Set protein prevents oxidative stress-induced apoptosis (2010)
- Authors:
- USP affiliated authors: CURTI, CARLOS - FCFRP ; POLIZELLO, ANA CRISTINA MORSELI - FCFRP ; LEOPOLDINO, ANDRÉIA MACHADO - FCFRP
- Unidade: FCFRP
- Subjects: APOPTOSE; NEOPLASIAS; ESTRESSE OXIDATIVO
- Language: Português
- Imprenta:
- Publisher place: Ribeirão Preto
- Date published: 2010
- Source:
- Título: Resumo
- Conference titles: Simpósio Internacional de Pós-Graduação e Pesquisa (SINPOSPq)
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ABNT
GODOY, Felipe Augusto et al. Set protein prevents oxidative stress-induced apoptosis. 2010, Anais.. Ribeirão Preto: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo, 2010. . Acesso em: 13 fev. 2026. -
APA
Godoy, F. A., Pestana, C. R., Polizello, A. C. M., Leopoldino, A. M., & Curti, C. (2010). Set protein prevents oxidative stress-induced apoptosis. In Resumo. Ribeirão Preto: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. -
NLM
Godoy FA, Pestana CR, Polizello ACM, Leopoldino AM, Curti C. Set protein prevents oxidative stress-induced apoptosis. Resumo. 2010 ;[citado 2026 fev. 13 ] -
Vancouver
Godoy FA, Pestana CR, Polizello ACM, Leopoldino AM, Curti C. Set protein prevents oxidative stress-induced apoptosis. Resumo. 2010 ;[citado 2026 fev. 13 ] - Different responses of two HNSCC cell lineages to oxidative stress
- Knockdown of SET protein sensitizes HNSCC-HN12 cells to UCN-01
- Epigenetic regulation by SET protein in HNSCC: histone acetylation versus DNA methylation
- SET/I2PP2A is an essential protein for cell migration in head and neck squamous cell carcinoma
- The accumulation of SET protein in head and neck squamous cell carcinoma (HNSCCs) increases basal autophagy and prevents mitochondrial dysfunction
- ATP production in HnRNP K-overexpressing HEK293 cells
- Potential modulation of PTEN phosphatase by PP2A and oxidative stress
- SET protein regulates expression of glutathione S-transferase pi 1 (GSTP-1) in oral carcinoma
- SET protein accumulation prevents oxidative stress-induced apoptosis/mitochondrial dysfunction with potential implication in tumorigenesis
- Neuroprotective effects of C-Phycocyanin in neuronal cell lines and transient ischemic rat retina
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