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Artigo de periodico
Cannabidiol as an adjunctive treatment for acute bipolar depression: a pilot study (2024)
Pinto, Jairo Vinicius Merege de Mello Cruz; Crippa, José Alexandre de Souza ; Ceresér, Keila Maria; Vianna-Sulzbach, Miréia Fortes; Silveira Júnior, Érico de Moura; Rosa, Gabriel Santana da; Silva, Manoella Guatimuzim Testa da; Hizo, Gabriel Henrique; Medeiros, Leonardo Simão; Oliveira, Carlos Eduardo Santana de; Bristot, Giovana; Campos, Alline Cristina de ; Guimarães, Francisco Silveira ; Hallak, Jaime Eduardo Cecilio ; Zuardi, Antônio Waldo ; Yatham, Lakshmi N.; Kapczinski, Flávio; Kauer-Sant\2019anna, Márcia
Fonte: The Canadian Journal of Psychiatry. Unidades: FMRP, ESALQ
Assuntos: TRANSTORNO BIPOLAR, HUMOR, DEPRESSÃO, CANNABIS, CANABINOIDES, ENSAIO CLÍNICO CONTROLADO RANDOMIZADO
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ABNT
PINTO, Jairo Vinicius Merege de Mello Cruz et al. Cannabidiol as an adjunctive treatment for acute bipolar depression: a pilot study. The Canadian Journal of Psychiatry, v. 69, n. 4, p. 242-251, 2024Tradução . . Disponível em: https://doi.org/10.1177/07067437231209650. Acesso em: 05 out. 2024.
APA
Pinto, J. V. M. de M. C., Crippa, J. A. de S., Ceresér, K. M., Vianna-Sulzbach, M. F., Silveira Júnior, É. de M., Rosa, G. S. da, et al. (2024). Cannabidiol as an adjunctive treatment for acute bipolar depression: a pilot study. The Canadian Journal of Psychiatry, 69( 4), 242-251. doi:10.1177/07067437231209650
NLM
Pinto JVM de MC, Crippa JA de S, Ceresér KM, Vianna-Sulzbach MF, Silveira Júnior É de M, Rosa GS da, Silva MGT da, Hizo GH, Medeiros LS, Oliveira CES de, Bristot G, Campos AC de, Guimarães FS, Hallak JEC, Zuardi AW, Yatham LN, Kapczinski F, Kauer-Sant\2019anna M. Cannabidiol as an adjunctive treatment for acute bipolar depression: a pilot study [Internet]. The Canadian Journal of Psychiatry. 2024 ; 69( 4): 242-251.[citado 2024 out. 05 ] Available from: https://doi.org/10.1177/07067437231209650
Vancouver
Pinto JVM de MC, Crippa JA de S, Ceresér KM, Vianna-Sulzbach MF, Silveira Júnior É de M, Rosa GS da, Silva MGT da, Hizo GH, Medeiros LS, Oliveira CES de, Bristot G, Campos AC de, Guimarães FS, Hallak JEC, Zuardi AW, Yatham LN, Kapczinski F, Kauer-Sant\2019anna M. Cannabidiol as an adjunctive treatment for acute bipolar depression: a pilot study [Internet]. The Canadian Journal of Psychiatry. 2024 ; 69( 4): 242-251.[citado 2024 out. 05 ] Available from: https://doi.org/10.1177/07067437231209650
Artigo de periodico-carta/editorial
Dr. Raphael Mechoulam, cannabis and cannabinoids research pioneer (November 5, 1930-March 9, 2023) and his legacy for Brazilian pharmacology [Editorial] (2023)
Crippa, José Alexandre de Souza ; Guimarães, Francisco Silveira ; Zuardi, Antônio Waldo ; Hallak, Jaime Eduardo Cecilio
Fonte: Brazilian Journal of Psychiatry. Unidade: FMRP
Assuntos: CANABINOIDES, CANNABIS, ALUCINOGÊNICOS
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ABNT
CRIPPA, José Alexandre de Souza et al. Dr. Raphael Mechoulam, cannabis and cannabinoids research pioneer (November 5, 1930-March 9, 2023) and his legacy for Brazilian pharmacology [Editorial]. Brazilian Journal of Psychiatry. São Paulo: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Disponível em: https://doi.org/10.47626/1516-4446-2023-0047. Acesso em: 05 out. 2024. , 2023
APA
Crippa, J. A. de S., Guimarães, F. S., Zuardi, A. W., & Hallak, J. E. C. (2023). Dr. Raphael Mechoulam, cannabis and cannabinoids research pioneer (November 5, 1930-March 9, 2023) and his legacy for Brazilian pharmacology [Editorial]. Brazilian Journal of Psychiatry. São Paulo: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. doi:10.47626/1516-4446-2023-0047
NLM
Crippa JA de S, Guimarães FS, Zuardi AW, Hallak JEC. Dr. Raphael Mechoulam, cannabis and cannabinoids research pioneer (November 5, 1930-March 9, 2023) and his legacy for Brazilian pharmacology [Editorial] [Internet]. Brazilian Journal of Psychiatry. 2023 ; 45( 3): 201-202.[citado 2024 out. 05 ] Available from: https://doi.org/10.47626/1516-4446-2023-0047
Vancouver
Crippa JA de S, Guimarães FS, Zuardi AW, Hallak JEC. Dr. Raphael Mechoulam, cannabis and cannabinoids research pioneer (November 5, 1930-March 9, 2023) and his legacy for Brazilian pharmacology [Editorial] [Internet]. Brazilian Journal of Psychiatry. 2023 ; 45( 3): 201-202.[citado 2024 out. 05 ] Available from: https://doi.org/10.47626/1516-4446-2023-0047
Artigo de periodico
Synovial fluid survivin and NETs as independent biomarkers in rheumatoid arthritis (2023)
Souza, Flávio Falcão Lima de; Schneider, Ayda Henriques ; Machado, Caio Cavalcante ; Almeida, Sérgio Couto Luna de; Silva, Tarcília Aparecida da; Cunha, Fernando de Queiroz ; Louzada Júnior, Paulo ; Oliveira, Renê Donizeti Ribeiro de
Fonte: Clinical and Experimental Rheumatology. Unidade: FMRP
Assuntos: BIOMARCADORES, ARTRITE REUMATOIDE, OSTEOARTRITE, ESTUDOS TRANSVERSAIS
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ABNT
SOUZA, Flávio Falcão Lima de et al. Synovial fluid survivin and NETs as independent biomarkers in rheumatoid arthritis. Clinical and Experimental Rheumatology, v. 41, n. 7, p. 1473-1479, 2023Tradução . . Disponível em: https://doi.org/10.55563/clinexprheumatol/flzpgb. Acesso em: 05 out. 2024.
