Hydroxi-3-methylglutaryl coenzyme a reductase inhibitor (simvastatin) decreases the production of nitric oxide and reverts the impaired vascular responsiveness observed during experimental septic shock (2003)
- Authors:
- USP affiliated authors: ROCHA, MARIA JOSE ALVES DA - FORP ; ELIAS, LUCILA LEICO KAGOHARA - FMRP ; RODRIGUES, JOSE ANTUNES - FMRP
- Unidades: FORP; FMRP
- Subjects: FISIOLOGIA CARDIOVASCULAR; ÓXIDO NÍTRICO
- Language: Inglês
- Imprenta:
- Publisher: SBFis
- Publisher place: Ribeirão Preto
- Date published: 2003
- Source:
- Título do periódico: Abstracts
- Conference titles: Congresso da Sociedade Brasileira de Fisiologia - SBFis
-
ABNT
GIUSTI-PAIVA, Alexandre et al. Hydroxi-3-methylglutaryl coenzyme a reductase inhibitor (simvastatin) decreases the production of nitric oxide and reverts the impaired vascular responsiveness observed during experimental septic shock. 2003, Anais.. Ribeirão Preto: SBFis, 2003. . Acesso em: 23 abr. 2024. -
APA
Giusti-Paiva, A., Martinez, M. R., Felix, J. V. C., Rocha, M. J. A. da, Elias, L. L. K., & Antunes-Rodrigues, J. (2003). Hydroxi-3-methylglutaryl coenzyme a reductase inhibitor (simvastatin) decreases the production of nitric oxide and reverts the impaired vascular responsiveness observed during experimental septic shock. In Abstracts. Ribeirão Preto: SBFis. -
NLM
Giusti-Paiva A, Martinez MR, Felix JVC, Rocha MJA da, Elias LLK, Antunes-Rodrigues J. Hydroxi-3-methylglutaryl coenzyme a reductase inhibitor (simvastatin) decreases the production of nitric oxide and reverts the impaired vascular responsiveness observed during experimental septic shock. Abstracts. 2003 ;[citado 2024 abr. 23 ] -
Vancouver
Giusti-Paiva A, Martinez MR, Felix JVC, Rocha MJA da, Elias LLK, Antunes-Rodrigues J. Hydroxi-3-methylglutaryl coenzyme a reductase inhibitor (simvastatin) decreases the production of nitric oxide and reverts the impaired vascular responsiveness observed during experimental septic shock. Abstracts. 2003 ;[citado 2024 abr. 23 ] - Time course of nitric oxide formation and secretion of vasopressin and oxytocin in an experimental model of polymicrobial sepsis
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