Nrf2 as a potential mediator of cardiovascular risk in metabolic diseases (2019)
- Authors:
- USP affiliated authors: PASSAGLIA, RITA DE CASSIA ALEIXO TOSTES - FMRP ; RODRIGUES, DANIEL - FMRP ; COSTA, RAFAEL MENEZES DA - FMRP ; PEREIRA, CAMILA ANDRÉ - FMRP ; SILVA, JOSIANE FERNANDES DA - FMRP ; ALVES, JULIANO VILELA - FMRP
- Unidade: FMRP
- DOI: 10.3389/fphar.2019.00382
- Subjects: ESTRESSE OXIDATIVO; DOENÇAS CARDIOVASCULARES; DOENÇAS METABÓLICAS
- Keywords: Oxidative stress; Cardiovascular risk; Metabolic diseases; Therapeutic target
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Frontiers in Pharmacology
- ISSN: 1663-9812
- Volume/Número/Paginação/Ano: v. 10, art. 382, 2019
- Status:
- Artigo publicado em periódico de acesso aberto (Gold Open Access)
- Versão do Documento:
- Versão publicada (Published version)
- Acessar versão aberta:
-
ABNT
COSTA, Rafael Menezes da et al. Nrf2 as a potential mediator of cardiovascular risk in metabolic diseases. Frontiers in Pharmacology, v. 10, 2019Tradução . . Disponível em: https://doi.org/10.3389/fphar.2019.00382. Acesso em: 28 mar. 2026. -
APA
Costa, R. M. da, Rodrigues, D., Pereira, C. A., Silva, J. F. da, Alves, J. V., Lobato, N. S., & Tostes, R. de C. A. (2019). Nrf2 as a potential mediator of cardiovascular risk in metabolic diseases. Frontiers in Pharmacology, 10. doi:10.3389/fphar.2019.00382 -
NLM
Costa RM da, Rodrigues D, Pereira CA, Silva JF da, Alves JV, Lobato NS, Tostes R de CA. Nrf2 as a potential mediator of cardiovascular risk in metabolic diseases [Internet]. Frontiers in Pharmacology. 2019 ; 10[citado 2026 mar. 28 ] Available from: https://doi.org/10.3389/fphar.2019.00382 -
Vancouver
Costa RM da, Rodrigues D, Pereira CA, Silva JF da, Alves JV, Lobato NS, Tostes R de CA. Nrf2 as a potential mediator of cardiovascular risk in metabolic diseases [Internet]. Frontiers in Pharmacology. 2019 ; 10[citado 2026 mar. 28 ] Available from: https://doi.org/10.3389/fphar.2019.00382 - Beclin-1-dependent autophagy protects perivascular adipose tissue function from hyperaldosteronism effects
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