APA
Souza, F. F. L. de, Schneider, A. H., Machado, C. C., Almeida, S. C. L. de, Silva, T. A. da, Cunha, F. de Q., et al. (2023). Synovial fluid survivin and NETs as independent biomarkers in rheumatoid arthritis. Clinical and Experimental Rheumatology, 41( 7), 1473-1479. doi:10.55563/clinexprheumatol/flzpgb
NLM
Souza FFL de, Schneider AH, Machado CC, Almeida SCL de, Silva TA da, Cunha F de Q, Louzada Júnior P, Oliveira RDR de. Synovial fluid survivin and NETs as independent biomarkers in rheumatoid arthritis [Internet]. Clinical and Experimental Rheumatology. 2023 ; 41( 7): 1473-1479.[citado 2024 out. 05 ] Available from: https://doi.org/10.55563/clinexprheumatol/flzpgb
Vancouver
Souza FFL de, Schneider AH, Machado CC, Almeida SCL de, Silva TA da, Cunha F de Q, Louzada Júnior P, Oliveira RDR de. Synovial fluid survivin and NETs as independent biomarkers in rheumatoid arthritis [Internet]. Clinical and Experimental Rheumatology. 2023 ; 41( 7): 1473-1479.[citado 2024 out. 05 ] Available from: https://doi.org/10.55563/clinexprheumatol/flzpgb
Artigo de periodico
Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19 (2023)
Formiga, Rodrigo de Oliveira; Amaral, Flávia C.; Souza, Camila F.; Mendes, Daniel A. G. B.; Wanderley, Carlos Wagner de Souza ; Lorenzini, Cristina B.; Santos, Adara A.; Antônia, Juliana; Faria, Lucas F.; Natale, Caio C.; Paula, Nicholas M.; Cunha, Fernando de Queiroz
Fonte: British Journal of Pharmacology. Unidade: FMRP
Assuntos: COVID-19, LIPOPOLISSACARÍDEOS, METALOPROTEINASES, NEUTRÓFILOS, ESPÉCIES REATIVAS DE OXIGÊNIO, SEPSE
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ABNT
FORMIGA, Rodrigo de Oliveira et al. Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19. British Journal of Pharmacology, v. 180, n. 11, p. 1460-1481, 2023Tradução . . Disponível em: https://doi.org/10.1111/bph.16013. Acesso em: 05 out. 2024.
APA
Formiga, R. de O., Amaral, F. C., Souza, C. F., Mendes, D. A. G. B., Wanderley, C. W. de S., Lorenzini, C. B., et al. (2023). Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19. British Journal of Pharmacology, 180( 11), 1460-1481. doi:10.1111/bph.16013
NLM
Formiga R de O, Amaral FC, Souza CF, Mendes DAGB, Wanderley CW de S, Lorenzini CB, Santos AA, Antônia J, Faria LF, Natale CC, Paula NM, Cunha F de Q. Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19 [Internet]. British Journal of Pharmacology. 2023 ; 180( 11): 1460-1481.[citado 2024 out. 05 ] Available from: https://doi.org/10.1111/bph.16013
Vancouver
Formiga R de O, Amaral FC, Souza CF, Mendes DAGB, Wanderley CW de S, Lorenzini CB, Santos AA, Antônia J, Faria LF, Natale CC, Paula NM, Cunha F de Q. Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19 [Internet]. British Journal of Pharmacology. 2023 ; 180( 11): 1460-1481.[citado 2024 out. 05 ] Available from: https://doi.org/10.1111/bph.16013
Artigo de periodico
FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility (2023)
Borges, Vanessa de Fátima; Galant, Leticia Selinger; Kanashiro, Alexandre ; Castanheira, Fernanda Vargas e Silva; Monteiro, Valter Vinícius Silva; Duarte, Diego Ângelo; Rodrigues, Filipe Camargo; Silva, Camila Meirelles de Souza; Schneider, Ayda Henriques ; Cebinelli, Guilherme Cesar Martelossi ; Lima, Mikhael Haruo Fernandes de; Viola, João Paulo de Biaso; Cunha, Thiago Mattar ; Costa Neto, Claudio Miguel da ; Alves Filho, José Carlos Farias ; Pupo, André Sampaio; Cunha, Fernando de Queiroz
Fonte: Inflammation Research. Unidade: FMRP
Assuntos: NEUTRÓFILOS, SEPSE, FÁRMACOS IMUNOSSUPRESSORES
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ABNT
BORGES, Vanessa de Fátima et al. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility. Inflammation Research, v. 72, n. 2, p. 203-215, 2023Tradução . . Disponível em: https://doi.org/10.1007/s00011-022-01669-w. Acesso em: 05 out. 2024.
