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Artigo de periodico
Pharmacological activation of nuclear factor erythroid 2-related factor-2 prevents hyperglycemia-induced renal oxidative damage: possible involvement of O-GlcNAcylation (2024)
Costa, Rafael Menezes da ; Dias, Mayara C.; Alves, Juliano Vilela ; Silva, João Lucas M.; Rodrigues, Daniel; Silva, Josiane Fernandes da ; Francescato, Heloisa Della Coletta; Ramalho, Leandra Naira Zambelli ; Coimbra, Terezila Machado ; Passaglia, Rita de Cassia Aleixo Tostes
Source: Biochemical Pharmacology. Unidade: FMRP
Subjects: RIM, HIPERGLICEMIA, ESTRESSE OXIDATIVO, ESPÉCIES REATIVAS DE OXIGÊNIO
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COSTA, Rafael Menezes da et al. Pharmacological activation of nuclear factor erythroid 2-related factor-2 prevents hyperglycemia-induced renal oxidative damage: possible involvement of O-GlcNAcylation. Biochemical Pharmacology, v. 220, p. 1-10, 2024Tradução . . Disponível em: https://doi.org/10.1016/j.bcp.2023.115982. Acesso em: 14 nov. 2024.
APA
Costa, R. M. da, Dias, M. C., Alves, J. V., Silva, J. L. M., Rodrigues, D., Silva, J. F. da, et al. (2024). Pharmacological activation of nuclear factor erythroid 2-related factor-2 prevents hyperglycemia-induced renal oxidative damage: possible involvement of O-GlcNAcylation. Biochemical Pharmacology, 220, 1-10. doi:10.1016/j.bcp.2023.115982
NLM
Costa RM da, Dias MC, Alves JV, Silva JLM, Rodrigues D, Silva JF da, Francescato HDC, Ramalho LNZ, Coimbra TM, Passaglia R de CAT. Pharmacological activation of nuclear factor erythroid 2-related factor-2 prevents hyperglycemia-induced renal oxidative damage: possible involvement of O-GlcNAcylation [Internet]. Biochemical Pharmacology. 2024 ; 220 1-10.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.bcp.2023.115982
Vancouver
Costa RM da, Dias MC, Alves JV, Silva JLM, Rodrigues D, Silva JF da, Francescato HDC, Ramalho LNZ, Coimbra TM, Passaglia R de CAT. Pharmacological activation of nuclear factor erythroid 2-related factor-2 prevents hyperglycemia-induced renal oxidative damage: possible involvement of O-GlcNAcylation [Internet]. Biochemical Pharmacology. 2024 ; 220 1-10.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.bcp.2023.115982
Artigo de periodico
Ethanol consumption favors pro-contractile phenotype of perivascular adipose tissue: a role for interleukin-6 (2023)
Simplicio, Janaina Aparecida ; Dourado, Thales de Mileto Henrique; Awata, Wanessa Mayumi Carvalho; Vale, Gabriel Tavares do ; Dias, Vinícius Ribeiro; Barros, Paula Rodrigues de ; Martinis, Bruno Spinosa de ; Tostes, Rita de Cássia Aleixo ; Tirapelli, Carlos Renato
Source: Life Sciences. Unidades: FFCLRP, FMRP, EERP
Subjects: ETANOL, INTERLEUCINA 6, TECIDO ADIPOSO, ESTRESSE OXIDATIVO, HIPERTENSÃO
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SIMPLICIO, Janaina Aparecida et al. Ethanol consumption favors pro-contractile phenotype of perivascular adipose tissue: a role for interleukin-6. Life Sciences, v. 319, p. 1-10, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2023.121526. Acesso em: 14 nov. 2024.
APA
Simplicio, J. A., Dourado, T. de M. H., Awata, W. M. C., Vale, G. T. do, Dias, V. R., Barros, P. R. de, et al. (2023). Ethanol consumption favors pro-contractile phenotype of perivascular adipose tissue: a role for interleukin-6. Life Sciences, 319, 1-10. doi:10.1016/j.lfs.2023.121526
NLM
Simplicio JA, Dourado T de MH, Awata WMC, Vale GT do, Dias VR, Barros PR de, Martinis BS de, Tostes R de CA, Tirapelli CR. Ethanol consumption favors pro-contractile phenotype of perivascular adipose tissue: a role for interleukin-6 [Internet]. Life Sciences. 2023 ; 319 1-10.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2023.121526
Vancouver
Simplicio JA, Dourado T de MH, Awata WMC, Vale GT do, Dias VR, Barros PR de, Martinis BS de, Tostes R de CA, Tirapelli CR. Ethanol consumption favors pro-contractile phenotype of perivascular adipose tissue: a role for interleukin-6 [Internet]. Life Sciences. 2023 ; 319 1-10.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2023.121526
Artigo de periodico-carta/editorial
Neurobehavioral toxicity induced by environmental contaminants [Editorial]: mechanisms of neurodegeneration and neuroprotection, volume II (2023)
Dorta, Daniel Junqueira ; Oliveira, Danielle Palma de ; Campos, Alline Cristina de ; Aschner, Michael
