Estradiol and angiotensin II crosstalk in hydromineral balance: role of the ERK1/2 and JNk signaling pathways (2016)
- Authors:
- USP affiliated authors: ELIAS, LUCILA LEICO KAGOHARA - FMRP ; RODRIGUES, JOSE ANTUNES - FMRP
- Unidade: FMRP
- DOI: 10.1016/j.neuroscience.2016.02.067
- Subjects: ANGIOTENSINA II; ESTRADIOL; NÚCLEO SUPRAQUIASMÁTICO; VASOPRESSINAS; OVARIECTOMIA
- Keywords: Sodium intake; Water intake; Vasopressin secretion; Oxytocin secretion; Ovariectomized rats
- Language: Inglês
- Imprenta:
- Publisher place: Kidlington
- Date published: 2016
- Source:
- Título: Neuroscience
- ISSN: 0306-4522
- Volume/Número/Paginação/Ano: v. 322, p. 525-538, 2016
- Este artigo NÃO possui versão em acesso aberto
-
Status: Nenhuma versão em acesso aberto identificada -
ABNT
ALMEIDA-PEREIRA, G. et al. Estradiol and angiotensin II crosstalk in hydromineral balance: role of the ERK1/2 and JNk signaling pathways. Neuroscience, v. 322, p. 525-538, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2016.02.067. Acesso em: 15 mar. 2026. -
APA
Almeida-Pereira, G., Coletti, R., Mecawi, A. S., Reis, L. C., Elias, L. L. K., & Antunes-Rodrigues, J. (2016). Estradiol and angiotensin II crosstalk in hydromineral balance: role of the ERK1/2 and JNk signaling pathways. Neuroscience, 322, 525-538. doi:10.1016/j.neuroscience.2016.02.067 -
NLM
Almeida-Pereira G, Coletti R, Mecawi AS, Reis LC, Elias LLK, Antunes-Rodrigues J. Estradiol and angiotensin II crosstalk in hydromineral balance: role of the ERK1/2 and JNk signaling pathways [Internet]. Neuroscience. 2016 ; 322 525-538.[citado 2026 mar. 15 ] Available from: https://doi.org/10.1016/j.neuroscience.2016.02.067 -
Vancouver
Almeida-Pereira G, Coletti R, Mecawi AS, Reis LC, Elias LLK, Antunes-Rodrigues J. Estradiol and angiotensin II crosstalk in hydromineral balance: role of the ERK1/2 and JNk signaling pathways [Internet]. Neuroscience. 2016 ; 322 525-538.[citado 2026 mar. 15 ] Available from: https://doi.org/10.1016/j.neuroscience.2016.02.067 - Central nitric oxide blocks vasopressin, oxytocin and atrial natriuretic peptide release and antidiuretic and natriuretic responses induced by central angiotensin II in conscious rats
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