Somatotroph pituitary adenoma with acromegaly and autosomal dominant polycystic kidney disease: SSTR5 polymorphism and PKD1 mutation (2013)
- Authors:
- Autor USP: TOLEDO, SERGIO PEREIRA DE ALMEIDA - FM
- Unidade: FM
- DOI: 10.1007/s11102-011-0325-0
- Subjects: NEOPLASIAS DAS GLÂNDULAS SUPRARRENAIS; ADENOMA; ACROMEGALIA; CISTOS; RIM (PATOLOGIA); IMUNOHISTOQUÍMICA; POLIMORFISMO
- Language: Inglês
- Imprenta:
- Publisher place: Norwell, MA
- Date published: 2013
- Source:
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: green
-
ABNT
SYRO, Luis V. et al. Somatotroph pituitary adenoma with acromegaly and autosomal dominant polycystic kidney disease: SSTR5 polymorphism and PKD1 mutation. Pituitary, v. 15, n. 3, p. 342-349, 2013Tradução . . Disponível em: http://link.springer.com/article/10.1007/s11102-011-0325-0. Acesso em: 04 jan. 2026. -
APA
Syro, L. V., Sundsbak, J. L., Scheithauer, B. W., Toledo, R. A., Camargo, M., Heyer, C. M., et al. (2013). Somatotroph pituitary adenoma with acromegaly and autosomal dominant polycystic kidney disease: SSTR5 polymorphism and PKD1 mutation. Pituitary, 15( 3), 342-349. doi:10.1007/s11102-011-0325-0 -
NLM
Syro LV, Sundsbak JL, Scheithauer BW, Toledo RA, Camargo M, Heyer CM, Sekiya T, Uribe H, Escobar JI, Vasquez M, Rotondo F, Toledo SPA, Kovacs K, Horvath E, Babovic-Vuksanovic D, Harris PC. Somatotroph pituitary adenoma with acromegaly and autosomal dominant polycystic kidney disease: SSTR5 polymorphism and PKD1 mutation [Internet]. Pituitary. 2013 ; 15( 3): 342-349.[citado 2026 jan. 04 ] Available from: http://link.springer.com/article/10.1007/s11102-011-0325-0 -
Vancouver
Syro LV, Sundsbak JL, Scheithauer BW, Toledo RA, Camargo M, Heyer CM, Sekiya T, Uribe H, Escobar JI, Vasquez M, Rotondo F, Toledo SPA, Kovacs K, Horvath E, Babovic-Vuksanovic D, Harris PC. Somatotroph pituitary adenoma with acromegaly and autosomal dominant polycystic kidney disease: SSTR5 polymorphism and PKD1 mutation [Internet]. Pituitary. 2013 ; 15( 3): 342-349.[citado 2026 jan. 04 ] Available from: http://link.springer.com/article/10.1007/s11102-011-0325-0 - Molecular analysis of p53 vhl and ret genes in pheochromocytoma
- CPHD patients with severe sexual infantilism caused by the PROP1 301-302 2-BASE pair deletion present adrenal insufficiency
- Impaired adrenocorticotropin-adrenal axis in combined pituytary hormone deficiency caused by a two-base pair deletion (301-302delAG) in the prophet of Pit-1 gene
- Lit/lit mice release growth hormone (GH) in response to GH releasing peptide-2 (GHRP-2) administration
- Impact of RET proto-oncogene analysis on the clinical management of multiple endocrine neoplasia type 2
- A HIF1a Regulatory Loop Links Hypoxia and Mitochondrial Signals in Pheochromocytomas
- Neoplasia endócrina múltipla
- Rastreamento de mutações no gene ret em indivíduos com baixa idade, sob risco para neoplasia endócrina múltipla, tipo 2 (NEM-2)
- Hugt frequency of hirschprung in a large family C620R-ret mutated family
- Peptide hormone receptor defects leading to abnormal phenotypes
Informações sobre o DOI: 10.1007/s11102-011-0325-0 (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas