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Artigo de periodico
The role of dendritic cells in COVID-19 infection (2023)
Wang, Xuying; Miller, Heather; Byazrova, Maria G. ; Candotti, Fabio ; Benlagha, Kamel ; Lei, Jiahui; Filatov, Alexander ; Liu, Chaohong ; Câmara, Niels Olsen Saraiva
Fonte: Emerging Microbes & Infections. Unidade: ICB
Assuntos: IMUNOLOGIA, CÉLULAS DENDRÍTICAS, CITOCINAS, COVID-19, INFECÇÕES POR VÍRUS DE RNA
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ABNT
WANG, Xuying et al. The role of dendritic cells in COVID-19 infection. Emerging Microbes & Infections, v. 12, n. 1, p. 1-9, 2023Tradução . . Disponível em: https://doi.org/10.1080/22221751.2023.2195019. Acesso em: 06 jul. 2024.
APA
Wang, X., Miller, H., Byazrova, M. G., Candotti, F., Benlagha, K., Lei, J., et al. (2023). The role of dendritic cells in COVID-19 infection. Emerging Microbes & Infections, 12( 1), 1-9. doi:10.1080/22221751.2023.2195019
NLM
Wang X, Miller H, Byazrova MG, Candotti F, Benlagha K, Lei J, Filatov A, Liu C, Câmara NOS. The role of dendritic cells in COVID-19 infection [Internet]. Emerging Microbes & Infections. 2023 ; 12( 1): 1-9.[citado 2024 jul. 06 ] Available from: https://doi.org/10.1080/22221751.2023.2195019
Vancouver
Wang X, Miller H, Byazrova MG, Candotti F, Benlagha K, Lei J, Filatov A, Liu C, Câmara NOS. The role of dendritic cells in COVID-19 infection [Internet]. Emerging Microbes & Infections. 2023 ; 12( 1): 1-9.[citado 2024 jul. 06 ] Available from: https://doi.org/10.1080/22221751.2023.2195019
Artigo de periodico
The role of Raptor in lymphocytes differentiation and function (2023)
Tang, Jianing; Yang, Lu; Guan, Fei; Miller, Heather; James, Louisa K.; Benlagha, Kamel ; Kubo, Masato ; Heegaard, Steffen ; Lee, Pamela ; Lei, Jiahui; Zeng, Hu ; He, Chengwei; Zhai, Zhimin ; Liu, Chaohong ; Câmara, Niels Olsen Saraiva
Fonte: Frontiers in Immunology. Unidade: ICB
Assuntos: LINFÓCITOS, METABOLISMO CELULAR, CITOCINAS, TRANSDUÇÃO DE SINAL CELULAR
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ABNT
TANG, Jianing et al. The role of Raptor in lymphocytes differentiation and function. Frontiers in Immunology, v. 14, 2023Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2023.1146628. Acesso em: 06 jul. 2024.
APA
Tang, J., Yang, L., Guan, F., Miller, H., James, L. K., Benlagha, K., et al. (2023). The role of Raptor in lymphocytes differentiation and function. Frontiers in Immunology, 14. doi:10.3389/fimmu.2023.1146628
NLM
Tang J, Yang L, Guan F, Miller H, James LK, Benlagha K, Kubo M, Heegaard S, Lee P, Lei J, Zeng H, He C, Zhai Z, Liu C, Câmara NOS. The role of Raptor in lymphocytes differentiation and function [Internet]. Frontiers in Immunology. 2023 ; 14[citado 2024 jul. 06 ] Available from: https://doi.org/10.3389/fimmu.2023.1146628
Vancouver
Tang J, Yang L, Guan F, Miller H, James LK, Benlagha K, Kubo M, Heegaard S, Lee P, Lei J, Zeng H, He C, Zhai Z, Liu C, Câmara NOS. The role of Raptor in lymphocytes differentiation and function [Internet]. Frontiers in Immunology. 2023 ; 14[citado 2024 jul. 06 ] Available from: https://doi.org/10.3389/fimmu.2023.1146628
Artigo de periodico
The role of B cells in COVID-19 infection and vaccination (2022)
Chen, Shiru; Guan, Fei; Candotti, Fabio ; Benlagha, Kamel ; Câmara, Niels Olsen Saraiva
Fonte: Frontiers in Immunology. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
CHEN, Shiru et al. The role of B cells in COVID-19 infection and vaccination. Frontiers in Immunology, v. 13, p. 1-13, 2022Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2022.988536. Acesso em: 06 jul. 2024.
APA
Chen, S., Guan, F., Candotti, F., Benlagha, K., & Câmara, N. O. S. (2022). The role of B cells in COVID-19 infection and vaccination. Frontiers in Immunology, 13, 1-13. doi:10.3389/fimmu.2022.988536
NLM
Chen S, Guan F, Candotti F, Benlagha K, Câmara NOS. The role of B cells in COVID-19 infection and vaccination [Internet]. Frontiers in Immunology. 2022 ; 13 1-13.[citado 2024 jul. 06 ] Available from: https://doi.org/10.3389/fimmu.2022.988536
Vancouver
Chen S, Guan F, Candotti F, Benlagha K, Câmara NOS. The role of B cells in COVID-19 infection and vaccination [Internet]. Frontiers in Immunology. 2022 ; 13 1-13.[citado 2024 jul. 06 ] Available from: https://doi.org/10.3389/fimmu.2022.988536
Artigo de periodico
Memory CD4+ and CD8+ T lymphocyte proliferation in vaccinated dairy cows with different histories of Staphylococcus aureus mastitis (2022)
Soares, Thais Cristine dos Santos ; Santos, Kamila Reis ; Lima, Daniel Magalhães ; Maia, Raysa Brenda Marques ; Sanchez, Eduardo Milton Ramos ; Reis, Luiza de Campos ; Gidlund, Magnus Ake ; Cunha, Adriano F. da ; Ramirez, Carla M. Ordinola ; Cerqueira, Mônica M. O. P. ; Heinemann, Marcos Bryan ; Libera, Alice Maria Melville Paiva Della ; Goto, Hiro ; Souza, Fernando Nogueira de
Fonte: Veterinary Immunology and Immunopathology. Unidades: ICB, FMVZ, FM
Assuntos: BOVINOS LEITEIROS, IMUNOLOGIA, MASTITE ANIMAL, STAPHYLOCOCCUS, LINFÓCITOS, MEMÓRIA ANIMAL, INFECÇÕES BACTERIANAS GRAM-POSITIVAS, INFECÇÕES ESTAFILOCÓCICAS, PROLIFERAÇÃO CELULAR
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ABNT
SOARES, Thais Cristine dos Santos et al. Memory CD4+ and CD8+ T lymphocyte proliferation in vaccinated dairy cows with different histories of Staphylococcus aureus mastitis. Veterinary Immunology and Immunopathology, v. 253, p. 1-5, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.vetimm.2022.110508. Acesso em: 06 jul. 2024.
APA
Soares, T. C. dos S., Santos, K. R., Lima, D. M., Maia, R. B. M., Sanchez, E. M. R., Reis, L. de C., et al. (2022). Memory CD4+ and CD8+ T lymphocyte proliferation in vaccinated dairy cows with different histories of Staphylococcus aureus mastitis. Veterinary Immunology and Immunopathology, 253, 1-5. doi:10.1016/j.vetimm.2022.110508
NLM
Soares TC dos S, Santos KR, Lima DM, Maia RBM, Sanchez EMR, Reis L de C, Gidlund MA, Cunha AF da, Ramirez CMO, Cerqueira MMOP, Heinemann MB, Libera AMMPD, Goto H, Souza FN de. Memory CD4+ and CD8+ T lymphocyte proliferation in vaccinated dairy cows with different histories of Staphylococcus aureus mastitis [Internet]. Veterinary Immunology and Immunopathology. 2022 ; 253 1-5.[citado 2024 jul. 06 ] Available from: https://doi.org/10.1016/j.vetimm.2022.110508
Vancouver
Soares TC dos S, Santos KR, Lima DM, Maia RBM, Sanchez EMR, Reis L de C, Gidlund MA, Cunha AF da, Ramirez CMO, Cerqueira MMOP, Heinemann MB, Libera AMMPD, Goto H, Souza FN de. Memory CD4+ and CD8+ T lymphocyte proliferation in vaccinated dairy cows with different histories of Staphylococcus aureus mastitis [Internet]. Veterinary Immunology and Immunopathology. 2022 ; 253 1-5.[citado 2024 jul. 06 ] Available from: https://doi.org/10.1016/j.vetimm.2022.110508
Artigo de periodico
Gut-kidney axis in IgA nephropathy: role on mesangial cell metabolism and inflammation (2022)
Luvizotto, Mateus Justi ; Silva, Luísa Menezes ; Woronik, Viktoria ; Monteiro, Renato C. ; Câmara, Niels Olsen Saraiva
Fonte: Frontiers in Cell and Developmental Biology. Unidades: FM, ICB
Assuntos: IMUNOLOGIA, NEFROPATIAS, IMUNOGLOBULINAS, GLOMÉRULOS RENAIS, CITOCINAS, GLOMERUNEFRITE, INFLAMAÇÃO
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ABNT
LUVIZOTTO, Mateus Justi et al. Gut-kidney axis in IgA nephropathy: role on mesangial cell metabolism and inflammation. Frontiers in Cell and Developmental Biology, v. 10, p. 1-16, 2022Tradução . . Disponível em: https://doi.org/10.3389/fcell.2022.993716. Acesso em: 06 jul. 2024.
