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Artigo de periodico
FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility (2023)
Borges, Vanessa de Fátima; Galant, Leticia Selinger; Kanashiro, Alexandre ; Castanheira, Fernanda Vargas e Silva; Monteiro, Valter Vinícius Silva; Duarte, Diego Ângelo; Rodrigues, Filipe Camargo; Silva, Camila Meirelles de Souza; Schneider, Ayda Henriques ; Cebinelli, Guilherme Cesar Martelossi ; Lima, Mikhael Haruo Fernandes de; Viola, João Paulo de Biaso; Cunha, Thiago Mattar ; Costa Neto, Claudio Miguel da ; Alves Filho, José Carlos Farias ; Pupo, André Sampaio; Cunha, Fernando de Queiroz
Source: Inflammation Research. Unidade: FMRP
Subjects: NEUTRÓFILOS, SEPSE, FÁRMACOS IMUNOSSUPRESSORES
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ABNT
BORGES, Vanessa de Fátima et al. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility. Inflammation Research, v. 72, n. 2, p. 203-215, 2023Tradução . . Disponível em: https://doi.org/10.1007/s00011-022-01669-w. Acesso em: 05 jul. 2024.
APA
Borges, V. de F., Galant, L. S., Kanashiro, A., Castanheira, F. V. e S., Monteiro, V. V. S., Duarte, D. Â., et al. (2023). FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility. Inflammation Research, 72( 2), 203-215. doi:10.1007/s00011-022-01669-w
NLM
Borges V de F, Galant LS, Kanashiro A, Castanheira FV e S, Monteiro VVS, Duarte DÂ, Rodrigues FC, Silva CM de S, Schneider AH, Cebinelli GCM, Lima MHF de, Viola JP de B, Cunha TM, Costa Neto CM da, Alves Filho JCF, Pupo AS, Cunha F de Q. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility [Internet]. Inflammation Research. 2023 ; 72( 2): 203-215.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1007/s00011-022-01669-w
Vancouver
Borges V de F, Galant LS, Kanashiro A, Castanheira FV e S, Monteiro VVS, Duarte DÂ, Rodrigues FC, Silva CM de S, Schneider AH, Cebinelli GCM, Lima MHF de, Viola JP de B, Cunha TM, Costa Neto CM da, Alves Filho JCF, Pupo AS, Cunha F de Q. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility [Internet]. Inflammation Research. 2023 ; 72( 2): 203-215.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1007/s00011-022-01669-w
Artigo de periodico
Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT (2022)
Lima, Mikhael Haruo Fernandes de; Hiroki, Carlos Hiroji; Borges, Vanessa de Fátima; Cebinelli, Guilherme Cesar Martelossi ; Santos, Jessica; Rosa, Marcos Henrique; Silva, Camila Meirelles de Souza; Wanderley, Carlos Wagner ; Gonçalves, Augusto Velozo; Quirino, Gustavo Fernando da Silva; Zamboni, Dario Simões ; Cunha, Thiago Mattar ; Alves Filho, José Carlos Farias ; Cunha, Fernando de Queiroz
Source: The Journal of Infectious Diseases. Unidade: FMRP
Subjects: IMUNOSSUPRESSÃO, SEPSE, MACRÓFAGOS, CITOMETRIA DE FLUXO
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ABNT
LIMA, Mikhael Haruo Fernandes de et al. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT. The Journal of Infectious Diseases, v. 225, n. 3, p. 531-541, 2022Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiab405. Acesso em: 05 jul. 2024.
APA
Lima, M. H. F. de, Hiroki, C. H., Borges, V. de F., Cebinelli, G. C. M., Santos, J., Rosa, M. H., et al. (2022). Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT. The Journal of Infectious Diseases, 225( 3), 531-541. doi:10.1093/infdis/jiab405
NLM
Lima MHF de, Hiroki CH, Borges V de F, Cebinelli GCM, Santos J, Rosa MH, Silva CM de S, Wanderley CW, Gonçalves AV, Quirino GF da S, Zamboni DS, Cunha TM, Alves Filho JCF, Cunha F de Q. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT [Internet]. The Journal of Infectious Diseases. 2022 ; 225( 3): 531-541.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1093/infdis/jiab405
Vancouver
Lima MHF de, Hiroki CH, Borges V de F, Cebinelli GCM, Santos J, Rosa MH, Silva CM de S, Wanderley CW, Gonçalves AV, Quirino GF da S, Zamboni DS, Cunha TM, Alves Filho JCF, Cunha F de Q. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT [Internet]. The Journal of Infectious Diseases. 2022 ; 225( 3): 531-541.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1093/infdis/jiab405
Artigo de periodico
Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy (2022)
Santos, Jeimison D.; Oliveira Neto, José Teles de ; Barros, Paula Rodrigues de; Damasceno, Luis Eduardo Alves; Lautherbach, Natalia; Assis, Ana Paula de; Silva, Carlos Alberto Aguiar da; Sorgi, Carlos Arterio ; Faccioli, Lucia Helena ; Kettelhut, Isis do Carmo ; Salgado, Hélio César ; Carneiro, Fernando Silva ; Alves-Filho, José Carlos ; Tostes, Rita de Cássia Aleixo
Source: American Journal of Physiology-Heart and Circulatory Physiology. Unidades: FMRP, FFCLRP, FCFRP
Subjects: DOENÇAS CARDIOVASCULARES, FARMACOTERAPIA, HORMÔNIOS, ESTEROIDES, TESTOSTERONA
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ABNT
SANTOS, Jeimison D. et al. Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy. American Journal of Physiology-Heart and Circulatory Physiology, v. 323, p. H322-H335, 2022Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00182.2022. Acesso em: 05 jul. 2024.