APA
Borges, V. de F., Galant, L. S., Kanashiro, A., Castanheira, F. V. e S., Monteiro, V. V. S., Duarte, D. Â., et al. (2023). FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility. Inflammation Research, 72( 2), 203-215. doi:10.1007/s00011-022-01669-w
NLM
Borges V de F, Galant LS, Kanashiro A, Castanheira FV e S, Monteiro VVS, Duarte DÂ, Rodrigues FC, Silva CM de S, Schneider AH, Cebinelli GCM, Lima MHF de, Viola JP de B, Cunha TM, Costa Neto CM da, Alves Filho JCF, Pupo AS, Cunha F de Q. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility [Internet]. Inflammation Research. 2023 ; 72( 2): 203-215.[citado 2024 out. 05 ] Available from: https://doi.org/10.1007/s00011-022-01669-w
Vancouver
Borges V de F, Galant LS, Kanashiro A, Castanheira FV e S, Monteiro VVS, Duarte DÂ, Rodrigues FC, Silva CM de S, Schneider AH, Cebinelli GCM, Lima MHF de, Viola JP de B, Cunha TM, Costa Neto CM da, Alves Filho JCF, Pupo AS, Cunha F de Q. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility [Internet]. Inflammation Research. 2023 ; 72( 2): 203-215.[citado 2024 out. 05 ] Available from: https://doi.org/10.1007/s00011-022-01669-w
Artigo de periodico
Quercetin decreases cardiac hypertrophic mediators and maladaptive coronary arterial remodeling in renovascular hypertensive rats without improving cardiac function (2023)
Rocha, Eduardo Vieira da; Falchetti, Francisco; Pernomian, Laena; Mello, Marcela Maria Blascke de ; Parente, Juliana Montenegro ; Nogueira, Renato Corrêa; Gomes, Beatriz Quaresemin; Bertozi, Giuliana; Sanches-Lopes, Jessica M.; Tanus-Santos, José Eduardo ; Castro, Michele Mazzaron de
Fonte: Naunyn-Schmiedeberg's Archives of Pharmacology. Unidades: FMRP, FFCLRP
Assuntos: PRESSÃO SANGUÍNEA, CARDIOMEGALIA, VASOS CORONÁRIOS, HIPERTENSÃO, FARMACOTERAPIA, NEFROPATIAS
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ABNT
ROCHA, Eduardo Vieira da et al. Quercetin decreases cardiac hypertrophic mediators and maladaptive coronary arterial remodeling in renovascular hypertensive rats without improving cardiac function. Naunyn-Schmiedeberg's Archives of Pharmacology, v. 396, n. 5, p. 939-949, 2023Tradução . . Disponível em: https://doi.org/10.1007/s00210-022-02349-6. Acesso em: 05 out. 2024.
APA
Rocha, E. V. da, Falchetti, F., Pernomian, L., Mello, M. M. B. de, Parente, J. M., Nogueira, R. C., et al. (2023). Quercetin decreases cardiac hypertrophic mediators and maladaptive coronary arterial remodeling in renovascular hypertensive rats without improving cardiac function. Naunyn-Schmiedeberg's Archives of Pharmacology, 396( 5), 939-949. doi:10.1007/s00210-022-02349-6
NLM
Rocha EV da, Falchetti F, Pernomian L, Mello MMB de, Parente JM, Nogueira RC, Gomes BQ, Bertozi G, Sanches-Lopes JM, Tanus-Santos JE, Castro MM de. Quercetin decreases cardiac hypertrophic mediators and maladaptive coronary arterial remodeling in renovascular hypertensive rats without improving cardiac function [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2023 ; 396( 5): 939-949.[citado 2024 out. 05 ] Available from: https://doi.org/10.1007/s00210-022-02349-6
Vancouver
Rocha EV da, Falchetti F, Pernomian L, Mello MMB de, Parente JM, Nogueira RC, Gomes BQ, Bertozi G, Sanches-Lopes JM, Tanus-Santos JE, Castro MM de. Quercetin decreases cardiac hypertrophic mediators and maladaptive coronary arterial remodeling in renovascular hypertensive rats without improving cardiac function [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2023 ; 396( 5): 939-949.[citado 2024 out. 05 ] Available from: https://doi.org/10.1007/s00210-022-02349-6
Artigo de periodico
Neostriatum neuronal TRPV1-signalling mediates striatal anandamide at high concentration facilitatory influence on neostriato-nigral dishinhibitory GABAergic connections (2023)
Silva, Juliana Almeida da ; Almada, Rafael Carvalho ; Falconi-Sobrinho, Luiz Luciano ; Pigatto, Glauce Regina; Hernandes, Paloma Molina; Coimbra, Norberto Cysne
Fonte: Brain Research Bulletin. Unidade: FMRP
Assuntos: ANTAGONISTAS, VIAS NEURAIS, MEDO, NEUROTRANSMISSORES
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ABNT
SILVA, Juliana Almeida da et al. Neostriatum neuronal TRPV1-signalling mediates striatal anandamide at high concentration facilitatory influence on neostriato-nigral dishinhibitory GABAergic connections. Brain Research Bulletin, v. 192, p. 128-141, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.brainresbull.2022.11.014. Acesso em: 05 out. 2024.
APA
Silva, J. A. da, Almada, R. C., Falconi-Sobrinho, L. L., Pigatto, G. R., Hernandes, P. M., & Coimbra, N. C. (2023). Neostriatum neuronal TRPV1-signalling mediates striatal anandamide at high concentration facilitatory influence on neostriato-nigral dishinhibitory GABAergic connections. Brain Research Bulletin, 192, 128-141. doi:10.1016/j.brainresbull.2022.11.014
NLM
Silva JA da, Almada RC, Falconi-Sobrinho LL, Pigatto GR, Hernandes PM, Coimbra NC. Neostriatum neuronal TRPV1-signalling mediates striatal anandamide at high concentration facilitatory influence on neostriato-nigral dishinhibitory GABAergic connections [Internet]. Brain Research Bulletin. 2023 ; 192 128-141.[citado 2024 out. 05 ] Available from: https://doi.org/10.1016/j.brainresbull.2022.11.014
Vancouver
Silva JA da, Almada RC, Falconi-Sobrinho LL, Pigatto GR, Hernandes PM, Coimbra NC. Neostriatum neuronal TRPV1-signalling mediates striatal anandamide at high concentration facilitatory influence on neostriato-nigral dishinhibitory GABAergic connections [Internet]. Brain Research Bulletin. 2023 ; 192 128-141.[citado 2024 out. 05 ] Available from: https://doi.org/10.1016/j.brainresbull.2022.11.014
Artigo de periodico
Levetiracetam attenuates adolescent stress-induced behavioral and electrophysiological changes associated with schizophrenia in adult rats (2023)
Cavichioli, Andreza M.; Silva, Thamyris Santos; Grace, Anthony A.; Guimarães, Francisco Silveira ; Gomes, Felipe Villela
Fonte: Schizophrenia Bulletin. Unidade: FMRP
Assuntos: POTENCIAIS DE AÇÃO, NEUROTRANSMISSORES, ESQUIZOFRENIA, HIPOCAMPO
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ABNT
CAVICHIOLI, Andreza M. et al. Levetiracetam attenuates adolescent stress-induced behavioral and electrophysiological changes associated with schizophrenia in adult rats. Schizophrenia Bulletin, v. 49, n. 1, p. 68-77, 2023Tradução . . Disponível em: https://doi.org/10.1093/schbul/sbac106. Acesso em: 05 out. 2024.
