Joint production of IL-22 participates in the initial phase of antigen-induced arthritis through IL-1β production (2015)
- Authors:
- USP affiliated authors: FERREIRA, SERGIO HENRIQUE - FMRP ; ALVES FILHO, JOSÉ CARLOS FARIAS - FMRP ; CUNHA, THIAGO MATTAR - FMRP ; CUNHA, FERNANDO DE QUEIROZ - FMRP
- Unidade: FMRP
- DOI: 10.1186/s13075-015-0759-2
- Subjects: ARTRITE REUMATOIDE; NEUTRÓFILOS; INTERLEUCINAS; MODELOS ANIMAIS DE DOENÇAS
- Language: Inglês
- Imprenta:
- Source:
- Título: Arthritis Research & Therapy
- ISSN: 1478-6354
- Volume/Número/Paginação/Ano: v. 17, art. 235, 2015
- Status:
- Artigo publicado em periódico de acesso aberto (Gold Open Access)
- Versão do Documento:
- Versão publicada (Published version)
- Acessar versão aberta:
-
ABNT
PINTO, Larissa G. et al. Joint production of IL-22 participates in the initial phase of antigen-induced arthritis through IL-1β production. Arthritis Research & Therapy, v. 17, 2015Tradução . . Disponível em: https://doi.org/10.1186/s13075-015-0759-2. Acesso em: 01 abr. 2026. -
APA
Pinto, L. G., Talbot, J., Peres, R. S., Franca, R. F., Ferreira, S. H., Ryffel, B., et al. (2015). Joint production of IL-22 participates in the initial phase of antigen-induced arthritis through IL-1β production. Arthritis Research & Therapy, 17. doi:10.1186/s13075-015-0759-2 -
NLM
Pinto LG, Talbot J, Peres RS, Franca RF, Ferreira SH, Ryffel B, Alves Filho JCF, Figueiredo F, Cunha TM, Cunha F de Q. Joint production of IL-22 participates in the initial phase of antigen-induced arthritis through IL-1β production [Internet]. Arthritis Research & Therapy. 2015 ; 17[citado 2026 abr. 01 ] Available from: https://doi.org/10.1186/s13075-015-0759-2 -
Vancouver
Pinto LG, Talbot J, Peres RS, Franca RF, Ferreira SH, Ryffel B, Alves Filho JCF, Figueiredo F, Cunha TM, Cunha F de Q. Joint production of IL-22 participates in the initial phase of antigen-induced arthritis through IL-1β production [Internet]. Arthritis Research & Therapy. 2015 ; 17[citado 2026 abr. 01 ] Available from: https://doi.org/10.1186/s13075-015-0759-2 - Medial plantar nerve ligation as a novel model of neuropathic pain in mice: pharmacological and molecular characterization
- Interleukin-10 limits intense acute swimming-induced muscle mechanical hyperalgesia in mice
- 5-lipoxygenase deficiency reduces acetaminophen-induced hepatotoxicity and lethality
- Mechanisms underlying the hyperalgesic responses triggered by joint activation of TLR4
- Nitroxyl inhibits overt pain-like behavior in mice: role of cGMP/PKG/ATP-sensitive potassium channel signaling pathway
- Targeting interleukin-1'beta' reduces intense acute swimming-induced muscle mechanical hyperalgesia in mice
- Spinal cord oligodendrocyte-derived alarmin IL-33 mediates neuropathic pain
- Lipopolysaccharide induces inflammatory hyperalgesia triggering a TLR4/MyD88-dependent cytokine cascade in the mice paw
- Pyrrolidine dithiocarbamate inhibits superoxide anion-induced pain and inflammation in the paw skin and spinal cord by targeting NF-kB and oxidative stress
- Toll like receptor 9 deficiency in neutrophil improves sepsis outcome preventing down-regulation of CXCR2
Informações sobre a disponibilidade de versões do artigo em acesso aberto coletadas automaticamente via oaDOI API (Unpaywall).
Por se tratar de integração com serviço externo, podem existir diferentes versões do trabalho (como preprints ou postprints), que podem diferir da versão publicada.
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