APA
Santos, J. D., Oliveira Neto, J. T. de, Barros, P. R. de, Damasceno, L. E. A., Lautherbach, N., Assis, A. P. de, et al. (2022). Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy. American Journal of Physiology-Heart and Circulatory Physiology, 323, H322-H335. doi:10.1152/ajpheart.00182.2022
NLM
Santos JD, Oliveira Neto JT de, Barros PR de, Damasceno LEA, Lautherbach N, Assis AP de, Silva CAA da, Sorgi CA, Faccioli LH, Kettelhut I do C, Salgado HC, Carneiro FS, Alves-Filho JC, Tostes R de CA. Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy [Internet]. American Journal of Physiology-Heart and Circulatory Physiology. 2022 ; 323 H322-H335.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1152/ajpheart.00182.2022
Vancouver
Santos JD, Oliveira Neto JT de, Barros PR de, Damasceno LEA, Lautherbach N, Assis AP de, Silva CAA da, Sorgi CA, Faccioli LH, Kettelhut I do C, Salgado HC, Carneiro FS, Alves-Filho JC, Tostes R de CA. Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy [Internet]. American Journal of Physiology-Heart and Circulatory Physiology. 2022 ; 323 H322-H335.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1152/ajpheart.00182.2022
Artigo de periodico
Meningeal dendritic cells drive neuropathic pain through elevation of the kynurenine metabolic pathway in mice (2022)
Maganin, Alexandre Gomes de Macedo ; Souza, Guilherme Rabelo de; Fonseca, Miriam D.; Lopes, Alexandre H.; Guimarães, Rafaela Mano ; Dagostin, André; Cecílio, Nerry T.; Mendes, Atlante Silva ; Gonçalves, William Antônio; Silva, Conceição Elidianne Aníbal; Gomes, Francisco Isaac Fernandes ; Marques, Lucas Maciel Mauriz ; Silva, Rangel L.; Arruda, Leticia M.; Santana, Denis A.; Lemos, Henrique; Huang, Lei; Ferreira, Marcela Davoli; Santana-Coelho, Danielle; Sant’Anna, Morena Brasil Martins; Kusuda, Ricardo; Talbot, Jhimmy; Pacholczyk, Gabriela; Buqui, Gabriela A.; Lopes, Norberto Peporine ; Alves Filho, José Carlos Farias ; Leão, Ricardo Maurício Xavier; O’Connor, Jason C.; Cunha, Fernando de Queiroz ; Mellor, Andrew; Cunha, Thiago Mattar
Source: Journal of Clinical Investigation. Unidades: FCFRP, FMRP, FCF
Subjects: CÉLULAS DENDRÍTICAS, METABÓLITOS, DOENÇAS DO SISTEMA NERVOSO PERIFÉRICO, NERVOS PERIFÉRICOS, DOR CRÔNICA
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ABNT
MAGANIN, Alexandre Gomes de Macedo et al. Meningeal dendritic cells drive neuropathic pain through elevation of the kynurenine metabolic pathway in mice. Journal of Clinical Investigation, v. 132, n. 23, p. 1-17, 2022Tradução . . Disponível em: https://doi.org/10.1172/JCI153805. Acesso em: 05 jul. 2024.