Source: Frontiers in Pharmacology. Unidades: FFCLRP, FCFRP, FMRP
Subjects: CONTAMINAÇÃO AMBIENTAL, METAIS PESADOS, ESPÉCIES REATIVAS DE OXIGÊNIO, ESTRESSE OXIDATIVO, INFLAMAÇÃO, EPIGÊNESE GENÉTICA
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DORTA, Daniel Junqueira et al. Neurobehavioral toxicity induced by environmental contaminants [Editorial]: mechanisms of neurodegeneration and neuroprotection, volume II. Frontiers in Pharmacology. Lausanne: Faculdade de Filosofia, Ciências e Letras de Ribeirão Preto, Universidade de São Paulo. Disponível em: https://doi.org/10.3389/fphar.2023.1255471. Acesso em: 14 nov. 2024. , 2023
APA
Dorta, D. J., Oliveira, D. P. de, Campos, A. C. de, & Aschner, M. (2023). Neurobehavioral toxicity induced by environmental contaminants [Editorial]: mechanisms of neurodegeneration and neuroprotection, volume II. Frontiers in Pharmacology. Lausanne: Faculdade de Filosofia, Ciências e Letras de Ribeirão Preto, Universidade de São Paulo. doi:10.3389/fphar.2023.1255471
NLM
Dorta DJ, Oliveira DP de, Campos AC de, Aschner M. Neurobehavioral toxicity induced by environmental contaminants [Editorial]: mechanisms of neurodegeneration and neuroprotection, volume II [Internet]. Frontiers in Pharmacology. 2023 ; 14 1-3.[citado 2024 nov. 14 ] Available from: https://doi.org/10.3389/fphar.2023.1255471
Vancouver
Dorta DJ, Oliveira DP de, Campos AC de, Aschner M. Neurobehavioral toxicity induced by environmental contaminants [Editorial]: mechanisms of neurodegeneration and neuroprotection, volume II [Internet]. Frontiers in Pharmacology. 2023 ; 14 1-3.[citado 2024 nov. 14 ] Available from: https://doi.org/10.3389/fphar.2023.1255471
Artigo de periodico
Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress (2022)
Costa, Rafael Menezes da ; Alves-Lopes, Rhéure ; Alves, Juliano Vilela ; Servian, Carolina P.; Mestriner, Fabiola Leslie Antunes Cardoso; Carneiro, Fernando Silva ; Lobato, Núbia de S.; Tostes, Rita de Cássia Aleixo
Source: Frontiers in Physiology. Unidade: FMRP
Subjects: OBESIDADE, ESTRESSE OXIDATIVO, TESTOSTERONA, DOENÇAS VASCULARES
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COSTA, Rafael Menezes da et al. Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress. Frontiers in Physiology, v. 13, p. 1-14, 2022Tradução . . Disponível em: https://doi.org/10.3389/fphys.2022.837603. Acesso em: 14 nov. 2024.
APA
Costa, R. M. da, Alves-Lopes, R., Alves, J. V., Servian, C. P., Mestriner, F. L. A. C., Carneiro, F. S., et al. (2022). Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress. Frontiers in Physiology, 13, 1-14. doi:10.3389/fphys.2022.837603
NLM
Costa RM da, Alves-Lopes R, Alves JV, Servian CP, Mestriner FLAC, Carneiro FS, Lobato N de S, Tostes R de CA. Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress [Internet]. Frontiers in Physiology. 2022 ; 13 1-14.[citado 2024 nov. 14 ] Available from: https://doi.org/10.3389/fphys.2022.837603
Vancouver
Costa RM da, Alves-Lopes R, Alves JV, Servian CP, Mestriner FLAC, Carneiro FS, Lobato N de S, Tostes R de CA. Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress [Internet]. Frontiers in Physiology. 2022 ; 13 1-14.[citado 2024 nov. 14 ] Available from: https://doi.org/10.3389/fphys.2022.837603
Artigo de periodico
Melatonin regulates antioxidant defense and inflammatory response by activating Nrf2–dependent mechanisms and inhibiting NFkappaB expression in middle-aged T. cruzi infected rats (2022)
Brazão, Vânia; Colato, Rafaela Pravato ; Santello, Fabricia Helena; Duarte, Andressa ; Goulart, Amanda; Oliveira, Pedro Alexandre Sampaio ; Silva, Carla Brigagão Pacheco da ; Tirapelli, Carlos Renato ; Costa, Rafael Menezes da ; Tostes, Rita de Cássia Aleixo ; Prado Júnior, José Clóvis do
Source: Experimental Gerontology. Unidades: EERP, FMRP, FCFRP
Subjects: ANTIOXIDANTES, DOENÇA DE CHAGAS, PERÓXIDO DE HIDROGÊNIO, MELATONINA, ESTRESSE OXIDATIVO
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BRAZÃO, Vânia et al. Melatonin regulates antioxidant defense and inflammatory response by activating Nrf2–dependent mechanisms and inhibiting NFkappaB expression in middle-aged T. cruzi infected rats. Experimental Gerontology, v. 167, p. 1-9, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.exger.2022.111895. Acesso em: 14 nov. 2024.
