Accumulation of the SET protein in HEK293T cells and mild oxidative stress: cell survival or deth signaling (2012)
- Authors:
- USP affiliated authors: LEOPOLDINO, ANDRÉIA MACHADO - FCFRP ; GARCIA, CRISTIANA BERNADELLI - FCFRP ; POLIZELLO, ANA CRISTINA MORSELI - FCFRP ; UYEMURA, SERGIO AKIRA - FCFRP ; CURTI, CARLOS - FCFRP
- Unidade: FCFRP
- DOI: 10.1007/s11010-011-1158-x
- Subjects: APOPTOSE; SOBREVIVÊNCIA CELULAR; ESTRESSE OXIDATIVO (EFEITOS DE DROGAS;GENÉTICA); FATORES DE TRANSCRIÇÃO (GENÉTICA;METABOLISMO)
- Language: Inglês
- Imprenta:
- Source:
- Título: Molecular and Cellular Biochemistry
- ISSN: 0300-8177
- Volume/Número/Paginação/Ano: v. 363, n. 1-2, p. 65-74, 2012
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
LEOPOLDINO, Andréia Machado et al. Accumulation of the SET protein in HEK293T cells and mild oxidative stress: cell survival or deth signaling. Molecular and Cellular Biochemistry, v. 363, n. 1-2, p. 65-74, 2012Tradução . . Disponível em: https://doi.org/10.1007/s11010-011-1158-x. Acesso em: 15 fev. 2026. -
APA
Leopoldino, A. M., Squarize, C. H., Garcia, C. B., Almeida, L. O., Pestana, C. R., Polizello, A. C. M., et al. (2012). Accumulation of the SET protein in HEK293T cells and mild oxidative stress: cell survival or deth signaling. Molecular and Cellular Biochemistry, 363( 1-2), 65-74. doi:10.1007/s11010-011-1158-x -
NLM
Leopoldino AM, Squarize CH, Garcia CB, Almeida LO, Pestana CR, Polizello ACM, Uyemura SA, Tajara EH, Gutkind JS, Curti C. Accumulation of the SET protein in HEK293T cells and mild oxidative stress: cell survival or deth signaling [Internet]. Molecular and Cellular Biochemistry. 2012 ; 363( 1-2): 65-74.[citado 2026 fev. 15 ] Available from: https://doi.org/10.1007/s11010-011-1158-x -
Vancouver
Leopoldino AM, Squarize CH, Garcia CB, Almeida LO, Pestana CR, Polizello ACM, Uyemura SA, Tajara EH, Gutkind JS, Curti C. Accumulation of the SET protein in HEK293T cells and mild oxidative stress: cell survival or deth signaling [Internet]. Molecular and Cellular Biochemistry. 2012 ; 363( 1-2): 65-74.[citado 2026 fev. 15 ] Available from: https://doi.org/10.1007/s11010-011-1158-x - Preliminary evidence for a resistance of HNSCC cells to oxidative stress and to hindering of apoptosis signaling by protein kinase B(AKT) phosphorylation
- Set/I2PPA2 plays relevant function in oral tumorigenesis by transcription and signaling regulation
- Accumulated SET protein up-regulates and interacts with hnRNPK, increasing its binding to nucleic acids, the Bcl-xS repression, and cellular proliferation
- Set protein prevents oxidative stress-induced apoptosis
- Evidence for involvement of SET/I2PP2A on cancer resistance to apoptosis induced by oxidative stress
- Vias alternativas mitocondriais: estudos da NADH desidrogenase alternativa externa de Aspergillus fumigatus
- SET/I2PP2A protein accumulation and ethanol detoxification: decrease of the ALDH2 levels and failure in DNA damage repair mechanism
- SET overexpression decreases cell detoxification efficiency: ALDH2 and GSTP1 are dowregulated, DDR is impaired and DNA damage accumulates
- Bioenergetica mitocondrial cardiaca na infeccao experimental pelo t. Cruzi em ratos: parametros criticos da fof1-atpase
- Heart mitochondrial energetics in the experimental infection by t. Cruzi in rats . (Iv) synthesis and hydrolysis of atp by fo f1 - atpase
Informações sobre o DOI: 10.1007/s11010-011-1158-x (Fonte: oaDOI API)
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