APA
Maganin, A. G. de M., Souza, G. R. de, Fonseca, M. D., Lopes, A. H., Guimarães, R. M., Dagostin, A., et al. (2022). Meningeal dendritic cells drive neuropathic pain through elevation of the kynurenine metabolic pathway in mice. Journal of Clinical Investigation, 132( 23), 1-17. doi:10.1172/JCI153805
NLM
Maganin AG de M, Souza GR de, Fonseca MD, Lopes AH, Guimarães RM, Dagostin A, Cecílio NT, Mendes AS, Gonçalves WA, Silva CEA, Gomes FIF, Marques LMM, Silva RL, Arruda LM, Santana DA, Lemos H, Huang L, Ferreira MD, Santana-Coelho D, Sant’Anna MBM, Kusuda R, Talbot J, Pacholczyk G, Buqui GA, Lopes NP, Alves Filho JCF, Leão RMX, O’Connor JC, Cunha F de Q, Mellor A, Cunha TM. Meningeal dendritic cells drive neuropathic pain through elevation of the kynurenine metabolic pathway in mice [Internet]. Journal of Clinical Investigation. 2022 ; 132( 23): 1-17.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1172/JCI153805
Vancouver
Maganin AG de M, Souza GR de, Fonseca MD, Lopes AH, Guimarães RM, Dagostin A, Cecílio NT, Mendes AS, Gonçalves WA, Silva CEA, Gomes FIF, Marques LMM, Silva RL, Arruda LM, Santana DA, Lemos H, Huang L, Ferreira MD, Santana-Coelho D, Sant’Anna MBM, Kusuda R, Talbot J, Pacholczyk G, Buqui GA, Lopes NP, Alves Filho JCF, Leão RMX, O’Connor JC, Cunha F de Q, Mellor A, Cunha TM. Meningeal dendritic cells drive neuropathic pain through elevation of the kynurenine metabolic pathway in mice [Internet]. Journal of Clinical Investigation. 2022 ; 132( 23): 1-17.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1172/JCI153805
Artigo de periodico
Cinnamoyloxy-mammeisin, a coumarin from propolis of stingless bees, attenuates Th17 cell differentiation and autoimmune inflammation via STAT3 inhibition (2022)
Franchin, Marcelo ; Rosalen, Pedro Luiz ; Prado, Douglas da Silva ; Paraluppi, Murilo César; Silva, Rangel Leal ; Damasceno, Luis Eduardo Alves ; morelo, David Fernando Colon ; Napimoga, Marcelo Henrique; Cunha, Fernando de Queiroz ; Alves Filho, José Carlos Farias ; Cunha, Thiago Mattar
Source: European Journal of Pharmacology. Unidades: CENA, FMRP
Subjects: DIFERENCIAÇÃO CELULAR, ENCEFALOMIELITE AGUDA DISSEMINADA, DOENÇAS AUTOIMUNES, INFLAMAÇÃO, FARMACOTERAPIA, INTERLEUCINAS, PRÓPOLIS
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ABNT
FRANCHIN, Marcelo et al. Cinnamoyloxy-mammeisin, a coumarin from propolis of stingless bees, attenuates Th17 cell differentiation and autoimmune inflammation via STAT3 inhibition. European Journal of Pharmacology, v. 929, p. 1-8, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2022.175127. Acesso em: 05 jul. 2024.
APA
Franchin, M., Rosalen, P. L., Prado, D. da S., Paraluppi, M. C., Silva, R. L., Damasceno, L. E. A., et al. (2022). Cinnamoyloxy-mammeisin, a coumarin from propolis of stingless bees, attenuates Th17 cell differentiation and autoimmune inflammation via STAT3 inhibition. European Journal of Pharmacology, 929, 1-8. doi:10.1016/j.ejphar.2022.175127
NLM
Franchin M, Rosalen PL, Prado D da S, Paraluppi MC, Silva RL, Damasceno LEA, morelo DFC, Napimoga MH, Cunha F de Q, Alves Filho JCF, Cunha TM. Cinnamoyloxy-mammeisin, a coumarin from propolis of stingless bees, attenuates Th17 cell differentiation and autoimmune inflammation via STAT3 inhibition [Internet]. European Journal of Pharmacology. 2022 ; 929 1-8.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1016/j.ejphar.2022.175127
Vancouver
Franchin M, Rosalen PL, Prado D da S, Paraluppi MC, Silva RL, Damasceno LEA, morelo DFC, Napimoga MH, Cunha F de Q, Alves Filho JCF, Cunha TM. Cinnamoyloxy-mammeisin, a coumarin from propolis of stingless bees, attenuates Th17 cell differentiation and autoimmune inflammation via STAT3 inhibition [Internet]. European Journal of Pharmacology. 2022 ; 929 1-8.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1016/j.ejphar.2022.175127
Artigo de periodico
Riboflavin did not provide anti-inflammatory or antioxidant effects in an experimental model of sepsis (2022)
Vale, Aline Halfeld Fernandes ; Nascimento, Daniele Carvalho ; Alvarez, Annie Rocío Piñeros ; Ferreira, Raphael G. ; Santos, Jeimison Duarte ; Aragon, Davi Casale ; Cunha, Fernando de Queiroz ; Ramalho, Fernando Silva ; Alves Filho, José Carlos Farias ; Carlotti, Ana Paula de Carvalho Panzeri
Source: Brazilian Journal of Medical and Biological Research. Unidade: FMRP
Subjects: ANTI-INFLAMATÓRIOS, ANTIOXIDANTES, INFERÊNCIA BAYESIANA, QUIMIOCINAS, CREATINA, FARMACOTERAPIA
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ABNT
VALE, Aline Halfeld Fernandes et al. Riboflavin did not provide anti-inflammatory or antioxidant effects in an experimental model of sepsis. Brazilian Journal of Medical and Biological Research, v. 55, p. 1-8, 2022Tradução . . Disponível em: https://doi.org/10.1590/1414-431X2022e12107. Acesso em: 05 jul. 2024.
