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Artigo de periodico
Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity (2021)
Nascimento, Daniele Carvalho ; Viacava, Paula Ramos; Ferreira, Raphael Gomes; Damaceno, Marina Alves; Piñeros, Annie Rocío Alvarez ; Melo, Paulo Henrique de; Donate, Paula Barbim ; Toller-Kawahisa, Juliana Escher; Zoppi, Daniel ; Veras, Flávio Protásio ; Peres, Raphael S. ; Silva, Luisa Menezes ; Caetité, Diego ; Oliveira, Antonio Edson Rocha; Castro, Icaro Maia Santos ; Kauffenstein, Gilles ; Nakaya, Helder Takashi Imoto ; Borges, Marcos de Carvalho ; Zamboni, Dario Simões ; Fonseca, Denise Morais da ; Paschoal, Jonas Augusto Rizzato ; Cunha, Thiago Mattar ; Quesniaux, Valerie ; Linden, Joel ; Cunha, Fernando Queiróz ; Ryffe, Bernhard; Alves-Filho, José Carlos
Source: Immunity. Unidades: FMRP, FCFRP, FCF, ICB, BIOINFORMÁTICA
Subjects: ADENOSINA, FÁRMACOS IMUNOSSUPRESSORES, SEPSE, CAMUNDONGOS, INTERLEUCINA 10, MACRÓFAGOS
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ABNT
NASCIMENTO, Daniele Carvalho et al. Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity. Immunity, v. 54, n. 9, p. 2024-2041, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.immuni.2021.08.005. Acesso em: 14 jun. 2024.
APA
Nascimento, D. C., Viacava, P. R., Ferreira, R. G., Damaceno, M. A., Piñeros, A. R. A., Melo, P. H. de, et al. (2021). Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity. Immunity, 54( 9), 2024-2041. doi:10.1016/j.immuni.2021.08.005
NLM
Nascimento DC, Viacava PR, Ferreira RG, Damaceno MA, Piñeros ARA, Melo PH de, Donate PB, Toller-Kawahisa JE, Zoppi D, Veras FP, Peres RS, Silva LM, Caetité D, Oliveira AER, Castro IMS, Kauffenstein G, Nakaya HTI, Borges M de C, Zamboni DS, Fonseca DM da, Paschoal JAR, Cunha TM, Quesniaux V, Linden J, Cunha FQ, Ryffe B, Alves-Filho JC. Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity [Internet]. Immunity. 2021 ; 54( 9): 2024-2041.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1016/j.immuni.2021.08.005
Vancouver
Nascimento DC, Viacava PR, Ferreira RG, Damaceno MA, Piñeros ARA, Melo PH de, Donate PB, Toller-Kawahisa JE, Zoppi D, Veras FP, Peres RS, Silva LM, Caetité D, Oliveira AER, Castro IMS, Kauffenstein G, Nakaya HTI, Borges M de C, Zamboni DS, Fonseca DM da, Paschoal JAR, Cunha TM, Quesniaux V, Linden J, Cunha FQ, Ryffe B, Alves-Filho JC. Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity [Internet]. Immunity. 2021 ; 54( 9): 2024-2041.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1016/j.immuni.2021.08.005
Artigo de periodico
Neuraminidase inhibitors rewire neutrophil function in vivo in murine sepsis and ex vivo in COVID-19 (2021)
Formiga, Rodrigo Oliveira; Amaral, Flávia Cardoso; Souza, Camila F.; Mendes, Daniel A. G. B.; Wanderley, Carlos Wagner de Souza; Lorenzini, Cristina B.; Santos, Adara A.; Antônia, Juliana; Faria, Lucas F.; Natale, Caio C.; Paula, Nicholas M.; Silva, Priscila C. S.; Fonseca, Fernanda R.; Aires, Luan; Heck, Nicoli; Starick, Márick R.; Barroso, Shana P. C.; Morrot, Alexandre; Weyenbergh, Johan Van; Sordi, Regina de; Alisson-Silva, Frederico; Mansur, Daniel S.; Cunha, Fernando de Queiroz ; Rocha, Edroaldo L.; Witko-Sarsat, Véronique; Burgel, Pierre-Régis; Martin, Clémance; Maurici, Rosemeri; Bafica, Andre; Macauley, Matthew S.; Spiller, Fernando
Source: bioRxiv. Unidade: FMRP
Subjects: GLICOSÍDEOS, SEPSE, NEUTRÓFILOS, COVID-19, ANTIVIRAIS
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ABNT
FORMIGA, Rodrigo Oliveira et al. Neuraminidase inhibitors rewire neutrophil function in vivo in murine sepsis and ex vivo in COVID-19. bioRxiv, p. 1-68, 2021Tradução . . Disponível em: https://doi.org/10.1101/2020.11.12.379115. Acesso em: 14 jun. 2024.
