Simvastatin decreases nitric oxide overproduction and reverts the impaired vascular responsiveness induced by endotoxic shock in rats (2004)
- Authors:
- USP affiliated authors: ROCHA, MARIA JOSE ALVES DA - FORP ; CARNIO, EVELIN CAPELLARI - EERP ; ELIAS, LUCILA LEICO KAGOHARA - FMRP ; RODRIGUES, JOSE ANTUNES - FMRP
- Unidades: FORP; EERP; FMRP
- Subjects: SEPSE; PRESSÃO SANGUÍNEA; LIPOPOLISSACARÍDEOS; ÓXIDO NÍTRICO (SÍNTESE)
- Language: Inglês
- Imprenta:
- Publisher place: Philadelphia
- Date published: 2004
- Source:
-
ABNT
GIUSTI-PAIVA, Alexandre et al. Simvastatin decreases nitric oxide overproduction and reverts the impaired vascular responsiveness induced by endotoxic shock in rats. SHOCK, v. 21, n. 3, p. 271-275, 2004Tradução . . Acesso em: 26 abr. 2024. -
APA
Giusti-Paiva, A., Martinez, M. R., Felix, J. V. C., Rocha, M. J. A. da, Cárnio, E. C., Elias, L. L. K., & Antunes-Rodrigues, J. (2004). Simvastatin decreases nitric oxide overproduction and reverts the impaired vascular responsiveness induced by endotoxic shock in rats. SHOCK, 21( 3), 271-275. -
NLM
Giusti-Paiva A, Martinez MR, Felix JVC, Rocha MJA da, Cárnio EC, Elias LLK, Antunes-Rodrigues J. Simvastatin decreases nitric oxide overproduction and reverts the impaired vascular responsiveness induced by endotoxic shock in rats. SHOCK. 2004 ; 21( 3): 271-275.[citado 2024 abr. 26 ] -
Vancouver
Giusti-Paiva A, Martinez MR, Felix JVC, Rocha MJA da, Cárnio EC, Elias LLK, Antunes-Rodrigues J. Simvastatin decreases nitric oxide overproduction and reverts the impaired vascular responsiveness induced by endotoxic shock in rats. SHOCK. 2004 ; 21( 3): 271-275.[citado 2024 abr. 26 ] - Hydroxi-3-methylglutaryl coenzyme a reductase inhibitor (simvastatin) decreases the production of nitric oxide and reverts the impaired vascular responsiveness observed during experimental septic shock
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- Vasopressin and oxytocin secretion following experimental septic shock in rats with AV3V lesion
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- Participation of iNOS-derived NO in hypothalamic activation and vasopressin release during polymicrobial sepsis
- Role of nitric oxide in lipopolysaccharide-induced release of vasopressin
- Neonatal endotoxin exposure changes neuroendocrine, cardiovascular function and mortality during polymicrobial sepsis in adult rats
- Role of AV3V in the hypothalamus activation and hormone secretion during experimental septic shock in rats
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