APA
Brazão, V., Colato, R. P., Santello, F. H., Duarte, A., Goulart, A., Oliveira, P. A. S., et al. (2022). Melatonin regulates antioxidant defense and inflammatory response by activating Nrf2–dependent mechanisms and inhibiting NFkappaB expression in middle-aged T. cruzi infected rats. Experimental Gerontology, 167, 1-9. doi:10.1016/j.exger.2022.111895
NLM
Brazão V, Colato RP, Santello FH, Duarte A, Goulart A, Oliveira PAS, Silva CBP da, Tirapelli CR, Costa RM da, Tostes R de CA, Prado Júnior JC do. Melatonin regulates antioxidant defense and inflammatory response by activating Nrf2–dependent mechanisms and inhibiting NFkappaB expression in middle-aged T. cruzi infected rats [Internet]. Experimental Gerontology. 2022 ; 167 1-9.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.exger.2022.111895
Vancouver
Brazão V, Colato RP, Santello FH, Duarte A, Goulart A, Oliveira PAS, Silva CBP da, Tirapelli CR, Costa RM da, Tostes R de CA, Prado Júnior JC do. Melatonin regulates antioxidant defense and inflammatory response by activating Nrf2–dependent mechanisms and inhibiting NFkappaB expression in middle-aged T. cruzi infected rats [Internet]. Experimental Gerontology. 2022 ; 167 1-9.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.exger.2022.111895
Artigo de periodico
Mitochondrial photobiomodulation as a neurotherapeutic strategy for epilepsy (2022)
Cardoso, Fabrízio dos Santos; Gonzalez-Lima, Francisco; Coimbra, Norberto Cysne
Source: Frontiers in Neurology. Unidade: FMRP
Subjects: EPILEPSIA, TERAPIA A LASER, MITOCÔNDRIAS, ESTRESSE OXIDATIVO, CONVULSÕES
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CARDOSO, Fabrízio dos Santos e GONZALEZ-LIMA, Francisco e COIMBRA, Norberto Cysne. Mitochondrial photobiomodulation as a neurotherapeutic strategy for epilepsy. Frontiers in Neurology, v. 13, p. 1-5, 2022Tradução . . Disponível em: https://doi.org/10.3389/fneur.2022.873496. Acesso em: 14 nov. 2024.
APA
Cardoso, F. dos S., Gonzalez-Lima, F., & Coimbra, N. C. (2022). Mitochondrial photobiomodulation as a neurotherapeutic strategy for epilepsy. Frontiers in Neurology, 13, 1-5. doi:10.3389/fneur.2022.873496
NLM
Cardoso F dos S, Gonzalez-Lima F, Coimbra NC. Mitochondrial photobiomodulation as a neurotherapeutic strategy for epilepsy [Internet]. Frontiers in Neurology. 2022 ; 13 1-5.[citado 2024 nov. 14 ] Available from: https://doi.org/10.3389/fneur.2022.873496
Vancouver
Cardoso F dos S, Gonzalez-Lima F, Coimbra NC. Mitochondrial photobiomodulation as a neurotherapeutic strategy for epilepsy [Internet]. Frontiers in Neurology. 2022 ; 13 1-5.[citado 2024 nov. 14 ] Available from: https://doi.org/10.3389/fneur.2022.873496
Artigo de periodico
Inducible nitric oxide synthase (iNOS) mediates ethanol-induced redox imbalance and upregulation of inflammatory cytokines in the kidney (2021)
Silva, Carla Beatriz Pereira da ; Ceron, Carla S.; Mendes, Atlante Silva; Martinis, Bruno Spinosa de ; Castro, Michele Mazzaron de; Tirapelli, Carlos Renato
Source: Canadian Journal of Physiology and Pharmacology. Unidades: FFCLRP, FMRP, EERP
Subjects: BEBIDAS ALCOÓLICAS, CREATINA, CITOCINAS, ETANOL, INFLAMAÇÃO, NEFROPATIAS, ESTRESSE OXIDATIVO, ESPÉCIES REATIVAS DE OXIGÊNIO
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ABNT
SILVA, Carla Beatriz Pereira da et al. Inducible nitric oxide synthase (iNOS) mediates ethanol-induced redox imbalance and upregulation of inflammatory cytokines in the kidney. Canadian Journal of Physiology and Pharmacology, v. 99, n. 10, p. 1016-1025, 2021Tradução . . Disponível em: https://doi.org/10.1139/cjpp-2021-0108. Acesso em: 14 nov. 2024.
APA
Silva, C. B. P. da, Ceron, C. S., Mendes, A. S., Martinis, B. S. de, Castro, M. M. de, & Tirapelli, C. R. (2021). Inducible nitric oxide synthase (iNOS) mediates ethanol-induced redox imbalance and upregulation of inflammatory cytokines in the kidney. Canadian Journal of Physiology and Pharmacology, 99( 10), 1016-1025. doi:10.1139/cjpp-2021-0108
NLM
Silva CBP da, Ceron CS, Mendes AS, Martinis BS de, Castro MM de, Tirapelli CR. Inducible nitric oxide synthase (iNOS) mediates ethanol-induced redox imbalance and upregulation of inflammatory cytokines in the kidney [Internet]. Canadian Journal of Physiology and Pharmacology. 2021 ; 99( 10): 1016-1025.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1139/cjpp-2021-0108
Vancouver
Silva CBP da, Ceron CS, Mendes AS, Martinis BS de, Castro MM de, Tirapelli CR. Inducible nitric oxide synthase (iNOS) mediates ethanol-induced redox imbalance and upregulation of inflammatory cytokines in the kidney [Internet]. Canadian Journal of Physiology and Pharmacology. 2021 ; 99( 10): 1016-1025.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1139/cjpp-2021-0108
Artigo de periodico
Nitrate and nitrite-based therapy to attenuate cardiovascular remodelling in arterial hypertension (2021)
Guimarães, Danielle Aparecida; Batista, Rose Inês Matos ; Tanus-Santos, José Eduardo
Source: Basic & Clinical Pharmacology & Toxicology. Unidade: FMRP
Subjects: FÁRMACOS (SISTEMA CARDIOVASCULAR), ARTÉRIAS, HIPERTENSÃO, HIPERTROFIA VENTRICULAR ESQUERDA, NITRATOS, ÓXIDO NÍTRICO, ESTRESSE OXIDATIVO, SISTEMA RENINA-ANGIOTENSINA
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ABNT
GUIMARÃES, Danielle Aparecida e BATISTA, Rose Inês Matos e TANUS-SANTOS, José Eduardo. Nitrate and nitrite-based therapy to attenuate cardiovascular remodelling in arterial hypertension. Basic & Clinical Pharmacology & Toxicology, v. 128, n. 1, p. 9-17, 2021Tradução . . Disponível em: https://doi.org/10.1111/bcpt.13474. Acesso em: 14 nov. 2024.
