Smoking-induced aggravation of experimental arthritis is dependent of aryl hydrocarbon receptor activation in Th17 cells (2018)
- Authors:
- USP affiliated authors: CUNHA, THIAGO MATTAR - FMRP ; ALVES FILHO, JOSÉ CARLOS FARIAS - FMRP ; LOUZADA JÚNIOR, PAULO - FMRP ; CUNHA, FERNANDO DE QUEIROZ - FMRP
- Unidade: FMRP
- DOI: 10.1186/s13075-018-1609-9
- Subjects: ARTRITE REUMATOIDE; FUMANTES; INFLAMAÇÃO; HIDROCARBONETOS
- Keywords: Rheumatoid arthritis; Cigarette smoke; Inflammation; Polycyclic aromatic hydrocarbons; Th17
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Arthritis Research & Therapy
- ISSN: 1478-6362
- Volume/Número/Paginação/Ano: v.20, n.1, art. 119, 2018
- Status:
- Artigo publicado em periódico de acesso aberto (Gold Open Access)
- Versão do Documento:
- Versão publicada (Published version)
- Acessar versão aberta:
-
ABNT
TALBOT, Jhimmy et al. Smoking-induced aggravation of experimental arthritis is dependent of aryl hydrocarbon receptor activation in Th17 cells. Arthritis Research & Therapy, v. 20, n. 1, 2018Tradução . . Disponível em: https://doi.org/10.1186/s13075-018-1609-9. Acesso em: 07 maio 2026. -
APA
Talbot, J., Peres, R. S., Pinto, L. G., Oliveira, R. D. R., Lima, K. A., Donate, P. B., et al. (2018). Smoking-induced aggravation of experimental arthritis is dependent of aryl hydrocarbon receptor activation in Th17 cells. Arthritis Research & Therapy, 20( 1). doi:10.1186/s13075-018-1609-9 -
NLM
Talbot J, Peres RS, Pinto LG, Oliveira RDR, Lima KA, Donate PB, Silva JR, Ryffel B, Cunha TM, Alves Filho JCF, Liew FY, Louzada Júnior P, Cunha F de Q. Smoking-induced aggravation of experimental arthritis is dependent of aryl hydrocarbon receptor activation in Th17 cells [Internet]. Arthritis Research & Therapy. 2018 ;20( 1):[citado 2026 maio 07 ] Available from: https://doi.org/10.1186/s13075-018-1609-9 -
Vancouver
Talbot J, Peres RS, Pinto LG, Oliveira RDR, Lima KA, Donate PB, Silva JR, Ryffel B, Cunha TM, Alves Filho JCF, Liew FY, Louzada Júnior P, Cunha F de Q. Smoking-induced aggravation of experimental arthritis is dependent of aryl hydrocarbon receptor activation in Th17 cells [Internet]. Arthritis Research & Therapy. 2018 ;20( 1):[citado 2026 maio 07 ] Available from: https://doi.org/10.1186/s13075-018-1609-9 - Pyrrolidine dithiocarbamate inhibits superoxide anion-induced pain and inflammation in the paw skin and spinal cord by targeting NF-kB and oxidative stress
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