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Artigo de periodico
Anti-cancer activity of a new dihydropyridine derivative, VdiE-2N, in head and neck squamous cell carcinoma (2018)
Goto, Renata Nishida; Sobral, Lays Martin ; Sousa, Lucas Oliveira ; Garcia, Cristiana Bernadelli; Lopes, Norberto Peporine ; Marín-Prida, Javier; Ochoa-Rodríguez, Estael; Verdecia-Reyes, Yamila; Pardo-Andreu, Gilberto Lázaro; Curti, Carlos ; Leopoldino, Andréia Machado
Source: European Journal of Pharmacology. Unidade: FCFRP
Subjects: ANTINEOPLÁSICOS, APOPTOSE, NEOPLASIAS DE CABEÇA E PESCOÇO, MITOCÔNDRIAS, NEOVASCULARIZAÇÃO PATOLÓGICA
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ABNT
GOTO, Renata Nishida et al. Anti-cancer activity of a new dihydropyridine derivative, VdiE-2N, in head and neck squamous cell carcinoma. European Journal of Pharmacology, v. 819, p. 198-206, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2017.12.009. Acesso em: 18 nov. 2024.
APA
Goto, R. N., Sobral, L. M., Sousa, L. O., Garcia, C. B., Lopes, N. P., Marín-Prida, J., et al. (2018). Anti-cancer activity of a new dihydropyridine derivative, VdiE-2N, in head and neck squamous cell carcinoma. European Journal of Pharmacology, 819, 198-206. doi:10.1016/j.ejphar.2017.12.009
NLM
Goto RN, Sobral LM, Sousa LO, Garcia CB, Lopes NP, Marín-Prida J, Ochoa-Rodríguez E, Verdecia-Reyes Y, Pardo-Andreu GL, Curti C, Leopoldino AM. Anti-cancer activity of a new dihydropyridine derivative, VdiE-2N, in head and neck squamous cell carcinoma [Internet]. European Journal of Pharmacology. 2018 ; 819 198-206.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.ejphar.2017.12.009
Vancouver
Goto RN, Sobral LM, Sousa LO, Garcia CB, Lopes NP, Marín-Prida J, Ochoa-Rodríguez E, Verdecia-Reyes Y, Pardo-Andreu GL, Curti C, Leopoldino AM. Anti-cancer activity of a new dihydropyridine derivative, VdiE-2N, in head and neck squamous cell carcinoma [Internet]. European Journal of Pharmacology. 2018 ; 819 198-206.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.ejphar.2017.12.009
Artigo de periodico
QSPR/QSAR-based Perturbation Theory approach and mechanistic electrochemical assays on carbon nanotubes with optimal properties against mitochondrial Fenton reaction experimentally induced by Fe2+-overload (2017)
Durruthy, Michael González; Castro, Micheli; Nunes, Silvana Manske; Lima, Juliane Ventura; Alberici, Luciane Carla; Naal, Zeki; Sawazaki, David T. Atique; Curti, Carlos; Ruas, Caroline Pires; Gelesky, Marcos A.; Roy, Kunal; González-Díaz, Humberto; Monserrat, José M.
Source: Carbon. Unidade: FCFRP
Subjects: NANOTUBOS DE CARBONO, MITOCÔNDRIAS, CURVAS DE DOSE-RESPOSTA, TEORIA DE PERTURBAÇÃO PARA OPERADORES LINEARES
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DURRUTHY, Michael González et al. QSPR/QSAR-based Perturbation Theory approach and mechanistic electrochemical assays on carbon nanotubes with optimal properties against mitochondrial Fenton reaction experimentally induced by Fe2+-overload. Carbon, v. 115, p. 312-330, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.carbon.2017.01.002. Acesso em: 18 nov. 2024.