APA
Guimarães, D. A., Batista, R. I. M., & Tanus-Santos, J. E. (2021). Nitrate and nitrite-based therapy to attenuate cardiovascular remodelling in arterial hypertension. Basic & Clinical Pharmacology & Toxicology, 128( 1), 9-17. doi:10.1111/bcpt.13474
NLM
Guimarães DA, Batista RIM, Tanus-Santos JE. Nitrate and nitrite-based therapy to attenuate cardiovascular remodelling in arterial hypertension [Internet]. Basic & Clinical Pharmacology & Toxicology. 2021 ; 128( 1): 9-17.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1111/bcpt.13474
Vancouver
Guimarães DA, Batista RIM, Tanus-Santos JE. Nitrate and nitrite-based therapy to attenuate cardiovascular remodelling in arterial hypertension [Internet]. Basic & Clinical Pharmacology & Toxicology. 2021 ; 128( 1): 9-17.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1111/bcpt.13474
Artigo de periodico
Aldosterone negatively regulates Nrf2 activity: an additional mechanism contributing to oxidative stress and vascular dysfunction by Aldosterone (2021)
Rodrigues, Daniel ; Costa, Tiago Januário da ; Silva, Josiane Fernandes da ; Oliveira Neto, José Teles de ; Alves, Juliano Vilela ; Fedoce, Aline Garcia ; Costa, Rafael Menezes da ; Tostes, Rita de Cássia Aleixo
Source: International Journal of Molecular Sciences. Unidade: FMRP
Subjects: ALDOSTERONA, DOENÇAS VASCULARES, ESTRESSE OXIDATIVO
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RODRIGUES, Daniel et al. Aldosterone negatively regulates Nrf2 activity: an additional mechanism contributing to oxidative stress and vascular dysfunction by Aldosterone. International Journal of Molecular Sciences, v. 22, n. 11, p. 1-16, 2021Tradução . . Disponível em: https://doi.org/10.3390/ijms22116154. Acesso em: 14 nov. 2024.
APA
Rodrigues, D., Costa, T. J. da, Silva, J. F. da, Oliveira Neto, J. T. de, Alves, J. V., Fedoce, A. G., et al. (2021). Aldosterone negatively regulates Nrf2 activity: an additional mechanism contributing to oxidative stress and vascular dysfunction by Aldosterone. International Journal of Molecular Sciences, 22( 11), 1-16. doi:10.3390/ijms22116154
NLM
Rodrigues D, Costa TJ da, Silva JF da, Oliveira Neto JT de, Alves JV, Fedoce AG, Costa RM da, Tostes R de CA. Aldosterone negatively regulates Nrf2 activity: an additional mechanism contributing to oxidative stress and vascular dysfunction by Aldosterone [Internet]. International Journal of Molecular Sciences. 2021 ; 22( 11): 1-16.[citado 2024 nov. 14 ] Available from: https://doi.org/10.3390/ijms22116154
Vancouver
Rodrigues D, Costa TJ da, Silva JF da, Oliveira Neto JT de, Alves JV, Fedoce AG, Costa RM da, Tostes R de CA. Aldosterone negatively regulates Nrf2 activity: an additional mechanism contributing to oxidative stress and vascular dysfunction by Aldosterone [Internet]. International Journal of Molecular Sciences. 2021 ; 22( 11): 1-16.[citado 2024 nov. 14 ] Available from: https://doi.org/10.3390/ijms22116154
Artigo de periodico
Lysophosphatidylcholine induces oxidative stress in human endothelial cells via NOX5 activation: implications in atherosclerosis (2021)
Silva, Josiane Fernandes da; Alves, Juliano Vilela; Silva Neto, Júlio Alves da; Costa, Rafael Menezes da; Neves, Karla B.; Lopes, Rheure Alves Moreira; Carmargo, Livia L.; Rios, Francisco J.; Montezano, Augusto C.; Touyz, Rhian M.; Tostes, Rita de Cássia Aleixo
Source: Clinical Science. Unidade: FMRP
Subjects: ARTERIOSCLEROSE, CÁLCIO, CÉLULAS CULTIVADAS, CÉLULAS ENDOTELIAIS, ESTRESSE OXIDATIVO, ESPÉCIES REATIVAS DE OXIGÊNIO
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ABNT
SILVA, Josiane Fernandes da et al. Lysophosphatidylcholine induces oxidative stress in human endothelial cells via NOX5 activation: implications in atherosclerosis. Clinical Science, v. 135, n. 15, p. 1845-1858, 2021Tradução . . Disponível em: https://doi.org/10.1042/CS20210468. Acesso em: 14 nov. 2024.
