and pro-inflammatory cytrokines during experimental periodontitis in rats. International Immunopharmacology, v. 9, n. 2, p. 216-222, 2009Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2008.11.010. Acesso em: 31 out. 2024.
APA
Napimoga, M. H., Benatti, B. B., Lima, F. O. de, Alves, P. M., Campos, A. C. de, Pena-dos-Santos, D. R., et al. (2009). Cannabidiol decreases bone resorption by inhibiting RANK/RANKL expression and pro-inflammatory cytrokines during experimental periodontitis in rats. International Immunopharmacology, 9( 2), 216-222. doi:10.1016/j.intimp.2008.11.010
NLM
Napimoga MH, Benatti BB, Lima FO de, Alves PM, Campos AC de, Pena-dos-Santos DR, Severino FP, Cunha F de Q, Guimarães FS. Cannabidiol decreases bone resorption by inhibiting RANK/RANKL expression and pro-inflammatory cytrokines during experimental periodontitis in rats [Internet]. International Immunopharmacology. 2009 ; 9( 2): 216-222.[citado 2024 out. 31 ] Available from: https://doi.org/10.1016/j.intimp.2008.11.010
Vancouver
Napimoga MH, Benatti BB, Lima FO de, Alves PM, Campos AC de, Pena-dos-Santos DR, Severino FP, Cunha F de Q, Guimarães FS. Cannabidiol decreases bone resorption by inhibiting RANK/RANKL expression and pro-inflammatory cytrokines during experimental periodontitis in rats [Internet]. International Immunopharmacology. 2009 ; 9( 2): 216-222.[citado 2024 out. 31 ] Available from: https://doi.org/10.1016/j.intimp.2008.11.010
Artigo de periodico
IL-4 regulates susceptibility to intestinal inflammation in murine food allergy (2009)
Cardoso, Cristina Ribeiro; Provinciatto, Pauline Rossetti; Godoi, Dannielle Fernandes; Ferreira, Beatriz Rossetti ; Teixeira, Gerlinde; Rossi, Marcos Antonio; Cunha, Fernando de Queiroz; Silva, João Santana da
Source: American Journal of Physiology. Unidades: EERP, FMRP
Subjects: HIPERSENSIBILIDADE ALIMENTAR, INTESTINOS, INFLAMAÇÃO
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ABNT
CARDOSO, Cristina Ribeiro et al. IL-4 regulates susceptibility to intestinal inflammation in murine food allergy. American Journal of Physiology, v. 296, n. 3, p. G593-G600, 2009Tradução . . Disponível em: https://doi.org/10.1152/ajpgi.90431.2008. Acesso em: 31 out. 2024.
APA
Cardoso, C. R., Provinciatto, P. R., Godoi, D. F., Ferreira, B. R., Teixeira, G., Rossi, M. A., et al. (2009). IL-4 regulates susceptibility to intestinal inflammation in murine food allergy. American Journal of Physiology, 296( 3), G593-G600. doi:10.1152/ajpgi.90431.2008
NLM
Cardoso CR, Provinciatto PR, Godoi DF, Ferreira BR, Teixeira G, Rossi MA, Cunha F de Q, Silva JS da. IL-4 regulates susceptibility to intestinal inflammation in murine food allergy [Internet]. American Journal of Physiology. 2009 ; 296( 3): G593-G600.[citado 2024 out. 31 ] Available from: https://doi.org/10.1152/ajpgi.90431.2008
Vancouver
Cardoso CR, Provinciatto PR, Godoi DF, Ferreira BR, Teixeira G, Rossi MA, Cunha F de Q, Silva JS da. IL-4 regulates susceptibility to intestinal inflammation in murine food allergy [Internet]. American Journal of Physiology. 2009 ; 296( 3): G593-G600.[citado 2024 out. 31 ] Available from: https://doi.org/10.1152/ajpgi.90431.2008
Artigo de periodico
Prostaglandin mediates IL-23/IL-17-induced neutrophil migration in inflammation by inhibiting IL-12 and IFN'upsilon' production (2009)
Lemos, Henrique de Paula; Grespan, Renata; Vieira, Sílvio Manfredo; Cunha, Thiago Mattar; Verri Júnior, Waldiceu Aparecido; Fernandes, Karla Simone S.; Souto, Fabrício Oliveira; Mclnnes, Iain B.; Ferreira, Sérgio Henrique; Liew, Foo Y.; Cunha, Fernando de Queiroz
Source: Proceedings of the Nacional Academy of Sciences. Unidade: FMRP
Subjects: ARTRITE REUMATOIDE, CITOCINAS, NEUTRÓFILOS
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ABNT
LEMOS, Henrique de Paula et al. Prostaglandin mediates IL-23/IL-17-induced neutrophil migration in inflammation by inhibiting IL-12 and IFN'upsilon' production. Proceedings of the Nacional Academy of Sciences, v. 106, n. 14, p. 5954-5959, 2009Tradução . . Disponível em: https://doi.org/10.1073/pnas.0812782106. Acesso em: 31 out. 2024.
