Set protein prevents oxidative stress-induced apoptosis (2010)
- Authors:
- USP affiliated authors: CURTI, CARLOS - FCFRP ; POLIZELLO, ANA CRISTINA MORSELI - FCFRP ; LEOPOLDINO, ANDRÉIA MACHADO - FCFRP
- Unidade: FCFRP
- Subjects: APOPTOSE; NEOPLASIAS; ESTRESSE OXIDATIVO
- Language: Português
- Imprenta:
- Publisher place: Ribeirão Preto
- Date published: 2010
- Source:
- Título: Resumo
- Conference titles: Simpósio Internacional de Pós-Graduação e Pesquisa (SINPOSPq)
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ABNT
GODOY, Felipe Augusto et al. Set protein prevents oxidative stress-induced apoptosis. 2010, Anais.. Ribeirão Preto: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo, 2010. . Acesso em: 29 dez. 2025. -
APA
Godoy, F. A., Pestana, C. R., Polizello, A. C. M., Leopoldino, A. M., & Curti, C. (2010). Set protein prevents oxidative stress-induced apoptosis. In Resumo. Ribeirão Preto: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. -
NLM
Godoy FA, Pestana CR, Polizello ACM, Leopoldino AM, Curti C. Set protein prevents oxidative stress-induced apoptosis. Resumo. 2010 ;[citado 2025 dez. 29 ] -
Vancouver
Godoy FA, Pestana CR, Polizello ACM, Leopoldino AM, Curti C. Set protein prevents oxidative stress-induced apoptosis. Resumo. 2010 ;[citado 2025 dez. 29 ] - SET protein regulates expression of glutathione S-transferase pi 1 (GSTP-1) in oral carcinoma
- Different responses of two HNSCC cell lineages to oxidative stress
- Knockdown of SET protein sensitizes HNSCC-HN12 cells to UCN-01
- SET protects mitochondrial function and induces autophagy in response to redox unbalance
- Epigenetic regulation by SET protein in HNSCC: histone acetylation versus DNA methylation
- ATP production in HnRNP K-overexpressing HEK293 cells
- Maternal protein restriction increases respiratory and sympathetic activities and sensitizes peripheral chemoreflex in male rat offspring
- SET/I2PP2A is an essential protein for cell migration in head and neck squamous cell carcinoma
- SET protects mitochondria from apoptosis signaling and favors autophagy in HNSCC cells exposed to a pro-oxidant
- SET protein accumulation prevents oxidative stress-induced apoptosis/mitochondrial dysfunction with potential implication in tumorigenesis
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