APA
Cavichioli, A. M., Silva, T. S., Grace, A. A., Guimarães, F. S., & Gomes, F. V. (2023). Levetiracetam attenuates adolescent stress-induced behavioral and electrophysiological changes associated with schizophrenia in adult rats. Schizophrenia Bulletin, 49( 1), 68-77. doi:10.1093/schbul/sbac106
NLM
Cavichioli AM, Silva TS, Grace AA, Guimarães FS, Gomes FV. Levetiracetam attenuates adolescent stress-induced behavioral and electrophysiological changes associated with schizophrenia in adult rats [Internet]. Schizophrenia Bulletin. 2023 ; 49( 1): 68-77.[citado 2024 out. 05 ] Available from: https://doi.org/10.1093/schbul/sbac106
Vancouver
Cavichioli AM, Silva TS, Grace AA, Guimarães FS, Gomes FV. Levetiracetam attenuates adolescent stress-induced behavioral and electrophysiological changes associated with schizophrenia in adult rats [Internet]. Schizophrenia Bulletin. 2023 ; 49( 1): 68-77.[citado 2024 out. 05 ] Available from: https://doi.org/10.1093/schbul/sbac106
Artigo de periodico
COVID-19-related hyperglycemia is associated with infection of hepatocytes and stimulation of gluconeogenesis (2023)
Barreto, Ester A.; Cruz, Amanda S.; Veras, Flávio Protásio ; Martins, Ronaldo Bragança ; Bernardelli, Rafaella S.; Paiva, Isadora Marques ; Lima, Thais Melquiades de; Singh, Youvika ; Guimaraes, Raphael C.; Damasceno, Samara; Pereira, Nayara; Alves, João Manoel; Gonçalves, Tiago T.; Forato, Julia; Muraro, Stéfanie P.; Souza, Gabriela F.; Batah, Sabrina Setembre ; Proença-Modena, José L.; Mori, Marcelo A.; Cunha, Fernando de Queiroz ; Louzada Júnior, Paulo ; Cunha, Thiago Mattar ; Nakaya, Helder Takashi Imoto ; Fabro, Alexandre Todorovic ; Oliveira, Renê Donizeti Ribeiro de; Arruda, Eurico ; Rea, Rosângela; Réa Neto, Álvaro; Silva, Miguel M Fernandes da; Leiria, Luiz Osório Silveira
Fonte: Proceedings of the National Academy of Sciences. Unidades: FMRP, FCF
Assuntos: GLICOSE, COVID-19, GLICOGENOSE, HIPERGLICEMIA, ESTUDOS RETROSPECTIVOS
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ABNT
BARRETO, Ester A. et al. COVID-19-related hyperglycemia is associated with infection of hepatocytes and stimulation of gluconeogenesis. Proceedings of the National Academy of Sciences, v. 120, n. 21, p. 1-10, 2023Tradução . . Disponível em: https://doi.org/10.1073/pnas.2217119120. Acesso em: 05 out. 2024.
APA
Barreto, E. A., Cruz, A. S., Veras, F. P., Martins, R. B., Bernardelli, R. S., Paiva, I. M., et al. (2023). COVID-19-related hyperglycemia is associated with infection of hepatocytes and stimulation of gluconeogenesis. Proceedings of the National Academy of Sciences, 120( 21), 1-10. doi:10.1073/pnas.2217119120
NLM
Barreto EA, Cruz AS, Veras FP, Martins RB, Bernardelli RS, Paiva IM, Lima TM de, Singh Y, Guimaraes RC, Damasceno S, Pereira N, Alves JM, Gonçalves TT, Forato J, Muraro SP, Souza GF, Batah SS, Proença-Modena JL, Mori MA, Cunha F de Q, Louzada Júnior P, Cunha TM, Nakaya HTI, Fabro AT, Oliveira RDR de, Arruda E, Rea R, Réa Neto Á, Silva MMF da, Leiria LOS. COVID-19-related hyperglycemia is associated with infection of hepatocytes and stimulation of gluconeogenesis [Internet]. Proceedings of the National Academy of Sciences. 2023 ; 120( 21): 1-10.[citado 2024 out. 05 ] Available from: https://doi.org/10.1073/pnas.2217119120
Vancouver
Barreto EA, Cruz AS, Veras FP, Martins RB, Bernardelli RS, Paiva IM, Lima TM de, Singh Y, Guimaraes RC, Damasceno S, Pereira N, Alves JM, Gonçalves TT, Forato J, Muraro SP, Souza GF, Batah SS, Proença-Modena JL, Mori MA, Cunha F de Q, Louzada Júnior P, Cunha TM, Nakaya HTI, Fabro AT, Oliveira RDR de, Arruda E, Rea R, Réa Neto Á, Silva MMF da, Leiria LOS. COVID-19-related hyperglycemia is associated with infection of hepatocytes and stimulation of gluconeogenesis [Internet]. Proceedings of the National Academy of Sciences. 2023 ; 120( 21): 1-10.[citado 2024 out. 05 ] Available from: https://doi.org/10.1073/pnas.2217119120
Artigo de periodico
Enoxaparin improves COVID-19 by reducing Neutrophils Extracellular Traps (NETs) production (2023)
Córneo, Emily S.; Veras, Flávio Protásio ; Gomes, Giovanni F.; Schneider, Ayda Henriques ; Silva, Bruna Manuella Souza; Almeida, Cícero José Luíz dos Ramos; Silva, Camila M.; Martins, Ronaldo B.; Batah, Sabrina Setembre ; Simon, Carla S.; Prestes, Gabriele da S.; Alves Filho, José Carlos Farias ; Arruda, Eurico ; Louzada Júnior, Paulo ; Oliveira, Renê Donizeti Ribeiro de; Fabro, Alexandre Todorovic ; Cunha, Thiago Mattar ; Cunha, Fernando de Queiroz ; Dal-Pizzol, Felipe
Fonte: Clinical Immunology. Unidades: FMRP, ESALQ
Assuntos: COVID-19, NEUTRÓFILOS, CORONAVIRUS
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ABNT
CÓRNEO, Emily S. et al. Enoxaparin improves COVID-19 by reducing Neutrophils Extracellular Traps (NETs) production. Clinical Immunology, v. 257, p. 1-12, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.clim.2023.109836. Acesso em: 05 out. 2024.