APA
Luvizotto, M. J., Silva, L. M., Woronik, V., Monteiro, R. C., & Câmara, N. O. S. (2022). Gut-kidney axis in IgA nephropathy: role on mesangial cell metabolism and inflammation. Frontiers in Cell and Developmental Biology, 10, 1-16. doi:10.3389/fcell.2022.993716
NLM
Luvizotto MJ, Silva LM, Woronik V, Monteiro RC, Câmara NOS. Gut-kidney axis in IgA nephropathy: role on mesangial cell metabolism and inflammation [Internet]. Frontiers in Cell and Developmental Biology. 2022 ; 10 1-16.[citado 2024 jul. 06 ] Available from: https://doi.org/10.3389/fcell.2022.993716
Vancouver
Luvizotto MJ, Silva LM, Woronik V, Monteiro RC, Câmara NOS. Gut-kidney axis in IgA nephropathy: role on mesangial cell metabolism and inflammation [Internet]. Frontiers in Cell and Developmental Biology. 2022 ; 10 1-16.[citado 2024 jul. 06 ] Available from: https://doi.org/10.3389/fcell.2022.993716
Artigo de periodico-carta/editorial
Harnessing Type I IFN immunity against SARS-CoV-2 with early administration of IFN-β (2021)
Vinh, Donald C. ; Abel, Laurent ; Bastard, Paul ; Cheng, Matthew P. ; Condino Neto, Antonio ; Gregersen, Peter K. ; Haerynck, Filomeen ; Cicalese, Maria-Pia ; Hagin, David; Palacín, Pere Soler ; Planas, Anna M. ; Pujol, Aurora ; Notarangelo, Luigi D. ; Zhang, Qian ; Su, Helen C. ; Casanova, Jean-Laurent ; Meyts, Isabelle
Fonte: Journal of Clinical Immunology. Unidade: ICB
Assuntos: IMUNOLOGIA, IMUNIDADE, IMUNODEFICIÊNCIA DE VARIÁVEL COMUM, IMUNOGENÉTICA, COVID-19, INTERFERON TIPO I, INFECÇÕES POR CORONAVIRUS, ESTUDOS DE COORTES
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ABNT
VINH, Donald C. et al. Harnessing Type I IFN immunity against SARS-CoV-2 with early administration of IFN-β. Journal of Clinical Immunology. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo. Disponível em: https://doi.org/10.1007/s10875-021-01068-6. Acesso em: 06 jul. 2024. , 2021
APA
Vinh, D. C., Abel, L., Bastard, P., Cheng, M. P., Condino Neto, A., Gregersen, P. K., et al. (2021). Harnessing Type I IFN immunity against SARS-CoV-2 with early administration of IFN-β. Journal of Clinical Immunology. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo. doi:10.1007/s10875-021-01068-6
NLM
Vinh DC, Abel L, Bastard P, Cheng MP, Condino Neto A, Gregersen PK, Haerynck F, Cicalese M-P, Hagin D, Palacín PS, Planas AM, Pujol A, Notarangelo LD, Zhang Q, Su HC, Casanova J-L, Meyts I. Harnessing Type I IFN immunity against SARS-CoV-2 with early administration of IFN-β [Internet]. Journal of Clinical Immunology. 2021 ; 1-18.[citado 2024 jul. 06 ] Available from: https://doi.org/10.1007/s10875-021-01068-6
Vancouver
Vinh DC, Abel L, Bastard P, Cheng MP, Condino Neto A, Gregersen PK, Haerynck F, Cicalese M-P, Hagin D, Palacín PS, Planas AM, Pujol A, Notarangelo LD, Zhang Q, Su HC, Casanova J-L, Meyts I. Harnessing Type I IFN immunity against SARS-CoV-2 with early administration of IFN-β [Internet]. Journal of Clinical Immunology. 2021 ; 1-18.[citado 2024 jul. 06 ] Available from: https://doi.org/10.1007/s10875-021-01068-6
Artigo de periodico
Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity (2021)
Nascimento, Daniele Carvalho ; Viacava, Paula Ramos; Ferreira, Raphael Gomes; Damaceno, Marina Alves; Piñeros, Annie Rocío Alvarez ; Melo, Paulo Henrique de; Donate, Paula Barbim ; Toller-Kawahisa, Juliana Escher; Zoppi, Daniel ; Veras, Flávio Protásio ; Peres, Raphael S. ; Silva, Luisa Menezes ; Caetité, Diego ; Oliveira, Antonio Edson Rocha; Castro, Icaro Maia Santos ; Kauffenstein, Gilles ; Nakaya, Helder Takashi Imoto ; Borges, Marcos de Carvalho ; Zamboni, Dario Simões ; Fonseca, Denise Morais da ; Paschoal, Jonas Augusto Rizzato ; Cunha, Thiago Mattar ; Quesniaux, Valerie ; Linden, Joel ; Cunha, Fernando Queiróz ; Ryffe, Bernhard; Alves-Filho, José Carlos
Fonte: Immunity. Unidades: FMRP, FCFRP, FCF, ICB, BIOINFORMÁTICA
Assuntos: ADENOSINA, FÁRMACOS IMUNOSSUPRESSORES, SEPSE, CAMUNDONGOS, INTERLEUCINA 10, MACRÓFAGOS
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ABNT
NASCIMENTO, Daniele Carvalho et al. Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity. Immunity, v. 54, n. 9, p. 2024-2041, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.immuni.2021.08.005. Acesso em: 06 jul. 2024.
APA
Nascimento, D. C., Viacava, P. R., Ferreira, R. G., Damaceno, M. A., Piñeros, A. R. A., Melo, P. H. de, et al. (2021). Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity. Immunity, 54( 9), 2024-2041. doi:10.1016/j.immuni.2021.08.005
NLM
Nascimento DC, Viacava PR, Ferreira RG, Damaceno MA, Piñeros ARA, Melo PH de, Donate PB, Toller-Kawahisa JE, Zoppi D, Veras FP, Peres RS, Silva LM, Caetité D, Oliveira AER, Castro IMS, Kauffenstein G, Nakaya HTI, Borges M de C, Zamboni DS, Fonseca DM da, Paschoal JAR, Cunha TM, Quesniaux V, Linden J, Cunha FQ, Ryffe B, Alves-Filho JC. Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity [Internet]. Immunity. 2021 ; 54( 9): 2024-2041.[citado 2024 jul. 06 ] Available from: https://doi.org/10.1016/j.immuni.2021.08.005
Vancouver
Nascimento DC, Viacava PR, Ferreira RG, Damaceno MA, Piñeros ARA, Melo PH de, Donate PB, Toller-Kawahisa JE, Zoppi D, Veras FP, Peres RS, Silva LM, Caetité D, Oliveira AER, Castro IMS, Kauffenstein G, Nakaya HTI, Borges M de C, Zamboni DS, Fonseca DM da, Paschoal JAR, Cunha TM, Quesniaux V, Linden J, Cunha FQ, Ryffe B, Alves-Filho JC. Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity [Internet]. Immunity. 2021 ; 54( 9): 2024-2041.[citado 2024 jul. 06 ] Available from: https://doi.org/10.1016/j.immuni.2021.08.005
Artigo de periodico
Inherited GATA2 deficiency is dominant by haploinsufficiency and displays incomplete clinical penetrance (2021)
Quintas, Carmen Oleaga ; Oliveira Júnior, Edgar Borges de; Rosain, Jérémie ; Rapaport, Franck ; Deswarte, Caroline ; Guérin, Antoine; Sajjath, Sairaj Munavar ; Zhou, Yu Jerry ; Marot, Stéphane; Lozano, Claire ; Branco, Lidia ; Hidalgo, Nuria Fernández ; Lew, Dukhee Betty ; Brunel, Anne Sophiehttps ; Thomas, Caroline; Launay, Elise ; Arias, Andrés Augusto ; Cuffel, Alexis; Monjo, Vanesa Cunill ; Neehus, Anna-Lena ; Marques, Laura; Roynard, Manon; Vélez, Marcela Moncada ; Gerçeker, Bengü; Colobran, Roger ; Vigué, Marie-Gabrielle; Herrera, Gabriela Lopez ; Ruiz, Laura Berron ; Méndez, Nora Hilda Segura ; Romanillos, Patricia O’Farrill ; Voyer, Tom Le; Puel, Anne ; Chantelot, Christine Bellanné ; Ramirez, Kacy A.; Diaz, Lazaro Lorenzo ; Alejo, Noé Ramirez ; Diego, Rebeca Pérez de ; Condino Neto, Antonio ; Mellouli, Fethi ; Gallego, Carlos Rodriguez; Witte, Torsten ; Restrepo, José Franco; Jobim, Mariana ; Dupuis, Stéphanie Boisson ; Jeziorski, Eric ; Fieschi, Claire ; Vogt, Guillaume ; Donadieu, Jean ; Pasquet, Marlène ; Vasconcelos, Julia; Ardeniz, Fatma Omur; Gallo, Mónica Martínez ; Campos, Regis A.; Jobim, Luiz Fernando; Barricarte, Rubén Martínez ; Liu, Kang; Cobat, Aurélie; Abel, Laurent ; Casanova, Jean-Laurent ; Bustamante, Jacinta
Fonte: Journal of Clinical Immunology. Unidade: ICB
Assuntos: IMUNOLOGIA, IMUNODEFICIÊNCIA DE VARIÁVEL COMUM, TUBERCULOSE, INTERLEUCINA 8, POLIMORFISMO, ALELOS, IMUNOGENÉTICA, MUTAÇÃO GENÉTICA, FENÓTIPOS
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ABNT
QUINTAS, Carmen Oleaga et al. Inherited GATA2 deficiency is dominant by haploinsufficiency and displays incomplete clinical penetrance. Journal of Clinical Immunology, p. 19 , 2021Tradução . . Disponível em: https://doi.org/10.1007/s10875-020-00930-3. Acesso em: 06 jul. 2024.
