Glyceraldehyde-3-phosphate Dehydrogenase (GAPDH) Phosphorylation by Protein Kinase Cδ (PKCδ) inhibits mitochondria elimination by lysosomal-like structures following ischemia and reoxygenation-induced injury (2013)
- Authors:
- Autor USP: FERREIRA, JULIO CESAR BATISTA - ICB
- Unidade: ICB
- DOI: 10.1074/jbc.M113.466870
- Assunto: ANATOMIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Journal of Biological Chemistry
- ISSN: 1083-351X
- Volume/Número/Paginação/Ano: v. 288, n. 26, p. 18947-18960, 2013
- Status:
- Artigo aberto em periódico híbrido (Hybrid Open Access)
- Versão do Documento:
- Versão publicada (Published version)
- Acessar versão aberta:
-
ABNT
YOUGALINGAM, Gouri et al. Glyceraldehyde-3-phosphate Dehydrogenase (GAPDH) Phosphorylation by Protein Kinase Cδ (PKCδ) inhibits mitochondria elimination by lysosomal-like structures following ischemia and reoxygenation-induced injury. Journal of Biological Chemistry, v. 288, n. 26, p. 18947-18960, 2013Tradução . . Disponível em: https://doi.org/10.1074/jbc.M113.466870. Acesso em: 02 abr. 2026. -
APA
Yougalingam, G., Hwang, S., Ferreira, J. C. B., & Mochly-Rosen, D. (2013). Glyceraldehyde-3-phosphate Dehydrogenase (GAPDH) Phosphorylation by Protein Kinase Cδ (PKCδ) inhibits mitochondria elimination by lysosomal-like structures following ischemia and reoxygenation-induced injury. Journal of Biological Chemistry, 288( 26), 18947-18960. doi:10.1074/jbc.M113.466870 -
NLM
Yougalingam G, Hwang S, Ferreira JCB, Mochly-Rosen D. Glyceraldehyde-3-phosphate Dehydrogenase (GAPDH) Phosphorylation by Protein Kinase Cδ (PKCδ) inhibits mitochondria elimination by lysosomal-like structures following ischemia and reoxygenation-induced injury [Internet]. Journal of Biological Chemistry. 2013 ; 288( 26): 18947-18960.[citado 2026 abr. 02 ] Available from: https://doi.org/10.1074/jbc.M113.466870 -
Vancouver
Yougalingam G, Hwang S, Ferreira JCB, Mochly-Rosen D. Glyceraldehyde-3-phosphate Dehydrogenase (GAPDH) Phosphorylation by Protein Kinase Cδ (PKCδ) inhibits mitochondria elimination by lysosomal-like structures following ischemia and reoxygenation-induced injury [Internet]. Journal of Biological Chemistry. 2013 ; 288( 26): 18947-18960.[citado 2026 abr. 02 ] Available from: https://doi.org/10.1074/jbc.M113.466870 - Acute inhibition of excessive mitochondrial fission after myocardial infarction prevents long-term cardiac dysfunction
- Targeting aldehyde dehydrogenase 2: new therapeutic opportunities
- Disruption of mitochondrial quality control in peripheral arterydisease: new therapeutic opportunities
- Treatment strategies for glucose-6-phosphate dehydrogenase deficiency: past and future perspectives
- In vivo measurement of aldehyde dehydrogenase-2 activity in rat liver ethanol model using dynamic MRSI of hyperpolarized [1-13C]pyruvate
- Induced pluripotent stem cells reprogramming: Epigenetics and applications in the regenerative medicine
- Targeting mitochondrial dysfunction and oxidative stress in heart failure: challenges and opportunities
- Impact of exercise training on redox signaling in cardiovascular diseases
- Redefinindo a fisiopatologia cardiovascular na busca de terapias mais eficazes
- Peripheral sensitization increases opioid receptor expression and activation by crotalphine in rats
Informações sobre a disponibilidade de versões do artigo em acesso aberto coletadas automaticamente via oaDOI API (Unpaywall).
Por se tratar de integração com serviço externo, podem existir diferentes versões do trabalho (como preprints ou postprints), que podem diferir da versão publicada.
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