APA
Córneo, E. S., Veras, F. P., Gomes, G. F., Schneider, A. H., Silva, B. M. S., Almeida, C. J. L. dos R., et al. (2023). Enoxaparin improves COVID-19 by reducing Neutrophils Extracellular Traps (NETs) production. Clinical Immunology, 257, 1-12. doi:10.1016/j.clim.2023.109836
NLM
Córneo ES, Veras FP, Gomes GF, Schneider AH, Silva BMS, Almeida CJL dos R, Silva CM, Martins RB, Batah SS, Simon CS, Prestes G da S, Alves Filho JCF, Arruda E, Louzada Júnior P, Oliveira RDR de, Fabro AT, Cunha TM, Cunha F de Q, Dal-Pizzol F. Enoxaparin improves COVID-19 by reducing Neutrophils Extracellular Traps (NETs) production [Internet]. Clinical Immunology. 2023 ; 257 1-12.[citado 2024 out. 05 ] Available from: https://doi.org/10.1016/j.clim.2023.109836
Vancouver
Córneo ES, Veras FP, Gomes GF, Schneider AH, Silva BMS, Almeida CJL dos R, Silva CM, Martins RB, Batah SS, Simon CS, Prestes G da S, Alves Filho JCF, Arruda E, Louzada Júnior P, Oliveira RDR de, Fabro AT, Cunha TM, Cunha F de Q, Dal-Pizzol F. Enoxaparin improves COVID-19 by reducing Neutrophils Extracellular Traps (NETs) production [Internet]. Clinical Immunology. 2023 ; 257 1-12.[citado 2024 out. 05 ] Available from: https://doi.org/10.1016/j.clim.2023.109836
Artigo de periodico
NLRP3 activation contributes to endothelin-1-induced erectile dysfunction (2023)
Fais, Rafael Sobrano; Costa, Rafael Menezes da ; Mendes, Allan Carvalho; Mestriner, Fabiola Leslie Antunes Cardoso; Comerma-Steffensen, Simón Gabriel; Tostes, Rita de Cássia Aleixo ; Simonsen, Ulf; Carneiro, Fernando Silva
Fonte: Journal of Cellular and Molecular Medicine. Unidade: FMRP
Assuntos: ANTAGONISTAS, ENDOTELINAS, DISFUNÇÃO ERÉTIL, ESPÉCIES REATIVAS DE OXIGÊNIO, INFLAMAÇÃO
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ABNT
FAIS, Rafael Sobrano et al. NLRP3 activation contributes to endothelin-1-induced erectile dysfunction. Journal of Cellular and Molecular Medicine, v. 27, n. 1, p. 1-14, 2023Tradução . . Disponível em: https://doi.org/10.1111/jcmm.17463. Acesso em: 05 out. 2024.
APA
Fais, R. S., Costa, R. M. da, Mendes, A. C., Mestriner, F. L. A. C., Comerma-Steffensen, S. G., Tostes, R. de C. A., et al. (2023). NLRP3 activation contributes to endothelin-1-induced erectile dysfunction. Journal of Cellular and Molecular Medicine, 27( 1), 1-14. doi:10.1111/jcmm.17463
NLM
Fais RS, Costa RM da, Mendes AC, Mestriner FLAC, Comerma-Steffensen SG, Tostes R de CA, Simonsen U, Carneiro FS. NLRP3 activation contributes to endothelin-1-induced erectile dysfunction [Internet]. Journal of Cellular and Molecular Medicine. 2023 ; 27( 1): 1-14.[citado 2024 out. 05 ] Available from: https://doi.org/10.1111/jcmm.17463
Vancouver
Fais RS, Costa RM da, Mendes AC, Mestriner FLAC, Comerma-Steffensen SG, Tostes R de CA, Simonsen U, Carneiro FS. NLRP3 activation contributes to endothelin-1-induced erectile dysfunction [Internet]. Journal of Cellular and Molecular Medicine. 2023 ; 27( 1): 1-14.[citado 2024 out. 05 ] Available from: https://doi.org/10.1111/jcmm.17463
Artigo de periodico
Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection (2023)
Moreira, Thaiane Pinto; Sousa, Carla Daiane Ferreira de; Costa, Victor Rodrigues de Melo; Queiroz Junior, Celso Martins; Santos, Franciele Martins; Bonilha, Caio Santos ; Ésper, Lísia Maria; Nogueira, Mauricio Lacerda ; Cunha, Thiago Mattar ; Teixeira, Mauro Martins ; Costa, Vivian Vasconcelos; Souza, Daniele da Glória de
Fonte: Immunology. Unidade: FMRP
Assuntos: ARTRALGIA, VÍRUS CHIKUNGUNYA, NECROSE, REPLICAÇÃO VIRAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MOREIRA, Thaiane Pinto et al. Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection. Immunology, v. 168, n. 3, p. 444-458, 2023Tradução . . Disponível em: https://doi.org/10.1111/imm.13583. Acesso em: 05 out. 2024.
APA
Moreira, T. P., Sousa, C. D. F. de, Costa, V. R. de M., Queiroz Junior, C. M., Santos, F. M., Bonilha, C. S., et al. (2023). Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection. Immunology, 168( 3), 444-458. doi:10.1111/imm.13583
NLM
Moreira TP, Sousa CDF de, Costa VR de M, Queiroz Junior CM, Santos FM, Bonilha CS, Ésper LM, Nogueira ML, Cunha TM, Teixeira MM, Costa VV, Souza D da G de. Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection [Internet]. Immunology. 2023 ; 168( 3): 444-458.[citado 2024 out. 05 ] Available from: https://doi.org/10.1111/imm.13583
Vancouver
Moreira TP, Sousa CDF de, Costa VR de M, Queiroz Junior CM, Santos FM, Bonilha CS, Ésper LM, Nogueira ML, Cunha TM, Teixeira MM, Costa VV, Souza D da G de. Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection [Internet]. Immunology. 2023 ; 168( 3): 444-458.[citado 2024 out. 05 ] Available from: https://doi.org/10.1111/imm.13583
Artigo de periodico
Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT (2022)
Lima, Mikhael Haruo Fernandes de; Hiroki, Carlos Hiroji; Borges, Vanessa de Fátima; Cebinelli, Guilherme Cesar Martelossi ; Santos, Jessica; Rosa, Marcos Henrique; Silva, Camila Meirelles de Souza; Wanderley, Carlos Wagner ; Gonçalves, Augusto Velozo; Quirino, Gustavo Fernando da Silva; Zamboni, Dario Simões ; Cunha, Thiago Mattar ; Alves Filho, José Carlos Farias ; Cunha, Fernando de Queiroz
Fonte: The Journal of Infectious Diseases. Unidade: FMRP
Assuntos: IMUNOSSUPRESSÃO, SEPSE, MACRÓFAGOS, CITOMETRIA DE FLUXO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LIMA, Mikhael Haruo Fernandes de et al. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT. The Journal of Infectious Diseases, v. 225, n. 3, p. 531-541, 2022Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiab405. Acesso em: 05 out. 2024.
