ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition (2004)
- Authors:
- Autor USP: KOWALTOWSKI, ALICIA JULIANA - IQ
- Unidade: IQ
- Subjects: BIOQUÍMICA; ADENOSINA TRIFOSFATO
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Abstracts
- Conference titles: Congress of the Brazilian Society for Cell Biology
-
ABNT
FACUNDO, Hebert Di Tarso Fernandes e DE PAULA, J. G. e KOWALTOWSKI, Alicia Juliana. ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition. 2004, Anais.. São Paulo: SBBC, 2004. . Acesso em: 23 abr. 2024. -
APA
Facundo, H. D. T. F., de Paula, J. G., & Kowaltowski, A. J. (2004). ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition. In Abstracts. São Paulo: SBBC. -
NLM
Facundo HDTF, de Paula JG, Kowaltowski AJ. ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition. Abstracts. 2004 ;[citado 2024 abr. 23 ] -
Vancouver
Facundo HDTF, de Paula JG, Kowaltowski AJ. ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition. Abstracts. 2004 ;[citado 2024 abr. 23 ] - Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- Diet, mitochondria and energy metabolism
- Efeito da morfologia mitocondrial sobre a capacidade de retenção de cálcio mitocondrial
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