APA
Quintas, C. O., Oliveira Júnior, E. B. de, Rosain, J., Rapaport, F., Deswarte, C., Guérin, A., et al. (2021). Inherited GATA2 deficiency is dominant by haploinsufficiency and displays incomplete clinical penetrance. Journal of Clinical Immunology, 19 . doi:10.1007/s10875-020-00930-3
NLM
Quintas CO, Oliveira Júnior EB de, Rosain J, Rapaport F, Deswarte C, Guérin A, Sajjath SM, Zhou YJ, Marot S, Lozano C, Branco L, Hidalgo NF, Lew DB, Brunel AS, Thomas C, Launay E, Arias AA, Cuffel A, Monjo VC, Neehus A-L, Marques L, Roynard M, Vélez MM, Gerçeker B, Colobran R, Vigué M-G, Herrera GL, Ruiz LB, Méndez NHS, Romanillos PO’F, Voyer TL, Puel A, Chantelot CB, Ramirez KA, Diaz LL, Alejo NR, Diego RP de, Condino Neto A, Mellouli F, Gallego CR, Witte T, Restrepo JF, Jobim M, Dupuis SB, Jeziorski E, Fieschi C, Vogt G, Donadieu J, Pasquet M, Vasconcelos J, Ardeniz FO, Gallo MM, Campos RA, Jobim LF, Barricarte RM, Liu K, Cobat A, Abel L, Casanova J-L, Bustamante J. Inherited GATA2 deficiency is dominant by haploinsufficiency and displays incomplete clinical penetrance [Internet]. Journal of Clinical Immunology. 2021 ;19 .[citado 2024 jul. 06 ] Available from: https://doi.org/10.1007/s10875-020-00930-3
Vancouver
Quintas CO, Oliveira Júnior EB de, Rosain J, Rapaport F, Deswarte C, Guérin A, Sajjath SM, Zhou YJ, Marot S, Lozano C, Branco L, Hidalgo NF, Lew DB, Brunel AS, Thomas C, Launay E, Arias AA, Cuffel A, Monjo VC, Neehus A-L, Marques L, Roynard M, Vélez MM, Gerçeker B, Colobran R, Vigué M-G, Herrera GL, Ruiz LB, Méndez NHS, Romanillos PO’F, Voyer TL, Puel A, Chantelot CB, Ramirez KA, Diaz LL, Alejo NR, Diego RP de, Condino Neto A, Mellouli F, Gallego CR, Witte T, Restrepo JF, Jobim M, Dupuis SB, Jeziorski E, Fieschi C, Vogt G, Donadieu J, Pasquet M, Vasconcelos J, Ardeniz FO, Gallo MM, Campos RA, Jobim LF, Barricarte RM, Liu K, Cobat A, Abel L, Casanova J-L, Bustamante J. Inherited GATA2 deficiency is dominant by haploinsufficiency and displays incomplete clinical penetrance [Internet]. Journal of Clinical Immunology. 2021 ;19 .[citado 2024 jul. 06 ] Available from: https://doi.org/10.1007/s10875-020-00930-3
Artigo de periodico
Autoantibodies neutralizing type I IFNs are present in ~4% of uninfected individuals over 70 years old and account for ~20% of COVID-19 deaths (2021)
Bastard, Paul ; Gervais, Adrian ; Voyer, Tom Le ; Rosain, Jérémie ; Philippot, Quentin; Manry, Jérémy ; Michailidis, Eleftherios ; Hoffmann, Hans-Heinrich ; Eto, Shohei; Garcia-Prat, Marina; Bizien, Lucy; Parra-Martínez, Alba; Yang, Rui; Liis, Haljasmägi; Migaud, Mélanie; Särekannu, Karita; Maslovskaja, Julia; Prost, Nicolas de; Tandjaoui-Lambiotte, Yacine; Luyt , Charles-Edouard; Amador-Borrero, Blanca; Alexandre, Gaudet; Poissy, Julien; Morel, Pascal; Richard, Pascale; Cognasse, Fabrice; Troya, Jesus; Trouillet-Assant, Sophie; Belot, Alexandre; Saker, Kahina; Garçon, Pierre; Rivière, Jacques G; Lagier, Jean-Christophe; Gentile, Stéphanie; Rosen, Lindsey B; Shaw, Elana; Morio, Tomohiro; Tanaka, Junko; Dalmau, David; Tharaux, Pierre-Louis; Sene, Damien; Stepanian, Alain; Megarbane, Bruno; Triantafyllia, Vasiliki; Fekkar, Arnaud; Heath, James R; Franco, José Luis; Anaya, Juan-Manuel; Solé-Violán, Jordi; Imberti, Luisa; Biondi, Andrea; Bonfanti, Paolo; Castagnoli, Riccardo; Delmonte, Ottavia M; Zhang, Yu; Snow, Andrew L; Holland, Steven M; Biggs, Catherine M; Moncada-Vélez, Marcela; Arias, Andrés Augusto; Lorenzo, Lazaro; Boucherit, Soraya; Coulibaly, Boubacar; Anglicheau, Dany; Planas, Anna M; Haerynck, Filomeen; Duvlis, Sotirija; Nussbaum, Robert L; Ozcelik, Tayfun; Keles, Sevgi; Bousfiha, Ahmed A; ElBakkouri, Jalila; Ramirez-Santana, Carolina; Paul, Stéphane; Pan-Hammarström, Qiang; Hammarström, Lennart; Dupont, Annabelle; Kurolap, Alina; Metz, Christine N; Aiuti, Alessandro; Casari, Giorgio; Lampasona, Vito; Ciceri, Fabio; Barreiros, Lucila A; Dominguez-Garrido, Elena; Vidigal, Mateus; Zatz, Mayana ; Beek, Diederik van de; Sahanic, Sabina; Tancevski, Ivan; Stepanovskyy, Yurii; Boyarchuk, Oksana; Nukui, Yoko; Tsumura, Miyuki; Vidaur, Loreto; Tangye, Stuart G; Burrel, Sonia; Duffy, Darragh; Quintana-Murci, Lluis; Klocperk, Adam; Kann, Nelli Y; Shcherbina, Anna; Lau, Yu-Lung; Leung, Daniel; Coulongeat, Matthieu; Marlet, Julien; Koning, Rutger; Reyes, Luis Felipe; Chauvineau-Grenier, Angélique; Vene, Fabienne; Monneret, Guillaume; Nussenzweig, Michel C; Arrestier, Romain; Boudhabhay, Idris; Baris-Feldman, Hagit; Hagin, David; Wauters, Joost; Meyts, Isabelle; Dyer, Adam H; Kennelly, Sean P; Bourke, Nollaig M; Halwani, Rabih; Sharif-Askari, Narjes Saheb; Dorgham, Karim; Sallette, Jérome; Sedkaoui, Souad Mehlal; AlKhater, Suzan; Rigo-Bonnin, Raúl; Morandeira, Francisco; Roussel, Lucie; Vinh, Donald C; Ostrowski, Sisse Rye; Condino Neto, Antonio ; Prando, Carolina ; Bondarenko, Anastasiia; Spaan, András N; Gilardin, Laurent; Fellay, Jacques; Lyonnet, Stanislas; Bilguvar, Kaya; Lifton, Richard P; Mane, Shrikant; Anderson, Mark S; Boisson, Bertrand; Béziat, Vivien; Zhang, Shen-Ying; Andreakos, Evangelos; Hermine, Olivier; Pujol, Aurora; Peterson, Pärt; Mogensen, Trine; H Rowen, Lee; Mond, James; Debette, Stéphanie; Lamballerie, Xavier de; Duval, Xavier; Mentré, France; Zins, Marie; Soler-Palacin, Pere; Colobran, Roger; Gorochov, Guy; Solanich, Xavier; Susen, Sophie; Martinez-Picado, Javier; Raoult, Didier; Vasse, Marc; Gregersen, Peter K; Piemonti, Lorenzo; Rodríguez-Gallego, Carlos; Notarangelo, Luigi D; Su, Helen C; Kisand, Kai; Okada, Satoshi; Puel, Anne; Jouanguy, Emmanuelle; Rice, Charles M; Tiberghien, Pierre; Zhang, Qian; Cobat, Aurélie; Abel, Laurent; Casanova, Jean-Laurent
Fonte: Science Immunology. Unidades: IB, ICB
Assuntos: COVID-19, INFECÇÕES POR CORONAVIRUS, ANTICORPOS, IMUNOLOGIA, HOMENS
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ABNT
BASTARD, Paul et al. Autoantibodies neutralizing type I IFNs are present in ~4% of uninfected individuals over 70 years old and account for ~20% of COVID-19 deaths. Science Immunology, v. 6, n. 62, p. 1-30, 2021Tradução . . Disponível em: https://doi.org/10.1126/sciimmunol.abl4340. Acesso em: 06 jul. 2024.