APA
Lima, M. H. F. de, Hiroki, C. H., Borges, V. de F., Cebinelli, G. C. M., Santos, J., Rosa, M. H., et al. (2022). Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT. The Journal of Infectious Diseases, 225( 3), 531-541. doi:10.1093/infdis/jiab405
NLM
Lima MHF de, Hiroki CH, Borges V de F, Cebinelli GCM, Santos J, Rosa MH, Silva CM de S, Wanderley CW, Gonçalves AV, Quirino GF da S, Zamboni DS, Cunha TM, Alves Filho JCF, Cunha F de Q. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT [Internet]. The Journal of Infectious Diseases. 2022 ; 225( 3): 531-541.[citado 2024 out. 05 ] Available from: https://doi.org/10.1093/infdis/jiab405
Vancouver
Lima MHF de, Hiroki CH, Borges V de F, Cebinelli GCM, Santos J, Rosa MH, Silva CM de S, Wanderley CW, Gonçalves AV, Quirino GF da S, Zamboni DS, Cunha TM, Alves Filho JCF, Cunha F de Q. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT [Internet]. The Journal of Infectious Diseases. 2022 ; 225( 3): 531-541.[citado 2024 out. 05 ] Available from: https://doi.org/10.1093/infdis/jiab405
Artigo de periodico
Anthrax toxins regulate pain signaling and can deliver molecular cargoes into ANTXR2+ DRG sensory neurons (2022)
Yang, Nicole J.; Isensee, Jörg; Neel, Dylan V; Quadros, Andreza Urba de; Zhang, Han-Xiong Bear; Lauzadis, Justas; Liu, Sai Man; Shiers, Stephanie; Belu, Andreea; Cunha, Thiago Mattar
Fonte: Nature Neuroscience. Unidade: FMRP
Assuntos: TOXINAS, BACTÉRIAS, GÂNGLIOS ESPINHAIS, CÉLULAS-TRONCO, NOCICEPTORES, DOR, PEPTÍDEOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
YANG, Nicole J. et al. Anthrax toxins regulate pain signaling and can deliver molecular cargoes into ANTXR2+ DRG sensory neurons. Nature Neuroscience, v. 25, n. 2, p. 168-179, 2022Tradução . . Disponível em: https://doi.org/10.1038/s41593-021-00973-8. Acesso em: 05 out. 2024.
APA
Yang, N. J., Isensee, J., Neel, D. V., Quadros, A. U. de, Zhang, H. -X. B., Lauzadis, J., et al. (2022). Anthrax toxins regulate pain signaling and can deliver molecular cargoes into ANTXR2+ DRG sensory neurons. Nature Neuroscience, 25( 2), 168-179. doi:10.1038/s41593-021-00973-8
NLM
Yang NJ, Isensee J, Neel DV, Quadros AU de, Zhang H-XB, Lauzadis J, Liu SM, Shiers S, Belu A, Cunha TM. Anthrax toxins regulate pain signaling and can deliver molecular cargoes into ANTXR2+ DRG sensory neurons [Internet]. Nature Neuroscience. 2022 ; 25( 2): 168-179.[citado 2024 out. 05 ] Available from: https://doi.org/10.1038/s41593-021-00973-8
Vancouver
Yang NJ, Isensee J, Neel DV, Quadros AU de, Zhang H-XB, Lauzadis J, Liu SM, Shiers S, Belu A, Cunha TM. Anthrax toxins regulate pain signaling and can deliver molecular cargoes into ANTXR2+ DRG sensory neurons [Internet]. Nature Neuroscience. 2022 ; 25( 2): 168-179.[citado 2024 out. 05 ] Available from: https://doi.org/10.1038/s41593-021-00973-8
Artigo de periodico
The carotid body detects circulating tumor necrosis factor-alpha to activate a sympathetic anti-inflammatory reflex (2022)
Katayama, Pedro L.; Leirão, Isabela P.; Kanashiro, Alexandre; Luiz, João Paulo Mesquita ; Cunha, Fernando de Queiroz ; Navegantes, Luiz Carlos Carvalho ; Menani, Jose Vanderlei; Zoccal, Daniel Breseghello; Colombari, Debora Simoes de Almeida ; Colombari, Eduardo
Fonte: Brain, Behavior, and Immunity. Unidade: FMRP
Assuntos: SISTEMA NERVOSO SIMPÁTICO, INFLAMAÇÃO, NEUROIMUNOMODULAÇÃO, ANTI-INFLAMATÓRIOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KATAYAMA, Pedro L. et al. The carotid body detects circulating tumor necrosis factor-alpha to activate a sympathetic anti-inflammatory reflex. Brain, Behavior, and Immunity, v. 102, p. 370-386, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.bbi.2022.03.014. Acesso em: 05 out. 2024.