APA
Vale, A. H. F., Nascimento, D. C., Alvarez, A. R. P., Ferreira, R. G., Santos, J. D., Aragon, D. C., et al. (2022). Riboflavin did not provide anti-inflammatory or antioxidant effects in an experimental model of sepsis. Brazilian Journal of Medical and Biological Research, 55, 1-8. doi:10.1590/1414-431X2022e12107
NLM
Vale AHF, Nascimento DC, Alvarez ARP, Ferreira RG, Santos JD, Aragon DC, Cunha F de Q, Ramalho FS, Alves Filho JCF, Carlotti AP de CP. Riboflavin did not provide anti-inflammatory or antioxidant effects in an experimental model of sepsis [Internet]. Brazilian Journal of Medical and Biological Research. 2022 ; 55 1-8.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1590/1414-431X2022e12107
Vancouver
Vale AHF, Nascimento DC, Alvarez ARP, Ferreira RG, Santos JD, Aragon DC, Cunha F de Q, Ramalho FS, Alves Filho JCF, Carlotti AP de CP. Riboflavin did not provide anti-inflammatory or antioxidant effects in an experimental model of sepsis [Internet]. Brazilian Journal of Medical and Biological Research. 2022 ; 55 1-8.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1590/1414-431X2022e12107
Artigo de periodico
SARS-CoV-2–triggered neutrophil extracellular traps mediate COVID-19 pathology (2020)
Veras, Flavio Protasio; Pontelli, Marjorie Cornejo ; Silva, Camila Meirelles de Souza; Toller-Kawahisa, Juliana E.; Lima, Mikhael de; Nascimento, Daniele Carvalho; Schneider, Ayda Henriques; Caetité, Diego; Tavares, Lucas Alves ; Paiva, Isadora M.; Rosales, Roberta Ribeiro Costa; morelo, David Fernando Colon ; Martins, Ronaldo Bragança ; Castro, Ítalo Araújo de ; Almeida, Glaucia Maria ; Lopes, Maria Isabel Fernandes; Benatti, Maíra Nilson; Bonjorno, Letícia Pastorelli; Giannini, Marcela Cavichioli; Luppino-Assad, Rodrigo; Almeida, Sérgio Luna; Vilar, Fernando; Santana, Rodrigo de Carvalho ; Bollela, Valdes Roberto ; Auxiliadora-Martins, Maria ; Borges, Marcos de Carvalho ; Miranda, Carlos Henrique ; Pazin Filho, Antônio ; Silva, Luis Lamberti Pinto da ; Cunha, Larissa Dias; Zamboni, Dario Simões ; Dal-Pizzol, Felipe; Leiria, Luiz Osório Silveira ; Siyuan, Li; Batah, Sabrina Setembre ; Fabro, Alexandre Todorovic ; Mauad, Thais ; Dolhnikoff, Marisa ; Duarte Neto, Amaro Nunes ; Saldiva, Paulo Hilário Nascimento ; Cunha, Thiago Mattar ; Alves Filho, José Carlos Farias ; Arruda, Eurico ; Louzada Júnior, Paulo ; Oliveira, Renê Donizeti; Cunha, Fernando de Queiroz
Source: Journal of Experimental Medicine. Unidades: FMRP, FM
Subjects: COVID-19, CÉLULAS EPITELIAIS, PATOLOGIA, VIROLOGIA, PNEUMONIA, NEUTRÓFILOS, ANGIOTENSINA II
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ABNT
VERAS, Flavio Protasio et al. SARS-CoV-2–triggered neutrophil extracellular traps mediate COVID-19 pathology. Journal of Experimental Medicine, v. 217, n. 12, 2020Tradução . . Disponível em: https://doi.org/10.1084/jem.20201129. Acesso em: 05 jul. 2024.