APA
Bastard, P., Gervais, A., Voyer, T. L., Rosain, J., Philippot, Q., Manry, J., et al. (2021). Autoantibodies neutralizing type I IFNs are present in ~4% of uninfected individuals over 70 years old and account for ~20% of COVID-19 deaths. Science Immunology, 6( 62), 1-30. doi:10.1126/sciimmunol.abl4340
NLM
Bastard P, Gervais A, Voyer TL, Rosain J, Philippot Q, Manry J, Michailidis E, Hoffmann H-H, Eto S, Garcia-Prat M, Bizien L, Parra-Martínez A, Yang R, Liis H, Migaud M, Särekannu K, Maslovskaja J, Prost N de, Tandjaoui-Lambiotte Y, Luyt C-E, Amador-Borrero B, Alexandre G, Poissy J, Morel P, Richard P, Cognasse F, Troya J, Trouillet-Assant S, Belot A, Saker K, Garçon P, Rivière JG, Lagier J-C, Gentile S, Rosen LB, Shaw E, Morio T, Tanaka J, Dalmau D, Tharaux P-L, Sene D, Stepanian A, Megarbane B, Triantafyllia V, Fekkar A, Heath JR, Franco JL, Anaya J-M, Solé-Violán J, Imberti L, Biondi A, Bonfanti P, Castagnoli R, Delmonte OM, Zhang Y, Snow AL, Holland SM, Biggs CM, Moncada-Vélez M, Arias AA, Lorenzo L, Boucherit S, Coulibaly B, Anglicheau D, Planas AM, Haerynck F, Duvlis S, Nussbaum RL, Ozcelik T, Keles S, Bousfiha AA, ElBakkouri J, Ramirez-Santana C, Paul S, Pan-Hammarström Q, Hammarström L, Dupont A, Kurolap A, Metz CN, Aiuti A, Casari G, Lampasona V, Ciceri F, Barreiros LA, Dominguez-Garrido E, Vidigal M, Zatz M, Beek D van de, Sahanic S, Tancevski I, Stepanovskyy Y, Boyarchuk O, Nukui Y, Tsumura M, Vidaur L, Tangye SG, Burrel S, Duffy D, Quintana-Murci L, Klocperk A, Kann NY, Shcherbina A, Lau Y-L, Leung D, Coulongeat M, Marlet J, Koning R, Reyes LF, Chauvineau-Grenier A, Vene F, Monneret G, Nussenzweig MC, Arrestier R, Boudhabhay I, Baris-Feldman H, Hagin D, Wauters J, Meyts I, Dyer AH, Kennelly SP, Bourke NM, Halwani R, Sharif-Askari NS, Dorgham K, Sallette J, Sedkaoui SM, AlKhater S, Rigo-Bonnin R, Morandeira F, Roussel L, Vinh DC, Ostrowski SR, Condino Neto A, Prando C, Bondarenko A, Spaan AN, Gilardin L, Fellay J, Lyonnet S, Bilguvar K, Lifton RP, Mane S, Anderson MS, Boisson B, Béziat V, Zhang S-Y, Andreakos E, Hermine O, Pujol A, Peterson P, Mogensen T, H Rowen L, Mond J, Debette S, Lamballerie X de, Duval X, Mentré F, Zins M, Soler-Palacin P, Colobran R, Gorochov G, Solanich X, Susen S, Martinez-Picado J, Raoult D, Vasse M, Gregersen PK, Piemonti L, Rodríguez-Gallego C, Notarangelo LD, Su HC, Kisand K, Okada S, Puel A, Jouanguy E, Rice CM, Tiberghien P, Zhang Q, Cobat A, Abel L, Casanova J-L. Autoantibodies neutralizing type I IFNs are present in ~4% of uninfected individuals over 70 years old and account for ~20% of COVID-19 deaths [Internet]. Science Immunology. 2021 ; 6( 62): 1-30.[citado 2024 jul. 06 ] Available from: https://doi.org/10.1126/sciimmunol.abl4340
Vancouver
Bastard P, Gervais A, Voyer TL, Rosain J, Philippot Q, Manry J, Michailidis E, Hoffmann H-H, Eto S, Garcia-Prat M, Bizien L, Parra-Martínez A, Yang R, Liis H, Migaud M, Särekannu K, Maslovskaja J, Prost N de, Tandjaoui-Lambiotte Y, Luyt C-E, Amador-Borrero B, Alexandre G, Poissy J, Morel P, Richard P, Cognasse F, Troya J, Trouillet-Assant S, Belot A, Saker K, Garçon P, Rivière JG, Lagier J-C, Gentile S, Rosen LB, Shaw E, Morio T, Tanaka J, Dalmau D, Tharaux P-L, Sene D, Stepanian A, Megarbane B, Triantafyllia V, Fekkar A, Heath JR, Franco JL, Anaya J-M, Solé-Violán J, Imberti L, Biondi A, Bonfanti P, Castagnoli R, Delmonte OM, Zhang Y, Snow AL, Holland SM, Biggs CM, Moncada-Vélez M, Arias AA, Lorenzo L, Boucherit S, Coulibaly B, Anglicheau D, Planas AM, Haerynck F, Duvlis S, Nussbaum RL, Ozcelik T, Keles S, Bousfiha AA, ElBakkouri J, Ramirez-Santana C, Paul S, Pan-Hammarström Q, Hammarström L, Dupont A, Kurolap A, Metz CN, Aiuti A, Casari G, Lampasona V, Ciceri F, Barreiros LA, Dominguez-Garrido E, Vidigal M, Zatz M, Beek D van de, Sahanic S, Tancevski I, Stepanovskyy Y, Boyarchuk O, Nukui Y, Tsumura M, Vidaur L, Tangye SG, Burrel S, Duffy D, Quintana-Murci L, Klocperk A, Kann NY, Shcherbina A, Lau Y-L, Leung D, Coulongeat M, Marlet J, Koning R, Reyes LF, Chauvineau-Grenier A, Vene F, Monneret G, Nussenzweig MC, Arrestier R, Boudhabhay I, Baris-Feldman H, Hagin D, Wauters J, Meyts I, Dyer AH, Kennelly SP, Bourke NM, Halwani R, Sharif-Askari NS, Dorgham K, Sallette J, Sedkaoui SM, AlKhater S, Rigo-Bonnin R, Morandeira F, Roussel L, Vinh DC, Ostrowski SR, Condino Neto A, Prando C, Bondarenko A, Spaan AN, Gilardin L, Fellay J, Lyonnet S, Bilguvar K, Lifton RP, Mane S, Anderson MS, Boisson B, Béziat V, Zhang S-Y, Andreakos E, Hermine O, Pujol A, Peterson P, Mogensen T, H Rowen L, Mond J, Debette S, Lamballerie X de, Duval X, Mentré F, Zins M, Soler-Palacin P, Colobran R, Gorochov G, Solanich X, Susen S, Martinez-Picado J, Raoult D, Vasse M, Gregersen PK, Piemonti L, Rodríguez-Gallego C, Notarangelo LD, Su HC, Kisand K, Okada S, Puel A, Jouanguy E, Rice CM, Tiberghien P, Zhang Q, Cobat A, Abel L, Casanova J-L. Autoantibodies neutralizing type I IFNs are present in ~4% of uninfected individuals over 70 years old and account for ~20% of COVID-19 deaths [Internet]. Science Immunology. 2021 ; 6( 62): 1-30.[citado 2024 jul. 06 ] Available from: https://doi.org/10.1126/sciimmunol.abl4340
Artigo de periodico
A global effort to dissect the human genetic basis of resistance to SARS-CoV-2 infection (2021)
Andreakos, Evangelos ; Abel, Laurent ; Vinh, Donald C. ; Kaja, Elżbieta ; Drolet, Beth A. ; Zhang, Qian ; O’Farrelly, Cliona ; Novelli, Giuseppe ; Gallego, Carlos Rodríguez ; Haerynck, Filomeen ; Prando, Carolina ; Pujol, Aurora ; Condino Neto, Antonio ; Zatz, Mayana
Fonte: Nature Immunology. Unidades: ICB, IB
Assuntos: IMUNOLOGIA, INFECÇÕES POR CORONAVIRUS, GENÉTICA MÉDICA, INTERFERON TIPO I, QUIMIOCINAS, PLASMODIUM
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ANDREAKOS, Evangelos et al. A global effort to dissect the human genetic basis of resistance to SARS-CoV-2 infection. Nature Immunology, p. 1-6, 2021Tradução . . Disponível em: https://doi.org/10.1038/s41590-021-01030-z. Acesso em: 06 jul. 2024.
