Chronic intermittent hypoxia enhances spontaneous synaptic transmission and depresses solitary tract evoked neurotransmission in nucleus of the solitary tract neurons of juvenile rats (2008)
- Authors:
- USP affiliated authors: MACHADO, BENEDITO HONÓRIO - FMRP ; LEÃO, RICARDO MAURICIO XAVIER - FMRP
- Unidade: FMRP
- Subjects: NEUROTRANSMISSORES; FISIOLOGIA DO SISTEMA NERVOSO
- Language: Inglês
- Imprenta:
- Publisher: FeSBE
- Publisher place: Águas de Lindóia
- Date published: 2008
- Conference titles: Reunião Anual de Federação de Sociedades de Biologia Experimental (FeSBE)
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ABNT
ALMADO, Carlos Eduardo Lopes et al. Chronic intermittent hypoxia enhances spontaneous synaptic transmission and depresses solitary tract evoked neurotransmission in nucleus of the solitary tract neurons of juvenile rats. 2008, Anais.. Águas de Lindóia: FeSBE, 2008. . Acesso em: 25 jan. 2026. -
APA
Almado, C. E. L., Accorsi-Mendonça, D., Machado, B. H., & Leão, R. M. X. (2008). Chronic intermittent hypoxia enhances spontaneous synaptic transmission and depresses solitary tract evoked neurotransmission in nucleus of the solitary tract neurons of juvenile rats. In . Águas de Lindóia: FeSBE. -
NLM
Almado CEL, Accorsi-Mendonça D, Machado BH, Leão RMX. Chronic intermittent hypoxia enhances spontaneous synaptic transmission and depresses solitary tract evoked neurotransmission in nucleus of the solitary tract neurons of juvenile rats. 2008 ;[citado 2026 jan. 25 ] -
Vancouver
Almado CEL, Accorsi-Mendonça D, Machado BH, Leão RMX. Chronic intermittent hypoxia enhances spontaneous synaptic transmission and depresses solitary tract evoked neurotransmission in nucleus of the solitary tract neurons of juvenile rats. 2008 ;[citado 2026 jan. 25 ] - Depressed TS-evoked glutamatergic transmission on NTS neurons of rats submitted to intermittent hypoxia is due to reduced number of functional synapses
- Depressed TS-evoked glutamatergic transmission on NTS neurons of rats submitted to intermittent hypoxia is due to reduced number of functional synapses
- Chronic intermittent hypoxia in rats depressess solitary tract evoked neurotransmision in second-but not in higher-order NTS neurons
- Inhibition of spontaneous neurotransmission in the nucleus of solitary tract of the rat by the cannabinoid agonist WIN 55212-2 is not via CB1 or CB2 receptors
- Intrinsic pacemaker activity of pre-sympathetic neurons of adult rats is not altered by chronic intermittent hypoxia (CIH)
- The cannabinoid agonist win 55,212-2 inhibits spontaneous neurotransmission in neurons from rat nucleus tractus solitarius(nts)
- Depressed TS-evoked glutamatergic transmission on NTS neurons of CIH rats is due to reduced number of functional synapses
- Chronic intermittent hypoxia (CIH) enhances spontaneous synaptic transmission in the nucleus tractus solitarii (NTS) neurons of juvenile rats
- Distinct involvement of adenosine receptors in hypoxia-induced neuronal responses in the Nucleus Tractus Solitarii (NTS)
- Intrinsic properties of rostral ventrolateral medulla presympathetic and bulbospinal respiratory neurons of juvenile rats are not affected by chronic intermittent hypoxia
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