Clinical Spectrum and Genotypes of Patients With Hyper-IgM Syndrome in Latin America (2012)
- Authors:
- USP affiliated author: CONDINO NETO, ANTONIO - ICB
- School: ICB
- Subject: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Publisher: The European Society for Immunodeficiencies
- Place of publication: Florença
- Date published: 2012
- Source:
- Título do periódico: Resumos
- Conference title: Biennial Meenting of the European Society for Immunodeficiencies (ESID)
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ABNT
CONDINO-NETO, Antonio; ESPINOSA ROSALES, F. Clinical Spectrum and Genotypes of Patients With Hyper-IgM Syndrome in Latin America. Anais.. Florença: The European Society for Immunodeficiencies, 2012. -
APA
Condino-Neto, A., & Espinosa Rosales, F. (2012). Clinical Spectrum and Genotypes of Patients With Hyper-IgM Syndrome in Latin America. In Resumos. Florença: The European Society for Immunodeficiencies. -
NLM
Condino-Neto A, Espinosa Rosales F. Clinical Spectrum and Genotypes of Patients With Hyper-IgM Syndrome in Latin America. Resumos. 2012 ; -
Vancouver
Condino-Neto A, Espinosa Rosales F. Clinical Spectrum and Genotypes of Patients With Hyper-IgM Syndrome in Latin America. Resumos. 2012 ; - Anhidrotic ectodermal dysplasia and T cell immunodeficiency
- The role of nuclear factor kappa B (NF-kB) on the NCF1 gene expression
- Essential role of nuclear factor-kappa B for NADPH oxidase activity in normal and anhidrotic ectodermal dysplasia leukocytes
- A single nucleotide polymorphism in the promoter region of the NCF-2 gene
- Essential role of nuclear factor-kappa B for NADPH oxidase activity, CYBB and NCF-1 gene expression in normal and anhidrotic ectodermal dysplasia leukocytes
- Brain abscess as first clinical manifestation of hypogammaglobulinemia
- Essential role of Nuclear Factor kappa B (NF-jB) on NCFL gene expression in normal, anhidrotic ectodermal dysplasia, and chronic granulomatous disease leukocytes
- Molecular genetic analysis of Brazilian patients with CD40L deficiency
- The online latin american registry of primary immunodeficiencies
- BAY 41-2272 aumenta a atividade microbicida in vitro de leucócitos mononucleares humanos
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