APA
Formiga, R. O., Amaral, F. C., Souza, C. F., Mendes, D. A. G. B., Wanderley, C. W. de S., Lorenzini, C. B., et al. (2021). Neuraminidase inhibitors rewire neutrophil function in vivo in murine sepsis and ex vivo in COVID-19. bioRxiv, 1-68. doi:10.1101/2020.11.12.379115
NLM
Formiga RO, Amaral FC, Souza CF, Mendes DAGB, Wanderley CW de S, Lorenzini CB, Santos AA, Antônia J, Faria LF, Natale CC, Paula NM, Silva PCS, Fonseca FR, Aires L, Heck N, Starick MR, Barroso SPC, Morrot A, Weyenbergh JV, Sordi R de, Alisson-Silva F, Mansur DS, Cunha F de Q, Rocha EL, Witko-Sarsat V, Burgel P-R, Martin C, Maurici R, Bafica A, Macauley MS, Spiller F. Neuraminidase inhibitors rewire neutrophil function in vivo in murine sepsis and ex vivo in COVID-19 [Internet]. bioRxiv. 2021 ; 1-68.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1101/2020.11.12.379115
Vancouver
Formiga RO, Amaral FC, Souza CF, Mendes DAGB, Wanderley CW de S, Lorenzini CB, Santos AA, Antônia J, Faria LF, Natale CC, Paula NM, Silva PCS, Fonseca FR, Aires L, Heck N, Starick MR, Barroso SPC, Morrot A, Weyenbergh JV, Sordi R de, Alisson-Silva F, Mansur DS, Cunha F de Q, Rocha EL, Witko-Sarsat V, Burgel P-R, Martin C, Maurici R, Bafica A, Macauley MS, Spiller F. Neuraminidase inhibitors rewire neutrophil function in vivo in murine sepsis and ex vivo in COVID-19 [Internet]. bioRxiv. 2021 ; 1-68.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1101/2020.11.12.379115
Artigo de periodico
Molecular basis of carrageenan-induced cytokines production in macrophages (2020)
Lopes, Alexandre Hashimoto Pereira; Silva, Rangel Leal; Fonseca, Miriam das Dores Mendes; Gomes, Francisco Isaac Fernandes; Maganin, Alexandre Gomes de Macedo; Ribeiro, Lucas S.; Marques, Lucas Maciel Mauriz; Cunha, Fernando de Queiroz ; Alves Filho, José Carlos Farias ; Zamboni, Dario Simões; Lopes, Norberto Peporine ; Franklin, Bernardo S.; Gombault, Aurélie; Ramalho, Fernando Silva ; Quesniaux, Valérie F. J.; Couillin, Isabelle; Ryffel, Bernhard; Cunha, Thiago Mattar
Source: Cell Communication and Signaling. Unidades: FCFRP, FMRP
Subjects: CARRAGENINA, MACRÓFAGOS, POLISSACARÍDEOS, CITOCINAS, INFLAMAÇÃO
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ABNT
LOPES, Alexandre Hashimoto Pereira et al. Molecular basis of carrageenan-induced cytokines production in macrophages. Cell Communication and Signaling, v. 18, n. 1, 2020Tradução . . Disponível em: https://doi.org/10.1186/s12964-020-00621-x. Acesso em: 14 jun. 2024.
