Mitochondrial ATP-sensitive 'K POT.+' channels prevent oxidative stress, permeability transition and cell death (2005)
- Authors:
- Autor USP: KOWALTOWSKI, ALICIA JULIANA - IQ
- Unidade: IQ
- Subjects: RADICAIS LIVRES; POTÁSSIO; CÁLCIO
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Journal of Bioenergetics and Biomembranes
- ISSN: 0145-479X
- Volume/Número/Paginação/Ano: v. 37, n. 2, p. 75-82, 2005
-
ABNT
FACUNDO, Hebert Di Tarso Fernandes e PAULA, Juliane Gomes de e KOWALTOWSKI, Alicia Juliana. Mitochondrial ATP-sensitive 'K POT.+' channels prevent oxidative stress, permeability transition and cell death. Journal of Bioenergetics and Biomembranes, v. 37, n. 2, p. 75-82, 2005Tradução . . Acesso em: 25 set. 2024. -
APA
Facundo, H. D. T. F., Paula, J. G. de, & Kowaltowski, A. J. (2005). Mitochondrial ATP-sensitive 'K POT.+' channels prevent oxidative stress, permeability transition and cell death. Journal of Bioenergetics and Biomembranes, 37( 2), 75-82. -
NLM
Facundo HDTF, Paula JG de, Kowaltowski AJ. Mitochondrial ATP-sensitive 'K POT.+' channels prevent oxidative stress, permeability transition and cell death. Journal of Bioenergetics and Biomembranes. 2005 ; 37( 2): 75-82.[citado 2024 set. 25 ] -
Vancouver
Facundo HDTF, Paula JG de, Kowaltowski AJ. Mitochondrial ATP-sensitive 'K POT.+' channels prevent oxidative stress, permeability transition and cell death. Journal of Bioenergetics and Biomembranes. 2005 ; 37( 2): 75-82.[citado 2024 set. 25 ] - Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
- Diet, mitochondria and energy metabolism
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