ROCK inhibition reduces the sensitivity of mutant p53 glioblastoma to genotoxic stress through a Rac1-driven ROS production (2023)
- Authors:
- USP affiliated authors: FORTI, FÁBIO LUÍS - IQ ; MAGALHÃES, YULI THAMIRES - IQ
- Unidade: IQ
- DOI: 10.1016/j.biocel.2023.106474
- Subjects: ACTINAS; RADIAÇÃO IONIZANTE; REPARAÇÃO DE DNA
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: International Journal of Biochemistry and Cell Biology
- ISSN: 1357-2725
- Volume/Número/Paginação/Ano: v. 164, p. 1-11, art. 106474, 2023
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
MAGALHÃES, Yuli Thamires e FORTI, Fabio Luis. ROCK inhibition reduces the sensitivity of mutant p53 glioblastoma to genotoxic stress through a Rac1-driven ROS production. International Journal of Biochemistry and Cell Biology, v. 164, p. 1-11, 2023Tradução . . Disponível em: https://dx.doi.org/10.1016/j.biocel.2023.106474. Acesso em: 27 dez. 2025. -
APA
Magalhães, Y. T., & Forti, F. L. (2023). ROCK inhibition reduces the sensitivity of mutant p53 glioblastoma to genotoxic stress through a Rac1-driven ROS production. International Journal of Biochemistry and Cell Biology, 164, 1-11. doi:10.1016/j.biocel.2023.106474 -
NLM
Magalhães YT, Forti FL. ROCK inhibition reduces the sensitivity of mutant p53 glioblastoma to genotoxic stress through a Rac1-driven ROS production [Internet]. International Journal of Biochemistry and Cell Biology. 2023 ; 164 1-11.[citado 2025 dez. 27 ] Available from: https://dx.doi.org/10.1016/j.biocel.2023.106474 -
Vancouver
Magalhães YT, Forti FL. ROCK inhibition reduces the sensitivity of mutant p53 glioblastoma to genotoxic stress through a Rac1-driven ROS production [Internet]. International Journal of Biochemistry and Cell Biology. 2023 ; 164 1-11.[citado 2025 dez. 27 ] Available from: https://dx.doi.org/10.1016/j.biocel.2023.106474 - The Rho GTPase pathway as a potential chemosensitizer of TMZ and cisplatin-resistant GBM through a p53-dependent mechanism
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- Investigação do fenótipo de resistência múltipla a quimioterápicos em linhagens de glioblastoma humano
- Enhanced Rho GTPase pathway activity promotes acquisition of glioblastoma chemoresistance
- Actin cytoskeleton disruption suppresses glioma stem cell-like phenotype and tumorigenicity in therapy-resistant glioblastoma
- The rho gtpase pathway: a potential sensitizer of resistant glioblastoma
- Downregulation of the Rho GTPase pathway abrogates resistance to ionizing radiation in wild-type p53 glioblastoma by suppressing DNA repair mechanisms
- GTPases, genome, actin: a hidden story in DNA damage response and repair mechanisms
Informações sobre o DOI: 10.1016/j.biocel.2023.106474 (Fonte: oaDOI API)
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