Progression of diet-induced diabetes in C57BL6J mice involves functional dissociation of ‘Ca POT.2+’ channels from secretory vesicles (2010)
- Authors:
- Collins, Stephan C. - University of Oxford (UO)
- Hoppa, Michael B. - University of Oxford (UO)
- Walker, Jonathan N. - University of Oxford (UO)
- Amisten, Stefan - University of Oxford (UO)
- Abdulkader, Fernando Rodrigues de Moraes
- Bengtsson, Martin - University of Oxford (UO)
- Fearnside, Jane - University of Oxford (UO)
- Ramracheya, Reshma - University of Oxford (UO)
- Toye, Ayo A. - University of Oxford (UO)
- Zhang, Quan - University of Oxford (UO)
- Clark, Anne - University of Oxford (UO)
- Gauguier, Dominique - University of Oxford (UO)
- Rorsman, Patrik - University of Oxford (UO)
- Autor USP: ABDULKADER, FERNANDO RODRIGUES DE MORAES - ICB
- Unidade: ICB
- Assunto: FISIOLOGIA (HUMANO)
- Language: Inglês
- Imprenta:
- Source:
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ABNT
COLLINS, Stephan C. et al. Progression of diet-induced diabetes in C57BL6J mice involves functional dissociation of ‘Ca POT.2+’ channels from secretory vesicles. Diabetes, v. 59, n. 5, p. 1192-1201, 2010Tradução . . Acesso em: 10 out. 2024. -
APA
Collins, S. C., Hoppa, M. B., Walker, J. N., Amisten, S., Abdulkader, F. R. de M., Bengtsson, M., et al. (2010). Progression of diet-induced diabetes in C57BL6J mice involves functional dissociation of ‘Ca POT.2+’ channels from secretory vesicles. Diabetes, 59( 5), 1192-1201. -
NLM
Collins SC, Hoppa MB, Walker JN, Amisten S, Abdulkader FR de M, Bengtsson M, Fearnside J, Ramracheya R, Toye AA, Zhang Q, Clark A, Gauguier D, Rorsman P. Progression of diet-induced diabetes in C57BL6J mice involves functional dissociation of ‘Ca POT.2+’ channels from secretory vesicles. Diabetes. 2010 ; 59( 5): 1192-1201.[citado 2024 out. 10 ] -
Vancouver
Collins SC, Hoppa MB, Walker JN, Amisten S, Abdulkader FR de M, Bengtsson M, Fearnside J, Ramracheya R, Toye AA, Zhang Q, Clark A, Gauguier D, Rorsman P. Progression of diet-induced diabetes in C57BL6J mice involves functional dissociation of ‘Ca POT.2+’ channels from secretory vesicles. Diabetes. 2010 ; 59( 5): 1192-1201.[citado 2024 out. 10 ] - Efeitos dos ácidos graxos em membranas biológicas
- Novel aspects of the molecular mechanisms controlling insulin secretion
- A via de síntese de colesterol regula a secreção de insulina em células INS-1E através da isoprenilação de proteínas e da organização estrutural e funcional da membrana
- Potenciais de superfície na interação entre ácidos graxos e bicamadas lipídicas planas
- Preparando soluções
- Estrutura, classificação e propriedades físico-químicas dos ácidos graxos
- KATP-channels and glucose-regulated glucagon secretion
- Calcium oscillations are inhibited by Glucagon Like Peptide-l and stimulated by adrenaline in human and mouse alpha-cells in intact islets
- Chewing over physiology integration
- Membrana Celular
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