PI3K signaling in mechanisms and treatments of pulmonary fibrosis following sepsis and acute lung injury (2022)
- Authors:
- Autor USP: ALVES FILHO, JOSÉ CARLOS FARIAS - FMRP
- Unidade: FMRP
- DOI: 10.3390/biomedicines10040756
- Subjects: FIBROSE PULMONAR; SEPSE; CICLO CELULAR; BIOMEDICINA; COLÁGENO; CITOCINAS; ENZIMAS; COVID-19
- Keywords: Pulmonary fibrosis; PI3K; Sepsis; ARDS; Inhibitor
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Biomedicines
- ISSN: 2227-9059
- Volume/Número/Paginação/Ano: v. 10, n. 4, art. 756, p. 1-10, 2022
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
MARGARIA, Jean Piero et al. PI3K signaling in mechanisms and treatments of pulmonary fibrosis following sepsis and acute lung injury. Biomedicines, v. 10, n. 4, p. 1-10, 2022Tradução . . Disponível em: https://doi.org/10.3390/biomedicines10040756. Acesso em: 28 fev. 2026. -
APA
Margaria, J. P., Moretta, L., Alves Filho, J. C. F., & Hirsch, E. (2022). PI3K signaling in mechanisms and treatments of pulmonary fibrosis following sepsis and acute lung injury. Biomedicines, 10( 4), 1-10. doi:10.3390/biomedicines10040756 -
NLM
Margaria JP, Moretta L, Alves Filho JCF, Hirsch E. PI3K signaling in mechanisms and treatments of pulmonary fibrosis following sepsis and acute lung injury [Internet]. Biomedicines. 2022 ; 10( 4): 1-10.[citado 2026 fev. 28 ] Available from: https://doi.org/10.3390/biomedicines10040756 -
Vancouver
Margaria JP, Moretta L, Alves Filho JCF, Hirsch E. PI3K signaling in mechanisms and treatments of pulmonary fibrosis following sepsis and acute lung injury [Internet]. Biomedicines. 2022 ; 10( 4): 1-10.[citado 2026 fev. 28 ] Available from: https://doi.org/10.3390/biomedicines10040756 - Therapeutic effects of treatment with anti-TLR2 and anti-TLR4 monoclonal antibodies in polymicrobial sepsis
- Nitric oxide-induced regulatory T cells inhibit Th 17 but not Th1 cell differentiation and function
- Papel dos receptores Toll-like na gênese da resposta inflamatória local e sistêmica na sepse experimental
- Pyruvate kinase M2: a potential target for regulating inflammation
- Inhalation of the prodrug PI3K inhibitor CL27c improves lung function in asthma and fibrosis
- IL-33 induces skin inflammation with mast cell and neutrophil activation
- ILC2s and basophils team up to orchestrate IL-33–induced atopic dermatitis [Comentário]
- O papel do receptor GPR91 na polarização de macrófagos
- SOCS1 is a negative regulator of metabolic reprogramming during sepsis
- Falência da migração de neutrófilos para o foco infeccioso na sepse polimicrobiana: papel do receptor Toll-like 4
Informações sobre o DOI: 10.3390/biomedicines10040756 (Fonte: oaDOI API)
Download do texto completo
| Tipo | Nome | Link | |
|---|---|---|---|
 |
003072825.pdf | Direct link |
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