APA
Lemos, H. de P., Grespan, R., Vieira, S. M., Cunha, T. M., Verri Júnior, W. A., Fernandes, K. S. S., et al. (2009). Prostaglandin mediates IL-23/IL-17-induced neutrophil migration in inflammation by inhibiting IL-12 and IFN'upsilon' production. Proceedings of the Nacional Academy of Sciences, 106( 14), 5954-5959. doi:10.1073/pnas.0812782106
NLM
Lemos H de P, Grespan R, Vieira SM, Cunha TM, Verri Júnior WA, Fernandes KSS, Souto FO, Mclnnes IB, Ferreira SH, Liew FY, Cunha F de Q. Prostaglandin mediates IL-23/IL-17-induced neutrophil migration in inflammation by inhibiting IL-12 and IFN'upsilon' production [Internet]. Proceedings of the Nacional Academy of Sciences. 2009 ; 106( 14): 5954-5959.[citado 2024 out. 31 ] Available from: https://doi.org/10.1073/pnas.0812782106
Vancouver
Lemos H de P, Grespan R, Vieira SM, Cunha TM, Verri Júnior WA, Fernandes KSS, Souto FO, Mclnnes IB, Ferreira SH, Liew FY, Cunha F de Q. Prostaglandin mediates IL-23/IL-17-induced neutrophil migration in inflammation by inhibiting IL-12 and IFN'upsilon' production [Internet]. Proceedings of the Nacional Academy of Sciences. 2009 ; 106( 14): 5954-5959.[citado 2024 out. 31 ] Available from: https://doi.org/10.1073/pnas.0812782106
Artigo de periodico
Quercetin reduces inflammatory pain: inhibition of oxidative stress and cytokine production (2009)
Valerio, Daniel Augusto; Georgetti, Sandra R.; Fonseca, Maria Jose Vieira; Ferreira, Sérgio Henrique; Cunha, Fernando de Queiroz; Verri Júnior, Waldiceu Aparecido; Magro, Danilo A.; Casagrande, Rúbia; Cunha, Thiago Mattar; Vicentini, Fabiana Testa Moura de Carvalho; Vieira, Sílvio Manfredo
Source: Journal of Natural Products. Unidades: FCFRP, FMRP
Subjects: FLAVONÓIDES, DOR, INFLAMAÇÃO, ESTRESSE OXIDATIVO, CITOCINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VALERIO, Daniel Augusto et al. Quercetin reduces inflammatory pain: inhibition of oxidative stress and cytokine production. Journal of Natural Products, v. 72, n. 11, p. 1975-1979, 2009Tradução . . Acesso em: 31 out. 2024.
APA
Valerio, D. A., Georgetti, S. R., Fonseca, M. J. V., Ferreira, S. H., Cunha, F. de Q., Verri Júnior, W. A., et al. (2009). Quercetin reduces inflammatory pain: inhibition of oxidative stress and cytokine production. Journal of Natural Products, 72( 11), 1975-1979.
NLM
Valerio DA, Georgetti SR, Fonseca MJV, Ferreira SH, Cunha F de Q, Verri Júnior WA, Magro DA, Casagrande R, Cunha TM, Vicentini FTM de C, Vieira SM. Quercetin reduces inflammatory pain: inhibition of oxidative stress and cytokine production. Journal of Natural Products. 2009 ; 72( 11): 1975-1979.[citado 2024 out. 31 ]
Vancouver
Valerio DA, Georgetti SR, Fonseca MJV, Ferreira SH, Cunha F de Q, Verri Júnior WA, Magro DA, Casagrande R, Cunha TM, Vicentini FTM de C, Vieira SM. Quercetin reduces inflammatory pain: inhibition of oxidative stress and cytokine production. Journal of Natural Products. 2009 ; 72( 11): 1975-1979.[citado 2024 out. 31 ]
Artigo de periodico
Regulation of chemokine receptor by toll-like receptor 2 is critical to neutrophil migration and resistance to polymicrobial sepsis (2009)
Alves-Filho, José Carlos; Freitas, Andressa; Souto, Fabrício Oliveira; Spiller, Fernando; Paula Neto, Heitor; Silva, João Santana da; Gazzinelli, Ricardo T.; Teixeira, Mauro Martins; Ferreira, Sérgio Henrique; Cunha, Fernando de Queiroz
Source: Proceedings of the National Academy of Sciences. Unidade: FMRP
Subjects: NEUTRÓFILOS, SEPSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ALVES-FILHO, José Carlos et al. Regulation of chemokine receptor by toll-like receptor 2 is critical to neutrophil migration and resistance to polymicrobial sepsis. Proceedings of the National Academy of Sciences, v. 106, n. 10, p. 4018-4023, 2009Tradução . . Disponível em: https://doi.org/10.1073/pnas.0900196106. Acesso em: 31 out. 2024.