APA
Andreakos, E., Abel, L., Vinh, D. C., Kaja, E., Drolet, B. A., Zhang, Q., et al. (2021). A global effort to dissect the human genetic basis of resistance to SARS-CoV-2 infection. Nature Immunology, 1-6. doi:10.1038/s41590-021-01030-z
NLM
Andreakos E, Abel L, Vinh DC, Kaja E, Drolet BA, Zhang Q, O’Farrelly C, Novelli G, Gallego CR, Haerynck F, Prando C, Pujol A, Condino Neto A, Zatz M. A global effort to dissect the human genetic basis of resistance to SARS-CoV-2 infection [Internet]. Nature Immunology. 2021 ; 1-6.[citado 2024 jul. 06 ] Available from: https://doi.org/10.1038/s41590-021-01030-z
Vancouver
Andreakos E, Abel L, Vinh DC, Kaja E, Drolet BA, Zhang Q, O’Farrelly C, Novelli G, Gallego CR, Haerynck F, Prando C, Pujol A, Condino Neto A, Zatz M. A global effort to dissect the human genetic basis of resistance to SARS-CoV-2 infection [Internet]. Nature Immunology. 2021 ; 1-6.[citado 2024 jul. 06 ] Available from: https://doi.org/10.1038/s41590-021-01030-z
Artigo de periodico-carta/editorial
Author Correction: a global effort to dissect the human genetic basis of resistance to SARS-CoV-2 infection (2021)
Andreakos, Evangelos ; Abel, Laurent ; Vinh, Donald C. ; Kaja, Elżbieta ; Drolet, Beth A. ; Zhang, Qian ; O’Farrelly, Cliona ; Novelli, Giuseppe ; Gallego, Carlos Rodríguez ; Haerynck, Filomeen ; Prando, Carolina ; Pujol, Aurora ; Condino Neto, Antonio ; Su, Helen C. ; Casanova, Jean-Laurent ; Spaan, András N. ; Zatz, Mayana
Fonte: Nature Immunology. Unidades: ICB, IB
Assuntos: IMUNOLOGIA, INFECÇÕES POR CORONAVIRUS, CORONAVIRUS, GENÉTICA MÉDICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ANDREAKOS, Evangelos et al. Author Correction: a global effort to dissect the human genetic basis of resistance to SARS-CoV-2 infection. Nature Immunology. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo. Disponível em: https://doi.org/10.1038/s41590-021-01096-9. Acesso em: 06 jul. 2024. , 2021
APA
Andreakos, E., Abel, L., Vinh, D. C., Kaja, E., Drolet, B. A., Zhang, Q., et al. (2021). Author Correction: a global effort to dissect the human genetic basis of resistance to SARS-CoV-2 infection. Nature Immunology. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo. doi:10.1038/s41590-021-01096-9
NLM
Andreakos E, Abel L, Vinh DC, Kaja E, Drolet BA, Zhang Q, O’Farrelly C, Novelli G, Gallego CR, Haerynck F, Prando C, Pujol A, Condino Neto A, Su HC, Casanova J-L, Spaan AN, Zatz M. Author Correction: a global effort to dissect the human genetic basis of resistance to SARS-CoV-2 infection [Internet]. Nature Immunology. 2021 ;1 .[citado 2024 jul. 06 ] Available from: https://doi.org/10.1038/s41590-021-01096-9
Vancouver
Andreakos E, Abel L, Vinh DC, Kaja E, Drolet BA, Zhang Q, O’Farrelly C, Novelli G, Gallego CR, Haerynck F, Prando C, Pujol A, Condino Neto A, Su HC, Casanova J-L, Spaan AN, Zatz M. Author Correction: a global effort to dissect the human genetic basis of resistance to SARS-CoV-2 infection [Internet]. Nature Immunology. 2021 ;1 .[citado 2024 jul. 06 ] Available from: https://doi.org/10.1038/s41590-021-01096-9
Artigo de periodico
Hematologically important mutations: X-linked chronic granulomatous disease (fourth update) (2021)
Roos, Dirk; Leeuwen, Karin van; Hsu, Amy P.; Priel, Debra Long ; Begtrup, Amber ; Brandon, Rhonda; Stasia, Marie José ; Bakri, Faris Ghalib ; Köker, Nezihe ; Köker, Mustafa Yavuz ; Madkaika, Manisha; Boer, Martin de ; Morato, Maria Bravo Garcia ; Shephard, Juan Luis Valdivieso; Roesler, Joachim ; Kanegane, Hirokazu ; Kawai, Toshinao ; Matteo, Gigliola Di ; Shahrooei, Mohammad ; Bustamante, Jacinta ; Rawat, Amit ; Vignesh, Pandiarajan ; Mortaz, Esmaeil ; Fayezi, Abbas; Cagdas, Deniz ; Tezcan, Ilhan ; Kitcharoensakkul, Maleewan ; Dinauer, Mary C. ; Meyts, Isabelle; Wolach, Baruch; Condino Neto, Antonio ; Zerbe, Christa S. ; Holland, Steven M.; Malech, Harry L. ; Gallin, John I.; Kuhns, Douglas B.
Fonte: Blood Cells, Molecules and Diseases. Unidade: ICB
Assuntos: IMUNOLOGIA, IMUNODEFICIÊNCIA DE VARIÁVEL COMUM, MUTAÇÃO GENÉTICA, DOENÇAS SANGUÍNEAS E LINFÁTICAS, SUPERÓXIDO DISMUTASE, ATIVAÇÃO ENZIMÁTICA, IMUNOGENÉTICA, ENZIMAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ROOS, Dirk et al. Hematologically important mutations: X-linked chronic granulomatous disease (fourth update). Blood Cells, Molecules and Diseases, v. 90, p. 1-36, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.bcmd.2021.102587. Acesso em: 06 jul. 2024.
APA
Roos, D., Leeuwen, K. van, Hsu, A. P., Priel, D. L., Begtrup, A., Brandon, R., et al. (2021). Hematologically important mutations: X-linked chronic granulomatous disease (fourth update). Blood Cells, Molecules and Diseases, 90, 1-36. doi:10.1016/j.bcmd.2021.102587
NLM
Roos D, Leeuwen K van, Hsu AP, Priel DL, Begtrup A, Brandon R, Stasia MJ, Bakri FG, Köker N, Köker MY, Madkaika M, Boer M de, Morato MBG, Shephard JLV, Roesler J, Kanegane H, Kawai T, Matteo GD, Shahrooei M, Bustamante J, Rawat A, Vignesh P, Mortaz E, Fayezi A, Cagdas D, Tezcan I, Kitcharoensakkul M, Dinauer MC, Meyts I, Wolach B, Condino Neto A, Zerbe CS, Holland SM, Malech HL, Gallin JI, Kuhns DB. Hematologically important mutations: X-linked chronic granulomatous disease (fourth update) [Internet]. Blood Cells, Molecules and Diseases. 2021 ; 90 1-36.[citado 2024 jul. 06 ] Available from: https://doi.org/10.1016/j.bcmd.2021.102587
Vancouver
Roos D, Leeuwen K van, Hsu AP, Priel DL, Begtrup A, Brandon R, Stasia MJ, Bakri FG, Köker N, Köker MY, Madkaika M, Boer M de, Morato MBG, Shephard JLV, Roesler J, Kanegane H, Kawai T, Matteo GD, Shahrooei M, Bustamante J, Rawat A, Vignesh P, Mortaz E, Fayezi A, Cagdas D, Tezcan I, Kitcharoensakkul M, Dinauer MC, Meyts I, Wolach B, Condino Neto A, Zerbe CS, Holland SM, Malech HL, Gallin JI, Kuhns DB. Hematologically important mutations: X-linked chronic granulomatous disease (fourth update) [Internet]. Blood Cells, Molecules and Diseases. 2021 ; 90 1-36.[citado 2024 jul. 06 ] Available from: https://doi.org/10.1016/j.bcmd.2021.102587
Artigo de periodico
Hematologically important mutations: the autosomal forms of chronic granulomatous disease (third update) (2021)
Roos, Dirk ; Leeuwen, Karin van ; Hsu, Amy P. ; Priel, Debra Long ; Begtrup, Amber ; Brandon, Rhonda; Rawat, Amit ; Vignesh, Pandiarajan ; Madkaikar, Manesha ; Stasia, Marie José ; Bakri, Faris Ghalib ; Boer, Martin de ; Roesler, Joachim ; Köker, Nezihe ; Köker, Mustafa Yavuz ; Jakobsen, Marianne ; Bustamante, Jacinta ; Morato, Maria Bravo Garcia ; Condino Neto, Antonio
Fonte: Blood Cells, Molecules and Diseases. Unidade: ICB
Assuntos: IMUNOLOGIA, DOENÇAS GENÉTICAS, IMUNODEFICIÊNCIA DE VARIÁVEL COMUM, IMUNOGENÉTICA, MUTAÇÃO GENÉTICA, POLIMORFISMO, INIBIDORES DE ENZIMAS, PERÓXIDO DE HIDROGÊNIO, APOPTOSE, HERANÇA GENÉTICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ROOS, Dirk et al. Hematologically important mutations: the autosomal forms of chronic granulomatous disease (third update). Blood Cells, Molecules and Diseases, v. 92, p. 1-16, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.bcmd.2021.102596. Acesso em: 06 jul. 2024.