APA
Veras, F. P., Pontelli, M. C., Silva, C. M. de S., Toller-Kawahisa, J. E., Lima, M. de, Nascimento, D. C., et al. (2020). SARS-CoV-2–triggered neutrophil extracellular traps mediate COVID-19 pathology. Journal of Experimental Medicine, 217( 12). doi:10.1084/jem.20201129
NLM
Veras FP, Pontelli MC, Silva CM de S, Toller-Kawahisa JE, Lima M de, Nascimento DC, Schneider AH, Caetité D, Tavares LA, Paiva IM, Rosales RRC, morelo DFC, Martins RB, Castro ÍA de, Almeida GM, Lopes MIF, Benatti MN, Bonjorno LP, Giannini MC, Luppino-Assad R, Almeida SL, Vilar F, Santana R de C, Bollela VR, Auxiliadora-Martins M, Borges M de C, Miranda CH, Pazin Filho A, Silva LLP da, Cunha LD, Zamboni DS, Dal-Pizzol F, Leiria LOS, Siyuan L, Batah SS, Fabro AT, Mauad T, Dolhnikoff M, Duarte Neto AN, Saldiva PHN, Cunha TM, Alves Filho JCF, Arruda E, Louzada Júnior P, Oliveira RD, Cunha F de Q. SARS-CoV-2–triggered neutrophil extracellular traps mediate COVID-19 pathology [Internet]. Journal of Experimental Medicine. 2020 ; 217( 12):[citado 2024 jul. 05 ] Available from: https://doi.org/10.1084/jem.20201129
Vancouver
Veras FP, Pontelli MC, Silva CM de S, Toller-Kawahisa JE, Lima M de, Nascimento DC, Schneider AH, Caetité D, Tavares LA, Paiva IM, Rosales RRC, morelo DFC, Martins RB, Castro ÍA de, Almeida GM, Lopes MIF, Benatti MN, Bonjorno LP, Giannini MC, Luppino-Assad R, Almeida SL, Vilar F, Santana R de C, Bollela VR, Auxiliadora-Martins M, Borges M de C, Miranda CH, Pazin Filho A, Silva LLP da, Cunha LD, Zamboni DS, Dal-Pizzol F, Leiria LOS, Siyuan L, Batah SS, Fabro AT, Mauad T, Dolhnikoff M, Duarte Neto AN, Saldiva PHN, Cunha TM, Alves Filho JCF, Arruda E, Louzada Júnior P, Oliveira RD, Cunha F de Q. SARS-CoV-2–triggered neutrophil extracellular traps mediate COVID-19 pathology [Internet]. Journal of Experimental Medicine. 2020 ; 217( 12):[citado 2024 jul. 05 ] Available from: https://doi.org/10.1084/jem.20201129
Artigo de periodico
Downregulation of IL-2 and IL-23 in cervical biopsies of cervical intraepithelial lesions: a cross-sectional study (2020)
Petrini, Caetano Galvão; Bastos, Larissa Brito; Duarte, Geraldo ; Melli, Patrícia Pereira dos Santos; Alves Filho, José Carlos Farias ; Quintana, Silvana Maria
Source: Acta Cytologica. Unidade: FMRP
Subjects: HPV, CITOCINAS, COLO UTERINO, BIOMARCADORES, ESTUDOS TRANSVERSAIS, INTERLEUCINA 2, PAPILLOMAVIRIDAE
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ABNT
PETRINI, Caetano Galvão et al. Downregulation of IL-2 and IL-23 in cervical biopsies of cervical intraepithelial lesions: a cross-sectional study. Acta Cytologica, v. 64, n. 5, p. 442-451, 2020Tradução . . Disponível em: https://doi.org/10.1159/000508015. Acesso em: 05 jul. 2024.
APA
Petrini, C. G., Bastos, L. B., Duarte, G., Melli, P. P. dos S., Alves Filho, J. C. F., & Quintana, S. M. (2020). Downregulation of IL-2 and IL-23 in cervical biopsies of cervical intraepithelial lesions: a cross-sectional study. Acta Cytologica, 64( 5), 442-451. doi:10.1159/000508015
NLM
Petrini CG, Bastos LB, Duarte G, Melli PP dos S, Alves Filho JCF, Quintana SM. Downregulation of IL-2 and IL-23 in cervical biopsies of cervical intraepithelial lesions: a cross-sectional study [Internet]. Acta Cytologica. 2020 ; 64( 5): 442-451.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1159/000508015
Vancouver
Petrini CG, Bastos LB, Duarte G, Melli PP dos S, Alves Filho JCF, Quintana SM. Downregulation of IL-2 and IL-23 in cervical biopsies of cervical intraepithelial lesions: a cross-sectional study [Internet]. Acta Cytologica. 2020 ; 64( 5): 442-451.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1159/000508015
Artigo de periodico
Cardiovascular and autonomic dysfunction in murine ligature-induced periodontitis (2020)
Ribeiro, Aline Barbosa ; Santos Junior, Nilton Nascimento dos; Luiz, João Paulo Mesquita ; Oliveira, Mauro de; Kanashiro, Alexandre; Taira, Thaise Mayumi ; Fukada, Sandra Yasuyo ; Alves Filho, José Carlos Farias ; Fazan Júnior, Rubens ; Salgado, Hélio César
Source: Scientific Reports. Unidades: FMRP, FCFRP, FORP
Subjects: PRESSÃO SANGUÍNEA, FREQUÊNCIA CARDÍACA, PERIODONTITE, MIOCÁRDIO, INFLAMAÇÃO
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ABNT
RIBEIRO, Aline Barbosa et al. Cardiovascular and autonomic dysfunction in murine ligature-induced periodontitis. Scientific Reports, v. 10, 2020Tradução . . Disponível em: https://doi.org/10.1038/s41598-020-63953-1. Acesso em: 05 jul. 2024.