APA
Lopes, A. H. P., Silva, R. L., Fonseca, M. das D. M., Gomes, F. I. F., Maganin, A. G. de M., Ribeiro, L. S., et al. (2020). Molecular basis of carrageenan-induced cytokines production in macrophages. Cell Communication and Signaling, 18( 1). doi:10.1186/s12964-020-00621-x
NLM
Lopes AHP, Silva RL, Fonseca M das DM, Gomes FIF, Maganin AG de M, Ribeiro LS, Marques LMM, Cunha F de Q, Alves Filho JCF, Zamboni DS, Lopes NP, Franklin BS, Gombault A, Ramalho FS, Quesniaux VFJ, Couillin I, Ryffel B, Cunha TM. Molecular basis of carrageenan-induced cytokines production in macrophages [Internet]. Cell Communication and Signaling. 2020 ; 18( 1):[citado 2024 jun. 14 ] Available from: https://doi.org/10.1186/s12964-020-00621-x
Vancouver
Lopes AHP, Silva RL, Fonseca M das DM, Gomes FIF, Maganin AG de M, Ribeiro LS, Marques LMM, Cunha F de Q, Alves Filho JCF, Zamboni DS, Lopes NP, Franklin BS, Gombault A, Ramalho FS, Quesniaux VFJ, Couillin I, Ryffel B, Cunha TM. Molecular basis of carrageenan-induced cytokines production in macrophages [Internet]. Cell Communication and Signaling. 2020 ; 18( 1):[citado 2024 jun. 14 ] Available from: https://doi.org/10.1186/s12964-020-00621-x
Artigo de periodico
MEK5/ERK5 signaling mediates IL‐4‐induced M2 macrophage differentiation through regulation of c‐Myc expression (2020)
Luiz, João Paulo Mesquita ; Toller‐Kawahisa, Juliana E.; Viacava, Paula Ramos; Nascimento, Daniele C.; Pereira, Priscilla T.; Saraiva, André L.; Prado, Douglas da Silva ; Le Bert, Marc; Giurisato, Emanuele; Tournier, Cathy; Cunha, Thiago Mattar ; Cunha, Fernando Queiróz ; Quesniaux, Valerie; Ryffel, Bernhard; Alves Filho, José Carlos Farias
Source: Journal of Leukocyte Biology. Unidade: FMRP
Subjects: DIFERENCIAÇÃO CELULAR, ANTÍGENOS, IMUNOLOGIA, REGULAÇÃO GÊNICA, EXPRESSÃO GÊNICA, INTERLEUCINA 4, MACRÓFAGOS, PROTEÍNAS PROTO-ONCOGÊNICAS
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ABNT
LUIZ, João Paulo Mesquita et al. MEK5/ERK5 signaling mediates IL‐4‐induced M2 macrophage differentiation through regulation of c‐Myc expression. Journal of Leukocyte Biology, v. 108, p. 1215-1223, 2020Tradução . . Disponível em: https://doi.org/10.1002/JLB.1MA0520-016R. Acesso em: 14 jun. 2024.
APA
Luiz, J. P. M., Toller‐Kawahisa, J. E., Viacava, P. R., Nascimento, D. C., Pereira, P. T., Saraiva, A. L., et al. (2020). MEK5/ERK5 signaling mediates IL‐4‐induced M2 macrophage differentiation through regulation of c‐Myc expression. Journal of Leukocyte Biology, 108, 1215-1223. doi:10.1002/JLB.1MA0520-016R
NLM
Luiz JPM, Toller‐Kawahisa JE, Viacava PR, Nascimento DC, Pereira PT, Saraiva AL, Prado D da S, Le Bert M, Giurisato E, Tournier C, Cunha TM, Cunha FQ, Quesniaux V, Ryffel B, Alves Filho JCF. MEK5/ERK5 signaling mediates IL‐4‐induced M2 macrophage differentiation through regulation of c‐Myc expression [Internet]. Journal of Leukocyte Biology. 2020 ; 108 1215-1223.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1002/JLB.1MA0520-016R
Vancouver
Luiz JPM, Toller‐Kawahisa JE, Viacava PR, Nascimento DC, Pereira PT, Saraiva AL, Prado D da S, Le Bert M, Giurisato E, Tournier C, Cunha TM, Cunha FQ, Quesniaux V, Ryffel B, Alves Filho JCF. MEK5/ERK5 signaling mediates IL‐4‐induced M2 macrophage differentiation through regulation of c‐Myc expression [Internet]. Journal of Leukocyte Biology. 2020 ; 108 1215-1223.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1002/JLB.1MA0520-016R
Artigo de periodico
Canonical PI3Kγ signaling in myeloid cells restricts Trypanosoma cruzi infection and dampens chagasic myocarditis (2018)
Silva, Maria C; Ferreira, Marcela Davoli; Medina, Tiago S; Costa, Renata Sesti; Silva, Grace K; Lopes, Carla D; Cardozo, Lucas Esteves; Gava, Fábio N; Lyroni, Konstantina; Dias, Fabricio C; Frade, Amanda F; Baron, Monique; Nakaya, Helder Takashi Imoto ; Figueiredo, Florêncio; Alves Filho, José Carlos Farias; Cunha, Fernando de Queiroz; Tsatsanis, Christos; Chevillard, Christophe; Cunha-Neto, Edécio; Hirsch, Emilio; Silva, João Santana da; Cunha, Thiago Mattar
Source: Nature Communications. Unidades: FCF, FMRP, FM
Subjects: TRYPANOSOMA CRUZI, MIOCARDIOPATIAS
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ABNT
SILVA, Maria C et al. Canonical PI3Kγ signaling in myeloid cells restricts Trypanosoma cruzi infection and dampens chagasic myocarditis. Nature Communications, v. 9, p. 1-14 art. 1513, 2018Tradução . . Disponível em: https://doi.org/10.1038/s41467-018-03986-3. Acesso em: 14 jun. 2024.
