Enhanced Rho GTPase pathway activity promotes acquisition of glioblastoma chemoresistance (2025)
- Authors:
- USP affiliated authors: FORTI, FÁBIO LUÍS - IQ ; MAGALHÃES, YULI THAMIRES - IQ ; BOELL, VIKTOR KALBERMATTER - IQ
- Unidade: IQ
- DOI: 10.1158/1535-7163.MCT-25-0039
- Subjects: RADIOTERAPIA; QUIMIOTERAPIA; POLIMERIZAÇÃO; CITOESQUELETO
- Language: Inglês
- Imprenta:
- Publisher place: Philadelphia
- Date published: 2025
- Source:
- Título: Molecular Cancer Therapeutics
- ISSN: 1535-7163
- Volume/Número/Paginação/Ano: In Press, 2025
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
MAGALHÃES, Yuli Thamires e BOELL, Viktor Kalbermatter e FORTI, Fabio Luis. Enhanced Rho GTPase pathway activity promotes acquisition of glioblastoma chemoresistance. Molecular Cancer Therapeutics, 2025Tradução . . Disponível em: https://dx.doi.org/10.1158/1535-7163.MCT-25-0039. Acesso em: 24 jan. 2026. -
APA
Magalhães, Y. T., Boell, V. K., & Forti, F. L. (2025). Enhanced Rho GTPase pathway activity promotes acquisition of glioblastoma chemoresistance. Molecular Cancer Therapeutics. doi:10.1158/1535-7163.MCT-25-0039 -
NLM
Magalhães YT, Boell VK, Forti FL. Enhanced Rho GTPase pathway activity promotes acquisition of glioblastoma chemoresistance [Internet]. Molecular Cancer Therapeutics. 2025 ;[citado 2026 jan. 24 ] Available from: https://dx.doi.org/10.1158/1535-7163.MCT-25-0039 -
Vancouver
Magalhães YT, Boell VK, Forti FL. Enhanced Rho GTPase pathway activity promotes acquisition of glioblastoma chemoresistance [Internet]. Molecular Cancer Therapeutics. 2025 ;[citado 2026 jan. 24 ] Available from: https://dx.doi.org/10.1158/1535-7163.MCT-25-0039 - Actin cytoskeleton disruption suppresses glioma stem cell-like phenotype and tumorigenicity in therapy-resistant glioblastoma
- The rho gtpase pathway: a potential sensitizer of resistant glioblastoma
- Downregulation of the Rho GTPase pathway abrogates resistance to ionizing radiation in wild-type p53 glioblastoma by suppressing DNA repair mechanisms
- DUSP12‐NAT10 interaction regulates DNA damage response and nucleolar stress in hepatocellular carcinoma
- The role of DUSP12 and cellular architecture in the response of hepatic cell lines to genotoxic agents
- The Rho GTPase pathway as a potential chemosensitizer of TMZ and cisplatin-resistant GBM through a p53-dependent mechanism
- Glioblastoma: descoberto mecanismo capaz de reverter resistência do tumor cerebral à radioterapia
- Rho pathway: a fragile point in the GBM resistance to radiotherapy
- ROCK inhibition reduces the sensitivity of mutant p53 glioblastoma to genotoxic stress through a Rac1-driven ROS production
- Knockdown of dual-specificity phosphatase 3 drives differentiation and polarization of myeloid leukemia cells into macrophages with reduced proliferative and DNA repair fitness
Informações sobre o DOI: 10.1158/1535-7163.MCT-25-0039 (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
