Inhibition of spontaneous neurotransmission in the nucleus of solitary tract of the rat by the cannabinoid agonist WIN 55212-2 is not via CB1 or CB2 receptors (2008)
- Authors:
- USP affiliated authors: MACHADO, BENEDITO HONÓRIO - FMRP ; LEÃO, RICARDO MAURICIO XAVIER - FMRP
- Unidade: FMRP
- DOI: 10.1016/j.brainres.2008.01.011
- Subjects: NEUROTRANSMISSORES; NEUROFARMACOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Brain Research
- ISSN: 0006-8993
- Volume/Número/Paginação/Ano: v. 1200, p. 1-9, 2008
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
MENDONÇA, Daniela Accorsi et al. Inhibition of spontaneous neurotransmission in the nucleus of solitary tract of the rat by the cannabinoid agonist WIN 55212-2 is not via CB1 or CB2 receptors. Brain Research, v. 1200, p. 1-9, 2008Tradução . . Disponível em: https://doi.org/10.1016/j.brainres.2008.01.011. Acesso em: 25 jan. 2026. -
APA
Mendonça, D. A., Almado, C. E. L., Dagostin, A. L. A., Machado, B. H., & Leão, R. M. X. (2008). Inhibition of spontaneous neurotransmission in the nucleus of solitary tract of the rat by the cannabinoid agonist WIN 55212-2 is not via CB1 or CB2 receptors. Brain Research, 1200, 1-9. doi:10.1016/j.brainres.2008.01.011 -
NLM
Mendonça DA, Almado CEL, Dagostin ALA, Machado BH, Leão RMX. Inhibition of spontaneous neurotransmission in the nucleus of solitary tract of the rat by the cannabinoid agonist WIN 55212-2 is not via CB1 or CB2 receptors [Internet]. Brain Research. 2008 ; 1200 1-9.[citado 2026 jan. 25 ] Available from: https://doi.org/10.1016/j.brainres.2008.01.011 -
Vancouver
Mendonça DA, Almado CEL, Dagostin ALA, Machado BH, Leão RMX. Inhibition of spontaneous neurotransmission in the nucleus of solitary tract of the rat by the cannabinoid agonist WIN 55212-2 is not via CB1 or CB2 receptors [Internet]. Brain Research. 2008 ; 1200 1-9.[citado 2026 jan. 25 ] Available from: https://doi.org/10.1016/j.brainres.2008.01.011 - Depressed TS-evoked glutamatergic transmission on NTS neurons of rats submitted to intermittent hypoxia is due to reduced number of functional synapses
- Depressed TS-evoked glutamatergic transmission on NTS neurons of rats submitted to intermittent hypoxia is due to reduced number of functional synapses
- Chronic intermittent hypoxia in rats depressess solitary tract evoked neurotransmision in second-but not in higher-order NTS neurons
- Intrinsic pacemaker activity of pre-sympathetic neurons of adult rats is not altered by chronic intermittent hypoxia (CIH)
- The cannabinoid agonist win 55,212-2 inhibits spontaneous neurotransmission in neurons from rat nucleus tractus solitarius(nts)
- Depressed TS-evoked glutamatergic transmission on NTS neurons of CIH rats is due to reduced number of functional synapses
- Chronic intermittent hypoxia (CIH) enhances spontaneous synaptic transmission in the nucleus tractus solitarii (NTS) neurons of juvenile rats
- Distinct involvement of adenosine receptors in hypoxia-induced neuronal responses in the Nucleus Tractus Solitarii (NTS)
- Intrinsic properties of rostral ventrolateral medulla presympathetic and bulbospinal respiratory neurons of juvenile rats are not affected by chronic intermittent hypoxia
- Chronic intermittent hypoxia enhances spontaneous synaptic transmission and depresses solitary tract evoked neurotransmission in nucleus of the solitary tract neurons of juvenile rats
Informações sobre o DOI: 10.1016/j.brainres.2008.01.011 (Fonte: oaDOI API)
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