APA
Silva, M. C., Ferreira, M. D., Medina, T. S., Costa, R. S., Silva, G. K., Lopes, C. D., et al. (2018). Canonical PI3Kγ signaling in myeloid cells restricts Trypanosoma cruzi infection and dampens chagasic myocarditis. Nature Communications, 9, 1-14 art. 1513. doi:10.1038/s41467-018-03986-3
NLM
Silva MC, Ferreira MD, Medina TS, Costa RS, Silva GK, Lopes CD, Cardozo LE, Gava FN, Lyroni K, Dias FC, Frade AF, Baron M, Nakaya HTI, Figueiredo F, Alves Filho JCF, Cunha F de Q, Tsatsanis C, Chevillard C, Cunha-Neto E, Hirsch E, Silva JS da, Cunha TM. Canonical PI3Kγ signaling in myeloid cells restricts Trypanosoma cruzi infection and dampens chagasic myocarditis [Internet]. Nature Communications. 2018 ; 9 1-14 art. 1513.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1038/s41467-018-03986-3
Vancouver
Silva MC, Ferreira MD, Medina TS, Costa RS, Silva GK, Lopes CD, Cardozo LE, Gava FN, Lyroni K, Dias FC, Frade AF, Baron M, Nakaya HTI, Figueiredo F, Alves Filho JCF, Cunha F de Q, Tsatsanis C, Chevillard C, Cunha-Neto E, Hirsch E, Silva JS da, Cunha TM. Canonical PI3Kγ signaling in myeloid cells restricts Trypanosoma cruzi infection and dampens chagasic myocarditis [Internet]. Nature Communications. 2018 ; 9 1-14 art. 1513.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1038/s41467-018-03986-3
Artigo de periodico
Succinate receptor deficiency attenuates arthritis by reducing dendritic cell traffic and expansion of Th17 cells in the lymph nodes (2018)
Saraiva, André L.; Veras, Flávio P.; Peres, Raphael S.; Talbot, Jhimmy; Lima, Kalil A. de; Luiz, João P.; Carballido, José M.; Cunha, Thiago Mattar; Cunha, Fernando de Queiroz; Ryffel, Bernhard; Alves Filho, José Carlos Farias
Source: The FASEB Journal. Unidade: FMRP
Subjects: DOENÇAS AUTOIMUNES, INFLAMAÇÃO, METABOLISMO
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ABNT
SARAIVA, André L. et al. Succinate receptor deficiency attenuates arthritis by reducing dendritic cell traffic and expansion of Th17 cells in the lymph nodes. The FASEB Journal, v. 32, n. 12, p. 6550-6558, 2018Tradução . . Disponível em: https://doi.org/10.1096/fj.201800285. Acesso em: 14 jun. 2024.
