BAY 41-2272 a potential pharmacological tool for treating phagocyte disorders (2008)
- Authors:
- Autor USP: CONDINO NETO, ANTONIO - ICB
- Unidade: ICB
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Clinical Immunology
- Volume/Número/Paginação/Ano: v. 127, suppl. 1, 2008
- Conference titles: Annual Meeting of Federation of Clinical Immunology Societies - FOCIS
-
ABNT
PEREIRA, Paulo Vitor et al. BAY 41-2272 a potential pharmacological tool for treating phagocyte disorders. Clinical Immunology. Orlando: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 23 jan. 2026. , 2008 -
APA
Pereira, P. V., Falcai, A., Oliveira, E., Aragão-Filho, W., Frazao, J., Marques, O., et al. (2008). BAY 41-2272 a potential pharmacological tool for treating phagocyte disorders. Clinical Immunology. Orlando: Instituto de Ciências Biomédicas, Universidade de São Paulo. -
NLM
Pereira PV, Falcai A, Oliveira E, Aragão-Filho W, Frazao J, Marques O, Errante P, Antunes E, Condino Neto A. BAY 41-2272 a potential pharmacological tool for treating phagocyte disorders. Clinical Immunology. 2008 ; 127[citado 2026 jan. 23 ] -
Vancouver
Pereira PV, Falcai A, Oliveira E, Aragão-Filho W, Frazao J, Marques O, Errante P, Antunes E, Condino Neto A. BAY 41-2272 a potential pharmacological tool for treating phagocyte disorders. Clinical Immunology. 2008 ; 127[citado 2026 jan. 23 ] - Neutrophils and monocytes from patients with CD40L deficiency present a defective respiratory burst response to several stimuli and this defect can be reversed by exogenous IFN-Gamma
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