APA
Alves-Filho, J. C., Freitas, A., Souto, F. O., Spiller, F., Paula Neto, H., Silva, J. S. da, et al. (2009). Regulation of chemokine receptor by toll-like receptor 2 is critical to neutrophil migration and resistance to polymicrobial sepsis. Proceedings of the National Academy of Sciences, 106( 10), 4018-4023. doi:10.1073/pnas.0900196106
NLM
Alves-Filho JC, Freitas A, Souto FO, Spiller F, Paula Neto H, Silva JS da, Gazzinelli RT, Teixeira MM, Ferreira SH, Cunha F de Q. Regulation of chemokine receptor by toll-like receptor 2 is critical to neutrophil migration and resistance to polymicrobial sepsis [Internet]. Proceedings of the National Academy of Sciences. 2009 ; 106( 10): 4018-4023.[citado 2024 out. 31 ] Available from: https://doi.org/10.1073/pnas.0900196106
Vancouver
Alves-Filho JC, Freitas A, Souto FO, Spiller F, Paula Neto H, Silva JS da, Gazzinelli RT, Teixeira MM, Ferreira SH, Cunha F de Q. Regulation of chemokine receptor by toll-like receptor 2 is critical to neutrophil migration and resistance to polymicrobial sepsis [Internet]. Proceedings of the National Academy of Sciences. 2009 ; 106( 10): 4018-4023.[citado 2024 out. 31 ] Available from: https://doi.org/10.1073/pnas.0900196106
Artigo de periodico
PPAR-'úpsilon' agonist rosiglitazone prevents inflammatory periodontal bone loss by inhibiting osteoclastogenesis (2009)
Hassumi, Márcio Y.; Silva Filho, Vilmar J.; Campos Júnior, Jozafá; Vieira, Sílvio Manfredo; Alves, Polyanna Miranda; Alves, José Bento; Kawai, Toshihisa; Gonçalves, Reginaldo Bruno; Napimoga, Marcelo H.; Cunha, Fernando de Queiroz
Source: International Immunopharmacology. Unidade: FMRP
Subjects: DOENÇAS PERIODONTAIS, REABSORÇÃO ÓSSEA ALVEOLAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
HASSUMI, Márcio Y. et al. PPAR-'úpsilon' agonist rosiglitazone prevents inflammatory periodontal bone loss by inhibiting osteoclastogenesis. International Immunopharmacology, v. 9, n. 10, p. 1150-1158, 2009Tradução . . Acesso em: 31 out. 2024.
APA
Hassumi, M. Y., Silva Filho, V. J., Campos Júnior, J., Vieira, S. M., Alves, P. M., Alves, J. B., et al. (2009). PPAR-'úpsilon' agonist rosiglitazone prevents inflammatory periodontal bone loss by inhibiting osteoclastogenesis. International Immunopharmacology, 9( 10), 1150-1158.
NLM
Hassumi MY, Silva Filho VJ, Campos Júnior J, Vieira SM, Alves PM, Alves JB, Kawai T, Gonçalves RB, Napimoga MH, Cunha F de Q. PPAR-'úpsilon' agonist rosiglitazone prevents inflammatory periodontal bone loss by inhibiting osteoclastogenesis. International Immunopharmacology. 2009 ; 9( 10): 1150-1158.[citado 2024 out. 31 ]
Vancouver
Hassumi MY, Silva Filho VJ, Campos Júnior J, Vieira SM, Alves PM, Alves JB, Kawai T, Gonçalves RB, Napimoga MH, Cunha F de Q. PPAR-'úpsilon' agonist rosiglitazone prevents inflammatory periodontal bone loss by inhibiting osteoclastogenesis. International Immunopharmacology. 2009 ; 9( 10): 1150-1158.[citado 2024 out. 31 ]
Artigo de periodico
Adjunctive benefits of systemic etoricoxib in non-surgical treatment of aggressive periodontitis: short-term evaluation (2008)
Azoubel, Maria Cecília F.; Sarmento, Viviane Almeida; Cangussú, Virna; Azoubel, Eduardo; Bittencourt, Sandro; Cunha, Fernando Queiróz; Ribeiro, Ronaldo de Albuquerque; Brito, Gerly Anne C.