APA
Ribeiro, A. B., Santos Junior, N. N. dos, Luiz, J. P. M., Oliveira, M. de, Kanashiro, A., Taira, T. M., et al. (2020). Cardiovascular and autonomic dysfunction in murine ligature-induced periodontitis. Scientific Reports, 10. doi:10.1038/s41598-020-63953-1
NLM
Ribeiro AB, Santos Junior NN dos, Luiz JPM, Oliveira M de, Kanashiro A, Taira TM, Fukada SY, Alves Filho JCF, Fazan Júnior R, Salgado HC. Cardiovascular and autonomic dysfunction in murine ligature-induced periodontitis [Internet]. Scientific Reports. 2020 ; 10[citado 2024 jul. 05 ] Available from: https://doi.org/10.1038/s41598-020-63953-1
Vancouver
Ribeiro AB, Santos Junior NN dos, Luiz JPM, Oliveira M de, Kanashiro A, Taira TM, Fukada SY, Alves Filho JCF, Fazan Júnior R, Salgado HC. Cardiovascular and autonomic dysfunction in murine ligature-induced periodontitis [Internet]. Scientific Reports. 2020 ; 10[citado 2024 jul. 05 ] Available from: https://doi.org/10.1038/s41598-020-63953-1
Artigo de periodico
IL-22 promotes IFN-γ-mediated immunity against histoplasma capsulatum Infection (2020)
Prado, Morgana Kelly Borges ; Fontanari, Caroline; Souza, Camila de Oliveira Silva e ; Gardinassi, Luiz Gustavo Araujo ; Zoccal, Karina Furlani ; Paula-Silva, Francisco Wanderley Garcia de ; Peti, Ana Paula Ferranti ; Sorgi, Carlos Arterio ; Meirelles, Alyne Fávero Galvão ; Ramos, Simone Gusmão ; Alves Filho, José Carlos Farias ; Faccioli, Lucia Helena
Source: Biomolecules. Unidades: FCFRP, FORP, FFCLRP, FMRP
Subjects: MICOSES, LINFÓCITOS T, CITOCINAS, ÓXIDO NÍTRICO
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ABNT
PRADO, Morgana Kelly Borges et al. IL-22 promotes IFN-γ-mediated immunity against histoplasma capsulatum Infection. Biomolecules, v. 10, n. 6, p. 1-18, 2020Tradução . . Disponível em: https://doi.org/10.3390/biom10060865. Acesso em: 05 jul. 2024.
APA
Prado, M. K. B., Fontanari, C., Souza, C. de O. S. e, Gardinassi, L. G. A., Zoccal, K. F., Paula-Silva, F. W. G. de, et al. (2020). IL-22 promotes IFN-γ-mediated immunity against histoplasma capsulatum Infection. Biomolecules, 10( 6), 1-18. doi:10.3390/biom10060865
NLM
Prado MKB, Fontanari C, Souza C de OS e, Gardinassi LGA, Zoccal KF, Paula-Silva FWG de, Peti APF, Sorgi CA, Meirelles AFG, Ramos SG, Alves Filho JCF, Faccioli LH. IL-22 promotes IFN-γ-mediated immunity against histoplasma capsulatum Infection [Internet]. Biomolecules. 2020 ;10( 6): 1-18.[citado 2024 jul. 05 ] Available from: https://doi.org/10.3390/biom10060865
Vancouver
Prado MKB, Fontanari C, Souza C de OS e, Gardinassi LGA, Zoccal KF, Paula-Silva FWG de, Peti APF, Sorgi CA, Meirelles AFG, Ramos SG, Alves Filho JCF, Faccioli LH. IL-22 promotes IFN-γ-mediated immunity against histoplasma capsulatum Infection [Internet]. Biomolecules. 2020 ;10( 6): 1-18.[citado 2024 jul. 05 ] Available from: https://doi.org/10.3390/biom10060865
Artigo de periodico
The citrus flavanone naringenin reduces gout-induced joint pain and inflammation in mice by inhibiting the activation of NFκB and macrophage release of IL-1β (2018)
Ruiz-Miyazawa, Kenji W.; Borghi, Sergio M.; Ribeiro, Felipe A. Pinho; Ferrari, Larissa Staurengo; Fattori, Victor; Fernandes, Glaura S. A.; Casella, Antonio M.; Alves Filho, José Carlos Farias ; Cunha, Thiago Mattar ; Cunha, Fernando de Queiroz ; Casagrande, Rubia; Verri, Waldiceu A.