APA
Saraiva, A. L., Veras, F. P., Peres, R. S., Talbot, J., Lima, K. A. de, Luiz, J. P., et al. (2018). Succinate receptor deficiency attenuates arthritis by reducing dendritic cell traffic and expansion of Th17 cells in the lymph nodes. The FASEB Journal, 32( 12), 6550-6558. doi:10.1096/fj.201800285
NLM
Saraiva AL, Veras FP, Peres RS, Talbot J, Lima KA de, Luiz JP, Carballido JM, Cunha TM, Cunha F de Q, Ryffel B, Alves Filho JCF. Succinate receptor deficiency attenuates arthritis by reducing dendritic cell traffic and expansion of Th17 cells in the lymph nodes [Internet]. The FASEB Journal. 2018 ; 32( 12): 6550-6558.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1096/fj.201800285
Vancouver
Saraiva AL, Veras FP, Peres RS, Talbot J, Lima KA de, Luiz JP, Carballido JM, Cunha TM, Cunha F de Q, Ryffel B, Alves Filho JCF. Succinate receptor deficiency attenuates arthritis by reducing dendritic cell traffic and expansion of Th17 cells in the lymph nodes [Internet]. The FASEB Journal. 2018 ; 32( 12): 6550-6558.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1096/fj.201800285
Artigo de periodico
Smoking-induced aggravation of experimental arthritis is dependent of aryl hydrocarbon receptor activation in Th17 cells (2018)
Talbot, Jhimmy; Peres, Raphael S.; Pinto, Larissa G.; Oliveira, Rene D. R.; Lima, Kalil A.; Donate, Paula B.; Silva, Jaqueline R.; Ryffel, Bernard; Cunha, Thiago Mattar; Alves Filho, José Carlos Farias; Liew, Foo Y.; Louzada Júnior, Paulo; Cunha, Fernando de Queiroz
Source: Arthritis Research & Therapy. Unidade: FMRP
Subjects: ARTRITE REUMATOIDE, FUMANTES, INFLAMAÇÃO, HIDROCARBONETOS
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ABNT
TALBOT, Jhimmy et al. Smoking-induced aggravation of experimental arthritis is dependent of aryl hydrocarbon receptor activation in Th17 cells. Arthritis Research & Therapy, v. 20, n. 1, 2018Tradução . . Disponível em: https://doi.org/10.1186/s13075-018-1609-9. Acesso em: 14 jun. 2024.
APA
Talbot, J., Peres, R. S., Pinto, L. G., Oliveira, R. D. R., Lima, K. A., Donate, P. B., et al. (2018). Smoking-induced aggravation of experimental arthritis is dependent of aryl hydrocarbon receptor activation in Th17 cells. Arthritis Research & Therapy, 20( 1). doi:10.1186/s13075-018-1609-9
NLM
Talbot J, Peres RS, Pinto LG, Oliveira RDR, Lima KA, Donate PB, Silva JR, Ryffel B, Cunha TM, Alves Filho JCF, Liew FY, Louzada Júnior P, Cunha F de Q. Smoking-induced aggravation of experimental arthritis is dependent of aryl hydrocarbon receptor activation in Th17 cells [Internet]. Arthritis Research & Therapy. 2018 ;20( 1):[citado 2024 jun. 14 ] Available from: https://doi.org/10.1186/s13075-018-1609-9
Vancouver
Talbot J, Peres RS, Pinto LG, Oliveira RDR, Lima KA, Donate PB, Silva JR, Ryffel B, Cunha TM, Alves Filho JCF, Liew FY, Louzada Júnior P, Cunha F de Q. Smoking-induced aggravation of experimental arthritis is dependent of aryl hydrocarbon receptor activation in Th17 cells [Internet]. Arthritis Research & Therapy. 2018 ;20( 1):[citado 2024 jun. 14 ] Available from: https://doi.org/10.1186/s13075-018-1609-9
Artigo de periodico
Spinal cord oligodendrocyte-derived alarmin IL-33 mediates neuropathic pain (2016)
Zarpelon, Ana C.; Rodrigues, Francielle C.; Lopes, Alexandre H.; Souza, Guilherme R.; Carvalho, Thacyana T.; Pinto, Larissa G.; Xu, Damo; Ferreira, Sérgio Henrique; Alves Filho, José Carlos Farias; McInnes, Iain B.; Ryffel, Bernhard; Quesniaux, Valérie F. J.; Reverchon, Flora; Mortaud, Stéphane; Menuet, Arnaud; Liew, Foo Y.; Cunha, Fernando de Queiroz; Cunha, Thiago Mattar; Verri Júnior, Waldiceu A.
Source: FASEB Journal. Unidade: FMRP
Subjects: INTERLEUCINAS, RECEPTORES, DOENÇAS DO SISTEMA NERVOSO, DOR, MODELOS ANIMAIS
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ABNT
ZARPELON, Ana C. et al. Spinal cord oligodendrocyte-derived alarmin IL-33 mediates neuropathic pain. FASEB Journal, v. 30, n. 1, p. 54-65, 2016Tradução . . Disponível em: https://doi.org/10.1096/fj.14-267146. Acesso em: 14 jun. 2024.