APA
Katayama, P. L., Leirão, I. P., Kanashiro, A., Luiz, J. P. M., Cunha, F. de Q., Navegantes, L. C. C., et al. (2022). The carotid body detects circulating tumor necrosis factor-alpha to activate a sympathetic anti-inflammatory reflex. Brain, Behavior, and Immunity, 102, 370-386. doi:10.1016/j.bbi.2022.03.014
NLM
Katayama PL, Leirão IP, Kanashiro A, Luiz JPM, Cunha F de Q, Navegantes LCC, Menani JV, Zoccal DB, Colombari DS de A, Colombari E. The carotid body detects circulating tumor necrosis factor-alpha to activate a sympathetic anti-inflammatory reflex [Internet]. Brain, Behavior, and Immunity. 2022 ; 102 370-386.[citado 2024 out. 05 ] Available from: https://doi.org/10.1016/j.bbi.2022.03.014
Vancouver
Katayama PL, Leirão IP, Kanashiro A, Luiz JPM, Cunha F de Q, Navegantes LCC, Menani JV, Zoccal DB, Colombari DS de A, Colombari E. The carotid body detects circulating tumor necrosis factor-alpha to activate a sympathetic anti-inflammatory reflex [Internet]. Brain, Behavior, and Immunity. 2022 ; 102 370-386.[citado 2024 out. 05 ] Available from: https://doi.org/10.1016/j.bbi.2022.03.014
Artigo de periodico
Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy (2022)
Santos, Jeimison D.; Oliveira Neto, José Teles de ; Barros, Paula Rodrigues de; Damasceno, Luis Eduardo Alves; Lautherbach, Natalia; Assis, Ana Paula de; Silva, Carlos Alberto Aguiar da; Sorgi, Carlos Arterio ; Faccioli, Lucia Helena ; Kettelhut, Isis do Carmo ; Salgado, Hélio César ; Carneiro, Fernando Silva ; Alves-Filho, José Carlos ; Tostes, Rita de Cássia Aleixo
Fonte: American Journal of Physiology-Heart and Circulatory Physiology. Unidades: FMRP, FFCLRP, FCFRP
Assuntos: DOENÇAS CARDIOVASCULARES, FARMACOTERAPIA, HORMÔNIOS, ESTEROIDES, TESTOSTERONA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANTOS, Jeimison D. et al. Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy. American Journal of Physiology-Heart and Circulatory Physiology, v. 323, p. H322-H335, 2022Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00182.2022. Acesso em: 05 out. 2024.
APA
Santos, J. D., Oliveira Neto, J. T. de, Barros, P. R. de, Damasceno, L. E. A., Lautherbach, N., Assis, A. P. de, et al. (2022). Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy. American Journal of Physiology-Heart and Circulatory Physiology, 323, H322-H335. doi:10.1152/ajpheart.00182.2022
NLM
Santos JD, Oliveira Neto JT de, Barros PR de, Damasceno LEA, Lautherbach N, Assis AP de, Silva CAA da, Sorgi CA, Faccioli LH, Kettelhut I do C, Salgado HC, Carneiro FS, Alves-Filho JC, Tostes R de CA. Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy [Internet]. American Journal of Physiology-Heart and Circulatory Physiology. 2022 ; 323 H322-H335.[citado 2024 out. 05 ] Available from: https://doi.org/10.1152/ajpheart.00182.2022
Vancouver
Santos JD, Oliveira Neto JT de, Barros PR de, Damasceno LEA, Lautherbach N, Assis AP de, Silva CAA da, Sorgi CA, Faccioli LH, Kettelhut I do C, Salgado HC, Carneiro FS, Alves-Filho JC, Tostes R de CA. Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy [Internet]. American Journal of Physiology-Heart and Circulatory Physiology. 2022 ; 323 H322-H335.[citado 2024 out. 05 ] Available from: https://doi.org/10.1152/ajpheart.00182.2022
Artigo de periodico
Intense acute swimming induces delayed-onset muscle soreness dependent on spinal cord neuroinflammation (2022)
Borghi, Sergio M.; Bussulo, Sylvia K. D.; Pinho-Ribeiro, Felipe A.; Fattori, Victor; Carvalho, Thacyana T.; Rasquel-Oliveira, Fernanda S.; Zaninelli, Tiago H.; Ferraz, Camila R.; Casella, Antônio M. B.; Cunha, Fernando de Queiroz ; Cunha, Thiago Mattar ; Casagrande, Rubia; Verri Júnior, Waldiceu A.
Fonte: Frontiers in Pharmacology. Unidade: FMRP
Assuntos: EXERCÍCIOS DE RESISTÊNCIA MUSCULAR, MEDULA ESPINHAL, INFLAMAÇÃO, DOR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BORGHI, Sergio M. et al. Intense acute swimming induces delayed-onset muscle soreness dependent on spinal cord neuroinflammation. Frontiers in Pharmacology, v. 12, p. 1-17, 2022Tradução . . Disponível em: https://doi.org/10.3389/fphar.2021.734091. Acesso em: 05 out. 2024.
APA
Borghi, S. M., Bussulo, S. K. D., Pinho-Ribeiro, F. A., Fattori, V., Carvalho, T. T., Rasquel-Oliveira, F. S., et al. (2022). Intense acute swimming induces delayed-onset muscle soreness dependent on spinal cord neuroinflammation. Frontiers in Pharmacology, 12, 1-17. doi:10.3389/fphar.2021.734091
NLM
Borghi SM, Bussulo SKD, Pinho-Ribeiro FA, Fattori V, Carvalho TT, Rasquel-Oliveira FS, Zaninelli TH, Ferraz CR, Casella AMB, Cunha F de Q, Cunha TM, Casagrande R, Verri Júnior WA. Intense acute swimming induces delayed-onset muscle soreness dependent on spinal cord neuroinflammation [Internet]. Frontiers in Pharmacology. 2022 ; 12 1-17.[citado 2024 out. 05 ] Available from: https://doi.org/10.3389/fphar.2021.734091
Vancouver
Borghi SM, Bussulo SKD, Pinho-Ribeiro FA, Fattori V, Carvalho TT, Rasquel-Oliveira FS, Zaninelli TH, Ferraz CR, Casella AMB, Cunha F de Q, Cunha TM, Casagrande R, Verri Júnior WA. Intense acute swimming induces delayed-onset muscle soreness dependent on spinal cord neuroinflammation [Internet]. Frontiers in Pharmacology. 2022 ; 12 1-17.[citado 2024 out. 05 ] Available from: https://doi.org/10.3389/fphar.2021.734091
Artigo de periodico
Context evoked morphine conditioned effects can be equivalent to morphine induced drug effects in terms of behavioral response and ERK activation in reward associated subcortical brain structures (2022)
Crespo, Luiz Gustavo Soares Carvalho; Leite Júnior, Joaquim Barbosa; Bastos, João Marcos de Mello; Samuels, Richard Ian; Coimbra, Norberto Cysne ; Carey, Robert J.; Carrera, Marinete Pinheiro
Fonte: Pharmacology Biochemistry and Behavior. Unidade: FMRP
Assuntos: CÉREBRO, CONDICIONAMENTO OPERANTE, MORFINA, ESTIMULAÇÃO CEREBRAL
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ABNT
CRESPO, Luiz Gustavo Soares Carvalho et al. Context evoked morphine conditioned effects can be equivalent to morphine induced drug effects in terms of behavioral response and ERK activation in reward associated subcortical brain structures. Pharmacology Biochemistry and Behavior, v. 214, p. 1-10, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.pbb.2022.173356. Acesso em: 05 out. 2024.
