Caloric restriction and redox state: does this diet increase or decrease oxidant production? (2011)
- Autor:
- Autor USP: KOWALTOWSKI, ALICIA JULIANA - IQ
- Unidade: IQ
- DOI: 10.1179/1351000211Y.0000000014
- Subjects: RADICAIS LIVRES; ENVELHECIMENTO
- Language: Inglês
- Imprenta:
- Publisher place: Reino Unido
- Date published: 2011
- Source:
- Título do periódico: Redox Report
- ISSN: 1351-0002
- Volume/Número/Paginação/Ano: v. 16, n. 6, p. 237-241, 2011
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: green
-
ABNT
KOWALTOWSKI, Alicia Juliana. Caloric restriction and redox state: does this diet increase or decrease oxidant production?. Redox Report, v. 16, n. 6, p. 237-241, 2011Tradução . . Disponível em: https://doi.org/10.1179/1351000211Y.0000000014. Acesso em: 19 set. 2024. -
APA
Kowaltowski, A. J. (2011). Caloric restriction and redox state: does this diet increase or decrease oxidant production? Redox Report, 16( 6), 237-241. doi:10.1179/1351000211Y.0000000014 -
NLM
Kowaltowski AJ. Caloric restriction and redox state: does this diet increase or decrease oxidant production? [Internet]. Redox Report. 2011 ; 16( 6): 237-241.[citado 2024 set. 19 ] Available from: https://doi.org/10.1179/1351000211Y.0000000014 -
Vancouver
Kowaltowski AJ. Caloric restriction and redox state: does this diet increase or decrease oxidant production? [Internet]. Redox Report. 2011 ; 16( 6): 237-241.[citado 2024 set. 19 ] Available from: https://doi.org/10.1179/1351000211Y.0000000014 - Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
- Diet, mitochondria and energy metabolism
Informações sobre o DOI: 10.1179/1351000211Y.0000000014 (Fonte: oaDOI API)
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