APA
Silva, J. F. da, Alves, J. V., Silva Neto, J. A. da, Costa, R. M. da, Neves, K. B., Lopes, R. A. M., et al. (2021). Lysophosphatidylcholine induces oxidative stress in human endothelial cells via NOX5 activation: implications in atherosclerosis. Clinical Science, 135( 15), 1845-1858. doi:10.1042/CS20210468
NLM
Silva JF da, Alves JV, Silva Neto JA da, Costa RM da, Neves KB, Lopes RAM, Carmargo L L, Rios FJ, Montezano AC, Touyz RM, Tostes R de CA. Lysophosphatidylcholine induces oxidative stress in human endothelial cells via NOX5 activation: implications in atherosclerosis [Internet]. Clinical Science. 2021 ; 135( 15): 1845-1858.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1042/CS20210468
Vancouver
Silva JF da, Alves JV, Silva Neto JA da, Costa RM da, Neves KB, Lopes RAM, Carmargo L L, Rios FJ, Montezano AC, Touyz RM, Tostes R de CA. Lysophosphatidylcholine induces oxidative stress in human endothelial cells via NOX5 activation: implications in atherosclerosis [Internet]. Clinical Science. 2021 ; 135( 15): 1845-1858.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1042/CS20210468
Trabalho de evento-resumo periodico
Sodium hydrogen sulfide improves renovascular hypertension-induced maladaptive cardiac remodeling and diastolic dysfunction by reducing oxidative stress and mmp activity (2021)
Pernomian, Laena ; Mello, Marcela Maria Blascke de ; Castro, Michele Mazzaron de
Source: Journal of Hypertension. Conference titles: International SHR symposium SHR: genetics and disease model guided novel therapeutic target discovery in hypertension. Unidade: FMRP
Subjects: HIPERTENSÃO, METALOPROTEINASES, ESTRESSE OXIDATIVO, ESPÉCIES REATIVAS DE OXIGÊNIO
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ABNT
PERNOMIAN, Laena e MELLO, Marcela Maria Blascke de e CASTRO, Michele Mazzaron de. Sodium hydrogen sulfide improves renovascular hypertension-induced maladaptive cardiac remodeling and diastolic dysfunction by reducing oxidative stress and mmp activity. Journal of Hypertension. Philadelphia: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Disponível em: https://doi.org/10.1097/01.hjh.0000749136.03071.6e. Acesso em: 14 nov. 2024. , 2021
APA
Pernomian, L., Mello, M. M. B. de, & Castro, M. M. de. (2021). Sodium hydrogen sulfide improves renovascular hypertension-induced maladaptive cardiac remodeling and diastolic dysfunction by reducing oxidative stress and mmp activity. Journal of Hypertension. Philadelphia: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. doi:10.1097/01.hjh.0000749136.03071.6e
NLM
Pernomian L, Mello MMB de, Castro MM de. Sodium hydrogen sulfide improves renovascular hypertension-induced maladaptive cardiac remodeling and diastolic dysfunction by reducing oxidative stress and mmp activity [Internet]. Journal of Hypertension. 2021 ; 39 e394.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1097/01.hjh.0000749136.03071.6e
Vancouver
Pernomian L, Mello MMB de, Castro MM de. Sodium hydrogen sulfide improves renovascular hypertension-induced maladaptive cardiac remodeling and diastolic dysfunction by reducing oxidative stress and mmp activity [Internet]. Journal of Hypertension. 2021 ; 39 e394.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1097/01.hjh.0000749136.03071.6e
Artigo de periodico
Matrix metalloproteinases and arterial hypertension: Role of oxidative stress and nitric oxide in vascular functional and structural alterations (2021)
Prado, Alejandro F.; Batista, Rose Inês Matos ; Tanus-Santos, José Eduardo ; Gerlach, Raquel Fernanda
Source: Biomolecules. Unidades: FMRP, FORP
Subjects: METALOPROTEINASES, HIPERTENSÃO, ESTRESSE OXIDATIVO, ÓXIDO NÍTRICO, FISIOPATOLOGIA, DOENÇAS CARDIOVASCULARES
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ABNT
PRADO, Alejandro F. et al. Matrix metalloproteinases and arterial hypertension: Role of oxidative stress and nitric oxide in vascular functional and structural alterations. Biomolecules, v. 11, n. 4, p. 1-17, 2021Tradução . . Disponível em: https://doi.org/10.3390/biom11040585. Acesso em: 14 nov. 2024.
APA
Prado, A. F., Batista, R. I. M., Tanus-Santos, J. E., & Gerlach, R. F. (2021). Matrix metalloproteinases and arterial hypertension: Role of oxidative stress and nitric oxide in vascular functional and structural alterations. Biomolecules, 11( 4), 1-17. doi:10.3390/biom11040585
NLM
Prado AF, Batista RIM, Tanus-Santos JE, Gerlach RF. Matrix metalloproteinases and arterial hypertension: Role of oxidative stress and nitric oxide in vascular functional and structural alterations [Internet]. Biomolecules. 2021 ; 11( 4): 1-17.[citado 2024 nov. 14 ] Available from: https://doi.org/10.3390/biom11040585
Vancouver
Prado AF, Batista RIM, Tanus-Santos JE, Gerlach RF. Matrix metalloproteinases and arterial hypertension: Role of oxidative stress and nitric oxide in vascular functional and structural alterations [Internet]. Biomolecules. 2021 ; 11( 4): 1-17.[citado 2024 nov. 14 ] Available from: https://doi.org/10.3390/biom11040585
Artigo de periodico
Exposure to low doses of malathion during juvenile and peripubertal periods impairs testicular and sperm parameters in rats: role of oxidative stress and testosterone (2020)
Erthal, Rafaela Pires; Staurengo-Ferrari, Larissa; Fattori, Victor; Luiz, Karen Gomes; Cunha, Fernando de Queiroz ; Pescim, Rodrigo Rossetto; Cecchini, Rubens; Verri Júnior, Waldiceu Aparecido; Guarnier, Flávia Alessandra; Fernandes, Glaura Scantamburlo Alves
Source: Reproductive Toxicology. Unidade: FMRP
Subjects: TESTOSTERONA, SÊMEN, ANTIPARASITÁRIOS, PUBERDADE, ESTRESSE OXIDATIVO
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ABNT
ERTHAL, Rafaela Pires et al. Exposure to low doses of malathion during juvenile and peripubertal periods impairs testicular and sperm parameters in rats: role of oxidative stress and testosterone. Reproductive Toxicology, v. 96, p. 17-26, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.reprotox.2020.05.013. Acesso em: 14 nov. 2024.