APA
Crespo, L. G. S. C., Leite Júnior, J. B., Bastos, J. M. de M., Samuels, R. I., Coimbra, N. C., Carey, R. J., & Carrera, M. P. (2022). Context evoked morphine conditioned effects can be equivalent to morphine induced drug effects in terms of behavioral response and ERK activation in reward associated subcortical brain structures. Pharmacology Biochemistry and Behavior, 214, 1-10. doi:10.1016/j.pbb.2022.173356
NLM
Crespo LGSC, Leite Júnior JB, Bastos JM de M, Samuels RI, Coimbra NC, Carey RJ, Carrera MP. Context evoked morphine conditioned effects can be equivalent to morphine induced drug effects in terms of behavioral response and ERK activation in reward associated subcortical brain structures [Internet]. Pharmacology Biochemistry and Behavior. 2022 ; 214 1-10.[citado 2024 out. 05 ] Available from: https://doi.org/10.1016/j.pbb.2022.173356
Vancouver
Crespo LGSC, Leite Júnior JB, Bastos JM de M, Samuels RI, Coimbra NC, Carey RJ, Carrera MP. Context evoked morphine conditioned effects can be equivalent to morphine induced drug effects in terms of behavioral response and ERK activation in reward associated subcortical brain structures [Internet]. Pharmacology Biochemistry and Behavior. 2022 ; 214 1-10.[citado 2024 out. 05 ] Available from: https://doi.org/10.1016/j.pbb.2022.173356
Artigo de periodico
Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress (2022)
Costa, Rafael Menezes da ; Alves-Lopes, Rhéure ; Alves, Juliano Vilela ; Servian, Carolina P.; Mestriner, Fabiola Leslie Antunes Cardoso; Carneiro, Fernando Silva ; Lobato, Núbia de S.; Tostes, Rita de Cássia Aleixo
Fonte: Frontiers in Physiology. Unidade: FMRP
Assuntos: OBESIDADE, ESTRESSE OXIDATIVO, TESTOSTERONA, DOENÇAS VASCULARES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Rafael Menezes da et al. Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress. Frontiers in Physiology, v. 13, p. 1-14, 2022Tradução . . Disponível em: https://doi.org/10.3389/fphys.2022.837603. Acesso em: 05 out. 2024.
APA
Costa, R. M. da, Alves-Lopes, R., Alves, J. V., Servian, C. P., Mestriner, F. L. A. C., Carneiro, F. S., et al. (2022). Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress. Frontiers in Physiology, 13, 1-14. doi:10.3389/fphys.2022.837603
NLM
Costa RM da, Alves-Lopes R, Alves JV, Servian CP, Mestriner FLAC, Carneiro FS, Lobato N de S, Tostes R de CA. Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress [Internet]. Frontiers in Physiology. 2022 ; 13 1-14.[citado 2024 out. 05 ] Available from: https://doi.org/10.3389/fphys.2022.837603
Vancouver
Costa RM da, Alves-Lopes R, Alves JV, Servian CP, Mestriner FLAC, Carneiro FS, Lobato N de S, Tostes R de CA. Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress [Internet]. Frontiers in Physiology. 2022 ; 13 1-14.[citado 2024 out. 05 ] Available from: https://doi.org/10.3389/fphys.2022.837603
Artigo de periodico
Paclitaxel binds and activates C5aR1: a new potential therapeutic target for the prevention of chemotherapy-induced peripheral neuropathy and hypersensitivity reactions (2022)
Brandolini, Laura; d’Angelo, Michele; Novelli, Rubina; Castelli, Vanessa; Giorgio, Cristina; Sirico, Anna; Cocchiaro, Pasquale; D’Egidio, Francesco; Benedetti, Elisabetta; Cristiano, Claudia; Bugatti, Antonella; Quadros, Andreza Urba de; Cunha, Thiago Mattar
Fonte: Cell Death & Disease. Unidade: FMRP
Assuntos: ANTINEOPLÁSICOS, DOENÇAS DO SISTEMA NERVOSO PERIFÉRICO, FARMACOTERAPIA, DOR CRÔNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BRANDOLINI, Laura et al. Paclitaxel binds and activates C5aR1: a new potential therapeutic target for the prevention of chemotherapy-induced peripheral neuropathy and hypersensitivity reactions. Cell Death & Disease, v. 13, n. 5, p. 1-15, 2022Tradução . . Disponível em: https://doi.org/10.1038/s41419-022-04964-w. Acesso em: 05 out. 2024.
APA
Brandolini, L., d’Angelo, M., Novelli, R., Castelli, V., Giorgio, C., Sirico, A., et al. (2022). Paclitaxel binds and activates C5aR1: a new potential therapeutic target for the prevention of chemotherapy-induced peripheral neuropathy and hypersensitivity reactions. Cell Death & Disease, 13( 5), 1-15. doi:10.1038/s41419-022-04964-w
NLM
Brandolini L, d’Angelo M, Novelli R, Castelli V, Giorgio C, Sirico A, Cocchiaro P, D’Egidio F, Benedetti E, Cristiano C, Bugatti A, Quadros AU de, Cunha TM. Paclitaxel binds and activates C5aR1: a new potential therapeutic target for the prevention of chemotherapy-induced peripheral neuropathy and hypersensitivity reactions [Internet]. Cell Death & Disease. 2022 ; 13( 5): 1-15.[citado 2024 out. 05 ] Available from: https://doi.org/10.1038/s41419-022-04964-w
Vancouver
Brandolini L, d’Angelo M, Novelli R, Castelli V, Giorgio C, Sirico A, Cocchiaro P, D’Egidio F, Benedetti E, Cristiano C, Bugatti A, Quadros AU de, Cunha TM. Paclitaxel binds and activates C5aR1: a new potential therapeutic target for the prevention of chemotherapy-induced peripheral neuropathy and hypersensitivity reactions [Internet]. Cell Death & Disease. 2022 ; 13( 5): 1-15.[citado 2024 out. 05 ] Available from: https://doi.org/10.1038/s41419-022-04964-w

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