Source: Journal of Periodontology. Unidade: FMRP
Subjects: PERIODONTITE, ANTI-INFLAMATÓRIOS, RASPAGEM DENTÁRIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
AZOUBEL, Maria Cecília F. et al. Adjunctive benefits of systemic etoricoxib in non-surgical treatment of aggressive periodontitis: short-term evaluation. Journal of Periodontology, v. 79, n. 9, p. 1719-1725, 2008Tradução . . Acesso em: 31 out. 2024.
APA
Azoubel, M. C. F., Sarmento, V. A., Cangussú, V., Azoubel, E., Bittencourt, S., Cunha, F. Q., et al. (2008). Adjunctive benefits of systemic etoricoxib in non-surgical treatment of aggressive periodontitis: short-term evaluation. Journal of Periodontology, 79( 9), 1719-1725.
NLM
Azoubel MCF, Sarmento VA, Cangussú V, Azoubel E, Bittencourt S, Cunha FQ, Ribeiro R de A, Brito GAC. Adjunctive benefits of systemic etoricoxib in non-surgical treatment of aggressive periodontitis: short-term evaluation. Journal of Periodontology. 2008 ; 79( 9): 1719-1725.[citado 2024 out. 31 ]
Vancouver
Azoubel MCF, Sarmento VA, Cangussú V, Azoubel E, Bittencourt S, Cunha FQ, Ribeiro R de A, Brito GAC. Adjunctive benefits of systemic etoricoxib in non-surgical treatment of aggressive periodontitis: short-term evaluation. Journal of Periodontology. 2008 ; 79( 9): 1719-1725.[citado 2024 out. 31 ]
Artigo de periodico
Detrimental role of endogenous nitric oxide in host defence against Sporothrix schenckii (2008)
Fernandes, Karla Simone S.; Helal Neto, Edward; Brito, Marcelly M. S.; Silva, João Santana da; Cunha, Fernando de Queiroz; Barja-Fidalga, Christina
Source: Immunology. Unidade: FMRP
Subjects: ÓXIDO NÍTRICO, FUNGOS, MACRÓFAGOS, FAGOCITOSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FERNANDES, Karla Simone S. et al. Detrimental role of endogenous nitric oxide in host defence against Sporothrix schenckii. Immunology, v. 123, n. 4, p. 469-479, 2008Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2567.2007.02712.x. Acesso em: 31 out. 2024.
APA
Fernandes, K. S. S., Helal Neto, E., Brito, M. M. S., Silva, J. S. da, Cunha, F. de Q., & Barja-Fidalga, C. (2008). Detrimental role of endogenous nitric oxide in host defence against Sporothrix schenckii. Immunology, 123( 4), 469-479. doi:10.1111/j.1365-2567.2007.02712.x
NLM
Fernandes KSS, Helal Neto E, Brito MMS, Silva JS da, Cunha F de Q, Barja-Fidalga C. Detrimental role of endogenous nitric oxide in host defence against Sporothrix schenckii [Internet]. Immunology. 2008 ; 123( 4): 469-479.[citado 2024 out. 31 ] Available from: https://doi.org/10.1111/j.1365-2567.2007.02712.x
Vancouver
Fernandes KSS, Helal Neto E, Brito MMS, Silva JS da, Cunha F de Q, Barja-Fidalga C. Detrimental role of endogenous nitric oxide in host defence against Sporothrix schenckii [Internet]. Immunology. 2008 ; 123( 4): 469-479.[citado 2024 out. 31 ] Available from: https://doi.org/10.1111/j.1365-2567.2007.02712.x
Artigo de periodico
Treatment with DF 2162, a non-competitive allosteric inhibitor of CXCR1/2, diminishes neutrophil influx and inflammatory hypernociception in mice (2008)
Cunha, T. M.; Barsante, M. M.; Guerrero, A. T.; Verri Júnior, Waldiceu Aparecido; Ferreira, Sérgio Henrique; Coelho, F. M.; Bertini, R.; Giacinto, C. Di; Allegretti, M.; Cunha, Fernando de Queiroz; Teixeira, M. M.