APA
Durruthy, M. G., Castro, M., Nunes, S. M., Lima, J. V., Alberici, L. C., Naal, Z., et al. (2017). QSPR/QSAR-based Perturbation Theory approach and mechanistic electrochemical assays on carbon nanotubes with optimal properties against mitochondrial Fenton reaction experimentally induced by Fe2+-overload. Carbon, 115, 312-330. doi:10.1016/j.carbon.2017.01.002
NLM
Durruthy MG, Castro M, Nunes SM, Lima JV, Alberici LC, Naal Z, Sawazaki DTA, Curti C, Ruas CP, Gelesky MA, Roy K, González-Díaz H, Monserrat JM. QSPR/QSAR-based Perturbation Theory approach and mechanistic electrochemical assays on carbon nanotubes with optimal properties against mitochondrial Fenton reaction experimentally induced by Fe2+-overload [Internet]. Carbon. 2017 ; 115 312-330.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.carbon.2017.01.002
Vancouver
Durruthy MG, Castro M, Nunes SM, Lima JV, Alberici LC, Naal Z, Sawazaki DTA, Curti C, Ruas CP, Gelesky MA, Roy K, González-Díaz H, Monserrat JM. QSPR/QSAR-based Perturbation Theory approach and mechanistic electrochemical assays on carbon nanotubes with optimal properties against mitochondrial Fenton reaction experimentally induced by Fe2+-overload [Internet]. Carbon. 2017 ; 115 312-330.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.carbon.2017.01.002
Artigo de periodico
Triacsin C reduces lipid droplet formation and induces mitochondrial biogenesis in primary rat hepatocytes (2017)
Dechandt, Carlos R. P.; Zuccolotto-dos-Reis, Felippe H.; Teodoro, Bruno G.; Fernandes, Anna Maria A. P.; Eberlin, Marcos N.; Kettelhut, Isis do Carmo; Curti, Carlos; Alberici, Luciane Carla
Source: Journal of Bioenergetics and Biomembranes. Unidades: FMRP, FCFRP
Subjects: CÉLULAS CULTIVADAS, MITOCÔNDRIAS, METABOLISMO DE GORDURA, FÍGADO GORDUROSO
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DECHANDT, Carlos R. P. et al. Triacsin C reduces lipid droplet formation and induces mitochondrial biogenesis in primary rat hepatocytes. Journal of Bioenergetics and Biomembranes, v. 49, n. 5, p. 399-411, 2017Tradução . . Disponível em: https://doi.org/10.1007/s10863-017-9725-9. Acesso em: 18 nov. 2024.
APA
Dechandt, C. R. P., Zuccolotto-dos-Reis, F. H., Teodoro, B. G., Fernandes, A. M. A. P., Eberlin, M. N., Kettelhut, I. do C., et al. (2017). Triacsin C reduces lipid droplet formation and induces mitochondrial biogenesis in primary rat hepatocytes. Journal of Bioenergetics and Biomembranes, 49( 5), 399-411. doi:10.1007/s10863-017-9725-9
NLM
Dechandt CRP, Zuccolotto-dos-Reis FH, Teodoro BG, Fernandes AMAP, Eberlin MN, Kettelhut I do C, Curti C, Alberici LC. Triacsin C reduces lipid droplet formation and induces mitochondrial biogenesis in primary rat hepatocytes [Internet]. Journal of Bioenergetics and Biomembranes. 2017 ; 49( 5): 399-411.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s10863-017-9725-9
Vancouver
Dechandt CRP, Zuccolotto-dos-Reis FH, Teodoro BG, Fernandes AMAP, Eberlin MN, Kettelhut I do C, Curti C, Alberici LC. Triacsin C reduces lipid droplet formation and induces mitochondrial biogenesis in primary rat hepatocytes [Internet]. Journal of Bioenergetics and Biomembranes. 2017 ; 49( 5): 399-411.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s10863-017-9725-9
Artigo de periodico
SET/I2PP2A overexpression induces phenotypic, molecular, and metabolic alterations in an oral keratinocyte cell line (2017)
Sobral, Lays M.; Coletta, Ricardo D.; Alberici, Luciane Carla; Curti, Carlos; Leopoldino, Andréia Machado
Source: The FEBS Journal. Unidade: FCFRP
Subjects: TOXICOLOGIA, CÉLULAS, METABOLISMO, MITOCÔNDRIAS, RESPIRAÇÃO
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ABNT
SOBRAL, Lays M. et al. SET/I2PP2A overexpression induces phenotypic, molecular, and metabolic alterations in an oral keratinocyte cell line. The FEBS Journal, v. 284, n. 17, p. 2774-2785, 2017Tradução . . Disponível em: https://doi.org/10.1111/febs.14148. Acesso em: 18 nov. 2024.