APA
Erthal, R. P., Staurengo-Ferrari, L., Fattori, V., Luiz, K. G., Cunha, F. de Q., Pescim, R. R., et al. (2020). Exposure to low doses of malathion during juvenile and peripubertal periods impairs testicular and sperm parameters in rats: role of oxidative stress and testosterone. Reproductive Toxicology, 96, 17-26. doi:10.1016/j.reprotox.2020.05.013
NLM
Erthal RP, Staurengo-Ferrari L, Fattori V, Luiz KG, Cunha F de Q, Pescim RR, Cecchini R, Verri Júnior WA, Guarnier FA, Fernandes GSA. Exposure to low doses of malathion during juvenile and peripubertal periods impairs testicular and sperm parameters in rats: role of oxidative stress and testosterone [Internet]. Reproductive Toxicology. 2020 ; 96 17-26.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.reprotox.2020.05.013
Vancouver
Erthal RP, Staurengo-Ferrari L, Fattori V, Luiz KG, Cunha F de Q, Pescim RR, Cecchini R, Verri Júnior WA, Guarnier FA, Fernandes GSA. Exposure to low doses of malathion during juvenile and peripubertal periods impairs testicular and sperm parameters in rats: role of oxidative stress and testosterone [Internet]. Reproductive Toxicology. 2020 ; 96 17-26.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.reprotox.2020.05.013
Artigo de periodico
One-week high-intensity interval training increases hippocampal plasticity and mitochondrial content without changes in redox state (2020)
Santos, Jonathas Rodrigo dos; Bortolanza, Mariza; Ferrari, Gustavo Duarte ; Lanfredi, Guilherme Pauperio ; Nascimento, Glauce Crivelaro do ; Azzolini, Ana Elisa Caleiro Seixas ; Del Bel, Elaine Aparecida ; Campos, Alline Cristina de; Faça, Vitor Marcel ; Vulczak, Anderson; Alberici, Luciane Carla
Source: Antioxidants. Unidades: FMRP, FORP, FCFRP
Subjects: EXERCÍCIO FÍSICO, ESTRESSE OXIDATIVO, NEUROGENÉTICA, CÉREBRO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANTOS, Jonathas Rodrigo dos et al. One-week high-intensity interval training increases hippocampal plasticity and mitochondrial content without changes in redox state. Antioxidants, v. 9, n. 5, 2020Tradução . . Disponível em: https://doi.org/10.3390/antiox9050445. Acesso em: 14 nov. 2024.
APA
Santos, J. R. dos, Bortolanza, M., Ferrari, G. D., Lanfredi, G. P., Nascimento, G. C. do, Azzolini, A. E. C. S., et al. (2020). One-week high-intensity interval training increases hippocampal plasticity and mitochondrial content without changes in redox state. Antioxidants, 9( 5). doi:10.3390/antiox9050445
NLM
Santos JR dos, Bortolanza M, Ferrari GD, Lanfredi GP, Nascimento GC do, Azzolini AECS, Del Bel EA, Campos AC de, Faça VM, Vulczak A, Alberici LC. One-week high-intensity interval training increases hippocampal plasticity and mitochondrial content without changes in redox state [Internet]. Antioxidants. 2020 ; 9( 5):[citado 2024 nov. 14 ] Available from: https://doi.org/10.3390/antiox9050445
Vancouver
Santos JR dos, Bortolanza M, Ferrari GD, Lanfredi GP, Nascimento GC do, Azzolini AECS, Del Bel EA, Campos AC de, Faça VM, Vulczak A, Alberici LC. One-week high-intensity interval training increases hippocampal plasticity and mitochondrial content without changes in redox state [Internet]. Antioxidants. 2020 ; 9( 5):[citado 2024 nov. 14 ] Available from: https://doi.org/10.3390/antiox9050445
Artigo de periodico
Cannabidiol prevents LPS-induced microglial inflammation by inhibiting ROS/NF-κB-dependent signaling and glucose consumption (2020)
Pereira, Maurício dos Santos; Guimarães, Francisco Silveira ; Del Bel, Elaine Aparecida ; Raisman-Vozari, Rita; Michel, Patrick P.
Source: Glia. Unidades: FMRP, FORP
Subjects: CANABINOIDES, GLICOSE, ESTRESSE OXIDATIVO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PEREIRA, Maurício dos Santos et al. Cannabidiol prevents LPS-induced microglial inflammation by inhibiting ROS/NF-κB-dependent signaling and glucose consumption. Glia, v. 68, n. 3, p. 561-573, 2020Tradução . . Disponível em: https://doi.org/10.1002/glia.23738. Acesso em: 14 nov. 2024.