Source: British Journal of Pharmacology. Unidade: FMRP
Subjects: ARTRITE, DOR, NEUTRÓFILOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CUNHA, T. M. et al. Treatment with DF 2162, a non-competitive allosteric inhibitor of CXCR1/2, diminishes neutrophil influx and inflammatory hypernociception in mice. British Journal of Pharmacology, v. 154, n. 2, p. 460-470, 2008Tradução . . Disponível em: https://doi.org/10.1038/bjp.2008.94. Acesso em: 31 out. 2024.
APA
Cunha, T. M., Barsante, M. M., Guerrero, A. T., Verri Júnior, W. A., Ferreira, S. H., Coelho, F. M., et al. (2008). Treatment with DF 2162, a non-competitive allosteric inhibitor of CXCR1/2, diminishes neutrophil influx and inflammatory hypernociception in mice. British Journal of Pharmacology, 154( 2), 460-470. doi:10.1038/bjp.2008.94
NLM
Cunha TM, Barsante MM, Guerrero AT, Verri Júnior WA, Ferreira SH, Coelho FM, Bertini R, Giacinto CD, Allegretti M, Cunha F de Q, Teixeira MM. Treatment with DF 2162, a non-competitive allosteric inhibitor of CXCR1/2, diminishes neutrophil influx and inflammatory hypernociception in mice [Internet]. British Journal of Pharmacology. 2008 ; 154( 2): 460-470.[citado 2024 out. 31 ] Available from: https://doi.org/10.1038/bjp.2008.94
Vancouver
Cunha TM, Barsante MM, Guerrero AT, Verri Júnior WA, Ferreira SH, Coelho FM, Bertini R, Giacinto CD, Allegretti M, Cunha F de Q, Teixeira MM. Treatment with DF 2162, a non-competitive allosteric inhibitor of CXCR1/2, diminishes neutrophil influx and inflammatory hypernociception in mice [Internet]. British Journal of Pharmacology. 2008 ; 154( 2): 460-470.[citado 2024 out. 31 ] Available from: https://doi.org/10.1038/bjp.2008.94
Artigo de periodico
Commensal microbiota is fundamental for the development of inflammatory pain (2008)
Amatal, F. A.; Sachs, D.; Costa, V. V.; Fagundes, C. T.; Cisalpino, D.; Cunha, T. M.; Ferreira, Sérgio Henrique; Cunha, Fernando de Queiroz; Silva, T. A.; Nicoli, J. R.; Vieira, L. Q.; Souza, D. G.; Teixeira, M. M.
Source: Proceedings of the National Academy of Sciences. Unidade: FMRP
Subjects: CITOCINAS, DOR, NOCICEPTORES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
AMATAL, F. A. et al. Commensal microbiota is fundamental for the development of inflammatory pain. Proceedings of the National Academy of Sciences, v. 105, n. 6, p. 2193-2197, 2008Tradução . . Disponível em: https://doi.org/10.1073/pnas.0711891105. Acesso em: 31 out. 2024.
APA
Amatal, F. A., Sachs, D., Costa, V. V., Fagundes, C. T., Cisalpino, D., Cunha, T. M., et al. (2008). Commensal microbiota is fundamental for the development of inflammatory pain. Proceedings of the National Academy of Sciences, 105( 6), 2193-2197. doi:10.1073/pnas.0711891105
NLM
Amatal FA, Sachs D, Costa VV, Fagundes CT, Cisalpino D, Cunha TM, Ferreira SH, Cunha F de Q, Silva TA, Nicoli JR, Vieira LQ, Souza DG, Teixeira MM. Commensal microbiota is fundamental for the development of inflammatory pain [Internet]. Proceedings of the National Academy of Sciences. 2008 ; 105( 6): 2193-2197.[citado 2024 out. 31 ] Available from: https://doi.org/10.1073/pnas.0711891105
Vancouver
Amatal FA, Sachs D, Costa VV, Fagundes CT, Cisalpino D, Cunha TM, Ferreira SH, Cunha F de Q, Silva TA, Nicoli JR, Vieira LQ, Souza DG, Teixeira MM. Commensal microbiota is fundamental for the development of inflammatory pain [Internet]. Proceedings of the National Academy of Sciences. 2008 ; 105( 6): 2193-2197.[citado 2024 out. 31 ] Available from: https://doi.org/10.1073/pnas.0711891105

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