APA
Sobral, L. M., Coletta, R. D., Alberici, L. C., Curti, C., & Leopoldino, A. M. (2017). SET/I2PP2A overexpression induces phenotypic, molecular, and metabolic alterations in an oral keratinocyte cell line. The FEBS Journal, 284( 17), 2774-2785. doi:10.1111/febs.14148
NLM
Sobral LM, Coletta RD, Alberici LC, Curti C, Leopoldino AM. SET/I2PP2A overexpression induces phenotypic, molecular, and metabolic alterations in an oral keratinocyte cell line [Internet]. The FEBS Journal. 2017 ; 284( 17): 2774-2785.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1111/febs.14148
Vancouver
Sobral LM, Coletta RD, Alberici LC, Curti C, Leopoldino AM. SET/I2PP2A overexpression induces phenotypic, molecular, and metabolic alterations in an oral keratinocyte cell line [Internet]. The FEBS Journal. 2017 ; 284( 17): 2774-2785.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1111/febs.14148
Artigo de periodico
Experimental–computational study of carbon nanotube effects on mitochondrial respiration: in silico nano-QSPR machine learning models based on new raman spectra transform with markov–shannon entropy invariants (2017)
González-Durruthy, Michael; Alberici, Luciane C.; Curti, Carlos; Naal, Zeki; Sawazaki, David Tatsuo A; Vázquez-Naya, José M.; González-Díaz, Humberto; Munteanu, Cristian R.
Source: Journal of Chemical Information and Modeling. Unidade: FCFRP
Subjects: ENTROPIA, REGRESSÃO LINEAR, MITOCÔNDRIAS, NANOTUBOS DE CARBONO, BIOMEDICINA, OXIGÊNIO
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ABNT
GONZÁLEZ-DURRUTHY, Michael et al. Experimental–computational study of carbon nanotube effects on mitochondrial respiration: in silico nano-QSPR machine learning models based on new raman spectra transform with markov–shannon entropy invariants. Journal of Chemical Information and Modeling, v. 57, n. 5, p. 1029-1044, 2017Tradução . . Disponível em: https://doi.org/10.1021/acs.jcim.6b00458. Acesso em: 18 nov. 2024.
APA
González-Durruthy, M., Alberici, L. C., Curti, C., Naal, Z., Sawazaki, D. T. A., Vázquez-Naya, J. M., et al. (2017). Experimental–computational study of carbon nanotube effects on mitochondrial respiration: in silico nano-QSPR machine learning models based on new raman spectra transform with markov–shannon entropy invariants. Journal of Chemical Information and Modeling, 57( 5), 1029-1044. doi:10.1021/acs.jcim.6b00458
NLM
González-Durruthy M, Alberici LC, Curti C, Naal Z, Sawazaki DTA, Vázquez-Naya JM, González-Díaz H, Munteanu CR. Experimental–computational study of carbon nanotube effects on mitochondrial respiration: in silico nano-QSPR machine learning models based on new raman spectra transform with markov–shannon entropy invariants [Internet]. Journal of Chemical Information and Modeling. 2017 ; 57( 5): 1029-1044.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1021/acs.jcim.6b00458
Vancouver
González-Durruthy M, Alberici LC, Curti C, Naal Z, Sawazaki DTA, Vázquez-Naya JM, González-Díaz H, Munteanu CR. Experimental–computational study of carbon nanotube effects on mitochondrial respiration: in silico nano-QSPR machine learning models based on new raman spectra transform with markov–shannon entropy invariants [Internet]. Journal of Chemical Information and Modeling. 2017 ; 57( 5): 1029-1044.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1021/acs.jcim.6b00458
Artigo de periodico
CR-LAAO antileukemic effect against Bcr-Abl+ cells is mediated by apoptosis and hydrogen peroxide (2016)
Burin, Sandra Mara; Ghisla, Sandro; Ouchida, Amanda Tomie; Aissa, Alexandre Ferro; Coelho, Maria Gabriela Berzoti; Costa, Tássia Rafaella; Marsola, Ana Paula Zambuzi Cardoso; Simões, Belinda Pinto; Antunes, Lusânia Maria Greggi; Curti, Carlos; Sampaio, Suely Vilela; Castro, Fabíola Attié de
Source: International Journal of Biological Macromolecules. Unidades: FMRP, FCFRP
Subjects: APOPTOSE, LEUCEMIA MIELOIDE, NEOPLASIAS, VENENOS DE ORIGEM ANIMAL, COBRAS, VIPERIDAE, CITOTOXICIDADE IMUNOLÓGICA
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BURIN, Sandra Mara et al. CR-LAAO antileukemic effect against Bcr-Abl+ cells is mediated by apoptosis and hydrogen peroxide. International Journal of Biological Macromolecules, v. 86, p. 309-320, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.ijbiomac.2016.01.069. Acesso em: 18 nov. 2024.
