Accumulation of the SET protein in HEK293T cells and mild oxidative stress: cell survival or deth signaling (2012)
- Autores:
- Autores USP: LEOPOLDINO, ANDRÉIA MACHADO - FCFRP ; GARCIA, CRISTIANA BERNADELLI - FCFRP ; POLIZELLO, ANA CRISTINA MORSELI - FCFRP ; UYEMURA, SERGIO AKIRA - FCFRP ; CURTI, CARLOS - FCFRP
- Unidade: FCFRP
- DOI: 10.1007/s11010-011-1158-x
- Assuntos: APOPTOSE; SOBREVIVÊNCIA CELULAR; ESTRESSE OXIDATIVO (EFEITOS DE DROGAS;GENÉTICA); FATORES DE TRANSCRIÇÃO (GENÉTICA;METABOLISMO)
- Idioma: Inglês
- Imprenta:
- Fonte:
- Título do periódico: Molecular and Cellular Biochemistry
- ISSN: 0300-8177
- Volume/Número/Paginação/Ano: v. 363, n. 1-2, p. 65-74, 2012
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
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ABNT
LEOPOLDINO, Andréia Machado et al. Accumulation of the SET protein in HEK293T cells and mild oxidative stress: cell survival or deth signaling. Molecular and Cellular Biochemistry, v. 363, n. 1-2, p. 65-74, 2012Tradução . . Disponível em: https://doi.org/10.1007/s11010-011-1158-x. Acesso em: 18 set. 2024. -
APA
Leopoldino, A. M., Squarize, C. H., Garcia, C. B., Almeida, L. O., Pestana, C. R., Polizello, A. C. M., et al. (2012). Accumulation of the SET protein in HEK293T cells and mild oxidative stress: cell survival or deth signaling. Molecular and Cellular Biochemistry, 363( 1-2), 65-74. doi:10.1007/s11010-011-1158-x -
NLM
Leopoldino AM, Squarize CH, Garcia CB, Almeida LO, Pestana CR, Polizello ACM, Uyemura SA, Tajara EH, Gutkind JS, Curti C. Accumulation of the SET protein in HEK293T cells and mild oxidative stress: cell survival or deth signaling [Internet]. Molecular and Cellular Biochemistry. 2012 ; 363( 1-2): 65-74.[citado 2024 set. 18 ] Available from: https://doi.org/10.1007/s11010-011-1158-x -
Vancouver
Leopoldino AM, Squarize CH, Garcia CB, Almeida LO, Pestana CR, Polizello ACM, Uyemura SA, Tajara EH, Gutkind JS, Curti C. Accumulation of the SET protein in HEK293T cells and mild oxidative stress: cell survival or deth signaling [Internet]. Molecular and Cellular Biochemistry. 2012 ; 363( 1-2): 65-74.[citado 2024 set. 18 ] Available from: https://doi.org/10.1007/s11010-011-1158-x - Preliminary evidence for a resistance of HNSCC cells to oxidative stress and to hindering of apoptosis signaling by protein kinase B(AKT) phosphorylation
- Accumulated SET protein up-regulates and interacts with hnRNPK, increasing its binding to nucleic acids, the Bcl-xS repression, and cellular proliferation
- Set/I2PPA2 plays relevant function in oral tumorigenesis by transcription and signaling regulation
- Set protein prevents oxidative stress-induced apoptosis
- Heart mitochondrial bioenergetics in the experimental infection by t. Cruzi in rats . (Ii) kinetic properties of fof1 - atpase
- Evidence for involvement of SET/I2PP2A on cancer resistance to apoptosis induced by oxidative stress
- Heart fof1-atpase changes during the acute phase of trypanosoma cruzi infection in rats
- Heart mitochondrial energetics in the experimental infection by t. Cruzi in rats . (Iv) synthesis and hydrolysis of atp by fo f1 - atpase
- SET overexpression decreases cell detoxification efficiency: ALDH2 and GSTP1 are dowregulated, DDR is impaired and DNA damage accumulates
- Bioenergetica mitocondrial cardiaca na infeccao experimental pelo t. Cruzi em ratos: parametros criticos da fof1-atpase
Informações sobre o DOI: 10.1007/s11010-011-1158-x (Fonte: oaDOI API)
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