Fructose 1,6-bisphosphate, a high-energy intermediate of glycolysis, attenuates experimental arthritis by activating anti-inflammatory adenosinergic pathway (2015)
- Authors:
- USP affiliated authors: LOUZADA JÚNIOR, PAULO - FMRP ; CUNHA, THIAGO MATTAR - FMRP ; PASCHOAL, JONAS AUGUSTO RIZZATO - FCFRP ; CUNHA, FERNANDO DE QUEIROZ - FMRP ; ALVES FILHO, JOSÉ CARLOS FARIAS - FMRP
- Unidades: FMRP; FCFRP
- DOI: 10.1038/srep15171
- Subjects: ADENOSINA; GLICOSE; INFLAMAÇÃO; ARTRITE REUMATOIDE
- Language: Inglês
- Imprenta:
- Source:
- Título: Scientific Reports
- ISSN: 2045-2322
- Volume/Número/Paginação/Ano: v. 5, art. 15171, 2015
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
VERAS, Flávio P et al. Fructose 1,6-bisphosphate, a high-energy intermediate of glycolysis, attenuates experimental arthritis by activating anti-inflammatory adenosinergic pathway. Scientific Reports, v. 5, 2015Tradução . . Disponível em: https://doi.org/10.1038/srep15171. Acesso em: 19 fev. 2026. -
APA
Veras, F. P., Peres, R. S., Saraiva, A. L. L., Pinto, L. G., Louzada Júnior, P., Cunha, T. M., et al. (2015). Fructose 1,6-bisphosphate, a high-energy intermediate of glycolysis, attenuates experimental arthritis by activating anti-inflammatory adenosinergic pathway. Scientific Reports, 5. doi:10.1038/srep15171 -
NLM
Veras FP, Peres RS, Saraiva ALL, Pinto LG, Louzada Júnior P, Cunha TM, Paschoal JAR, Cunha F de Q, Alves Filho JCF. Fructose 1,6-bisphosphate, a high-energy intermediate of glycolysis, attenuates experimental arthritis by activating anti-inflammatory adenosinergic pathway [Internet]. Scientific Reports. 2015 ; 5[citado 2026 fev. 19 ] Available from: https://doi.org/10.1038/srep15171 -
Vancouver
Veras FP, Peres RS, Saraiva ALL, Pinto LG, Louzada Júnior P, Cunha TM, Paschoal JAR, Cunha F de Q, Alves Filho JCF. Fructose 1,6-bisphosphate, a high-energy intermediate of glycolysis, attenuates experimental arthritis by activating anti-inflammatory adenosinergic pathway [Internet]. Scientific Reports. 2015 ; 5[citado 2026 fev. 19 ] Available from: https://doi.org/10.1038/srep15171 - Smoking-induced aggravation of experimental arthritis is dependent of aryl hydrocarbon receptor activation in Th17 cells
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Informações sobre o DOI: 10.1038/srep15171 (Fonte: oaDOI API)
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