APA
Burin, S. M., Ghisla, S., Ouchida, A. T., Aissa, A. F., Coelho, M. G. B., Costa, T. R., et al. (2016). CR-LAAO antileukemic effect against Bcr-Abl+ cells is mediated by apoptosis and hydrogen peroxide. International Journal of Biological Macromolecules, 86, 309-320. doi:10.1016/j.ijbiomac.2016.01.069
NLM
Burin SM, Ghisla S, Ouchida AT, Aissa AF, Coelho MGB, Costa TR, Marsola APZC, Simões BP, Antunes LMG, Curti C, Sampaio SV, Castro FA de. CR-LAAO antileukemic effect against Bcr-Abl+ cells is mediated by apoptosis and hydrogen peroxide [Internet]. International Journal of Biological Macromolecules. 2016 ; 86 309-320.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.ijbiomac.2016.01.069
Vancouver
Burin SM, Ghisla S, Ouchida AT, Aissa AF, Coelho MGB, Costa TR, Marsola APZC, Simões BP, Antunes LMG, Curti C, Sampaio SV, Castro FA de. CR-LAAO antileukemic effect against Bcr-Abl+ cells is mediated by apoptosis and hydrogen peroxide [Internet]. International Journal of Biological Macromolecules. 2016 ; 86 309-320.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.ijbiomac.2016.01.069
Artigo de periodico
SET overexpression in HEK293 cells regulates mitochondrial uncoupling proteins levels within a mitochondrial fission/reduced autophagic flux scenario (2015)
Almeida, Luciana O; Goto, Renata N; Cavalcanti Neto, Marinaldo P.; Sousa, Lucas Oliveira; Curti, Carlos; Leopoldino, Andréia Machado
Source: Biochemical and Biophysical Research Communications. Unidade: FCFRP
Subjects: MITOCÔNDRIAS, PROTEÍNAS, NEOPLASIAS, DOENÇA DE ALZHEIMER
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ABNT
ALMEIDA, Luciana O et al. SET overexpression in HEK293 cells regulates mitochondrial uncoupling proteins levels within a mitochondrial fission/reduced autophagic flux scenario. Biochemical and Biophysical Research Communications, v. 458, n. 2, p. 300-306, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.bbrc.2015.01.104. Acesso em: 18 nov. 2024.
APA
Almeida, L. O., Goto, R. N., Cavalcanti Neto, M. P., Sousa, L. O., Curti, C., & Leopoldino, A. M. (2015). SET overexpression in HEK293 cells regulates mitochondrial uncoupling proteins levels within a mitochondrial fission/reduced autophagic flux scenario. Biochemical and Biophysical Research Communications, 458( 2), 300-306. doi:10.1016/j.bbrc.2015.01.104
NLM
Almeida LO, Goto RN, Cavalcanti Neto MP, Sousa LO, Curti C, Leopoldino AM. SET overexpression in HEK293 cells regulates mitochondrial uncoupling proteins levels within a mitochondrial fission/reduced autophagic flux scenario [Internet]. Biochemical and Biophysical Research Communications. 2015 ; 458( 2): 300-306.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.bbrc.2015.01.104
Vancouver
Almeida LO, Goto RN, Cavalcanti Neto MP, Sousa LO, Curti C, Leopoldino AM. SET overexpression in HEK293 cells regulates mitochondrial uncoupling proteins levels within a mitochondrial fission/reduced autophagic flux scenario [Internet]. Biochemical and Biophysical Research Communications. 2015 ; 458( 2): 300-306.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.bbrc.2015.01.104
Artigo de periodico
The cytotoxic effects of brown Cuban propolis depend on the nemorosone content and may be mediated by mitochondrial uncoupling (2015)
Andreu, Gilberto L. Pardo; Reis, Felippe H. Z; Dalalio, Felipe M; Figueredo, Yanier Nuñez; Rubio, Osmany Cuesta; Uyemura, Sérgio Akira; Curti, Carlos; Alberici, Luciane Carla
Source: Chemico-Biological Interactions. Unidade: FCFRP
Subjects: PRÓPOLIS, MITOCÔNDRIAS, TESTES DE TOXICIDADE, TOXICOLOGIA
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ABNT
ANDREU, Gilberto L. Pardo et al. The cytotoxic effects of brown Cuban propolis depend on the nemorosone content and may be mediated by mitochondrial uncoupling. Chemico-Biological Interactions, v. 228, p. 28-34, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.cbi.2015.01.010. Acesso em: 18 nov. 2024.