APA
Roos, D., Leeuwen, K. van, Hsu, A. P., Priel, D. L., Begtrup, A., Brandon, R., et al. (2021). Hematologically important mutations: the autosomal forms of chronic granulomatous disease (third update). Blood Cells, Molecules and Diseases, 92, 1-16. doi:10.1016/j.bcmd.2021.102596
NLM
Roos D, Leeuwen K van, Hsu AP, Priel DL, Begtrup A, Brandon R, Rawat A, Vignesh P, Madkaikar M, Stasia MJ, Bakri FG, Boer M de, Roesler J, Köker N, Köker MY, Jakobsen M, Bustamante J, Morato MBG, Condino Neto A. Hematologically important mutations: the autosomal forms of chronic granulomatous disease (third update) [Internet]. Blood Cells, Molecules and Diseases. 2021 ; 92 1-16.[citado 2024 jul. 06 ] Available from: https://doi.org/10.1016/j.bcmd.2021.102596
Vancouver
Roos D, Leeuwen K van, Hsu AP, Priel DL, Begtrup A, Brandon R, Rawat A, Vignesh P, Madkaikar M, Stasia MJ, Bakri FG, Boer M de, Roesler J, Köker N, Köker MY, Jakobsen M, Bustamante J, Morato MBG, Condino Neto A. Hematologically important mutations: the autosomal forms of chronic granulomatous disease (third update) [Internet]. Blood Cells, Molecules and Diseases. 2021 ; 92 1-16.[citado 2024 jul. 06 ] Available from: https://doi.org/10.1016/j.bcmd.2021.102596
Artigo de periodico
Atypical prolonged viral shedding with Intra-Host SARS-CoV-2 evolution in a mildly affected symptomatic patient (2021)
Cunha, Marielton dos Passos ; Vilela, Ana Paula Pessoa ; Molina, Camila Vieira ; Acuña, Stephanie Maia ; Muxel, Sandra Marcia ; Barroso, Vinícius de Morais ; Baroni, Sabrina ; Oliveira, Lilian Gomes de ; Angelo, Yan de Souza ; Peron, Jean Pierre Schatzmann ; Góes, Luiz Gustavo Bentim ; Campos, Angélica Cristine de Almeida ; Minóprio, Paola
Fonte: Frontiers in Medicine. Unidades: ICB, FMRP
Assuntos: IMUNOLOGIA, CORONAVIRUS, INFECÇÕES POR CORONAVIRUS, RNA VIRAL, VÍRUS DE RNA, DIVERSIDADE GENÉTICA, DOENÇAS RESPIRATÓRIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CUNHA, Marielton dos Passos et al. Atypical prolonged viral shedding with Intra-Host SARS-CoV-2 evolution in a mildly affected symptomatic patient. Frontiers in Medicine, v. 8, p. 1-11, 2021Tradução . . Disponível em: https://doi.org/10.3389/fmed.2021.760170. Acesso em: 06 jul. 2024.
APA
Cunha, M. dos P., Vilela, A. P. P., Molina, C. V., Acuña, S. M., Muxel, S. M., Barroso, V. de M., et al. (2021). Atypical prolonged viral shedding with Intra-Host SARS-CoV-2 evolution in a mildly affected symptomatic patient. Frontiers in Medicine, 8, 1-11. doi:10.3389/fmed.2021.760170
NLM
Cunha M dos P, Vilela APP, Molina CV, Acuña SM, Muxel SM, Barroso V de M, Baroni S, Oliveira LG de, Angelo Y de S, Peron JPS, Góes LGB, Campos AC de A, Minóprio P. Atypical prolonged viral shedding with Intra-Host SARS-CoV-2 evolution in a mildly affected symptomatic patient [Internet]. Frontiers in Medicine. 2021 ; 8 1-11.[citado 2024 jul. 06 ] Available from: https://doi.org/10.3389/fmed.2021.760170
Vancouver
Cunha M dos P, Vilela APP, Molina CV, Acuña SM, Muxel SM, Barroso V de M, Baroni S, Oliveira LG de, Angelo Y de S, Peron JPS, Góes LGB, Campos AC de A, Minóprio P. Atypical prolonged viral shedding with Intra-Host SARS-CoV-2 evolution in a mildly affected symptomatic patient [Internet]. Frontiers in Medicine. 2021 ; 8 1-11.[citado 2024 jul. 06 ] Available from: https://doi.org/10.3389/fmed.2021.760170
Artigo de periodico
CD163+ tumor‐associated macrophage accumulation in breast cancer patients reflects both local differentiation signals and systemic skewing of monocytes (2020)
Ramos, Rodrigo Nalio; Rodriguez, Céline ; Hubert, Margaux ; Ardin, Maude; Treilleux, Isabelle ; Ries, Carola H.; Lavergne, Emilie ; Chabaud, Sylvie; Colombe, Amélie ; Trédan, Olivier ; Guedes, Henrique Gomes; Laginha, Fábio ; Richer, Wilfrid ; Piaggio, Eliane ; Barbuto, José Alexandre Marzagão ; Caux, Christophe ; Caux, Christine Ménétrier ; Vermare, Nathalie Bendriss
Fonte: Clinical & Translational Immunology. Unidade: ICB
Assuntos: IMUNOLOGIA, CITOMETRIA DE FLUXO, FENÓTIPOS, NEOPLASIAS MAMÁRIAS, CÉLULAS CULTIVADAS DE TUMOR, CITOCINAS, FÁRMACOS IMUNOSSUPRESSORES, MACRÓFAGOS, MONÓCITOS
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ABNT
RAMOS, Rodrigo Nalio et al. CD163+ tumor‐associated macrophage accumulation in breast cancer patients reflects both local differentiation signals and systemic skewing of monocytes. Clinical & Translational Immunology, v. 9, p. 18 , 2020Tradução . . Disponível em: https://doi.org/10.1002/cti2.1108. Acesso em: 06 jul. 2024.
APA
Ramos, R. N., Rodriguez, C., Hubert, M., Ardin, M., Treilleux, I., Ries, C. H., et al. (2020). CD163+ tumor‐associated macrophage accumulation in breast cancer patients reflects both local differentiation signals and systemic skewing of monocytes. Clinical & Translational Immunology, 9, 18 . doi:10.1002/cti2.1108
NLM
Ramos RN, Rodriguez C, Hubert M, Ardin M, Treilleux I, Ries CH, Lavergne E, Chabaud S, Colombe A, Trédan O, Guedes HG, Laginha F, Richer W, Piaggio E, Barbuto JAM, Caux C, Caux CM, Vermare NB. CD163+ tumor‐associated macrophage accumulation in breast cancer patients reflects both local differentiation signals and systemic skewing of monocytes [Internet]. Clinical & Translational Immunology. 2020 ; 9 18 .[citado 2024 jul. 06 ] Available from: https://doi.org/10.1002/cti2.1108
Vancouver
Ramos RN, Rodriguez C, Hubert M, Ardin M, Treilleux I, Ries CH, Lavergne E, Chabaud S, Colombe A, Trédan O, Guedes HG, Laginha F, Richer W, Piaggio E, Barbuto JAM, Caux C, Caux CM, Vermare NB. CD163+ tumor‐associated macrophage accumulation in breast cancer patients reflects both local differentiation signals and systemic skewing of monocytes [Internet]. Clinical & Translational Immunology. 2020 ; 9 18 .[citado 2024 jul. 06 ] Available from: https://doi.org/10.1002/cti2.1108
Artigo de periodico
Flow cytometry contributions for the diagnosis and immunopathological characterization of primary immunodeficiency diseases with immune dysregulation (2019)
Marques, Otavio Cabral ; Schimke, Lena Friederike; Oliveira Junior, Edgar Borges de ; Khawanky, Nadia El ; Ramos, Rodrigo Nalio; Al-Ramadi, Basel K. ; Segundo, Gesmar Rodrigues Silva ; Ochs, Hans D.; Condino Neto, Antonio
Fonte: Frontiers in Immunology. Unidade: ICB
Assuntos: IMUNOLOGIA, CITOMETRIA DE FLUXO, DOENÇAS IMUNOLÓGICAS, IMUNODEFICIÊNCIA DE VARIÁVEL COMUM, MUTAÇÃO GENÉTICA, SEQUENCIAMENTO GENÉTICO, GENOMAS, FENÓTIPOS, AUTOIMUNIDADE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MARQUES, Otavio Cabral et al. Flow cytometry contributions for the diagnosis and immunopathological characterization of primary immunodeficiency diseases with immune dysregulation. Frontiers in Immunology, v. 10, p. 23 , 2019Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2019.02742. Acesso em: 06 jul. 2024.
