Depressed TS-evoked glutamatergic transmission on NTS neurons of rats submitted to intermittent hypoxia is due to reduced number of functional synapses (2011)
- Authors:
- USP affiliated authors: MACHADO, BENEDITO HONÓRIO - FMRP ; LEÃO, RICARDO MAURICIO XAVIER - FMRP
- Unidade: FMRP
- Subjects: FISIOLOGIA; RATOS; NEUROTRANSMISSORES
- Language: Inglês
- Imprenta:
- Publisher place: Heidelberg
- Date published: 2011
- Source:
- Título: Clinical Autonomic Research
- ISSN: 0959-9851
- Volume/Número/Paginação/Ano: v. 21, n. 4, p. 262-263, res. P118, 2011
- Conference titles: Joint Meeting of The International Society for Autonomic Neuroscience and The American Autonomic Society (ISAN/AAS)
-
ABNT
ALMADO, C. E. e MACHADO, Benedito Honório e LEÃO, Ricardo Maurício Xavier. Depressed TS-evoked glutamatergic transmission on NTS neurons of rats submitted to intermittent hypoxia is due to reduced number of functional synapses. Clinical Autonomic Research. Heidelberg: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 26 jan. 2026. , 2011 -
APA
Almado, C. E., Machado, B. H., & Leão, R. M. X. (2011). Depressed TS-evoked glutamatergic transmission on NTS neurons of rats submitted to intermittent hypoxia is due to reduced number of functional synapses. Clinical Autonomic Research. Heidelberg: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. -
NLM
Almado CE, Machado BH, Leão RMX. Depressed TS-evoked glutamatergic transmission on NTS neurons of rats submitted to intermittent hypoxia is due to reduced number of functional synapses. Clinical Autonomic Research. 2011 ; 21( 4): 262-263.[citado 2026 jan. 26 ] -
Vancouver
Almado CE, Machado BH, Leão RMX. Depressed TS-evoked glutamatergic transmission on NTS neurons of rats submitted to intermittent hypoxia is due to reduced number of functional synapses. Clinical Autonomic Research. 2011 ; 21( 4): 262-263.[citado 2026 jan. 26 ] - Chronic intermittent hypoxia in rats depressess solitary tract evoked neurotransmision in second-but not in higher-order NTS neurons
- Inhibition of spontaneous neurotransmission in the nucleus of solitary tract of the rat by the cannabinoid agonist WIN 55212-2 is not via CB1 or CB2 receptors
- Intrinsic pacemaker activity of pre-sympathetic neurons of adult rats is not altered by chronic intermittent hypoxia (CIH)
- The cannabinoid agonist win 55,212-2 inhibits spontaneous neurotransmission in neurons from rat nucleus tractus solitarius(nts)
- Depressed TS-evoked glutamatergic transmission on NTS neurons of CIH rats is due to reduced number of functional synapses
- Chronic intermittent hypoxia (CIH) enhances spontaneous synaptic transmission in the nucleus tractus solitarii (NTS) neurons of juvenile rats
- Distinct involvement of adenosine receptors in hypoxia-induced neuronal responses in the Nucleus Tractus Solitarii (NTS)
- Intrinsic properties of rostral ventrolateral medulla presympathetic and bulbospinal respiratory neurons of juvenile rats are not affected by chronic intermittent hypoxia
- Chronic intermittent hypoxia enhances spontaneous synaptic transmission and depresses solitary tract evoked neurotransmission in nucleus of the solitary tract neurons of juvenile rats
- Chronic intermittent hypoxia depresses afferent neurotransmission in NTS neurons by a reduction in the number of active synapses
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