APA
Andreu, G. L. P., Reis, F. H. Z., Dalalio, F. M., Figueredo, Y. N., Rubio, O. C., Uyemura, S. A., et al. (2015). The cytotoxic effects of brown Cuban propolis depend on the nemorosone content and may be mediated by mitochondrial uncoupling. Chemico-Biological Interactions, 228, 28-34. doi:10.1016/j.cbi.2015.01.010
NLM
Andreu GLP, Reis FHZ, Dalalio FM, Figueredo YN, Rubio OC, Uyemura SA, Curti C, Alberici LC. The cytotoxic effects of brown Cuban propolis depend on the nemorosone content and may be mediated by mitochondrial uncoupling [Internet]. Chemico-Biological Interactions. 2015 ; 228 28-34.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.cbi.2015.01.010
Vancouver
Andreu GLP, Reis FHZ, Dalalio FM, Figueredo YN, Rubio OC, Uyemura SA, Curti C, Alberici LC. The cytotoxic effects of brown Cuban propolis depend on the nemorosone content and may be mediated by mitochondrial uncoupling [Internet]. Chemico-Biological Interactions. 2015 ; 228 28-34.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.cbi.2015.01.010
Artigo de periodico
Release of NO from a nitrosyl ruthenium complex through oxidation of mitochondrial NADH and effects on mitochondria (2012)
Rodrigues, Fernando P.; Pestana, Cezar R.; Polizello, Ana Cristina Morseli; Pardo-Andreu, Gilberto L.; Uyemura, Sérgio Akira; Santos, Antônio Cardozo dos; Alberici, Luciane Carla; Silva, Roberto Santana da; Curti, Carlos
Source: Nitric Oxide. Unidade: FCFRP
Subjects: MITOCÔNDRIAS, RUTÊNIO, ÓXIDO NÍTRICO
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ABNT
RODRIGUES, Fernando P. et al. Release of NO from a nitrosyl ruthenium complex through oxidation of mitochondrial NADH and effects on mitochondria. Nitric Oxide, v. 26, n. 3, p. 174-181, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.niox.2012.02.001. Acesso em: 18 nov. 2024.
APA
Rodrigues, F. P., Pestana, C. R., Polizello, A. C. M., Pardo-Andreu, G. L., Uyemura, S. A., Santos, A. C. dos, et al. (2012). Release of NO from a nitrosyl ruthenium complex through oxidation of mitochondrial NADH and effects on mitochondria. Nitric Oxide, 26( 3), 174-181. doi:10.1016/j.niox.2012.02.001
NLM
Rodrigues FP, Pestana CR, Polizello ACM, Pardo-Andreu GL, Uyemura SA, Santos AC dos, Alberici LC, Silva RS da, Curti C. Release of NO from a nitrosyl ruthenium complex through oxidation of mitochondrial NADH and effects on mitochondria [Internet]. Nitric Oxide. 2012 ; 26( 3): 174-181.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.niox.2012.02.001
Vancouver
Rodrigues FP, Pestana CR, Polizello ACM, Pardo-Andreu GL, Uyemura SA, Santos AC dos, Alberici LC, Silva RS da, Curti C. Release of NO from a nitrosyl ruthenium complex through oxidation of mitochondrial NADH and effects on mitochondria [Internet]. Nitric Oxide. 2012 ; 26( 3): 174-181.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.niox.2012.02.001
Artigo de periodico
The anti-cancer agent guttiferone-A permeabilizes mitochondrial membrane: ensuing energetic and oxidative stress implications (2011)
Pardo-Andreu, Gilberto L.; Nuñez-Figueredo, Yanier; Tudelli, Valeria G.; Cuesta-Rubio, Osmany; Rodrigues, Fernando P.; Pestana, Cezar R.; Uyemura, Sérgio Akira; Leopoldino, Andréia Machado; Alberici, Luciane Carla; Curti, Carlos
Source: Toxicology and Applied Pharmacology. Unidade: FCFRP
Subjects: MITOCÔNDRIAS, ESTRESSE OXIDATIVO, TOXICOLOGIA
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ABNT
PARDO-ANDREU, Gilberto L. et al. The anti-cancer agent guttiferone-A permeabilizes mitochondrial membrane: ensuing energetic and oxidative stress implications. Toxicology and Applied Pharmacology, v. 253, n. 3, p. 282-289, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.taap.2011.04.011. Acesso em: 18 nov. 2024.