APA
Marques, O. C., Schimke, L. F., Oliveira Junior, E. B. de, Khawanky, N. E., Ramos, R. N., Al-Ramadi, B. K., et al. (2019). Flow cytometry contributions for the diagnosis and immunopathological characterization of primary immunodeficiency diseases with immune dysregulation. Frontiers in Immunology, 10, 23 . doi:10.3389/fimmu.2019.02742
NLM
Marques OC, Schimke LF, Oliveira Junior EB de, Khawanky NE, Ramos RN, Al-Ramadi BK, Segundo GRS, Ochs HD, Condino Neto A. Flow cytometry contributions for the diagnosis and immunopathological characterization of primary immunodeficiency diseases with immune dysregulation [Internet]. Frontiers in Immunology. 2019 ;10 23 .[citado 2024 jul. 06 ] Available from: https://doi.org/10.3389/fimmu.2019.02742
Vancouver
Marques OC, Schimke LF, Oliveira Junior EB de, Khawanky NE, Ramos RN, Al-Ramadi BK, Segundo GRS, Ochs HD, Condino Neto A. Flow cytometry contributions for the diagnosis and immunopathological characterization of primary immunodeficiency diseases with immune dysregulation [Internet]. Frontiers in Immunology. 2019 ;10 23 .[citado 2024 jul. 06 ] Available from: https://doi.org/10.3389/fimmu.2019.02742
Artigo de periodico
The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression (2018)
Amano, Mariane Tami ; Castoldi, Angela ; Andrade-Oliveira, Vinicius ; Latância, Marcela Teatin ; Terra, Fernanda Fernandes ; Correa-Costa, Matheus ; Breda, Cristiane Naffah Souza ; Felizardo, Raphael José Ferreira ; Pereira, Welbert Oliveira de ; Silva, Marina Burgos da ; Miyagi, Mariana Yasue Saito ; Aguiar, Cristhiane Favero ; Hiyane, Meire Ioshie ; Silva, João Santana da ; Moura, Ivan Cruz ; Câmara, Niels Olsen Saraiva
Fonte: International Immunopharmacology. Unidades: ICB, FMRP, FCF, BIOTECNOLOGIA
Assuntos: INSUFICIÊNCIA RENAL AGUDA, NEFROPATIAS, MACRÓFAGOS, IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
AMANO, Mariane Tami et al. The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression. International Immunopharmacology, v. 64, p. 151-161, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2018.08.020. Acesso em: 06 jul. 2024.
APA
Amano, M. T., Castoldi, A., Andrade-Oliveira, V., Latância, M. T., Terra, F. F., Correa-Costa, M., et al. (2018). The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression. International Immunopharmacology, 64, 151-161. doi:10.1016/j.intimp.2018.08.020
NLM
Amano MT, Castoldi A, Andrade-Oliveira V, Latância MT, Terra FF, Correa-Costa M, Breda CNS, Felizardo RJF, Pereira WO de, Silva MB da, Miyagi MYS, Aguiar CF, Hiyane MI, Silva JS da, Moura IC, Câmara NOS. The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression [Internet]. International Immunopharmacology. 2018 ; 64 151-161.[citado 2024 jul. 06 ] Available from: https://doi.org/10.1016/j.intimp.2018.08.020
Vancouver
Amano MT, Castoldi A, Andrade-Oliveira V, Latância MT, Terra FF, Correa-Costa M, Breda CNS, Felizardo RJF, Pereira WO de, Silva MB da, Miyagi MYS, Aguiar CF, Hiyane MI, Silva JS da, Moura IC, Câmara NOS. The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression [Internet]. International Immunopharmacology. 2018 ; 64 151-161.[citado 2024 jul. 06 ] Available from: https://doi.org/10.1016/j.intimp.2018.08.020
Artigo de periodico
Inhibition of hypoxia-associated response and kynurenine production in response to hyperbaric oxygen as mechanisms involved in protection against experimental cerebral malaria (2018)
Bastos, Marcele F. ; Kayano, Ana Carolina A. V. ; Silva Filho, João Luiz ; Santos, João Conrado K. dos ; Judice, Carla ; Blanco, Yara C.; Shryock, Nathaniel; Sercundes, Michelle Klein ; Ortolan, Luana S. ; Francelin, Carolina ; Leite, Juliana A. ; Oliveira, Rafaella; Elias, Rosa Maria ; Câmara, Niels Olsen Saraiva ; Lopes, Stefanie C. P. ; Albrecht, Letusa ; Farias, Alessandro S. ; Vicente, Cristina Pontes; Werneck, Claudio C. ; Giorgio, Selma ; Verinaud, Liana ; Epiphanio, Sabrina ; Marinho, Claudio Romero Farias ; Flawani, Pritesh; Amino, Rogerio ; Aliberti, Júlio ; Costa, Fabio T. M.
Fonte: Faseb Journal. Unidades: ICB, FCF
Assuntos: ANÓXIA, MALÁRIA, PLASMODIUM BERGHEI, OXIGENAÇÃO HIPERBÁRICA, METABÓLITOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BASTOS, Marcele F. et al. Inhibition of hypoxia-associated response and kynurenine production in response to hyperbaric oxygen as mechanisms involved in protection against experimental cerebral malaria. Faseb Journal, v. 32, n. 8, p. 4470-4481, 2018Tradução . . Disponível em: https://doi.org/10.1096/fj.201700844R. Acesso em: 06 jul. 2024.
