Dual-specificity phosphatases as new biomarkers for gliomas (2025)
- Authors:
- USP affiliated authors: FORTI, FÁBIO LUÍS - IQ ; FORTI, SOFIA FIGUEIREDO - PRÓ-G ; BOELL, VIKTOR KALBERMATTER - IQ
- Unidades: IQ; PRÓ-G
- Subjects: BIOMARCADORES; GLIOMA
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Publisher: Sociedade Brasileira de Bioquímica e Biologia Celular
- Publisher place: São Paulo
- Date published: 2025
- Source:
- Título: Abstracts
- Conference titles: Annual Meeting of the Brazilian Society for Biochemistry and Molecular Biology/SBBQ
-
ABNT
FORTI, Sofia Figueiredo e BOELL, Viktor Kalbermatter e FORTI, Fabio Luis. Dual-specificity phosphatases as new biomarkers for gliomas. 2025, Anais.. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Celular, 2025. Disponível em: https://www2.sbbq.org.br/reuniao/2025/images/resumos.pdf. Acesso em: 10 fev. 2026. -
APA
Forti, S. F., Boell, V. K., & Forti, F. L. (2025). Dual-specificity phosphatases as new biomarkers for gliomas. In Abstracts. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Celular. Recuperado de https://www2.sbbq.org.br/reuniao/2025/images/resumos.pdf -
NLM
Forti SF, Boell VK, Forti FL. Dual-specificity phosphatases as new biomarkers for gliomas [Internet]. Abstracts. 2025 ;[citado 2026 fev. 10 ] Available from: https://www2.sbbq.org.br/reuniao/2025/images/resumos.pdf -
Vancouver
Forti SF, Boell VK, Forti FL. Dual-specificity phosphatases as new biomarkers for gliomas [Internet]. Abstracts. 2025 ;[citado 2026 fev. 10 ] Available from: https://www2.sbbq.org.br/reuniao/2025/images/resumos.pdf - Fosfatases de especificidade dual: possíveis novos biomarcadores para gliomas
- The role of DUSP12 and cellular architecture in the response of hepatic cell lines to genotoxic agents
- Intrinsically disordered regions in atypical dual-specificity phosphatases: a review
- Enhanced Rho GTPase pathway activity promotes acquisition of glioblastoma chemoresistance
- DUSP3 modulates IRES-dependent translation of mRNAs through dephosphorylation of the HNRNPC protein in cells under genotoxic stimulus
- Downregulation of the Rho GTPase pathway abrogates resistance to ionizing radiation in wild-type p53 glioblastoma by suppressing DNA repair mechanisms
- Actin cytoskeleton disruption suppresses glioma stem cell-like phenotype and tumorigenicity in therapy-resistant glioblastoma
- The rho gtpase pathway: a potential sensitizer of resistant glioblastoma
- DUSP12‐NAT10 interaction regulates DNA damage response and nucleolar stress in hepatocellular carcinoma
- Knockdown of dual-specificity phosphatase 3 drives differentiation and polarization of myeloid leukemia cells into macrophages with reduced proliferative and DNA repair fitness
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