Glyceraldehyde-3-phosphate dehydrogenase (GAPDH) protein-protein interaction inhibitor reveals a non-catalytic role for GAPDH oligomerization in cell death (2016)
- Authors:
- Autor USP: FERREIRA, JULIO CESAR BATISTA - ICB
- Unidade: ICB
- DOI: 10.1074/jbc.M115.711630
- Assunto: ANATOMIA
- Language: Inglês
- Imprenta:
- Source:
- Título: International Journal of Biological Chemistry
- ISSN: 2152-2561
- Volume/Número/Paginação/Ano: v. 291, n. 26, p. 13608-13621, 2016
- Status:
- Artigo aberto em periódico híbrido (Hybrid Open Access)
- Versão do Documento:
- Versão publicada (Published version)
- Acessar versão aberta:
-
ABNT
QVIT, Nir et al. Glyceraldehyde-3-phosphate dehydrogenase (GAPDH) protein-protein interaction inhibitor reveals a non-catalytic role for GAPDH oligomerization in cell death. International Journal of Biological Chemistry, v. 291, n. 26, p. 13608-13621, 2016Tradução . . Disponível em: https://doi.org/10.1074/jbc.M115.711630. Acesso em: 26 mar. 2026. -
APA
Qvit, N., Joshi, A. U., Cunningham, A. D., Ferreira, J. C. B., & Mochly-Rosen,. (2016). Glyceraldehyde-3-phosphate dehydrogenase (GAPDH) protein-protein interaction inhibitor reveals a non-catalytic role for GAPDH oligomerization in cell death. International Journal of Biological Chemistry, 291( 26), 13608-13621. doi:10.1074/jbc.M115.711630 -
NLM
Qvit N, Joshi AU, Cunningham AD, Ferreira JCB, Mochly-Rosen. Glyceraldehyde-3-phosphate dehydrogenase (GAPDH) protein-protein interaction inhibitor reveals a non-catalytic role for GAPDH oligomerization in cell death [Internet]. International Journal of Biological Chemistry. 2016 ; 291( 26): 13608-13621.[citado 2026 mar. 26 ] Available from: https://doi.org/10.1074/jbc.M115.711630 -
Vancouver
Qvit N, Joshi AU, Cunningham AD, Ferreira JCB, Mochly-Rosen. Glyceraldehyde-3-phosphate dehydrogenase (GAPDH) protein-protein interaction inhibitor reveals a non-catalytic role for GAPDH oligomerization in cell death [Internet]. International Journal of Biological Chemistry. 2016 ; 291( 26): 13608-13621.[citado 2026 mar. 26 ] Available from: https://doi.org/10.1074/jbc.M115.711630 - Acute inhibition of excessive mitochondrial fission after myocardial infarction prevents long-term cardiac dysfunction
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