APA
Bastos, M. F., Kayano, A. C. A. V., Silva Filho, J. L., Santos, J. C. K. dos, Judice, C., Blanco, Y. C., et al. (2018). Inhibition of hypoxia-associated response and kynurenine production in response to hyperbaric oxygen as mechanisms involved in protection against experimental cerebral malaria. Faseb Journal, 32( 8), 4470-4481. doi:10.1096/fj.201700844R
NLM
Bastos MF, Kayano ACAV, Silva Filho JL, Santos JCK dos, Judice C, Blanco YC, Shryock N, Sercundes MK, Ortolan LS, Francelin C, Leite JA, Oliveira R, Elias RM, Câmara NOS, Lopes SCP, Albrecht L, Farias AS, Vicente CP, Werneck CC, Giorgio S, Verinaud L, Epiphanio S, Marinho CRF, Flawani P, Amino R, Aliberti J, Costa FTM. Inhibition of hypoxia-associated response and kynurenine production in response to hyperbaric oxygen as mechanisms involved in protection against experimental cerebral malaria [Internet]. Faseb Journal. 2018 ; 32( 8): 4470-4481.[citado 2024 jul. 06 ] Available from: https://doi.org/10.1096/fj.201700844R
Vancouver
Bastos MF, Kayano ACAV, Silva Filho JL, Santos JCK dos, Judice C, Blanco YC, Shryock N, Sercundes MK, Ortolan LS, Francelin C, Leite JA, Oliveira R, Elias RM, Câmara NOS, Lopes SCP, Albrecht L, Farias AS, Vicente CP, Werneck CC, Giorgio S, Verinaud L, Epiphanio S, Marinho CRF, Flawani P, Amino R, Aliberti J, Costa FTM. Inhibition of hypoxia-associated response and kynurenine production in response to hyperbaric oxygen as mechanisms involved in protection against experimental cerebral malaria [Internet]. Faseb Journal. 2018 ; 32( 8): 4470-4481.[citado 2024 jul. 06 ] Available from: https://doi.org/10.1096/fj.201700844R
Artigo de periodico
Tuberculosis and impaired IL-23–dependent IFN-γ immunity in humans homozygous for a common TYK2 missense variant (2018)
Boisson-Dupuis, Stéphanie; Ramires-Alejo, Noe; Li, Zhi; Patin, Etienne; Rao, Geetha; Kerner, Gaspard; Lim, Che Kang; Krementsov, Dimitry N.; Hernandez, Nicholas; Ma, Cidy S.; Zhang, Qian; Markle, Janet; Martinez-Barricarte, Ruben; Payne, Kathryn; Fisch, Robert; Deswarte, Caroline; Halpern, Joshua; Bouaziz, Matthieu; Mulwa, Jeanette; Sivanesan, Durga; Lazarov, Tomi; Naves, Rodrigo; Garcia, Patricia; Itan, Yuval; Boisson, Bertrand; Checci, Alix; Jabot-Hanin, Fabienne; Cobat, Aurélie; Guennoun, Andrea; Jackson, Carolyn C.; Pekcan, Sevai; Caliskaner, Zafer; Inostroza, Jaime; Costa-Carvalho, Beatriz Tavares; Albuquerque, José Antonio Tavares de; Gacia-Ortiz, Humberto; Orozco, Lorena; Ozcelik, Tayfun; Abid, Ahmed; Rhorfi, Ismail Abderahmani; Souhi, Hicham; Amrani, Hicham Naji; Zegmout, Adil; Geissmann, Frédéric; Michnick, Stephen W.; Muller-Fleckenstein, Ingrid; Fleckenstein, Bernhard; Puel, Anne; Ciancanelli, Michael J.; Marr, Nico; Abolhassani, Hassan; Balcells, Maria Elvira; Strickler, Alexis; Abarca, Katia; Teuscher, Cory; Ochs, Hans D.; Reisli, Ismail; Sayar, Esra H.; El-Baghdadi, Jamila; Bustamante, Jacinta; Hammarströn, Lennart; Tangye, Stuart G.; Pellegrini, Sandra; Quintana-Murci, Lluis; Abel, Laurent; Casanova, Jean-Laurent; Condino-Neto, Antonio
Fonte: Science Immunology. Unidade: ICB
Assuntos: TUBERCULOSE, VARIAÇÃO GENÉTICA, IMUNIDADE, CITOCINAS, DIFERENCIAÇÃO CELULAR, ATIVAÇÃO ENZIMÁTICA, IMUNOGENÉTICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BOISSON-DUPUIS, Stéphanie et al. Tuberculosis and impaired IL-23–dependent IFN-γ immunity in humans homozygous for a common TYK2 missense variant. Science Immunology, v. 3, 2018Tradução . . Disponível em: https://doi.org/10.1126/sciimmunol.aau8714. Acesso em: 06 jul. 2024.
APA
Boisson-Dupuis, S., Ramires-Alejo, N., Li, Z., Patin, E., Rao, G., Kerner, G., et al. (2018). Tuberculosis and impaired IL-23–dependent IFN-γ immunity in humans homozygous for a common TYK2 missense variant. Science Immunology, 3. doi:10.1126/sciimmunol.aau8714
NLM
Boisson-Dupuis S, Ramires-Alejo N, Li Z, Patin E, Rao G, Kerner G, Lim CK, Krementsov DN, Hernandez N, Ma CS, Zhang Q, Markle J, Martinez-Barricarte R, Payne K, Fisch R, Deswarte C, Halpern J, Bouaziz M, Mulwa J, Sivanesan D, Lazarov T, Naves R, Garcia P, Itan Y, Boisson B, Checci A, Jabot-Hanin F, Cobat A, Guennoun A, Jackson CC, Pekcan S, Caliskaner Z, Inostroza J, Costa-Carvalho BT, Albuquerque JAT de, Gacia-Ortiz H, Orozco L, Ozcelik T, Abid A, Rhorfi IA, Souhi H, Amrani HN, Zegmout A, Geissmann F, Michnick SW, Muller-Fleckenstein I, Fleckenstein B, Puel A, Ciancanelli MJ, Marr N, Abolhassani H, Balcells ME, Strickler A, Abarca K, Teuscher C, Ochs HD, Reisli I, Sayar EH, El-Baghdadi J, Bustamante J, Hammarströn L, Tangye SG, Pellegrini S, Quintana-Murci L, Abel L, Casanova J-L, Condino-Neto A. Tuberculosis and impaired IL-23–dependent IFN-γ immunity in humans homozygous for a common TYK2 missense variant [Internet]. Science Immunology. 2018 ; 3[citado 2024 jul. 06 ] Available from: https://doi.org/10.1126/sciimmunol.aau8714
Vancouver
Boisson-Dupuis S, Ramires-Alejo N, Li Z, Patin E, Rao G, Kerner G, Lim CK, Krementsov DN, Hernandez N, Ma CS, Zhang Q, Markle J, Martinez-Barricarte R, Payne K, Fisch R, Deswarte C, Halpern J, Bouaziz M, Mulwa J, Sivanesan D, Lazarov T, Naves R, Garcia P, Itan Y, Boisson B, Checci A, Jabot-Hanin F, Cobat A, Guennoun A, Jackson CC, Pekcan S, Caliskaner Z, Inostroza J, Costa-Carvalho BT, Albuquerque JAT de, Gacia-Ortiz H, Orozco L, Ozcelik T, Abid A, Rhorfi IA, Souhi H, Amrani HN, Zegmout A, Geissmann F, Michnick SW, Muller-Fleckenstein I, Fleckenstein B, Puel A, Ciancanelli MJ, Marr N, Abolhassani H, Balcells ME, Strickler A, Abarca K, Teuscher C, Ochs HD, Reisli I, Sayar EH, El-Baghdadi J, Bustamante J, Hammarströn L, Tangye SG, Pellegrini S, Quintana-Murci L, Abel L, Casanova J-L, Condino-Neto A. Tuberculosis and impaired IL-23–dependent IFN-γ immunity in humans homozygous for a common TYK2 missense variant [Internet]. Science Immunology. 2018 ; 3[citado 2024 jul. 06 ] Available from: https://doi.org/10.1126/sciimmunol.aau8714
Artigo de periodico
Recombinant BCG expressing LTAK63 adjuvant induces superior protection against Mycobacterium tuberculosis (2017)
Nascimento, Ivan Pereira; Rodriguez, Dúnia; Santos, Carina Carvalho dos; Amaral, Eduardo Pinheiro; Rofatto, Henrique Krambeck; Junqueira-Kipnis, Ana Paula; Gonçalves, Eduardo Dantas Casillo; Lima, Maria Regina D'Império ; Hirata, Mario Hiroyuki ; Silva, Célio Lopes; Winter, Nathalie; Gicquel, Brigitte; Mills, Kingston H G; Pizza, Mariagrazia; Rappuoli, Rino; Leite, Luciana C. C
Fonte: Scientific Reports. Unidades: ICB, FCF, FMRP
Assuntos: IMUNOLOGIA, TUBERCULOSE, MYCOBACTERIUM TUBERCULOSIS, ADJUVANTES IMUNOLÓGICOS, PROTEÍNAS RECOMBINANTES, VACINAS, CITOCININAS, CAMUNDONGOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NASCIMENTO, Ivan Pereira et al. Recombinant BCG expressing LTAK63 adjuvant induces superior protection against Mycobacterium tuberculosis. Scientific Reports, v. 7, n. 1, p. 1-11, 2017Tradução . . Disponível em: https://doi.org/10.1038/s41598-017-02003-9. Acesso em: 06 jul. 2024.
APA
Nascimento, I. P., Rodriguez, D., Santos, C. C. dos, Amaral, E. P., Rofatto, H. K., Junqueira-Kipnis, A. P., et al. (2017). Recombinant BCG expressing LTAK63 adjuvant induces superior protection against Mycobacterium tuberculosis. Scientific Reports, 7( 1), 1-11. doi:10.1038/s41598-017-02003-9
NLM
Nascimento IP, Rodriguez D, Santos CC dos, Amaral EP, Rofatto HK, Junqueira-Kipnis AP, Gonçalves EDC, Lima MRD'I, Hirata MH, Silva CL, Winter N, Gicquel B, Mills KHG, Pizza M, Rappuoli R, Leite LCC. Recombinant BCG expressing LTAK63 adjuvant induces superior protection against Mycobacterium tuberculosis [Internet]. Scientific Reports. 2017 ; 7( 1): 1-11.[citado 2024 jul. 06 ] Available from: https://doi.org/10.1038/s41598-017-02003-9
Vancouver
Nascimento IP, Rodriguez D, Santos CC dos, Amaral EP, Rofatto HK, Junqueira-Kipnis AP, Gonçalves EDC, Lima MRD'I, Hirata MH, Silva CL, Winter N, Gicquel B, Mills KHG, Pizza M, Rappuoli R, Leite LCC. Recombinant BCG expressing LTAK63 adjuvant induces superior protection against Mycobacterium tuberculosis [Internet]. Scientific Reports. 2017 ; 7( 1): 1-11.[citado 2024 jul. 06 ] Available from: https://doi.org/10.1038/s41598-017-02003-9

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