APA
Pereira, M. dos S., Guimarães, F. S., Del Bel, E. A., Raisman-Vozari, R., & Michel, P. P. (2020). Cannabidiol prevents LPS-induced microglial inflammation by inhibiting ROS/NF-κB-dependent signaling and glucose consumption. Glia, 68( 3), 561-573. doi:10.1002/glia.23738
NLM
Pereira M dos S, Guimarães FS, Del Bel EA, Raisman-Vozari R, Michel PP. Cannabidiol prevents LPS-induced microglial inflammation by inhibiting ROS/NF-κB-dependent signaling and glucose consumption [Internet]. Glia. 2020 ; 68( 3): 561-573.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1002/glia.23738
Vancouver
Pereira M dos S, Guimarães FS, Del Bel EA, Raisman-Vozari R, Michel PP. Cannabidiol prevents LPS-induced microglial inflammation by inhibiting ROS/NF-κB-dependent signaling and glucose consumption [Internet]. Glia. 2020 ; 68( 3): 561-573.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1002/glia.23738
Artigo de periodico
Acute restraint stress increases blood pressure and oxidative stress in the cardiorenal system of rats: a role for AT1 receptors (2020)
Vale, Gabriel Tavares do; Leoni, Drieli; Sousa, Arthur Henrique de; Gonzaga, Natália de Almeida; Uliana, Daniela L; La Gata, Davi C; Resstel, Leonardo Barbosa Moraes; Padovan, Cláudia Maria ; Tirapelli, Carlos Renato
Source: Stress. Unidades: FMRP, FFCLRP, EERP
Subjects: ESTRESSE, ESTRESSE OXIDATIVO, ESPÉCIES REATIVAS DE OXIGÊNIO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VALE, Gabriel Tavares do et al. Acute restraint stress increases blood pressure and oxidative stress in the cardiorenal system of rats: a role for AT1 receptors. Stress, v. 23, n. 3, p. 328-337, 2020Tradução . . Disponível em: https://doi.org/10.1080/10253890.2019.1675627. Acesso em: 14 nov. 2024.
APA
Vale, G. T. do, Leoni, D., Sousa, A. H. de, Gonzaga, N. de A., Uliana, D. L., La Gata, D. C., et al. (2020). Acute restraint stress increases blood pressure and oxidative stress in the cardiorenal system of rats: a role for AT1 receptors. Stress, 23( 3), 328-337. doi:10.1080/10253890.2019.1675627
NLM
Vale GT do, Leoni D, Sousa AH de, Gonzaga N de A, Uliana DL, La Gata DC, Resstel LBM, Padovan CM, Tirapelli CR. Acute restraint stress increases blood pressure and oxidative stress in the cardiorenal system of rats: a role for AT1 receptors [Internet]. Stress. 2020 ; 23( 3): 328-337.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1080/10253890.2019.1675627
Vancouver
Vale GT do, Leoni D, Sousa AH de, Gonzaga N de A, Uliana DL, La Gata DC, Resstel LBM, Padovan CM, Tirapelli CR. Acute restraint stress increases blood pressure and oxidative stress in the cardiorenal system of rats: a role for AT1 receptors [Internet]. Stress. 2020 ; 23( 3): 328-337.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1080/10253890.2019.1675627
Artigo de periodico
Matrix metalloproteinase (MMP)-2 activation by oxidative stress decreases aortic calponin-1 levels during hypertrophic remodeling in early hypertension (2019)
Mello, Marcela M. Blascke de; Parente, Juliana Montenegro ; Schulz, Richard; Castro, Michele Mazzaron de
Source: Vascular Pharmacology. Unidade: FMRP
Subjects: HIPERTENSÃO, METALOPROTEINASES, ESTRESSE OXIDATIVO, POLIPEPTÍDEOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MELLO, Marcela M. Blascke de et al. Matrix metalloproteinase (MMP)-2 activation by oxidative stress decreases aortic calponin-1 levels during hypertrophic remodeling in early hypertension. Vascular Pharmacology, v. 116, p. 36-44, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2018.10.002. Acesso em: 14 nov. 2024.
APA
Mello, M. M. B. de, Parente, J. M., Schulz, R., & Castro, M. M. de. (2019). Matrix metalloproteinase (MMP)-2 activation by oxidative stress decreases aortic calponin-1 levels during hypertrophic remodeling in early hypertension. Vascular Pharmacology, 116, 36-44. doi:10.1016/j.vph.2018.10.002
NLM
Mello MMB de, Parente JM, Schulz R, Castro MM de. Matrix metalloproteinase (MMP)-2 activation by oxidative stress decreases aortic calponin-1 levels during hypertrophic remodeling in early hypertension [Internet]. Vascular Pharmacology. 2019 ; 116 36-44.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.vph.2018.10.002
Vancouver
Mello MMB de, Parente JM, Schulz R, Castro MM de. Matrix metalloproteinase (MMP)-2 activation by oxidative stress decreases aortic calponin-1 levels during hypertrophic remodeling in early hypertension [Internet]. Vascular Pharmacology. 2019 ; 116 36-44.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.vph.2018.10.002
Artigo de periodico
Chronic ethanol consumption induces micturition dysfunction and alters the oxidative state of the urinary bladder (2019)
Vale, Gabriel Tavares do; Sousa, Arthur Henrique de; Gonzaga, Natália de Almeida; Oliveira, Mariana G. de; Justo, Alberto F. O; Alexandre, Eduardo C.; Tanus-Santos, José Eduardo ; Antunes, Edson; Tirapelli, Carlos Renato
Source: Canadian Journal of Physiology and Pharmacology. Unidades: FMRP, EERP
Subjects: ETANOL, ESTRESSE OXIDATIVO, BEXIGA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VALE, Gabriel Tavares do et al. Chronic ethanol consumption induces micturition dysfunction and alters the oxidative state of the urinary bladder. Canadian Journal of Physiology and Pharmacology, v. 97, n. 12, p. 1103-1114, 2019Tradução . . Disponível em: https://doi.org/10.1139/cjpp-2019-0143. Acesso em: 14 nov. 2024.