Source: Journal of Functional Foods. Unidade: FMRP
Subjects: ARTRITE, DOR, INFLAMAÇÃO, RATOS, MACRÓFAGOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RUIZ-MIYAZAWA, Kenji W. et al. The citrus flavanone naringenin reduces gout-induced joint pain and inflammation in mice by inhibiting the activation of NFκB and macrophage release of IL-1β. Journal of Functional Foods, v. 48, p. 106-116, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.jff.2018.06.025. Acesso em: 05 jul. 2024.
APA
Ruiz-Miyazawa, K. W., Borghi, S. M., Ribeiro, F. A. P., Ferrari, L. S., Fattori, V., Fernandes, G. S. A., et al. (2018). The citrus flavanone naringenin reduces gout-induced joint pain and inflammation in mice by inhibiting the activation of NFκB and macrophage release of IL-1β. Journal of Functional Foods, 48, 106-116. doi:10.1016/j.jff.2018.06.025
NLM
Ruiz-Miyazawa KW, Borghi SM, Ribeiro FAP, Ferrari LS, Fattori V, Fernandes GSA, Casella AM, Alves Filho JCF, Cunha TM, Cunha F de Q, Casagrande R, Verri WA. The citrus flavanone naringenin reduces gout-induced joint pain and inflammation in mice by inhibiting the activation of NFκB and macrophage release of IL-1β [Internet]. Journal of Functional Foods. 2018 ; 48 106-116.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1016/j.jff.2018.06.025
Vancouver
Ruiz-Miyazawa KW, Borghi SM, Ribeiro FAP, Ferrari LS, Fattori V, Fernandes GSA, Casella AM, Alves Filho JCF, Cunha TM, Cunha F de Q, Casagrande R, Verri WA. The citrus flavanone naringenin reduces gout-induced joint pain and inflammation in mice by inhibiting the activation of NFκB and macrophage release of IL-1β [Internet]. Journal of Functional Foods. 2018 ; 48 106-116.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1016/j.jff.2018.06.025
Artigo de periodico
A crucial role for IL-6 in the CNS of rats during fever induced by the injection of live E. coli (2012)
Soares, Denis de Melo; Figueiredo, Maria Jose; Martins, Juliano Manvailer; Machado, Renes de Resende; Sorgi, Carlos Arterio; Faccioli, Lucia Helena; Alves-Filho, José Carlos; Cunha, Fernando de Queiroz; Souza, Glória Emília Petto de
Source: Medical Microbiology and Immunology. Unidades: FCFRP, FMRP
Subjects: ESCHERICHIA COLI, FEBRE, CITOCINAS, INFECÇÕES BACTERIANAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SOARES, Denis de Melo et al. A crucial role for IL-6 in the CNS of rats during fever induced by the injection of live E. coli. Medical Microbiology and Immunology, v. 201, n. 1, p. 47-60, 2012Tradução . . Disponível em: https://doi.org/10.1007/s00430-011-0204-3. Acesso em: 05 jul. 2024.
APA
Soares, D. de M., Figueiredo, M. J., Martins, J. M., Machado, R. de R., Sorgi, C. A., Faccioli, L. H., et al. (2012). A crucial role for IL-6 in the CNS of rats during fever induced by the injection of live E. coli. Medical Microbiology and Immunology, 201( 1), 47-60. doi:10.1007/s00430-011-0204-3
NLM
Soares D de M, Figueiredo MJ, Martins JM, Machado R de R, Sorgi CA, Faccioli LH, Alves-Filho JC, Cunha F de Q, Souza GEP de. A crucial role for IL-6 in the CNS of rats during fever induced by the injection of live E. coli [Internet]. Medical Microbiology and Immunology. 2012 ; 201( 1): 47-60.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1007/s00430-011-0204-3
Vancouver
Soares D de M, Figueiredo MJ, Martins JM, Machado R de R, Sorgi CA, Faccioli LH, Alves-Filho JC, Cunha F de Q, Souza GEP de. A crucial role for IL-6 in the CNS of rats during fever induced by the injection of live E. coli [Internet]. Medical Microbiology and Immunology. 2012 ; 201( 1): 47-60.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1007/s00430-011-0204-3
Artigo de periodico
IL-33 attenuates EAE by suppressing IL-17 and IFN-y production and inducing alternatively activated macrophages (2012)
Jiang, Hui-Rong; Milovanovic, Marija; Allan, Debbie; Niedbala, Wanda; Besnard, Anne-Galle; Fukada, Sandra Y.; Alves Filho, José Carlos Farias; Togbe, Dieudonnée; Goodyear, Carl S.; Linington, Christopher; Xu, Damo; Lukic, Miodrag L.; Liew, Foo Y.
