Actin cytoskeleton disruption suppresses glioma stem cell-like phenotype and tumorigenicity in therapy-resistant glioblastoma (2025)
- Authors:
- USP affiliated authors: FORTI, FÁBIO LUÍS - IQ ; MAGALHÃES, YULI THAMIRES - IQ ; BOELL, VIKTOR KALBERMATTER - IQ
- Unidade: IQ
- Subjects: CITOESQUELETO; CÉLULAS-TRONCO
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Publisher: Sociedade Brasileira de Bioquímica e Biologia Celular
- Publisher place: São Paulo
- Date published: 2025
- Source:
- Título: Abstracts
- Conference titles: Annual Meeting of the Brazilian Society for Biochemistry and Molecular Biology/SBBQ
-
ABNT
MAGALHÃES, Yuli Thamires e BOELL, Viktor Kalbermatter e FORTI, Fabio Luis. Actin cytoskeleton disruption suppresses glioma stem cell-like phenotype and tumorigenicity in therapy-resistant glioblastoma. 2025, Anais.. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Celular, 2025. Disponível em: https://www2.sbbq.org.br/reuniao/2025/images/resumos.pdf. Acesso em: 28 dez. 2025. -
APA
Magalhães, Y. T., Boell, V. K., & Forti, F. L. (2025). Actin cytoskeleton disruption suppresses glioma stem cell-like phenotype and tumorigenicity in therapy-resistant glioblastoma. In Abstracts. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Celular. Recuperado de https://www2.sbbq.org.br/reuniao/2025/images/resumos.pdf -
NLM
Magalhães YT, Boell VK, Forti FL. Actin cytoskeleton disruption suppresses glioma stem cell-like phenotype and tumorigenicity in therapy-resistant glioblastoma [Internet]. Abstracts. 2025 ;[citado 2025 dez. 28 ] Available from: https://www2.sbbq.org.br/reuniao/2025/images/resumos.pdf -
Vancouver
Magalhães YT, Boell VK, Forti FL. Actin cytoskeleton disruption suppresses glioma stem cell-like phenotype and tumorigenicity in therapy-resistant glioblastoma [Internet]. Abstracts. 2025 ;[citado 2025 dez. 28 ] Available from: https://www2.sbbq.org.br/reuniao/2025/images/resumos.pdf - Enhanced Rho GTPase pathway activity promotes acquisition of glioblastoma chemoresistance
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- ROCK inhibition reduces the sensitivity of mutant p53 glioblastoma to genotoxic stress through a Rac1-driven ROS production
- Knockdown of dual-specificity phosphatase 3 drives differentiation and polarization of myeloid leukemia cells into macrophages with reduced proliferative and DNA repair fitness
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