APA
Pardo-Andreu, G. L., Nuñez-Figueredo, Y., Tudelli, V. G., Cuesta-Rubio, O., Rodrigues, F. P., Pestana, C. R., et al. (2011). The anti-cancer agent guttiferone-A permeabilizes mitochondrial membrane: ensuing energetic and oxidative stress implications. Toxicology and Applied Pharmacology, 253( 3), 282-289. doi:10.1016/j.taap.2011.04.011
NLM
Pardo-Andreu GL, Nuñez-Figueredo Y, Tudelli VG, Cuesta-Rubio O, Rodrigues FP, Pestana CR, Uyemura SA, Leopoldino AM, Alberici LC, Curti C. The anti-cancer agent guttiferone-A permeabilizes mitochondrial membrane: ensuing energetic and oxidative stress implications [Internet]. Toxicology and Applied Pharmacology. 2011 ; 253( 3): 282-289.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.taap.2011.04.011
Vancouver
Pardo-Andreu GL, Nuñez-Figueredo Y, Tudelli VG, Cuesta-Rubio O, Rodrigues FP, Pestana CR, Uyemura SA, Leopoldino AM, Alberici LC, Curti C. The anti-cancer agent guttiferone-A permeabilizes mitochondrial membrane: ensuing energetic and oxidative stress implications [Internet]. Toxicology and Applied Pharmacology. 2011 ; 253( 3): 282-289.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.taap.2011.04.011
Artigo de periodico
A strong protective action of guttiferone-A, a naturally occurring prenylated benzophenone, against iron-induced neuronal cell damage (2011)
Figueredo, Yanier Núñez; García-Pupo, Laura; Rubio, Osmany Cuesta; Hernández, René Delgado; Naal, Zeki; Curti, Carlos; Andreu, Gilberto L. Pardo
Source: Journal of Pharmacological Sciences. Unidade: FCFRP
Subjects: ANTIOXIDANTES, FARMACOLOGIA, ESTRESSE OXIDATIVO
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ABNT
FIGUEREDO, Yanier Núñez et al. A strong protective action of guttiferone-A, a naturally occurring prenylated benzophenone, against iron-induced neuronal cell damage. Journal of Pharmacological Sciences, v. 116, n. 1, p. 36-46, 2011Tradução . . Acesso em: 18 nov. 2024.
APA
Figueredo, Y. N., García-Pupo, L., Rubio, O. C., Hernández, R. D., Naal, Z., Curti, C., & Andreu, G. L. P. (2011). A strong protective action of guttiferone-A, a naturally occurring prenylated benzophenone, against iron-induced neuronal cell damage. Journal of Pharmacological Sciences, 116( 1), 36-46.
NLM
Figueredo YN, García-Pupo L, Rubio OC, Hernández RD, Naal Z, Curti C, Andreu GLP. A strong protective action of guttiferone-A, a naturally occurring prenylated benzophenone, against iron-induced neuronal cell damage. Journal of Pharmacological Sciences. 2011 ; 116( 1): 36-46.[citado 2024 nov. 18 ]
Vancouver
Figueredo YN, García-Pupo L, Rubio OC, Hernández RD, Naal Z, Curti C, Andreu GLP. A strong protective action of guttiferone-A, a naturally occurring prenylated benzophenone, against iron-induced neuronal cell damage. Journal of Pharmacological Sciences. 2011 ; 116( 1): 36-46.[citado 2024 nov. 18 ]

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