APA
Vale, G. T. do, Sousa, A. H. de, Gonzaga, N. de A., Oliveira, M. G. de, Justo, A. F. O., Alexandre, E. C., et al. (2019). Chronic ethanol consumption induces micturition dysfunction and alters the oxidative state of the urinary bladder. Canadian Journal of Physiology and Pharmacology, 97( 12), 1103-1114. doi:10.1139/cjpp-2019-0143
NLM
Vale GT do, Sousa AH de, Gonzaga N de A, Oliveira MG de, Justo AFO, Alexandre EC, Tanus-Santos JE, Antunes E, Tirapelli CR. Chronic ethanol consumption induces micturition dysfunction and alters the oxidative state of the urinary bladder [Internet]. Canadian Journal of Physiology and Pharmacology. 2019 ; 97( 12): 1103-1114.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1139/cjpp-2019-0143
Vancouver
Vale GT do, Sousa AH de, Gonzaga N de A, Oliveira MG de, Justo AFO, Alexandre EC, Tanus-Santos JE, Antunes E, Tirapelli CR. Chronic ethanol consumption induces micturition dysfunction and alters the oxidative state of the urinary bladder [Internet]. Canadian Journal of Physiology and Pharmacology. 2019 ; 97( 12): 1103-1114.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1139/cjpp-2019-0143
Artigo de periodico
Nrf2 as a potential mediator of cardiovascular risk in metabolic diseases (2019)
Costa, Rafael Menezes da; Rodrigues, Daniel; Pereira, Camila André; Silva, Josiane F. da; Alves, Juliano Vilela; Lobato, Nubia Souza; Tostes, Rita de Cássia Aleixo
Source: Frontiers in Pharmacology. Unidade: FMRP
Subjects: ESTRESSE OXIDATIVO, DOENÇAS CARDIOVASCULARES, DOENÇAS METABÓLICAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Rafael Menezes da et al. Nrf2 as a potential mediator of cardiovascular risk in metabolic diseases. Frontiers in Pharmacology, v. 10, 2019Tradução . . Disponível em: https://doi.org/10.3389/fphar.2019.00382. Acesso em: 14 nov. 2024.
APA
Costa, R. M. da, Rodrigues, D., Pereira, C. A., Silva, J. F. da, Alves, J. V., Lobato, N. S., & Tostes, R. de C. A. (2019). Nrf2 as a potential mediator of cardiovascular risk in metabolic diseases. Frontiers in Pharmacology, 10. doi:10.3389/fphar.2019.00382
NLM
Costa RM da, Rodrigues D, Pereira CA, Silva JF da, Alves JV, Lobato NS, Tostes R de CA. Nrf2 as a potential mediator of cardiovascular risk in metabolic diseases [Internet]. Frontiers in Pharmacology. 2019 ; 10[citado 2024 nov. 14 ] Available from: https://doi.org/10.3389/fphar.2019.00382
Vancouver
Costa RM da, Rodrigues D, Pereira CA, Silva JF da, Alves JV, Lobato NS, Tostes R de CA. Nrf2 as a potential mediator of cardiovascular risk in metabolic diseases [Internet]. Frontiers in Pharmacology. 2019 ; 10[citado 2024 nov. 14 ] Available from: https://doi.org/10.3389/fphar.2019.00382
Artigo de periodico
Chronic ethanol consumption increases reactive oxygen species generation and the synthesis of pro-inflammatory proteins in the heart through TNFR1-dependent mechanisms (2019)
Nakashima, Marcelo A.; Silva, Carla Beatriz Pereira da ; Gonzaga, Natália de Almeida; Simplicio, Janaina A.; Omoto, Ana Carolina Mieko ; Tirapelli, Luis Fernando; Tanus-Santos, José Eduardo; Tirapelli, Carlos Renato
Source: Cytokine. Unidades: FMRP, EERP
Subjects: ESTRESSE OXIDATIVO, ETANOL, CITOTOXINAS, SEPSE, INTERLEUCINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NAKASHIMA, Marcelo A. et al. Chronic ethanol consumption increases reactive oxygen species generation and the synthesis of pro-inflammatory proteins in the heart through TNFR1-dependent mechanisms. Cytokine, v. 121, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.cyto.2019.154734. Acesso em: 14 nov. 2024.
APA
Nakashima, M. A., Silva, C. B. P. da, Gonzaga, N. de A., Simplicio, J. A., Omoto, A. C. M., Tirapelli, L. F., et al. (2019). Chronic ethanol consumption increases reactive oxygen species generation and the synthesis of pro-inflammatory proteins in the heart through TNFR1-dependent mechanisms. Cytokine, 121. doi:10.1016/j.cyto.2019.154734
NLM
Nakashima MA, Silva CBP da, Gonzaga N de A, Simplicio JA, Omoto ACM, Tirapelli LF, Tanus-Santos JE, Tirapelli CR. Chronic ethanol consumption increases reactive oxygen species generation and the synthesis of pro-inflammatory proteins in the heart through TNFR1-dependent mechanisms [Internet]. Cytokine. 2019 ; 121[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.cyto.2019.154734
Vancouver
Nakashima MA, Silva CBP da, Gonzaga N de A, Simplicio JA, Omoto ACM, Tirapelli LF, Tanus-Santos JE, Tirapelli CR. Chronic ethanol consumption increases reactive oxygen species generation and the synthesis of pro-inflammatory proteins in the heart through TNFR1-dependent mechanisms [Internet]. Cytokine. 2019 ; 121[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.cyto.2019.154734

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