Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state (2018)
- Authors:
- Autor USP: KOWALTOWSKI, ALÍCIA JULIANA - IQ
- Unidade: IQ
- Subjects: ANTIOXIDANTES; ESTRESSE OXIDATIVO; MITOCÔNDRIAS
- Language: Inglês
- Imprenta:
- Source:
- Título: Resumos
- Conference titles: Reunião Anual da Federação de Sociedades de Biologia Experimental/FeSBE
-
ABNT
DAVID, Cicera Edna Barbosa et al. Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state. 2018, Anais.. São Paulo: FeSBE, 2018. . Acesso em: 16 mar. 2026. -
APA
David, C. E. B., Lucas, A. M. B., Araújo, M. T. S., Coelho, B. N., Soares Neto, J. B., Portela, B. R. C., et al. (2018). Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state. In Resumos. São Paulo: FeSBE. -
NLM
David CEB, Lucas AMB, Araújo MTS, Coelho BN, Soares Neto JB, Portela BRC, Varela ALN, Kowaltowski AJ, Facundo HDTF. Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state. Resumos. 2018 ;[citado 2026 mar. 16 ] -
Vancouver
David CEB, Lucas AMB, Araújo MTS, Coelho BN, Soares Neto JB, Portela BRC, Varela ALN, Kowaltowski AJ, Facundo HDTF. Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state. Resumos. 2018 ;[citado 2026 mar. 16 ] - Cross-talk between mitochondria and NADPH oxidase: effects of mild mitochondrial dysfunction on Angiotensin II-Mediated increase in nox isoform expression and activity in vascular smooth muscle cells
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