Source: European Journal of Immunology. Unidade: FMRP
Subjects: MACRÓFAGOS, DOENÇAS AUTOIMUNES (EXPERIMENTAÇÃO)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
JIANG, Hui-Rong et al. IL-33 attenuates EAE by suppressing IL-17 and IFN-y production and inducing alternatively activated macrophages. European Journal of Immunology, v. 42, n. 7, p. 1804-1814, 2012Tradução . . Disponível em: https://doi.org/10.1002/eji.201141947. Acesso em: 05 jul. 2024.
APA
Jiang, H. -R., Milovanovic, M., Allan, D., Niedbala, W., Besnard, A. -G., Fukada, S. Y., et al. (2012). IL-33 attenuates EAE by suppressing IL-17 and IFN-y production and inducing alternatively activated macrophages. European Journal of Immunology, 42( 7), 1804-1814. doi:10.1002/eji.201141947
NLM
Jiang H-R, Milovanovic M, Allan D, Niedbala W, Besnard A-G, Fukada SY, Alves Filho JCF, Togbe D, Goodyear CS, Linington C, Xu D, Lukic ML, Liew FY. IL-33 attenuates EAE by suppressing IL-17 and IFN-y production and inducing alternatively activated macrophages [Internet]. European Journal of Immunology. 2012 ; 42( 7): 1804-1814.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1002/eji.201141947
Vancouver
Jiang H-R, Milovanovic M, Allan D, Niedbala W, Besnard A-G, Fukada SY, Alves Filho JCF, Togbe D, Goodyear CS, Linington C, Xu D, Lukic ML, Liew FY. IL-33 attenuates EAE by suppressing IL-17 and IFN-y production and inducing alternatively activated macrophages [Internet]. European Journal of Immunology. 2012 ; 42( 7): 1804-1814.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1002/eji.201141947
Artigo de periodico
Regulation of type 17 helper T-cell function by nitric oxide during inflammation (2011)
Niedbala, Wanda; Alves-Filho, José Carlos; Fukada, Sandra Y.; Vieira, Sílvio Manfredo; Mitani, Akio; Sonego, Fabiane; Mirchandani, Ananda; Nascimento, Daniele C.; Cunha, Fernando de Queiroz; Liew, Foo Y.
Source: Proceedings National Academy of Sciences. Unidade: FMRP
Subjects: ÓXIDO NÍTRICO, DOENÇAS, SISTEMA IMUNE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NIEDBALA, Wanda et al. Regulation of type 17 helper T-cell function by nitric oxide during inflammation. Proceedings National Academy of Sciences, v. 108, n. 22, p. 9220-9225, 2011Tradução . . Disponível em: https://doi.org/10.1073/pnas.1100667108. Acesso em: 05 jul. 2024.
APA
Niedbala, W., Alves-Filho, J. C., Fukada, S. Y., Vieira, S. M., Mitani, A., Sonego, F., et al. (2011). Regulation of type 17 helper T-cell function by nitric oxide during inflammation. Proceedings National Academy of Sciences, 108( 22), 9220-9225. doi:10.1073/pnas.1100667108
NLM
Niedbala W, Alves-Filho JC, Fukada SY, Vieira SM, Mitani A, Sonego F, Mirchandani A, Nascimento DC, Cunha F de Q, Liew FY. Regulation of type 17 helper T-cell function by nitric oxide during inflammation [Internet]. Proceedings National Academy of Sciences. 2011 ; 108( 22): 9220-9225.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1073/pnas.1100667108
Vancouver
Niedbala W, Alves-Filho JC, Fukada SY, Vieira SM, Mitani A, Sonego F, Mirchandani A, Nascimento DC, Cunha F de Q, Liew FY. Regulation of type 17 helper T-cell function by nitric oxide during inflammation [Internet]. Proceedings National Academy of Sciences. 2011 ; 108( 22): 9220-9225.[citado 2024 jul. 05 ] Available from: https://doi.org/10.1073/pnas.1100667108

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