APA
Zarpelon, A. C., Rodrigues, F. C., Lopes, A. H., Souza, G. R., Carvalho, T. T., Pinto, L. G., et al. (2016). Spinal cord oligodendrocyte-derived alarmin IL-33 mediates neuropathic pain. FASEB Journal, 30( 1), 54-65. doi:10.1096/fj.14-267146
NLM
Zarpelon AC, Rodrigues FC, Lopes AH, Souza GR, Carvalho TT, Pinto LG, Xu D, Ferreira SH, Alves Filho JCF, McInnes IB, Ryffel B, Quesniaux VFJ, Reverchon F, Mortaud S, Menuet A, Liew FY, Cunha F de Q, Cunha TM, Verri Júnior WA. Spinal cord oligodendrocyte-derived alarmin IL-33 mediates neuropathic pain [Internet]. FASEB Journal. 2016 ; 30( 1): 54-65.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1096/fj.14-267146
Vancouver
Zarpelon AC, Rodrigues FC, Lopes AH, Souza GR, Carvalho TT, Pinto LG, Xu D, Ferreira SH, Alves Filho JCF, McInnes IB, Ryffel B, Quesniaux VFJ, Reverchon F, Mortaud S, Menuet A, Liew FY, Cunha F de Q, Cunha TM, Verri Júnior WA. Spinal cord oligodendrocyte-derived alarmin IL-33 mediates neuropathic pain [Internet]. FASEB Journal. 2016 ; 30( 1): 54-65.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1096/fj.14-267146
Artigo de periodico
Joint production of IL-22 participates in the initial phase of antigen-induced arthritis through IL-1β production (2015)
Pinto, Larissa G.; Talbot, Jhimmy; Peres, Raphael S.; Franca, Rafael F.; Ferreira, Sérgio Henrique; Ryffel, Bernhard; Alves Filho, José Carlos Farias; Figueiredo, Florêncio; Cunha, Thiago Mattar; Cunha, Fernando de Queiroz
Source: Arthritis Research & Therapy. Unidade: FMRP
Subjects: ARTRITE REUMATOIDE, NEUTRÓFILOS, INTERLEUCINAS, MODELOS ANIMAIS DE DOENÇAS
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ABNT
PINTO, Larissa G. et al. Joint production of IL-22 participates in the initial phase of antigen-induced arthritis through IL-1β production. Arthritis Research & Therapy, v. 17, 2015Tradução . . Disponível em: https://doi.org/10.1186/s13075-015-0759-2. Acesso em: 14 jun. 2024.
APA
Pinto, L. G., Talbot, J., Peres, R. S., Franca, R. F., Ferreira, S. H., Ryffel, B., et al. (2015). Joint production of IL-22 participates in the initial phase of antigen-induced arthritis through IL-1β production. Arthritis Research & Therapy, 17. doi:10.1186/s13075-015-0759-2
NLM
Pinto LG, Talbot J, Peres RS, Franca RF, Ferreira SH, Ryffel B, Alves Filho JCF, Figueiredo F, Cunha TM, Cunha F de Q. Joint production of IL-22 participates in the initial phase of antigen-induced arthritis through IL-1β production [Internet]. Arthritis Research & Therapy. 2015 ; 17[citado 2024 jun. 14 ] Available from: https://doi.org/10.1186/s13075-015-0759-2
Vancouver
Pinto LG, Talbot J, Peres RS, Franca RF, Ferreira SH, Ryffel B, Alves Filho JCF, Figueiredo F, Cunha TM, Cunha F de Q. Joint production of IL-22 participates in the initial phase of antigen-induced arthritis through IL-1β production [Internet]. Arthritis Research & Therapy. 2015 ; 17[citado 2024 jun. 14 ] Available from: https://doi.org/10.1186/s13075-015-0759-2
Trabalho de evento-resumo
Unraveling the pathogenesis of Legionella longbeachae infection: a role of type IV secretion system to induce mice lethality via pulmonary inflammation (2015)
Massis, Liliana Moura; Castanheira, Fernanda Vargas; Escoll, Pedro; Assis, Mariana Aprigio; Cunha, Fernando de Queiroz; Burcheiser, Carmen; Zamboni, Dario Simões
Source: Abstracts. Conference titles: INNATE IMMUNITY 2015. Unidade: FMRP
Subjects: LEGIONELLA, PNEUMONIA, TRANSPORTE BIOLÓGICO
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ABNT
MASSIS, Liliana Moura et al. Unraveling the pathogenesis of Legionella longbeachae infection: a role of type IV secretion system to induce mice lethality via pulmonary inflammation . 2015, Anais.. Guarujá: SBI, 2015. Disponível em: https://icongresso.itarget.com.br/useradm/anais/?clt=imu.3&lng=I. Acesso em: 14 jun. 2024.
