Clinical Spectrum and Genotypes of Patients With Hyper-IgM Syndrome in Latin America (2012)
- Authors:
- Autor USP: CONDINO NETO, ANTONIO - ICB
- Unidade: ICB
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Publisher: The European Society for Immunodeficiencies
- Publisher place: Florença
- Date published: 2012
- Source:
- Título do periódico: Resumos
- Conference titles: Biennial Meenting of the European Society for Immunodeficiencies (ESID)
-
ABNT
CONDINO-NETO, Antonio e ESPINOSA ROSALES, F. Clinical Spectrum and Genotypes of Patients With Hyper-IgM Syndrome in Latin America. 2012, Anais.. Florença: The European Society for Immunodeficiencies, 2012. . Acesso em: 23 abr. 2024. -
APA
Condino-Neto, A., & Espinosa Rosales, F. (2012). Clinical Spectrum and Genotypes of Patients With Hyper-IgM Syndrome in Latin America. In Resumos. Florença: The European Society for Immunodeficiencies. -
NLM
Condino-Neto A, Espinosa Rosales F. Clinical Spectrum and Genotypes of Patients With Hyper-IgM Syndrome in Latin America. Resumos. 2012 ;[citado 2024 abr. 23 ] -
Vancouver
Condino-Neto A, Espinosa Rosales F. Clinical Spectrum and Genotypes of Patients With Hyper-IgM Syndrome in Latin America. Resumos. 2012 ;[citado 2024 abr. 23 ] - Anhidrotic ectodermal dysplasia and T cell immunodeficiency
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- Essential role of nuclear factor-kappa B for NADPH oxidase activity in normal and anhidrotic ectodermal dysplasia leukocytes
- A single nucleotide polymorphism in the promoter region of the NCF-2 gene
- Essential role of nuclear factor-kappa B for NADPH oxidase activity, CYBB and NCF-1 gene expression in normal and anhidrotic ectodermal dysplasia leukocytes
- An unusual case of a female patient presenting with recurrent infections due to mycobacteria, viruses, and pyogenic bacteria and defective TLRs response
- Critical issues and needs in management of primary immunodeficiency diseases in Latin America
- Brain abscess as first clinical manifestation of hypogammaglobulinemia
- Essential role of Nuclear Factor kappa B (NF-jB) on NCFL gene expression in normal, anhidrotic ectodermal dysplasia, and chronic granulomatous disease leukocytes
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