APA
Massis, L. M., Castanheira, F. V., Escoll, P., Assis, M. A., Cunha, F. de Q., Burcheiser, C., & Zamboni, D. S. (2015). Unraveling the pathogenesis of Legionella longbeachae infection: a role of type IV secretion system to induce mice lethality via pulmonary inflammation . In Abstracts. Guarujá: SBI. Recuperado de https://icongresso.itarget.com.br/useradm/anais/?clt=imu.3&lng=I
NLM
Massis LM, Castanheira FV, Escoll P, Assis MA, Cunha F de Q, Burcheiser C, Zamboni DS. Unraveling the pathogenesis of Legionella longbeachae infection: a role of type IV secretion system to induce mice lethality via pulmonary inflammation [Internet]. Abstracts. 2015 ;[citado 2024 jun. 14 ] Available from: https://icongresso.itarget.com.br/useradm/anais/?clt=imu.3&lng=I
Vancouver
Massis LM, Castanheira FV, Escoll P, Assis MA, Cunha F de Q, Burcheiser C, Zamboni DS. Unraveling the pathogenesis of Legionella longbeachae infection: a role of type IV secretion system to induce mice lethality via pulmonary inflammation [Internet]. Abstracts. 2015 ;[citado 2024 jun. 14 ] Available from: https://icongresso.itarget.com.br/useradm/anais/?clt=imu.3&lng=I
Artigo de periodico
Peripheral NLCR4 inflammasome participates in the genesis of acute inflammatory pain (2015)
Lopes, Alexandre H.; Talbot, Jhimmy; Silva, Rangel L.; Lima, Jonilson B.; França, Rafael O.; Verri Júnior, Waldiceu Aparecido; Mascarenhas, Danielle Pini Alves; Ryffel, Bernhard; Cunha, Fernando de Queiroz; Zamboni, Dario Simões; Cunha, Thiago Mattar
Source: Pain. Unidade: FMRP
Subjects: APOPTOSE, PROTEÍNAS DE LIGAÇÃO DO CÁLCIO, INFLAMAÇÃO, MODELOS ANIMAIS DE DOENÇAS
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ABNT
LOPES, Alexandre H. et al. Peripheral NLCR4 inflammasome participates in the genesis of acute inflammatory pain. Pain, v. 156, n. 3, p. 451-459, 2015Tradução . . Disponível em: https://doi.org/10.1097/01.j.pain.0000460322.72396.53. Acesso em: 14 jun. 2024.
APA
Lopes, A. H., Talbot, J., Silva, R. L., Lima, J. B., França, R. O., Verri Júnior, W. A., et al. (2015). Peripheral NLCR4 inflammasome participates in the genesis of acute inflammatory pain. Pain, 156( 3), 451-459. doi:10.1097/01.j.pain.0000460322.72396.53
NLM
Lopes AH, Talbot J, Silva RL, Lima JB, França RO, Verri Júnior WA, Mascarenhas DPA, Ryffel B, Cunha F de Q, Zamboni DS, Cunha TM. Peripheral NLCR4 inflammasome participates in the genesis of acute inflammatory pain [Internet]. Pain. 2015 ; 156( 3): 451-459.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1097/01.j.pain.0000460322.72396.53
Vancouver
Lopes AH, Talbot J, Silva RL, Lima JB, França RO, Verri Júnior WA, Mascarenhas DPA, Ryffel B, Cunha F de Q, Zamboni DS, Cunha TM. Peripheral NLCR4 inflammasome participates in the genesis of acute inflammatory pain [Internet]. Pain. 2015 ; 156( 3): 451-459.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1097/01.j.pain.0000460322.72396.53

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